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Fertility and Sterility, ISSN 0015-0282, 2010, Volume 93, Issue 6, pp. 1750 - 1773
Journal Article
Scientific Reports, ISSN 2045-2322, 10/2016, Volume 6, Issue 1, pp. 34713 - 34713
Effective pulp-capping materials must have antibacterial properties and induce dentin bridge formation; however, many current materials do not satisfy clinical... 
MINERAL TRIOXIDE AGGREGATE | BIOCOMPATIBILITY | IN-VITRO | REPAIR | CALCIUM HYDROXIDE | MULTIDISCIPLINARY SCIENCES | PORTLAND-CEMENT | TISSUE | TEETH | PROLIFERATION | CYTOTOXICITY | Molar - surgery | Sialoglycoproteins - genetics | Humans | Sialoglycoproteins - metabolism | Alkaline Phosphatase - metabolism | Osteocalcin - genetics | Dental Cements - pharmacology | Stem Cells - cytology | Phosphoproteins - metabolism | Young Adult | Osteonectin - genetics | Molar - cytology | Tooth Extraction | Methacrylates - pharmacology | Biological Assay | Osteoblasts - cytology | Wound Healing - drug effects | Extracellular Matrix Proteins - metabolism | Osteogenesis - genetics | Cell Survival - drug effects | Alkaline Phosphatase - genetics | Bicuspid - cytology | Osteocalcin - metabolism | Extracellular Matrix Proteins - genetics | Osteoblasts - drug effects | Osteogenesis - drug effects | Dental Pulp - cytology | Osteonectin - metabolism | Dental Pulp - drug effects | Phosphoproteins - genetics | Cell Adhesion - drug effects | Gene Expression Regulation - drug effects | Cell Differentiation - drug effects | Bicuspid - surgery | Adolescent | Stem Cells - drug effects | Primary Cell Culture | Quaternary Ammonium Compounds - pharmacology | Dentin | Wound healing | Leukocyte migration | Calcium | Stem cells | Biocompatibility | Cell migration | Osteogenesis | Dental pulp | Pulp | Index Medicus
Journal Article
Journal Article
Stroke, ISSN 0039-2499, 2001, Volume 32, Issue 4, pp. 1036 - 1042
Background and Purpose-Approximately 6% of human beings harbor an unruptured intracranial aneurysm. Each year in the United States, >30 000 people suffer a... 
Cerebral aneurysm | Stroke | Gene expression | Hemorrhagic | SYSTEM | LOCALIZATION | AORTIC-ANEURYSM | cerebral aneurysm | stroke, hemorrhagic | CLINICAL NEUROLOGY | CEREBRAL ANEURYSMS | FIBRONECTIN EXPRESSION | PRESSURE | PATHWAY | PERIPHERAL VASCULAR DISEASE | EXTRACELLULAR-MATRIX | PROTEIN SPARC | gene expression | CATHEPSIN-B | Inflammation - pathology | Cell Adhesion Molecules - genetics | Humans | Glycoproteins - metabolism | Child, Preschool | RNA, Messenger - metabolism | Osteonectin - genetics | Regeneration - genetics | Middle Cerebral Artery - metabolism | Tissue Inhibitor of Metalloproteinase-3 - metabolism | Cell Cycle Proteins - genetics | Female | Membrane Proteins - metabolism | Wound Healing - genetics | Extracellular Matrix Proteins - metabolism | Expressed Sequence Tags | Tissue Inhibitor of Metalloproteinase-3 - genetics | Calcium-Binding Proteins - metabolism | Glycoproteins - genetics | Gene Expression | Intracranial Aneurysm - genetics | Membrane Proteins - genetics | Extracellular Matrix Proteins - genetics | Gene Frequency | Cell Cycle Proteins - metabolism | Osteonectin - metabolism | Gene Expression Profiling - methods | Middle Cerebral Artery - pathology | Intracranial Aneurysm - metabolism | Cell Adhesion Molecules - metabolism | Cerebral Angiography | Intracranial Aneurysm - pathology | Calcium-Binding Proteins - genetics | Index Medicus
Journal Article
Arthritis Research and Therapy, ISSN 1478-6354, 04/2010, Volume 12, Issue 2, pp. R60 - R60
Journal Article
Cancer Letters, ISSN 0304-3835, 2013, Volume 341, Issue 2, pp. 195 - 203
Highlights • EF31 and UBS109 are more potent inhibitors of DNA methylation than curcumin. • EF31 and UBS109 inhibit DNA methylation through NF-κB and HSP90... 
Hematology, Oncology and Palliative Medicine | UBS109 | DNA methylation | Curcumin | Pancreatic cancer | EF31 | CELLS | APOPTOSIS | SPARC | ANGIOGENESIS | DUCTAL ADENOCARCINOMA | TRANSCRIPTION | INTRAEPITHELIAL NEOPLASIA | ABERRANT METHYLATION | INHIBITION | ONCOLOGY | CPG ISLANDS | Immunohistochemistry | Curcumin - analogs & derivatives | Pancreatic Neoplasms - metabolism | Cadherins - metabolism | Humans | DNA (Cytosine-5-)-Methyltransferases - metabolism | Dose-Response Relationship, Drug | Pancreatic Neoplasms - drug therapy | Osteonectin - genetics | Cytosine - metabolism | Female | HSP90 Heat-Shock Proteins - genetics | Gene Expression Regulation, Neoplastic - drug effects | Cadherins - genetics | Cell Survival - drug effects | Pyridines | DNA (Cytosine-5-)-Methyltransferase 1 | Curcumin - pharmacology | Osteonectin - metabolism | Pancreatic Neoplasms - genetics | Piperidones - pharmacology | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Xenograft Model Antitumor Assays | DNA (Cytosine-5-)-Methyltransferases - genetics | Animals | Mice, Nude | Cell Line, Tumor | HSP90 Heat-Shock Proteins - metabolism | Cell Proliferation - drug effects | Mice | DNA Methylation - drug effects | Development and progression | Genetic aspects | Methylation | DNA | Cancer | Proteins | Cyclin-dependent kinases | Cell cycle | Epigenetics | Pancreas | Kinases | Gene expression | Cancer therapies | Deoxyribonucleic acid--DNA | Apoptosis | Index Medicus
Journal Article
Journal Article
Journal Article
Journal of Pathology, ISSN 0022-3417, 01/2011, Volume 223, Issue 1, pp. 72 - 80
Endometrial carcinosarcomas (ECSs) undergo a true epithelial‐mesenchymal transition (EMT). The molecular determinants of the EMT in vivo are unclear, although... 
miRNA | endometrial carcinosarcoma | differentiation | epithelial‐mesenchymal transition | E‐cadherin | epithelial-mesenchymal transition | E-cadherin | CANCER-CELLS | STEM-CELLS | PHENOTYPE | PATHOLOGY | MIR-200 FAMILY | REPRESSORS ZEB1 | EMT | BHLH FACTORS | ONCOLOGY | DISEASE | EXPRESSION | PROGRESSION | Carcinosarcoma - pathology | Carcinosarcoma - metabolism | Cadherins - metabolism | Oligonucleotide Array Sequence Analysis - methods | Homeodomain Proteins - metabolism | Humans | Endometrial Neoplasms - metabolism | Gene Expression Regulation, Neoplastic | Gene Expression Profiling - methods | Epithelial-Mesenchymal Transition - genetics | Neoplasm Proteins - metabolism | Zinc Finger E-box Binding Homeobox 2 | Transcription Factors - metabolism | Endometrial Neoplasms - genetics | Reverse Transcriptase Polymerase Chain Reaction - methods | RNA, Neoplasm - genetics | Endometrial Neoplasms - pathology | Female | Carcinosarcoma - genetics | MicroRNAs - genetics | Repressor Proteins - metabolism | Zinc Finger E-box-Binding Homeobox 1 | Index Medicus | Immunohistochemistry | Vimentin | Transcription factors | Mesenchyme | Transcription | Data processing | Transforming growth factor- beta 1 | Cadherin | Polymerase chain reaction | caveolin-1 | Molecular modelling | N-Cadherin | ECS | Repressors | E-Cadherin | Osteonectin | Endometrium
Journal Article
Spine, ISSN 0362-2436, 11/2005, Volume 30, Issue 22, pp. 2510 - 2515
Journal Article