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Atherosclerosis, ISSN 0021-9150, 2014, Volume 236, Issue 2, pp. 327 - 337
Abstract Atherosclerosis is a chronic inflammatory process of the vessel wall with systemic correlates. It is now well established that patients' outcome is... 
Cardiovascular | Osteopontin | Coronary artery disease | Atherosclerosis | CARDIAC & CARDIOVASCULAR SYSTEMS | ACCELERATED ATHEROSCLEROSIS | PLASMA OSTEOPONTIN | MESSENGER-RNA | THROMBIN-CLEAVED OSTEOPONTIN | ENDOTHELIAL-CELLS | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | CHRONIC KIDNEY-DISEASE | UP-REGULATION | CORONARY-ARTERY-DISEASE | DENSITY-LIPOPROTEIN INCREASES | Osteopontin - blood | Osteopontin - genetics | Smoking - adverse effects | Osteopontin - deficiency | Prognosis | Apolipoproteins E - deficiency | Atherosclerosis - genetics | Humans | Renal Insufficiency, Chronic - complications | Molecular Targeted Therapy | Coronary Artery Disease - blood | Osteopontin - secretion | Peptide Fragments - physiology | Renal Insufficiency, Chronic - metabolism | Atherosclerosis - etiology | Plaque, Atherosclerotic - pathology | Osteopontin - physiology | Integrins - physiology | Diabetic Angiopathies - metabolism | Atherosclerosis - physiopathology | Risk Factors | Cells, Cultured | Rats | Mice, Transgenic | Inflammation | Calcinosis - blood | Protein Isoforms - physiology | Mice, Knockout | Atherosclerosis - blood | Smoking - metabolism | Animals | Coronary Artery Disease - complications | Biomarkers | Calcinosis - prevention & control | Mice | Protein Processing, Post-Translational | Matrix Metalloproteinases - metabolism | Thrombin - metabolism | Blood circulation disorders | Analysis | Integrins
Journal Article
Journal of cellular physiology, ISSN 1097-4652, 2018, Volume 234, Issue 6, pp. 8988 - 8997
Liver biopsy is the current reliable way of evaluating liver fibrosis. However, no specific sera biomarker could be applied in clinical diagnosis. As the... 
thrombin‐cleaved osteopontin (Thr‐OPN) | full‐length osteopontin (FL‐OPN) | integrins | liver fibrosis | hepatic stellate cells (HSCs) | full-length osteopontin (FL-OPN) | thrombin-cleaved osteopontin (Thr-OPN) | FIBROSIS | DOMAIN | PHYSIOLOGY | SVVYGLR MOTIF | COLLAGEN | ATHEROSCLEROSIS | CYTOKINE | CELL BIOLOGY | ALPHABETA | DISEASE | Osteopontin - genetics | Up-Regulation | Liver - pathology | Cell Proliferation | Osteopontin - deficiency | Carbon Tetrachloride | Humans | Hepatic Stellate Cells - metabolism | Male | Osteopontin - metabolism | Case-Control Studies | NF-kappa B | Liver Cirrhosis, Experimental - pathology | Chemical and Drug Induced Liver Injury - pathology | Chemical and Drug Induced Liver Injury - etiology | Hepatic Stellate Cells - pathology | Peptide Fragments - metabolism | Liver - metabolism | Mice, Inbred C57BL | Cells, Cultured | Integrin alpha Chains - metabolism | Mice, Knockout | Animals | Integrin alpha4 - metabolism | Chemical and Drug Induced Liver Injury - metabolism | Liver Cirrhosis, Experimental - chemically induced | Thrombin - metabolism | Liver Cirrhosis, Experimental - metabolism | Cell Movement | Mitogen-Activated Protein Kinases - metabolism | Hepatitis | Analysis | Liver | Thrombin | Mitogens | Liver cirrhosis | Integrins | Animal models | Peptides | Migration | Activation | Proteins | Signal transduction | Rodents | Biocompatibility | Hepatitis B virus | Stellate cells | Liver diseases | Osteopontin | MAP kinase | Patients | Cirrhosis | Biopsy | Fibrosis | Biomarkers | Mice | Diagnostic systems | Hepatitis B | Bile | Neutralization | Original | Original s
Journal Article
Laboratory investigation, ISSN 1530-0307, 2018, Volume 99, Issue 3, pp. 331 - 345
Osteopontin (OPN) is critical for ischemia-induced neovascularization. Unlike rodents, humans express three OPN isoforms (a, b, and c); however, the roles of... 
MEDICINE, RESEARCH & EXPERIMENTAL | ANGIOGENESIS | PHOSPHORYLATION | TRANSGLUTAMINASE | O-GLYCOSYLATION | GROWTH | CELL-ADHESION | PATHOLOGY | IDENTIFICATION | SPLICE VARIANTS | EXPRESSION | POSTTRANSLATIONAL MODIFICATION | Osteopontin - genetics | Recombinant Proteins - therapeutic use | Osteopontin - deficiency | Humans | Osteopontin - therapeutic use | Male | Ischemia - genetics | Arterial Occlusive Diseases - pathology | Vascular Remodeling - physiology | Arterial Occlusive Diseases - physiopathology | Ischemia - pathology | Disease Models, Animal | Osteopontin - physiology | Macrophages - physiology | Macrophages - pathology | Cell Survival | Mice, Inbred C57BL | Recombinant Proteins - genetics | Ischemia - physiopathology | Protein Isoforms - physiology | Mice, Knockout | Animals | Vascular Remodeling - genetics | Mice | Neovascularization, Physiologic | Apoptosis | Arterial Occlusive Diseases - therapy | Cell Movement | Protein Isoforms - genetics | Wheel running | Polarization | Leukocyte migration | Arterioles | Macrophages | Arteries | Accumulation | Cell adhesion & migration | Vascularization | Biomedical materials | Ischemia | Rodents | Surgery | Biocompatibility | Cell survival | Therapeutic applications | Osteopontin | Survival | Resistance | Perfusion | Isoforms | In vivo methods and tests | Conductance | Cell migration | Migration | Human Osteopontin Isoforms | Peripheral Artery Disease | Collateral Vessel Formation | Inflammation | OPNb | OPNa | Arteriogenesis | OPNc | Limb Ischemia | Neovascularization | Macrophage
Journal Article
Journal Article
PloS one, ISSN 1932-6203, 06/2018, Volume 13, Issue 6, p. e0198790
Journal Article
Immunology, ISSN 0019-2805, 2011, Volume 132, Issue 1, pp. 39 - 48
Journal Article