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Molecular and cellular biology, ISSN 0270-7306, 2007, Volume 27, Issue 13, pp. 4953 - 4967
Article Usage Stats Services MCB Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley... 
CELLS | OXIDATIVE STRESS | ACTIVATED PROTEIN-KINASE | PGC-1-ALPHA | BIOCHEMISTRY & MOLECULAR BIOLOGY | GROWTH | GENE-EXPRESSION | TRANSCRIPTIONAL COACTIVATOR | DIFFERENTIATION | MICROARRAY DATA | TRANSGENIC MICE | CELL BIOLOGY | Gene Expression Regulation, Enzymologic - drug effects | Acetyltransferases - metabolism | Multienzyme Complexes - metabolism | Adipocytes - drug effects | Glucose Intolerance | AMP-Activated Protein Kinases | Oxidative Phosphorylation - drug effects | Adipose Tissue, White - cytology | Body Composition - drug effects | Isoenzymes - metabolism | Adenosine Triphosphate - metabolism | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Trans-Activators - genetics | Food Deprivation | p38 Mitogen-Activated Protein Kinases - metabolism | Homeostasis - drug effects | Phosphorylation - drug effects | Protein-Serine-Threonine Kinases - metabolism | Fibroblasts - metabolism | Isoenzymes - genetics | Hydrogen Peroxide - pharmacology | Protein-Serine-Threonine Kinases - genetics | Mitochondria - metabolism | Multienzyme Complexes - genetics | Macrophages - cytology | Mitochondria - drug effects | Feeding Behavior - drug effects | Polyamines - metabolism | Macrophages - metabolism | Organ Size - drug effects | Animals | Adipocytes - metabolism | Fibroblasts - drug effects | Adipose Tissue, White - enzymology | Adipose Tissue, White - growth & development | Glucose - metabolism | Macrophages - drug effects | Trans-Activators - metabolism | Mice | Transcription Factors | Adipose Tissue, White - drug effects | Energy Metabolism - drug effects | White/cytology/drug effects/enzymology/growth | Hydrogen Peroxide/pharmacology | Macrophages/cytology/drug effects/metabolism | Mitochondria/drug effects/metabolism | Fibroblasts/drug effects/metabolism | p38 Mitogen-Activated Protein Kinases/metabolism | MEDICIN OCH HÄLSOVETENSKAP | Homeostasis/drug effects | Multienzyme Complexes/genetics/metabolism | Protein-Serine-Threonine Kinases/genetics/metabolism | Trans-Activators/genetics/metabolism | Enzymologic/drug effects | Glucose/metabolism | Adipose Tissue | Feeding Behavior/drug effects | Organ Size/drug effects | Oxidative Phosphorylation/drug effects | MEDICAL AND HEALTH SCIENCES | development | Acetyltransferases/metabolism | Adipocytes/drug effects/metabolism | Gene Expression Regulation | Adenosine Triphosphate/metabolism | Body Composition/drug effects | Polyamines/metabolism | Energy Metabolism/drug effects | Isoenzymes/genetics/metabolism | Phosphorylation/drug effects
Journal Article
Molecular Endocrinology, ISSN 1944-9917, 2013, Volume 27, Issue 5, pp. 814 - 827
Testis size and sperm production are directly correlated to the total number of adult Sertoli cells (SCs... 
SIGNAL-TRANSDUCTION | MALE-FERTILITY | SEMINIFEROUS EPITHELIUM | SPERM PRODUCTION | GRANULOSA-CELLS | MOUSE | ENDOCRINOLOGY & METABOLISM | FOLLICLE-STIMULATING-HORMONE | POSTNATAL-DEVELOPMENT | RAT TESTIS | GROWTH-FACTOR-I | Leydig Cells - cytology | Receptor, IGF Type 1 - metabolism | Cell Proliferation | Spermatogenesis - genetics | Thyroid Hormones - pharmacology | Cell Count | Germ Cells - drug effects | Humans | Male | Gene Expression Profiling | Leydig Cells - drug effects | Spermatozoa - metabolism | Cell Differentiation - genetics | Leydig Cells - metabolism | Receptor, Insulin - genetics | Seminiferous Tubules - metabolism | Sertoli Cells - cytology | Organ Size - genetics | Female | Germ Cells - cytology | Seminiferous Tubules - cytology | Germ Cells - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Sertoli Cells - metabolism | Spermatogenesis - drug effects | Follicle Stimulating Hormone - metabolism | Mice, Inbred C57BL | Signal Transduction - genetics | Mutation - genetics | Spermatozoa - drug effects | Receptor, IGF Type 1 - genetics | Cell Shape - drug effects | Fetus - cytology | Organ Size - drug effects | Animals | Signal Transduction - drug effects | Cell Differentiation - drug effects | Fetus - embryology | Receptor, Insulin - metabolism | Mice | Spermatozoa - cytology | Seminiferous Tubules - drug effects | Life Sciences | Biochemistry, Molecular Biology | Original Research
Journal Article
Neuropsychopharmacology, ISSN 0893-133X, 07/2007, Volume 32, Issue 7, pp. 1490 - 1503
.... Here, we examined in rats the effect of chronic social stress and concomitant antidepressant treatment on cell proliferation in the medial prefrontal cortex (mPFC... 
NG2 | Stereology | Glia | Cell number | Neurogenesis | Hippocampus | hippocampus | CHRONIC PSYCHOSOCIAL STRESS | PSYCHIATRY | stereology | ANTIDEPRESSANT TREATMENT | glia | NEUROSCIENCES | cell number | NEURONAL SIZE | ELECTROCONVULSIVE SEIZURES | TRANSCRANIAL MAGNETIC STIMULATION | MOOD DISORDERS | neurogenesis | PHARMACOLOGY & PHARMACY | RAT HIPPOCAMPUS | MAJOR DEPRESSIVE DISORDER | HIPPOCAMPAL NEUROGENESIS | LIFE EVENTS | Rats, Wistar | Cell Count | Prefrontal Cortex - physiopathology | Body Weight - drug effects | Bromodeoxyuridine | Depressive Disorder - drug therapy | Male | Stem Cells - metabolism | Dentate Gyrus - physiopathology | Neuroglia - drug effects | Prefrontal Cortex - pathology | Prefrontal Cortex - drug effects | Dentate Gyrus - drug effects | Social Behavior | Functional Laterality - drug effects | Functional Laterality - physiology | Neurons - metabolism | Neurons - drug effects | Cell Differentiation - physiology | Cell Survival - physiology | Cell Survival - drug effects | Fluoxetine - pharmacology | Stress, Psychological - drug therapy | Rats | Organ Size - physiology | Organ Size - drug effects | Animals | Cell Differentiation - drug effects | Depressive Disorder - physiopathology | Stem Cells - drug effects | Neuroglia - metabolism | Cell Proliferation - drug effects | Body Weight - physiology | Serotonin Uptake Inhibitors - pharmacology | Chronic Disease | Stress, Psychological - physiopathology
Journal Article
Nature medicine, ISSN 1546-170X, 2014, Volume 20, Issue 11, pp. 1279 - 1288
..., whereas vertebral fracture risk is determined mainly by trabecular bone mass (2-4). Currently used anti-resorptive drugs reduce the risk of vertebral fractures... 
MEDICINE, RESEARCH & EXPERIMENTAL | ACTIVATION | METAANALYSIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | PROLIFERATION | BETA-CATENIN | CELL BIOLOGY | MINERAL DENSITY | SOST GENE | MASS | GENOME-WIDE | DIFFERENTIATION | HOMODIMERIZATION | Osteocytes - drug effects | Humans | Wnt Proteins - deficiency | RNA, Messenger - metabolism | Wnt Proteins - metabolism | Cell Lineage - drug effects | Fractures, Bone - genetics | Cell Differentiation - genetics | RANK Ligand - pharmacology | Wnt Proteins - genetics | Skull - pathology | Gene Deletion | Organ Size - genetics | Osteogenesis - genetics | Cell Line | Osteoclasts - pathology | Disease Susceptibility | Osteocytes - metabolism | Mice, Inbred C57BL | Osteoblasts - drug effects | Osteogenesis - drug effects | RNA, Messenger - genetics | Cells, Cultured | Signal Transduction - genetics | Osteoclasts - metabolism | Fractures, Bone - metabolism | Aging - pathology | Gene Expression Regulation - drug effects | Osteoblasts - pathology | Organ Size - drug effects | Animals | Signal Transduction - drug effects | Cell Differentiation - drug effects | Osteocytes - pathology | Fractures, Bone - pathology | Osteoprotegerin - metabolism | Osteoblasts - metabolism | Fractures, Bone - prevention & control | Osteoclasts - drug effects | Physiological aspects | Fractures | Research | Wnt proteins | Analysis | Osteoclasts (Biology) | Osteoporosis | Signal transduction | Bones | Glycoproteins | Porosity | Clinical Medicine | Endokrinologi och diabetes | Klinisk medicin | Endocrinology and Diabetes
Journal Article
American journal of physiology. Heart and circulatory physiology, ISSN 0363-6135, 05/2010, Volume 298, Issue 5, pp. H1454 - H1465
Ye Y, Keyes KT, Zhang C, Perez-Polo JR, Lin Y, Birnbaum Y. The myocardial infarct size-limiting effect of sitagliptin is PKA-dependent, whereas the protective effect of pioglitazone is partially dependent on PKA... 
Protein kinase A | Adenosine 3′-5′-cyclic monophosphate | PROTEIN-KINASE-A | ISOLATED RAT-HEART | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | NITRIC-OXIDE SYNTHASE | ELEMENT-BINDING PROTEIN | STRESS STIMULATES PHOSPHORYLATION | AMERICAN-HEART-ASSOCIATION | ISCHEMIA-REPERFUSION INJURY | GLUCAGON-LIKE PEPTIDE-1 | adenosine 3 '-5 '-cyclic monophosphate | PERIPHERAL VASCULAR DISEASE | protein kinase A | NF-KAPPA-B | INHIBITOR-ASSOCIATED ANGIOEDEMA | Body Weight - drug effects | Pyrazines - therapeutic use | Protective Agents - therapeutic use | Dipeptidyl-Peptidase IV Inhibitors - pharmacology | Thiazolidinediones - therapeutic use | Culture Media | Hypertrophy, Left Ventricular - pathology | Myocardial Infarction - pathology | Myocardium - metabolism | Sitagliptin Phosphate | Cyclic AMP - metabolism | Triazoles - therapeutic use | Hypoglycemic Agents - therapeutic use | Glucagon-Like Peptide 1 - metabolism | Myocardium - pathology | Phospholipases A2 - metabolism | Blotting, Western | Myocytes, Cardiac - pathology | Cyclic AMP-Dependent Protein Kinases - physiology | Organ Size - drug effects | Animals | Myocardial Infarction - drug therapy | Myocytes, Cardiac - drug effects | Prostaglandin-Endoperoxide Synthases - metabolism | Eicosanoids - metabolism | Mice | Blood Glucose - metabolism | In Vitro Techniques | Phosphates | Development and progression | Sitagliptin | Dosage and administration | Properties | Drug therapy | Protein kinases | Health aspects | Heart attack
Journal Article
Journal of bone and mineral research, ISSN 0884-0431, 2009, Volume 24, Issue 4, pp. 578 - 588
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2011, Volume 108, Issue 9, pp. 3755 - 3760
.... The influence of GHRH antagonists on animal models of BPH has not been investigated. We evaluated the effects of the GHRH antagonists JMR-132 given at doses of 40 µg/d, MIA-313 at 20... 
Hormone antagonists | Receptors | Cell growth | Cytokines | Epithelial cells | Genes | Prostatic hyperplasia | Hormones | Prostate | Apoptosis | Rodent benign prostatic hyperplasia model | Prostatic hypertrophy | Chronic prostatic inflammation | Prostatic cell death | CYCLOOXYGENASE-2 | MULTIDISCIPLINARY SCIENCES | prostatic cell death | PROLIFERATION | rodent benign prostatic hyperplasia model | COMBINATION | SPLICE VARIANTS | CANCER | INHIBITION | THERAPY | prostatic hypertrophy | CELL LUNG-CARCINOMA | INFLAMMATION | chronic prostatic inflammation | EXPRESSION | Immunohistochemistry | Transcription, Genetic - drug effects | Prostatic Hyperplasia - pathology | Sermorelin - administration & dosage | Apoptosis - drug effects | Humans | Receptors, Androgen - metabolism | Male | NF-kappa B - metabolism | Receptors, Neuropeptide - metabolism | Prostate - metabolism | Inflammation - complications | Prostate - pathology | Interleukin-1beta - metabolism | Receptors, Pituitary Hormone-Regulating Hormone - metabolism | Inflammation Mediators - metabolism | Sermorelin - pharmacology | Prostate - drug effects | Sermorelin - analogs & derivatives | Gene Expression Regulation, Neoplastic - drug effects | Receptors, Pituitary Hormone-Regulating Hormone - genetics | Rats | Receptors, Neuropeptide - genetics | Signal Transduction - genetics | Down-Regulation - drug effects | Growth Hormone-Releasing Hormone - genetics | Prostatic Hyperplasia - genetics | Cell Division - drug effects | Prostate-Specific Antigen - blood | Growth Hormone-Releasing Hormone - metabolism | Organ Size - drug effects | Alternative Splicing - drug effects | Animals | Signal Transduction - drug effects | Cyclooxygenase 2 - metabolism | Inflammation - genetics | Prostatic Hyperplasia - blood | Prostatic Hyperplasia - enzymology | Growth Hormone-Releasing Hormone - antagonists & inhibitors | Insulin-Like Growth Factor I - metabolism | Genetic aspects | Polypeptides | Somatotropin releasing hormone | Health aspects | Hypertrophy | Biological Sciences
Journal Article
Nature medicine, ISSN 1546-170X, 2012, Volume 18, Issue 7, pp. 1095 - 1101
Insulin-like growth factor 1 (IGF-1), the most abundant growth factor in the bone matrix, maintains bone mass in adulthood. We now report that IGF-1 released... 
MEDICINE, RESEARCH & EXPERIMENTAL | LIFE-SPAN | PHYSIOLOGY | RESORPTION | BIOCHEMISTRY & MOLECULAR BIOLOGY | OSTEOPOROSIS | CELL BIOLOGY | INSULIN-RECEPTOR | OSTEOBLAST PROGENITORS | RELEVANCE | FRACTURES | POSTMENOPAUSAL WOMEN | GROWTH-FACTOR-I | Bone Matrix - metabolism | Immunohistochemistry | Mesenchymal Stromal Cells - enzymology | Bone and Bones - pathology | Receptor, IGF Type 1 - metabolism | Insulin-Like Growth Factor I - pharmacology | Femur - pathology | TOR Serine-Threonine Kinases - metabolism | Cell Count | Humans | Aging - drug effects | Insulin-Like Growth Factor Binding Protein 3 - pharmacology | Phosphatidylinositol 3-Kinases - metabolism | Femur - diagnostic imaging | Insulin Receptor Substrate Proteins - metabolism | X-Ray Microtomography | Bone and Bones - drug effects | Insulin-Like Growth Factor Binding Protein 3 - metabolism | Bone Matrix - drug effects | Bone and Bones - metabolism | Insulin-Like Growth Factor I - administration & dosage | Bone Resorption - metabolism | Insulin-Like Growth Factor Binding Protein 3 - administration & dosage | Receptor, IGF Type 1 - deficiency | Proto-Oncogene Proteins c-akt - metabolism | Femur - growth & development | Mesenchymal Stromal Cells - drug effects | Bone Resorption - blood | Osteoblasts - enzymology | Osteoblasts - drug effects | Osteogenesis - drug effects | Rats | Enzyme Activation - drug effects | Mice, Knockout | Bone Resorption - diagnostic imaging | Osteoblasts - pathology | Organ Size - drug effects | Animals | Cell Differentiation - drug effects | Bone Resorption - pathology | Mice | Mesenchymal Stromal Cells - pathology | Insulin-Like Growth Factor I - metabolism | Aging - metabolism | Physiological aspects | Research | Insulin-like growth factor 1 | Protein kinases | Stem cells | Bone marrow | Insulin-like growth factors
Journal Article