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Autophagy, ISSN 1554-8627, 09/2012, Volume 8, Issue 9, pp. 1333 - 1341
Photodynamic therapy (PDT) involves photosensitizing agents that, in the presence of oxygen and light, initiate formation of cytotoxic reactive oxygen species... 
apoptosis | lysosomes | autophagy | subcellular localization | photodynamic therapy | Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | Lysosomes | Photodynamic therapy | Autophagy | Subcellular localization | Apoptosis | CANCER-CELLS | ACTIVATION | AUTOPHAGIC RESPONSES | WST11 | CELL BIOLOGY | CYTOCHROME-C | BCL-2 | PHOTOSENSITIZER NPE6 | REACTIVE OXYGEN | MEMBRANE PERMEABILIZATION | Apoptosis - drug effects | Apoptosis - radiation effects | Microtubule-Associated Proteins - metabolism | Fluorescence | Phagosomes - radiation effects | Protein Transport - drug effects | Recombinant Fusion Proteins - metabolism | Vacuoles - drug effects | Cell Shape - radiation effects | Lysosomes - radiation effects | Photosensitizing Agents - pharmacology | Cell Nucleus - metabolism | Lysosomes - metabolism | Cell Nucleus - radiation effects | Light | Phagosomes - ultrastructure | Microtubule-Associated Proteins - deficiency | Phagosomes - drug effects | Lysosomes - drug effects | Cell Survival - drug effects | Green Fluorescent Proteins - metabolism | Permeability - drug effects | Subcellular Fractions - drug effects | Bacteriochlorophylls - pharmacology | Porphyrins - pharmacology | Cell Nucleus - ultrastructure | Amines - metabolism | Cell Survival - radiation effects | Subcellular Fractions - metabolism | Cell Shape - drug effects | Vacuoles - radiation effects | Autophagy-Related Protein 7 | Animals | Permeability - radiation effects | Protein Transport - radiation effects | Subcellular Fractions - radiation effects | Bacteriochlorophylls - chemistry | Cell Line, Tumor | Vacuoles - metabolism | Mice | Cell Nucleus - drug effects | Basic Research Paper
Journal Article
Genes and Development, ISSN 0890-9369, 07/2011, Volume 25, Issue 13, pp. 1439 - 1450
Leaves originate from stem cells located at the shoot apical meristem. The meristem is shielded from the environment by older leaves, and leaf initiation is... 
Auxin | Cytokinin | Light signaling | Shoot apical meristem | Organ initiation | Stem cells | stem cells | light signaling | auxin | ARABIDOPSIS SHOOT | RED-LIGHT | DEVELOPMENTAL BIOLOGY | PHYLLOTAXIS | LEAF DEVELOPMENT | CELL BIOLOGY | cytokinin | POLAR AUXIN TRANSPORT | shoot apical meristem | PHYTOCHROME-B | GENETICS & HEREDITY | GENE-EXPRESSION | organ initiation | ROOT-MERISTEM | Plant Shoots - growth & development | Cytokinins - pharmacology | Organogenesis - drug effects | Lycopersicon esculentum - cytology | Lycopersicon esculentum - growth & development | Light | Plant Shoots - drug effects | Plant Stems - cytology | Plant Stems - radiation effects | Membrane Transport Proteins - metabolism | Plant Leaves - drug effects | Signal Transduction - radiation effects | Lycopersicon esculentum - drug effects | Gene Expression Regulation, Plant - radiation effects | Indoleacetic Acids - metabolism | Plant Shoots - radiation effects | Plant Leaves - radiation effects | Lycopersicon esculentum - radiation effects | Organogenesis - radiation effects | Plant Shoots - cytology | Plant Leaves - cytology | Plant Stems - drug effects | Indoleacetic Acids - pharmacology | Plant Leaves - growth & development | Protein Transport - radiation effects | Cytokinins - metabolism | Plant Growth Regulators - pharmacology | Cytokinins | Plant genetics | Research | Research Paper
Journal Article
Autophagy, ISSN 1554-8627, 09/2011, Volume 7, Issue 9, pp. 1045 - 1051
Multiple stress pathways result in the induction of autophagy and apoptosis. Current methods (e.g., protein gel blot, microscopy) do not offer quantitative... 
Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | Autophagy | Multispectral imaging cytometry | Influenza | New methodology | Apoptosis | influenza | CROSS-TALK | new methodology | autophagy | BECLIN 1 | multispectral imaging cytometry | apoptosis | CELL-DEATH | CELL BIOLOGY | Leukocytes, Mononuclear - metabolism | Image Cytometry - methods | Apoptosis - drug effects | Apoptosis - radiation effects | Autophagy - radiation effects | Microtubule-Associated Proteins - metabolism | Humans | Autophagy - drug effects | Lysosomes - radiation effects | Chloroquine - pharmacology | Lysosomes - metabolism | Ultraviolet Rays | Time Factors | Leukocytes, Mononuclear - radiation effects | Signal Transduction - radiation effects | Fibroblasts - metabolism | Lysosomes - drug effects | Leukocytes, Mononuclear - drug effects | Animals | Fibroblasts - radiation effects | Signal Transduction - drug effects | Embryo, Mammalian - cytology | Fibroblasts - drug effects | Leukocytes, Mononuclear - cytology | Fibroblasts - cytology | Mice | Autophagy/radiation effects | Signal Transduction/radiation effects | Image Cytometry/methods | Autophagy/drug effects | Life Sciences | Lysosomes/drug effects | Immunology | Fibroblasts/metabolism | Leukocytes, Mononuclear/radiation effects | Fibroblasts/drug effects | Leukocytes, Mononuclear/metabolism | Apoptosis/radiation effects | Leukocytes, Mononuclear/drug effects | Fibroblasts/cytology | Apoptosis/drug effects | Fibroblasts/radiation effects | Lysosomes/metabolism | Leukocytes, Mononuclear/cytology | Signal Transduction/drug effects | Chloroquine/pharmacology | Embryo, Mammalian/cytology | Lysosomes/radiation effects | Microtubule-Associated Proteins/metabolism
Journal Article
Autophagy, ISSN 1554-8627, 05/2009, Volume 5, Issue 4, pp. 472 - 483
Malignant gliomas are resistant to many kinds of treatments including chemotherapy, radiotherapy, and other adjuvant therapies. Autophagy is a novel response... 
Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | Arsenic trioxide | Malignant gliomas | Mitotic arrest | Autophagy | Radiation | MALIGNANT GLIOMA-CELLS | autophagy | arsenic trioxide | mitotic arrest | INDUCED APOPTOSIS | CYCLE ARREST | MYELOID-LEUKEMIA CELLS | MITOCHONDRIAL-MEMBRANE | DEATH PATHWAYS | CELL BIOLOGY | COLON-CANCER CELLS | radiation | malignant gliomas | PROTEIN EXPRESSION | IONIZING-RADIATION | UP-REGULATION | Cytoplasmic Vesicles - drug effects | Apoptosis - drug effects | Apoptosis - radiation effects | Autophagy - radiation effects | Membrane Potential, Mitochondrial - radiation effects | Microtubule-Associated Proteins - metabolism | Humans | Phosphatidylinositol 3-Kinases - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Membrane Potential, Mitochondrial - drug effects | Autophagy - drug effects | Time Factors | Proto-Oncogene Proteins c-akt - metabolism | Radiation, Ionizing | MAP Kinase Signaling System - radiation effects | Arsenic Trioxide | Mitosis - radiation effects | Arsenicals - pharmacology | Combined Modality Therapy | Mitotic Index | Oxides - pharmacology | MAP Kinase Signaling System - drug effects | Mitosis - drug effects | Cytoplasmic Vesicles - ultrastructure | Acridine Orange | Cell Line, Tumor | Cytoplasmic Vesicles - radiation effects | Dose-Response Relationship, Radiation | Microscopy, Fluorescence
Journal Article
Journal Article
Developmental Biology, ISSN 0012-1606, 01/2012, Volume 361, Issue 2, pp. 263 - 276
Journal Article
Cell Cycle, ISSN 1538-4101, 05/2008, Volume 7, Issue 9, pp. 1238 - 1245
It is now well accepted that parental whole body irradiation causes transgenerational genome and epigenome instability in the offspring. The majority of human... 
Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | H2AX | Transgeneration effect | Germ cells | DNA damage | DNA methylation | Epigenetics | Radiation exposure | Bystander effect | bystander effect | CELLS | METHYLATION | STRAND BREAK REPAIR | transgeneration effect | HISTONE H2AX | radiation exposure | X-IRRADIATION | epigenetics | CELL BIOLOGY | INDUCED GENOMIC INSTABILITY | METHYLTRANSFERASES | IN-VIVO | MICE | IONIZING-RADIATION | germ cells | Rats, Long-Evans | Epigenesis, Genetic - radiation effects | Male | Bone Marrow - radiation effects | Testis - radiation effects | Abnormalities, Radiation-Induced - metabolism | Thymus Gland - radiation effects | Thymus Gland - pathology | DNA Damage - genetics | Bystander Effect - genetics | Abnormalities, Radiation-Induced - genetics | Genomic Instability - genetics | Germ Cells - radiation effects | Bystander Effect - radiation effects | Genomic Instability - radiation effects | Bone Marrow - physiopathology | Spermatocytes - pathology | Thymus Gland - physiopathology | Rats | Mutation - genetics | Abnormalities, Radiation-Induced - physiopathology | Cranial Irradiation - adverse effects | Germ Cells - pathology | Animals | Testis - pathology | Epigenesis, Genetic - genetics | Bone Marrow - pathology | Sex Factors | Testis - physiopathology | DNA Damage - radiation effects | Spermatocytes - radiation effects | Cell Proliferation - radiation effects
Journal Article
Autophagy, ISSN 1554-8627, 11/2012, Volume 8, Issue 11, pp. 1628 - 1642
Accidental or therapeutic exposure to ionizing radiation has severe physiological consequences and can result in cell death. We previously demonstrated that... 
BECN1 | CD47 | autophagosome | ATG7 | p62 | ionizing radiation | MAP1A/1BLC3 | ATG5 | Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | Ionizing radiation | Autophagosome | APOPTOSIS | THROMBOSPONDIN-1 | DEATH | TRANSLATION | RADIATION | CELL BIOLOGY | ISCHEMIA-REPERFUSION INJURY | MESSENGER-RNA | BREAST-TUMOR CELLS | RIBOSOME ENTRY SITE | INTEGRATED STRESS-RESPONSE | Whole-Body Irradiation | Human Umbilical Vein Endothelial Cells - metabolism | Microtubule-Associated Proteins - genetics | Apoptosis - radiation effects | Microtubule-Associated Proteins - metabolism | Sequestosome-1 Protein | Gene Silencing - radiation effects | Humans | Up-Regulation - radiation effects | Radiation Protection | Human Umbilical Vein Endothelial Cells - cytology | Apoptosis Regulatory Proteins - genetics | Phagosomes - ultrastructure | Membrane Proteins - metabolism | Autophagy - genetics | Radiation, Ionizing | Beclin-1 | CD47 Antigen - genetics | Ubiquitin-Activating Enzymes - genetics | Jurkat Cells | Membrane Proteins - genetics | Mice, Inbred C57BL | Phagosomes - metabolism | CD47 Antigen - metabolism | Ubiquitin-Activating Enzymes - metabolism | Apoptosis Regulatory Proteins - metabolism | Autophagy-Related Protein 7 | Organ Specificity - radiation effects | Animals | Autophagy-Related Protein 5 | Gene Expression Regulation - radiation effects | Mice | Adaptor Proteins, Signal Transducing - metabolism | Human Umbilical Vein Endothelial Cells - radiation effects | MAP1A | Basic Research Paper | 1BLC3
Journal Article
Journal Article
Cell Cycle, ISSN 1538-4101, 05/2008, Volume 7, Issue 9, pp. 1197 - 1204
The mammalian circadian system has been implicated in the regulation of various biological processes including those involved in genotoxic stress responses and... 
Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | Aging | CLOCK/BMAL1 transcriptional complex | Ionizing radiation | Circadian | Carcinogenesis | MOUSE | TRANSCRIPTION | CELL BIOLOGY | GENOTOXIC STRESS RESPONSES | BREAST-CANCER | circadian | MUTANT MICE | carcinogenesis | GENE | IN-VIVO | LOW-DOSE RADIATION | CELL-CYCLE | aging | EXPRESSION | ionizing radiation | Gamma Rays - adverse effects | Apoptosis - radiation effects | Immunity, Innate - genetics | Apoptosis - genetics | Male | Aging, Premature - genetics | Down-Regulation - radiation effects | Aging - genetics | Cell Transformation, Neoplastic - genetics | Trans-Activators - genetics | Female | Aging, Premature - metabolism | Gene Expression Regulation, Neoplastic - genetics | Chronobiology Disorders - genetics | Genetic Predisposition to Disease - genetics | Circadian Rhythm - genetics | Chronobiology Disorders - physiopathology | Lymphoid Tissue - metabolism | Lymphoid Tissue - radiation effects | Aging - radiation effects | Mutation - genetics | Cell Transformation, Neoplastic - metabolism | Down-Regulation - genetics | Phenotype | Animals | CLOCK Proteins | Immunity, Innate - radiation effects | Gene Expression Regulation, Neoplastic - radiation effects | Mice | Aging - metabolism | Cell Proliferation - radiation effects | Chronobiology Disorders - metabolism | BMAL1 transcriptional complex | CLOCK
Journal Article
Cancer Biology & Therapy, ISSN 1538-4047, 09/2012, Volume 13, Issue 11, pp. 1072 - 1081
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2016, Volume 11, Issue 3, p. e0150923
Journal Article