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Nature, ISSN 0028-0836, 02/2011, Volume 470, Issue 7332, pp. 105 - 110
Studies in embryonic development have guided successful efforts to direct the differentiation of human embryonic and induced pluripotent stem cells (PSCs) into... 
VIVO | NEUROGENIN-3 | MULTIDISCIPLINARY SCIENCES | BETA-CELLS | ENDODERM | DEFINED CONDITIONS | GENERATION | SPECIFICATION | FATE | EFFICIENT DIFFERENTIATION | ENDOCRINE-CELLS | Body Patterning - drug effects | Intestines - drug effects | Wnt Proteins - pharmacology | Embryonic Stem Cells - cytology | Humans | Endoderm - embryology | Intestines - embryology | Organogenesis - drug effects | Intestines - anatomy & histology | Basic Helix-Loop-Helix Transcription Factors - metabolism | Time Factors | Endoderm - cytology | Fibroblast Growth Factor 4 - pharmacology | Cell Culture Techniques | Culture Media - chemistry | Induced Pluripotent Stem Cells - cytology | Basic Helix-Loop-Helix Transcription Factors - genetics | Induced Pluripotent Stem Cells - drug effects | Cells, Cultured | Nerve Tissue Proteins - genetics | Nerve Tissue Proteins - metabolism | Microvilli - drug effects | Activins - pharmacology | Embryonic Stem Cells - drug effects | Cell Differentiation - drug effects | Intercellular Signaling Peptides and Proteins - pharmacology | Endoderm - drug effects | Culture Media - pharmacology | Morphogenesis - drug effects | Wnt3 Protein | Wnt3A Protein | Intestines - cytology | Physiological aspects | Intestines | Genetic aspects | Health aspects | Endoderm | Stem cells | Proteins | Studies | Index Medicus | posterior endoderm | intestine | FGF | transplantation | colon | drug transport | Wnt | progenitor cell
Journal Article
Journal Article
Autophagy, ISSN 1554-8627, 05/2012, Volume 8, Issue 5, pp. 812 - 825
Our study first proposed that curcumin could protect human endothelial cells from the damage caused by oxidative stress via autophagy. Furthermore, our results... 
autophagy | endothelial cell | FOXO1 | oxidative stress | curcumin | Proteins | Binding | Landes | Calcium | Biology | Bioscience | Cell | Cycle | Organogenesis | Cancer | Oxidative stress | Endothelial cell | Curcumin | Autophagy | TOR Serine-Threonine Kinases - metabolism | Apoptosis - drug effects | Humans | Phosphatidylinositol 3-Kinases - metabolism | Protein Transport - drug effects | Autophagy - drug effects | Gene Knockdown Techniques | Proto-Oncogene Proteins c-bcl-2 - metabolism | Cell Nucleus - metabolism | Forkhead Transcription Factors - metabolism | Protective Agents - pharmacology | Protein Binding - drug effects | Cytoprotection - drug effects | Membrane Proteins - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Beclin-1 | Hydrogen Peroxide - toxicity | rab GTP-Binding Proteins - metabolism | Cell Survival - drug effects | Human Umbilical Vein Endothelial Cells - drug effects | Curcumin - pharmacology | Down-Regulation - drug effects | Ubiquitin-Activating Enzymes - metabolism | Apoptosis Regulatory Proteins - metabolism | Up-Regulation - drug effects | Acetylation - drug effects | Autophagy-Related Protein 7 | Human Umbilical Vein Endothelial Cells - enzymology | Signal Transduction - drug effects | Models, Biological | Human Umbilical Vein Endothelial Cells - pathology | Forkhead Box Protein O1 | Oxidative Stress - drug effects | Cell Nucleus - drug effects | CELL BIOLOGY | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 04/2014, Volume 508, Issue 7494, pp. 123 - 7
Journal Article
Autophagy, ISSN 1554-8627, 09/2012, Volume 8, Issue 9, pp. 1333 - 1341
Photodynamic therapy (PDT) involves photosensitizing agents that, in the presence of oxygen and light, initiate formation of cytotoxic reactive oxygen species... 
apoptosis | lysosomes | autophagy | subcellular localization | photodynamic therapy | Proteins | Binding | Landes | Calcium | Biology | Bioscience | Cell | Cycle | Organogenesis | Cancer | Lysosomes | Photodynamic therapy | Autophagy | Subcellular localization | Apoptosis | CANCER-CELLS | ACTIVATION | AUTOPHAGIC RESPONSES | WST11 | CELL BIOLOGY | CYTOCHROME-C | BCL-2 | PHOTOSENSITIZER NPE6 | REACTIVE OXYGEN | MEMBRANE PERMEABILIZATION | Apoptosis - drug effects | Apoptosis - radiation effects | Microtubule-Associated Proteins - metabolism | Fluorescence | Phagosomes - radiation effects | Protein Transport - drug effects | Recombinant Fusion Proteins - metabolism | Vacuoles - drug effects | Cell Shape - radiation effects | Lysosomes - radiation effects | Photosensitizing Agents - pharmacology | Cell Nucleus - metabolism | Lysosomes - metabolism | Cell Nucleus - radiation effects | Light | Phagosomes - ultrastructure | Microtubule-Associated Proteins - deficiency | Phagosomes - drug effects | Lysosomes - drug effects | Cell Survival - drug effects | Green Fluorescent Proteins - metabolism | Permeability - drug effects | Subcellular Fractions - drug effects | Bacteriochlorophylls - pharmacology | Porphyrins - pharmacology | Cell Nucleus - ultrastructure | Amines - metabolism | Cell Survival - radiation effects | Subcellular Fractions - metabolism | Cell Shape - drug effects | Vacuoles - radiation effects | Autophagy-Related Protein 7 | Animals | Permeability - radiation effects | Protein Transport - radiation effects | Subcellular Fractions - radiation effects | Bacteriochlorophylls - chemistry | Cell Line, Tumor | Vacuoles - metabolism | Mice | Cell Nucleus - drug effects | Index Medicus | Basic Research Paper
Journal Article
Cancer Biology & Therapy, ISSN 1538-4047, 03/2011, Volume 11, Issue 5, pp. 464 - 473
Curcumin is a polyphenolic compound derived from the Indian spice turmeric. We used nanoparticle-encapsulated curcumin to treat medulloblastoma and... 
Proteins | Binding | Landes | Calcium | Biology | Bioscience | Cell | Cycle | Organogenesis | Cancer | Hedgehog | Medulloblastoma | Nanocurcumin | Glioblastoma | IGF | Curcumin | NanoCurc | NEURAL PROGENITORS | SIGNALING PATHWAYS | ACTIVATION | nanocurcumin | medulloblastoma | PANCREATIC-CANCER | nanoCurc (TM) | MEDULLOBLASTOMA CELLS | INSULIN | glioblastoma | hedgehog | ONCOLOGY | FACTOR-I RECEPTOR | NF-KAPPA-B | EXPRESSION | GLIOBLASTOMA CELLS | curcumin | Apoptosis - drug effects | Neoplastic Stem Cells - drug effects | Curcumin - chemistry | Humans | Nanocapsules | Receptors, Notch - genetics | Glycoproteins - drug effects | Antineoplastic Agents - administration & dosage | Curcumin - administration & dosage | Hedgehog Proteins - genetics | Medulloblastoma - pathology | Glioblastoma - metabolism | Antineoplastic Agents - pharmacology | Polymers | Neoplastic Stem Cells - physiology | Tumor Stem Cell Assay | Curcumin - pharmacology | Hedgehog Proteins - analysis | STAT3 Transcription Factor - analysis | Medulloblastoma - metabolism | Antigens, CD - drug effects | Antineoplastic Agents - chemistry | Down-Regulation - drug effects | AC133 Antigen | Peptides - drug effects | Receptors, Notch - analysis | Mitosis - drug effects | Signal Transduction - drug effects | Glioblastoma - pathology | Somatomedins - genetics | Cell Line, Tumor | Cell Proliferation - drug effects | Glioblastoma - drug therapy | Medulloblastoma - drug therapy | Index Medicus | nanoCurc | Research Paper
Journal Article
Cell Cycle, ISSN 1538-4101, 08/2012, Volume 11, Issue 15, pp. 2843 - 2855
Sorafenib, a multikinase inhibitor, recently received FDA approval for the treatment of advanced hepatocellular carcinoma (HCC). However, as the clinical... 
YAP1 | global gene expression analysis | microarray | Acheron/LARP6 | mini-chromosome maintenance genes | sorafenib | cell cycle | HCC | Dickkopf1 | Harakiri | Proteins | Binding | Landes | Calcium | Biology | Bioscience | Cell | Cycle | Organogenesis | Cancer | Sorafenib | Cell cycle | Global gene expression analysis | Mini-chromosome maintenance genes | Microarray | ADVANCED HEPATOCELLULAR-CARCINOMA | DOWN-REGULATION | HEPATOMA-CELLS | MULTIKINASE INHIBITOR | RAF/MEK/ERK PATHWAY | INHIBITOR BAY-43-9006 | CELL BIOLOGY | HUMAN-LEUKEMIA-CELLS | REFRACTORY SOLID TUMORS | PHASE-I | SIGNAL TRANSDUCER | Niacinamide - analogs & derivatives | Cell Cycle - genetics | Transcription, Genetic - drug effects | Apoptosis - drug effects | DNA Replication - drug effects | Humans | Apoptosis - genetics | Benzenesulfonates - therapeutic use | Gene Expression Profiling | Phenylurea Compounds | DNA Repair - genetics | Benzenesulfonates - pharmacology | Carcinoma, Hepatocellular - drug therapy | Biological Transport - drug effects | Biological Transport - genetics | Protein Biosynthesis - genetics | Pyridines - therapeutic use | Cell Survival - drug effects | Cell Adhesion - genetics | DNA Repair - drug effects | Liver Neoplasms - drug therapy | Signal Transduction - genetics | Cell Adhesion - drug effects | Mitogen-Activated Protein Kinases - antagonists & inhibitors | Signal Transduction - drug effects | Neovascularization, Pathologic - drug therapy | Protein Kinase Inhibitors - therapeutic use | Cell Line, Tumor | DNA Replication - genetics | Neovascularization, Pathologic - genetics | Protein Biosynthesis - drug effects | Cell Proliferation - drug effects | Protein Kinase Inhibitors - pharmacology | Pyridines - pharmacology | Transcription, Genetic - genetics | Cell Cycle - drug effects | Carcinoma, Hepatocellular - metabolism | Index Medicus
Journal Article
Journal Article
Development (Cambridge), ISSN 0950-1991, 05/2017, Volume 144, Issue 9, pp. 1607 - 1618
During early gonadogenesis, proliferating cells in the coelomic epithelium (CE) give rise to most of the somatic cells in both XX and XY gonads. Previous... 
NOTCH | NUMB | Mouse gonad | Cell polarity | Asymmetric cell division | Coelomic epithelium | PROGENITOR CELLS | MOUSE TESTIS | FETAL TESTIS | DEVELOPMENTAL BIOLOGY | DROSOPHILA | XY GONAD | CORTICAL NEUROGENESIS | GERM-CELLS | SEX DETERMINATION | GENE-EXPRESSION | SRY | Embryo, Mammalian - drug effects | Gonads - embryology | Leydig Cells - cytology | LIM-Homeodomain Proteins - metabolism | Receptors, Notch - metabolism | Cell Count | Gonads - drug effects | Gonads - pathology | Male | Receptors, Notch - genetics | Leydig Cells - drug effects | Organogenesis - drug effects | Cell Lineage - drug effects | Embryo, Mammalian - metabolism | Sertoli Cells - drug effects | Cell Differentiation - genetics | Epithelium - embryology | Leydig Cells - metabolism | Sertoli Cells - cytology | Female | Cell Polarity - drug effects | Membrane Proteins - metabolism | Cell Death - drug effects | Asymmetric Cell Division - drug effects | Sertoli Cells - metabolism | Epithelium - metabolism | Membrane Proteins - genetics | Dipeptides - pharmacology | Cells, Cultured | Gene Expression Regulation, Developmental - drug effects | Mutation - genetics | Nerve Tissue Proteins - genetics | Nerve Tissue Proteins - metabolism | Transcription Factors - metabolism | Cell Polarity - genetics | Gonads - metabolism | Phenotype | Animals | Signal Transduction - drug effects | Cell Differentiation - drug effects | Embryo, Mammalian - cytology | Models, Biological | Organogenesis - genetics | Mice | Cell Cycle - drug effects | Numbl protein | Germ cells | Cell fate | Somatic cells | Cell division | Cell lineage | Zebrafish | Polarity | NUMB protein | Epithelium | Cell surface | Gonads | Index Medicus | 203 | Stem Cells and Regeneration
Journal Article
Autophagy, ISSN 1554-8627, 09/2012, Volume 8, Issue 9, pp. 1357 - 1370
Journal Article
Development (Cambridge), ISSN 0950-1991, 03/2016, Volume 143, Issue 6, pp. 1041 - 1054
Heart valve development proceeds through coordinated steps by which endocardial cushions (ECs) form thin, elongated and stratified valves. Wnt signaling and... 
Lef1 | Tenascin | Mitral valve | Spongiosa | Wnt/β-Catenin signaling | EMT | Outflow tract | Heart valves | Cushion mesenchyme | Chordae tendineae | Atrioventricular canal | Axin2 | Versican | DEVELOPING MOUSE HEART | DEVELOPMENTAL BIOLOGY | Wnt/beta-catenin signaling | BETA-CATENIN | AMERICAN-HEART-ASSOCIATION | CARDIAC-VALVE FORMATION | NOONAN-SYNDROME | EPITHELIAL-MESENCHYMAL TRANSITION | ATRIOVENTRICULAR CUSHION TRANSFORMATION | EMBRYONIC HEART | NEURAL CREST | Body Patterning - drug effects | Extracellular Matrix - metabolism | Epithelial-Mesenchymal Transition - drug effects | Heart Valves - metabolism | Organogenesis - drug effects | Epithelial-Mesenchymal Transition - genetics | Axin Protein - metabolism | Mitral Valve - metabolism | Mitral Valve - drug effects | Myocardium - metabolism | Heart Valves - drug effects | Heart Valves - embryology | Extracellular Matrix - drug effects | Gene Expression Regulation, Developmental - drug effects | Mice, Transgenic | Fibroblast Growth Factors - pharmacology | Animals | Wnt Signaling Pathway - drug effects | Endocardial Cushions - drug effects | Organogenesis - genetics | Mitral Valve - embryology | Cell Proliferation - drug effects | Embryonic Development - drug effects | Body Patterning - genetics | Endocardial Cushions - cytology | Index Medicus | β-catenin signaling | Wnt
Journal Article