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Cancer Letters, ISSN 0304-3835, 2017, Volume 402, pp. 110 - 116
Abstract Silencing of tumor suppressor and DNA repair genes through methylation plays a role in cancer development, growth and response to therapy in... 
Hematology, Oncology and Palliative Medicine | DNA methylation | HSP90 | Ganetespib | Pancreatic cancer | Colorectal cancer | HYPERMETHYLATION | TUMOR-SUPPRESSOR GENES | CELL-LINES | THERAPY | ONCOLOGY | METHYLTRANSFERASES | PATHWAY | COLORECTAL-CANCER | EXPRESSION | Colonic Neoplasms - genetics | Pancreatic Neoplasms - metabolism | Colonic Neoplasms - drug therapy | Humans | RNA, Messenger - metabolism | Colonic Neoplasms - metabolism | DNA (Cytosine-5-)-Methyltransferases - metabolism | Pancreatic Neoplasms - drug therapy | Osteonectin - genetics | Transfection | Female | Antineoplastic Agents - pharmacology | Epigenesis, Genetic - drug effects | Gene Expression Regulation, Neoplastic - drug effects | DNA (Cytosine-5-)-Methyltransferase 1 | HCT116 Cells | Pancreatic Neoplasms - pathology | RNA, Messenger - genetics | Cyclin-Dependent Kinase Inhibitor p16 - genetics | Osteonectin - metabolism | Pancreatic Neoplasms - genetics | HT29 Cells | Triazoles - pharmacology | Xenograft Model Antitumor Assays | DNA (Cytosine-5-)-Methyltransferases - genetics | Animals | 5-Methylcytosine - metabolism | Signal Transduction - drug effects | HSP90 Heat-Shock Proteins - antagonists & inhibitors | Mice, Nude | Colonic Neoplasms - pathology | MutL Protein Homolog 1 - genetics | Cyclin-Dependent Kinase Inhibitor p16 - metabolism | HSP90 Heat-Shock Proteins - metabolism | Cell Proliferation - drug effects | Mice, Inbred BALB C | MutL Protein Homolog 1 - metabolism | DNA Methylation - drug effects | Proteins | Epigenetic inheritance | Colon cancer | Methyltransferases | Analysis | Heat shock proteins | Development and progression | Methylation | Biotechnology | Hsp90 protein | Transcription | Genes | Genomes | Kinases | DNA repair | Genotype & phenotype | Cell growth | Cell cycle | Cytosine | Inhibition | Colon | Deoxyribonucleic acid--DNA | Enzymes | DNMT1 protein | Cell division | Tumor cell lines | Gene expression | Gene silencing | Epigenetics | DNA methyltransferase | Tumor suppressor genes | Tumors | Heat shock | Cancer | Index Medicus
Journal Article
Pain, ISSN 0304-3959, 06/2012, Volume 153, Issue 6, pp. 1167 - 1179
Journal Article
American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 06/2015, Volume 308, Issue 12, pp. C972 - C982
To investigate the role of secreted protein acidic and rich in cysteine (SPARC) in age-related cardiac inflammation, we studied six groups of mice: young (3-5... 
Heart | Secreted protein acidic and rich in cysteine | Aging | Inflammation | Macrophage | FIBROSIS | PHYSIOLOGY | SPARC | secreted protein acidic and rich in cysteine | HEART-FAILURE | macrophage | heart | NEUTROPHIL RECRUITMENT | DIASTOLIC DYSFUNCTION | CELL BIOLOGY | inflammation | MATRICELLULAR PROTEIN | MAJOR SHAREHOLDERS | EXTRACELLULAR-MATRIX | aging | CARDIOVASCULAR-DISEASE ENTERPRISES | EXPRESSION | Receptors, CCR2 - genetics | Myocardium - immunology | Age Factors | Ventricular Function, Left | Chemokine CX3CL1 - metabolism | RNA, Messenger - metabolism | Osteonectin - deficiency | Osteonectin - genetics | Aging - genetics | Myocardium - metabolism | Inflammation Mediators - metabolism | Myocarditis - prevention & control | Chemokine CCL5 - metabolism | Myocarditis - immunology | Myocarditis - genetics | Receptors, CXCR3 - metabolism | Osteonectin - immunology | Mice, Inbred C57BL | Cells, Cultured | Gene Expression Regulation | Macrophages, Peritoneal - immunology | Osteonectin - metabolism | Chemokine CX3CL1 - genetics | Myocardium - pathology | Chemotaxis | Aging - pathology | Mice, Knockout | Phenotype | Receptors, CCR2 - metabolism | Animals | Receptors, CXCR3 - genetics | Chemokine CCL5 - genetics | Myocarditis - metabolism | Myocarditis - pathology | Macrophages, Peritoneal - metabolism | Myocarditis - physiopathology | Aging - metabolism | Physiological aspects | Cysteine | Macrophages | Health aspects | Proteins | Immunohistochemistry | Ultrasonic imaging | Cytokines | Rodents | Index Medicus | Call for Papers
Journal Article
Molecular Pain, ISSN 1744-8069, 8/2011, Volume 7, Issue 1, pp. 65 - 65
Background: The extracellular matrix protein SPARC (Secreted Protein, Acidic, Rich in Cysteine) has been linked to degeneration of the intervertebral discs and... 
Back pain | SPARC | Disc degeneration | Intervertebral disc | Aging | DNA methylation | Epigenetics | Gene expression | NUCLEUS PULPOSUS | intervertebral disc | CHROMATIN | MOUSE | CPG METHYLATION | NEUROSCIENCES | epigenetics | DEGENERATION | NEUROPATHIC PAIN | back pain | LUMBAR SPINE | GENE-EXPRESSION | disc degeneration | MICE | aging | gene expression | Gene Silencing - drug effects | Humans | Aging - drug effects | Male | Promoter Regions, Genetic - genetics | Intervertebral Disc Degeneration - pathology | Osteonectin - deficiency | Osteonectin - genetics | Aging - genetics | Low Back Pain - complications | Low Back Pain - genetics | Adult | Behavior, Animal - drug effects | Female | Intervertebral Disc - pathology | Low Back Pain - pathology | Intervertebral Disc Degeneration - genetics | Mice, Inbred C57BL | Osteonectin - metabolism | Chronic Pain - genetics | DNA Methylation - genetics | Intervertebral Disc - metabolism | Chronic Pain - pathology | Intervertebral Disc Degeneration - complications | Chronic Pain - complications | Gene Expression Regulation - drug effects | Azacitidine - pharmacology | Animals | Intervertebral Disc - drug effects | Mice | DNA Methylation - drug effects | Medicine | Medical research | Data collection | Behavior | Experiments | Deoxyribonucleic acid--DNA | Manuscripts | Index Medicus
Journal Article
Nephrology Dialysis Transplantation, ISSN 0931-0509, 1/2009, Volume 24, Issue 1, pp. 62 - 72
Journal Article
Cell Reports, ISSN 2211-1247, 09/2014, Volume 8, Issue 6, pp. 1905 - 1918
Journal Article
Journal Article
Cancer Research, ISSN 0008-5472, 07/2011, Volume 71, Issue 14, pp. 4908 - 4919
Secreted protein acidic and rich in cysteine (SPARC) participates in the regulation of morphogenesis and cellular differentiation through its modulation of... 
MATRIX | STEM-CELLS | ACTIVATION | INHIBITION | ONCOLOGY | NOTCH PATHWAY | STAT3 | CENTRAL-NERVOUS-SYSTEM | TUMOR-GROWTH | EXPRESSION | CANCER-THERAPY | Neurons - pathology | RNA, Small Interfering - genetics | DNA, Complementary - administration & dosage | Humans | Brain Neoplasms - pathology | DNA, Complementary - genetics | Medulloblastoma - therapy | Brain Neoplasms - metabolism | Osteonectin - genetics | Transfection | Medulloblastoma - pathology | Plasmids - genetics | Adenoviridae - genetics | Biomarkers, Tumor - metabolism | Medulloblastoma - genetics | Cell Differentiation - physiology | Interleukin-6 - metabolism | STAT3 Transcription Factor - metabolism | Brain Neoplasms - genetics | Osteonectin - metabolism | Medulloblastoma - metabolism | Random Allocation | Receptor, Notch1 - metabolism | Osteonectin - biosynthesis | Xenograft Model Antitumor Assays | Animals | Brain Neoplasms - therapy | Cell Line, Tumor | Mice | RNA, Small Interfering - administration & dosage | Genetic Therapy - methods | Enhancer-of-split protein | Cysteine | Nestin | Phosphorylation | Transcription factors | Stat3 protein | Interleukin 6 | Morphogenesis | Signal transduction | Medulloblastoma | Osteonectin | Neurofilaments | Helix-loop-helix proteins | Notch protein | Immunoreactivity | Differentiation | Cancer | Tumors | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 2010, Volume 5, Issue 11, pp. e15074 - e15074
Background: Despite many attempts to establish pre-treatment prognostic markers to understand the clinical biology of esophageal adenocarcinoma (EAC),... 
SURVIVAL | MATRIX | HEPATOCELLULAR-CARCINOMA | OSTEOPONTIN | SPARC | MATRICELLULAR PROTEIN | GROWTH | BIOLOGY | EXPRESSION | CANCER | PROGRESSION | Osteopontin - genetics | Multivariate Analysis | Prognosis | Humans | Middle Aged | Kaplan-Meier Estimate | Proportional Hazards Models | Male | Gene Expression Profiling | Reverse Transcriptase Polymerase Chain Reaction | Adenocarcinoma - diagnosis | Osteonectin - genetics | Esophageal Neoplasms - genetics | Adult | Esophageal Neoplasms - diagnosis | Female | Adenocarcinoma - genetics | Aged | Biomarkers, Tumor - genetics | Oligonucleotide Array Sequence Analysis - statistics & numerical data | Cluster Analysis | Adenocarcinoma | Cancer patients | DNA microarrays | Genes | Genomics | Development and progression | Genomes | Biological markers | Gene expression | Esophageal cancer | Formaldehyde | Tumors | Oncology | Biology | Statistical methods | Metastasis | Paraffin | Tissues | Epidemiology | Subgroups | Proteins | Cell growth | Endoscopes | Surgery | Hepatology | Gastroenterology | Extracellular matrix | Bioindicators | Pretreatment | Bioinformatics | Deoxyribonucleic acid--DNA | Statistical analysis | Regression analysis | Patients | Esophagus | Polymerase chain reaction | Biopsy | Medical prognosis | Biomarkers | Prostate | Cancer | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article