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Cellular Signalling, ISSN 0898-6568, 03/2015, Volume 27, Issue 3, pp. 532 - 544
In addition to providing skeletal support, the bone is an endocrine organ that produces osteocalcin, whose uncarboxylated form (GluOC) increases insulin... 
Osteoblast | Osteocalcin | Adipose tissue | PPARγ | Cell signaling | Adiponectin | MAP KINASE | DEPENDENT PROTEIN-KINASE | GAMMA PPAR-GAMMA | GLUCOSE-UTILIZATION | PPAR gamma | PEPTIDE-1 SECRETION | CELL BIOLOGY | GPRC6A RECEPTOR | CYCLIC-AMP | INSULIN-RESISTANCE | GENE-EXPRESSION | ENERGY-METABOLISM | Nitriles - pharmacology | Receptors, G-Protein-Coupled - metabolism | rap1 GTP-Binding Proteins - antagonists & inhibitors | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | Adipocytes - cytology | Osteocalcin - pharmacology | Adipocytes - drug effects | PPAR gamma - metabolism | Adipose Tissue - metabolism | RNA Interference | Adiponectin - metabolism | Cyclic AMP-Dependent Protein Kinases - antagonists & inhibitors | Phosphorylation - drug effects | Cyclic AMP - metabolism | Cyclic AMP-Dependent Protein Kinases - metabolism | rap1 GTP-Binding Proteins - metabolism | Butadienes - pharmacology | Mice, Inbred C57BL | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | 3T3-L1 Cells | Gene Expression Regulation - drug effects | Cyclic AMP Response Element-Binding Protein - genetics | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Signal Transduction - drug effects | Adipocytes - metabolism | Cyclic AMP Response Element-Binding Protein - antagonists & inhibitors | Cyclic AMP Response Element-Binding Protein - metabolism | Receptors, G-Protein-Coupled - antagonists & inhibitors | Mice | Receptors, G-Protein-Coupled - genetics | Adipose Tissue - drug effects | rap1 GTP-Binding Proteins - genetics | Mitogen-Activated Protein Kinase 1 - metabolism | Protein binding
Journal Article
Journal Article
Journal Article
Journal of Bone and Mineral Research, ISSN 0884-0431, 04/2011, Volume 26, Issue 4, pp. 730 - 738
Mesenchymal stem cells (MSCs) cultured on extracellular matrices with different stiffness have been shown to possess diverse lineage commitment owing to the... 
mesenchymal stem cell | matrix stiffness | integrin | mechanotransduction | osteogenic differentiation | MATRIX STIFFNESS | OSTEOBLAST DIFFERENTIATION | GROWTH-HORMONE | INTEGRIN | ENDOCRINOLOGY & METABOLISM | OSTEOGENIC DIFFERENTIATION | FOCAL ADHESION KINASE | LINEAGE | MESENCHYMAL STEM CELL | RHOA | MECHANOTRANSDUCTION | RNA, Small Interfering - genetics | Osteogenesis - physiology | Gene Expression - drug effects | Gene Expression - genetics | Humans | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | Collagen Type I - chemistry | Osteocalcin - genetics | Acrylic Resins - chemical synthesis | Integrin alpha2 - metabolism | Mechanotransduction, Cellular - physiology | rho-Associated Kinases - antagonists & inhibitors | Elasticity - physiology | Mesenchymal Stromal Cells - cytology | Collagen Type I - genetics | rho-Associated Kinases - metabolism | Extracellular Matrix - physiology | Myosin-Light-Chain Phosphatase - metabolism | Osteoblasts - cytology | Phosphorylation - drug effects | Cell Differentiation - physiology | Focal Adhesion Kinase 1 - metabolism | Mesenchymal Stromal Cells - drug effects | Calcification, Physiologic - drug effects | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | Osteoblasts - drug effects | Calcification, Physiologic - physiology | Mesenchymal Stromal Cells - metabolism | Acrylic Resins - chemistry | beta Catenin - metabolism | Biomechanical Phenomena | Integrin alpha2 - genetics | Mitogen-Activated Protein Kinase 3 - metabolism | Cell Differentiation - drug effects | Protein Kinase Inhibitors - pharmacology | Osteoblasts - metabolism | Focal Adhesion Kinase 1 - antagonists & inhibitors | Core Binding Factor Alpha 1 Subunit - genetics | Mitogen-Activated Protein Kinase 1 - metabolism
Journal Article
Journal of Cellular Physiology, ISSN 0021-9541, 12/2008, Volume 217, Issue 3, pp. 819 - 827
Findings from animal studies have suggested that bone remodeling is under beta‐adrenergic control. However, the level of adrenergic inhibition required to... 
PROPRANOLOL | PHYSIOLOGY | SYMPATHETIC-NERVOUS-SYSTEM | ADULT RATS | CANCELLOUS BONE | OSTEOBLAST-LIKE CELLS | BETA-ADRENERGIC RECEPTORS | ADRENERGIC-RECEPTORS | SEROTONIN 5-HYDROXYTRYPTAMINE | OSTEOCLAST DIFFERENTIATION | ADRENOCEPTOR ANTAGONISTS | CELL BIOLOGY | Propranolol - therapeutic use | Spine - drug effects | Dose-Response Relationship, Drug | Adrenergic beta-Antagonists - pharmacology | Heart Rate - drug effects | Osteocalcin - blood | Propranolol - pharmacology | Propranolol - administration & dosage | Polymerase Chain Reaction | Female | Blood Pressure - drug effects | Heart Function Tests | Adrenergic beta-Antagonists - administration & dosage | Tibia - physiopathology | Adrenergic beta-Antagonists - therapeutic use | Heart - physiopathology | Bone Resorption - physiopathology | Echocardiography | Ovariectomy | Bone Resorption - drug therapy | Rats | Spine - physiopathology | Femur - drug effects | Femur - physiopathology | Tibia - drug effects | Bone Resorption - prevention & control | Microscopy, Confocal | Biomechanical Phenomena | Bone Density - drug effects | Animals | Heart - drug effects | Insulin-Like Growth Factor I - metabolism | Heart | Blood Pressure | Femur | Spine | Adrenergic beta-Antagonists | Life Sciences | Tibia | Propranolol | Heart Rate | Osteocalcin | Bone Resorption | Bone Density | Biochemistry, Molecular Biology | Insulin-Like Growth Factor I | Biomechanics | Molecular biology
Journal Article
Journal Article
Journal Article
Journal Article