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Journal of Biological Chemistry, ISSN 0021-9258, 05/2006, Volume 281, Issue 21, pp. 14529 - 14532
Journal Article
The Plant Cell, ISSN 1040-4651, 3/2012, Volume 24, Issue 3, pp. 1202 - 1216
Journal Article
Journal Article
Plant Physiology, ISSN 0032-0889, 8/2016, Volume 171, Issue 4, pp. 2432 - 2444
Journal Article
Cancer Cell, ISSN 1535-6108, 2005, Volume 7, Issue 5, pp. 433 - 444
Checkpoints that control Myc-mediated proliferation and apoptosis are bypassed during tumorigenesis. Genes encoding polyamine biosynthetic enzymes are... 
WILD-TYPE P53 | BCL-X-L | NEOPLASTIC PHENOTYPE | ONCOLOGY | INDUCED APOPTOSIS | HUMAN-MELANOMA CELLS | C-MYC | CELL-CYCLE PROGRESSION | TRANSCRIPTIONAL TARGET | MOUSE SKIN | TRANSGENIC MICE | CELL BIOLOGY | Ornithine Decarboxylase - metabolism | Gene Expression - drug effects | Apoptosis - drug effects | Eflornithine - pharmacology | Cyclin-Dependent Kinase Inhibitor p16 | Lymphoma - metabolism | Lymphoma - drug therapy | Mice, Mutant Strains | Tumor Suppressor Proteins - genetics | Cell Cycle Proteins - genetics | Gene Expression Regulation, Neoplastic - drug effects | Spleen - pathology | B-Lymphocytes - metabolism | Ornithine Decarboxylase - genetics | Tumor Suppressor Proteins - metabolism | Cyclin-Dependent Kinase Inhibitor p21 | Mice, Inbred C57BL | Cell Cycle Proteins - metabolism | Lymphoma - genetics | Mice, Transgenic | Survival Rate | Proto-Oncogene Proteins c-myc - metabolism | B-Lymphocytes - chemistry | Cyclin-Dependent Kinase Inhibitor p27 | Mice, Knockout | Polyamines - metabolism | B-Lymphocytes - drug effects | Ornithine Decarboxylase Inhibitors | Organ Size - drug effects | Animals | Heterozygote | Cell Proliferation - drug effects | Mice | Proto-Oncogene Proteins c-myc - genetics | Tumor Suppressor Protein p14ARF - genetics | Tumor Suppressor Protein p14ARF - metabolism | Prevention | Enzymes | Enzyme inhibitors | Developmental biology | Oncology, Experimental | Lymphomas | Ornithine decarboxylase | Research | Polyamines | Cancer | Mice; Transgenic | Proto-Oncogene Proteins c-myc/genetics/metabolism | Spleen/pathology | Mice; Inbred C57BL | Cell Proliferation/drug effects | Organ Size/drug effects | Gene Expression/drug effects | Gene Expression Regulation; Neoplastic/drug effects | B-Lymphocytes/chemistry/drug effects/metabolism | Apoptosis/drug effects | Cell Cycle Proteins/genetics/metabolism | Eflornithine/pharmacology | Mice; Knockout | Polyamines/metabolism | Tumor Suppressor Protein p14ARF/genetics/metabolism | Mice; Mutant Strains | Ornithine Decarboxylase/antagonists & inhibitors/genetics/metabolism | Lymphoma/drug therapy/genetics/metabolism | Tumor Suppressor Proteins/genetics/metabolism
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2015, Volume 10, Issue 5, p. e0127246
Background Neuroblastoma (NB) is the most common cancer in infancy and most frequent cause of death from extracranial solid tumors in children. Ornithine... 
DIAMINE EXPORTER | CELLS | MULTIDISCIPLINARY SCIENCES | ALPHA-DIFLUOROMETHYLORNITHINE | DOUBLE-BLIND | N-MYC | C-MYC | EFLORNITHINE COMBINATION THERAPY | ORNITHINE-DECARBOXYLASE GENE | CANCER | ONCOGENE ADDICTION | Ornithine Decarboxylase Inhibitors - pharmacokinetics | Recurrence | Ornithine Decarboxylase - metabolism | Ornithine Decarboxylase Inhibitors - adverse effects | Humans | Neuroblastoma - enzymology | Child, Preschool | Eflornithine - pharmacokinetics | Eflornithine - adverse effects | Eflornithine - pharmacology | Infant | Male | Eflornithine - therapeutic use | Ornithine Decarboxylase Inhibitors - pharmacology | Ornithine Decarboxylase Inhibitors - therapeutic use | Female | Safety | Child | Etoposide - adverse effects | Neuroblastoma - genetics | Etoposide - pharmacology | Etoposide - therapeutic use | Treatment Outcome | Neuroblastoma - urine | Polyamines - metabolism | Phenotype | Polyamines - urine | Neuroblastoma - drug therapy | Adolescent | Difluoromethylornithine | Dosage and administration | Genetic aspects | Research | Neuroblastoma | Single nucleotide polymorphisms | Drug therapy | Polyamines | Health aspects | Therapy | Pediatrics | Transcription factors | Toxicity | Colorectal cancer | Clinical trials | Oncology | Biosynthesis | Single-nucleotide polymorphism | Epidemiology | Cancer therapies | Regulatory approval | Cell growth | Cell cycle | Children | Drug dosages | Significance | Medical research | Enzymes | Statistical analysis | Etoposide | Pharmacology | Ornithine decarboxylase | Gene expression | Addictions | Patients | Survival | Loci | Substrates | Children & youth | Medicine | Disease prevention | Chemotherapy | Gene amplification | Hospitals | Addiction | Alleles | Solid tumors | Pharmacokinetics | Ornithine | Tumors | Eflornithine | Cancer
Journal Article