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PLOS PATHOGENS, ISSN 1553-7366, 04/2019, Volume 15, Issue 4, p. e1007744
Staphylococcus aureus is able to infect virtually all organ systems and is a frequently isolated etiologic agent of osteomyelitis, a common and debilitating... 
INTERLEUKIN-1 | BACTERIAL-INFECTIONS | MICE DEFICIENT | RESORBING ACTIVITY | MACROPHAGE | RECEPTOR | MICROBIOLOGY | NEUTROPHIL RECRUITMENT | VIROLOGY | IL-1-BETA | DIFFERENTIATION | ACTIVATING FACTOR | PARASITOLOGY | Receptors, Interleukin-1 Type I - physiology | Signal Transduction | Staphylococcal Infections - immunology | Mice, Inbred C57BL | Cells, Cultured | Male | Bone Resorption - immunology | Osteomyelitis - metabolism | Osteoclasts - metabolism | Osteomyelitis - immunology | Mice, Knockout | Bone Resorption - microbiology | Animals | Osteoclasts - immunology | Osteomyelitis - microbiology | Myeloid Differentiation Factor 88 - physiology | Bone Resorption - metabolism | Female | Staphylococcus aureus - immunology | Cell Differentiation | Mice | Staphylococcal Infections - microbiology | Osteoclasts - microbiology | Staphylococcal Infections - metabolism | Osteomyelitis | Interleukins | Health aspects | Antibacterial agents | Staphylococcus aureus | Staphylococcus aureus infections | Osteoprogenitor cells | Pediatrics | Animal models | Destruction | Crosstalk | Interleukin | Homeostasis | Infections | Interleukin 1 receptors | Bone (trabecular) | Remodeling | Osteolysis | Antiinfectives and antibacterials | Immunity | Critical components | Bone resorption | Signal transduction | Biomedical materials | Immunology | Etiology | Interleukin 1 | Biocompatibility | Bone loss | Biomedical engineering | Immune system | Immune response | Bacterial infections | Cytokines | Bone remodelling | Adapters | Staphylococcus infections | Inflammation | Bone turnover | Osteoclastogenesis | Pathology | Signaling | Infectious diseases | Antibiotics | MyD88 protein | Osteoclasts | Toxins | Adapter proteins
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