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Molecular and Cellular Biology, ISSN 0270-7306, 11/2009, Volume 29, Issue 21, pp. 5843 - 5857
Journal Article
Archives of Oral Biology, ISSN 0003-9969, 2009, Volume 54, Issue 8, pp. 764 - 771
Journal Article
Journal of Immunology, ISSN 0022-1767, 04/2012, Volume 188, Issue 7, pp. 3513 - 3521
Rheumatoid arthritis (RA) is an autoimmune synovitis characterized by the formation of pannus and the destruction of cartilage and bone in the synovial joints.... 
CELL LUNG-CANCER | RHEUMATOID-ARTHRITIS | SIGNALING PATHWAYS | SYNOVIAL MEMBRANES | ANGIOGENESIS | C-FOS | GENE-EXPRESSION | IMMUNOLOGY | NF-KAPPA-B | OSTEOCLAST DIFFERENTIATION | ERLOTINIB | Erlotinib Hydrochloride | Receptor, Epidermal Growth Factor - genetics | Arthritis, Experimental - drug therapy | Fibroblasts - enzymology | Synovial Membrane - enzymology | Humans | Synovial Membrane - pathology | Male | Cyclooxygenase 2 - biosynthesis | Neovascularization, Pathologic - enzymology | Cyclooxygenase 2 - genetics | Mice, Inbred DBA | Drug Evaluation, Preclinical | Proto-Oncogene Proteins c-sis - pharmacology | Receptor, Epidermal Growth Factor - physiology | Cytokines - genetics | Receptor, Epidermal Growth Factor - biosynthesis | Human Umbilical Vein Endothelial Cells - drug effects | Arthritis, Rheumatoid - enzymology | Cell Division - drug effects | Gene Expression Regulation - drug effects | Animals | Human Umbilical Vein Endothelial Cells - enzymology | Neovascularization, Pathologic - drug therapy | Protein Kinase Inhibitors - therapeutic use | Quinazolines - therapeutic use | Fibroblasts - drug effects | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Mice | Osteoclasts - enzymology | Arthritis, Experimental - enzymology | Quinazolines - pharmacology | Endothelial Cells - enzymology | Cytokines - biosynthesis | Osteoclasts - drug effects | Endothelial Cells - drug effects | Synovial Fibroblast | Epidermal Growth Factor Receptor | Rheumatoid Arthritis | Erlotinib
Journal Article
Journal Article
Journal Article
Cancer Research, ISSN 0008-5472, 05/2010, Volume 70, Issue 9, pp. 3494 - 3504
The tropism of breast cancer cells for bone and their tendency to induce an osteolytic phenotype are a result of interactions between breast cancer cells and... 
LYSYL OXIDASE | LUNG-CANCER | TGF-BETA | COLLAGENASE-3 MMP-13 | METASTASIS | HORMONE-RELATED PROTEIN | ONCOLOGY | GENE-EXPRESSION | MATRIX-METALLOPROTEINASE-9 | HUMAN BREAST-CANCER | SQUAMOUS-CELL CARCINOMAS | Bone and Bones - pathology | Up-Regulation | Adenocarcinoma - pathology | Osteoprotegerin - biosynthesis | Bone Neoplasms - secondary | Gene Expression Profiling | Mammary Neoplasms, Experimental - metabolism | Matrix Metalloproteinase 13 - biosynthesis | Bone Neoplasms - metabolism | RNA, Messenger - biosynthesis | Matrix Metalloproteinase 9 - metabolism | Matrix Metalloproteinase 9 - genetics | Bone and Bones - metabolism | Mammary Neoplasms, Experimental - pathology | Female | Mammary Neoplasms, Experimental - enzymology | Osteolysis - pathology | Matrix Metalloproteinase 9 - biosynthesis | Osteoclasts - pathology | Bone Neoplasms - enzymology | Signal Transduction | Osteolysis - genetics | Matrix Metalloproteinase 13 - genetics | RNA, Messenger - genetics | Adenocarcinoma - enzymology | Bone and Bones - enzymology | RANK Ligand - biosynthesis | Osteolysis - metabolism | Matrix Metalloproteinase 13 - metabolism | Animals | Osteolysis - enzymology | Cell Line, Tumor | Matrix Metalloproteinase Inhibitors | Mice | Mice, Inbred BALB C | Osteoclasts - enzymology | Enzyme Activation | Transforming Growth Factor beta - metabolism | Index Medicus
Journal Article
Journal Article
Journal of Immunology, ISSN 0022-1767, 08/2010, Volume 185, Issue 4, pp. 2570 - 2579
Sphingosine kinase 1 (SphK1) is an enzyme that converts sphingosine to bioactive sphingosine-1-phosphate. Recent in vitro data suggest a potential role of... 
RHEUMATOID-ARTHRITIS | TUMOR-NECROSIS-FACTOR | ROLES | SPHINGOSINE-1-PHOSPHATE | DISEASE | BIOACTIVE SPHINGOLIPIDS | INDUCTION | IMMUNOLOGY | 1-PHOSPHATE RECEPTORS | EXPRESSION | MODULATION | Foot Joints - metabolism | Immunohistochemistry | Tumor Necrosis Factor-alpha - metabolism | Lysophospholipids - metabolism | Ankle Joint - metabolism | Arthritis - physiopathology | Synovial Membrane - enzymology | Humans | Tumor Necrosis Factor-alpha - genetics | Synovial Membrane - pathology | Immunoblotting | Male | Gene Expression Profiling | Cyclooxygenase 2 - genetics | Foot Joints - enzymology | Female | Sphingosine - metabolism | Synovitis - genetics | Foot Joints - pathology | Joints - metabolism | Severity of Illness Index | Joints - enzymology | Osteoclasts - pathology | Ankle Joint - pathology | Arthritis - pathology | Phosphotransferases (Alcohol Group Acceptor) - deficiency | Suppressor of Cytokine Signaling Proteins - genetics | Ankle Joint - enzymology | Joints - pathology | Mice, Transgenic | Phosphotransferases (Alcohol Group Acceptor) - genetics | Sphingolipids - metabolism | Lysophospholipids - blood | Reverse Transcriptase Polymerase Chain Reaction | Arthritis - enzymology | Osteoclasts - metabolism | Synovial Membrane - metabolism | Mice, Knockout | Synovitis - pathology | Suppressor of Cytokine Signaling 3 Protein | Sphingosine - analogs & derivatives | Animals | Synovitis - enzymology | Cyclooxygenase 2 - metabolism | Tumor Necrosis Factor-alpha - physiology | Sphingosine - blood | Mice | Suppressor of Cytokine Signaling Proteins - metabolism
Journal Article
Journal Article
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 01/2014, Volume 443, Issue 3, pp. 944 - 949
Journal Article
Journal of Cellular Physiology, ISSN 0021-9541, 09/2017, Volume 232, Issue 9, pp. 2396 - 2406
Bone infection is a common and serious complication in the orthopedics field, which often leads to excessive bone destruction and non‐union. Osteoclast is the... 
MAP Kinase | osteoclast | bone resorption | staphylococcal protein A | PATHWAYS | PHYSIOLOGY | SUPPRESSION | CELL BIOLOGY | FRACTURE-FIXATION | OSTEOMYELITIS | DESTRUCTION | PANTON-VALENTINE LEUKOCIDIN | OSTEOBLASTS | AUREUS | TNF-ALPHA | DIFFERENTIATION | Bone and Bones - pathology | RANK Ligand - metabolism | Staphylococcal Infections - pathology | Humans | Case-Control Studies | Bone Resorption - microbiology | Osteomyelitis - pathology | Cell Differentiation | Staphylococcal Infections - microbiology | Osteoclasts - microbiology | Staphylococcus aureus - metabolism | Osteomyelitis - enzymology | Osteoclasts - pathology | Signal Transduction | NFATC Transcription Factors - metabolism | Proto-Oncogene Proteins c-fos - metabolism | Bone and Bones - microbiology | Bone and Bones - enzymology | Staphylococcal Infections - enzymology | Staphylococcus aureus - pathogenicity | Bone Resorption - enzymology | Host-Pathogen Interactions | Animals | Osteomyelitis - microbiology | Bone Resorption - pathology | RAW 264.7 Cells | Mice | Osteoclasts - enzymology | Enzyme Activation | Staphylococcal Protein A - metabolism | Bone Remodeling | Apoptosis | Mitogen-Activated Protein Kinases - metabolism | Medicine, Experimental | Medical research | Development and progression | Health aspects | Staphylococcus aureus infections | Destruction | MAP kinase | Staphylococcus infections | Activation | Nonunion | Infections | Protein A | Bone diseases | Bone resorption | Osteoclastogenesis | Biomedical materials | Orthopedics | Biocompatibility | Osteoclasts | Bone | Differentiation | TRANCE protein | Bone loss | In vitro methods and tests | c-Fos protein | Original | Original s
Journal Article