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World journal of gastroenterology : WJG, ISSN 1007-9327, 2015, Volume 21, Issue 37, pp. 10573 - 10583
Journal Article
Journal Article
PloS one, ISSN 1932-6203, 06/2013, Volume 8, Issue 6, p. e68197
Free fatty acids (FFAs), elevated in metabolic syndrome and diabetes, play a crucial role in the development of atherosclerotic cardiovascular disease, and... 
SUPPLEMENTATION | ARTERY ENDOTHELIAL-CELLS | PROTEIN-KINASE | MULTIDISCIPLINARY SCIENCES | DISEASE | CALCIUM | CALCIFICATION | RISK | FREE FATTY-ACID | DEPENDENT ACTIVATION | EXPRESSION | Calcinosis - genetics | Carotid Arteries - metabolism | Osteopontin - genetics | Palmitic Acid - metabolism | Coenzyme A Ligases - genetics | Muscle, Smooth, Vascular - metabolism | Calcium - metabolism | Gene Expression - genetics | Homeodomain Proteins - metabolism | Humans | Eicosapentaenoic Acid - genetics | Myocytes, Smooth Muscle - pathology | NF-kappa B - metabolism | Osteopontin - metabolism | Aorta - metabolism | Promoter Regions, Genetic - genetics | Coenzyme A Ligases - metabolism | Cell Differentiation - genetics | Caspases - metabolism | Bone Morphogenetic Protein 2 - metabolism | Calcinosis - metabolism | Plaque, Atherosclerotic - pathology | Cell Differentiation - physiology | Myocytes, Smooth Muscle - metabolism | Bone Morphogenetic Protein 2 - genetics | Macrophages - pathology | Plaque, Atherosclerotic - metabolism | Caspases - genetics | Cells, Cultured | Eicosapentaenoic Acid - metabolism | Homeodomain Proteins - genetics | Aorta - pathology | Osteoblasts - pathology | Macrophages - metabolism | Muscle, Smooth, Vascular - pathology | NF-kappa B - genetics | Carotid Arteries - pathology | Calcinosis - pathology | Osteoblasts - metabolism | Immunohistochemistry | Smooth muscle | Cardiovascular disease | Palmitic acid | Osteoblasts | Proteins | Mineralization | Biocompatibility | Aorta | Inhibition | NF-κB protein | Cytokines | Osteopontin | Metabolism | Gene expression | Fatty acids | Studies | Osteoblastogenesis | Inhibitors | Arteriosclerosis | Calcification | Cardiovascular diseases | Metabolic disorders | Calcification (ectopic) | Health sciences | Oxidative stress | Calcium | Laboratories | Chains | Lipids | Activation | Kinases | Macrophages | Carotid arteries | Arteries | Msx2 protein | Rodents | Atherosclerosis | Tumor necrosis factor-TNF | Carotid artery | Deposition | Plaques | University graduates | Bone morphogenetic protein 2 | Diabetes mellitus | Muscles | Caspase | siRNA | Medicine | Antibiotics | Kidney diseases | Diabetes | Differentiation | Eicosapentaenoic acid | Apoptosis | Pharmaceuticals
Journal Article
Journal of molecular and cellular cardiology, ISSN 0022-2828, 2016, Volume 91, pp. 228 - 237
Journal Article
Journal of bone and mineral research, ISSN 0884-0431, 2011, Volume 26, Issue 4, pp. 777 - 791
Mesenchymal stem cell commitment to an osteoprogenitor lineage requires the activity of Runx2, a molecule implicated in the etiopathology of multiple... 
CLEIDOCRANIAL DYSPLASIA | CRANIOFACIAL DEVELOPMENT | TRANSGENIC ANIMAL | NELL‐1 | RUNX2 | NELL-1 | craniofacial development | transgenic animal | cleidocranial dysplasia | CELLS | OSTEOBLAST DIFFERENTIATION | BONE MORPHOGENETIC PROTEIN-2 | CRANIOSYNOSTOSIS-ASSOCIATED GENE | CBFA1-DEFICIENT MICE | BIOLOGY | ENDOCRINOLOGY & METABOLISM | CBFA1 | TRANSCRIPTION FACTOR | EXPRESSION | RNA, Small Interfering - genetics | Osteogenesis - physiology | Core Binding Factor Alpha 1 Subunit - deficiency | Gene Expression - genetics | Alkaline Phosphatase - metabolism | Osteocalcin - genetics | Extracellular Signal-Regulated MAP Kinases - antagonists & inhibitors | Glycoproteins - pharmacology | Core Binding Factor Alpha 1 Subunit - metabolism | Skull - pathology | Skull - abnormalities | Craniofacial Abnormalities - pathology | p38 Mitogen-Activated Protein Kinases - metabolism | Cleidocranial Dysplasia - metabolism | Phosphorylation - drug effects | Craniofacial Abnormalities - genetics | Animals, Newborn | Glycoproteins - genetics | Skull - metabolism | Alkaline Phosphatase - genetics | JNK Mitogen-Activated Protein Kinases - antagonists & inhibitors | Calcification, Physiologic - drug effects | Femur - metabolism | Calcification, Physiologic - physiology | Mice, Transgenic | Mice, Knockout | Cleidocranial Dysplasia - genetics | Skull - cytology | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Sp7 Transcription Factor | Dura Mater - metabolism | Mice | Calcium-Binding Proteins - pharmacology | Calcium-Binding Proteins - genetics | Osteopontin - genetics | Glycoproteins - metabolism | JNK Mitogen-Activated Protein Kinases - metabolism | Osteopontin - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Tibia - metabolism | Osteoblasts - cytology | Cell Differentiation - physiology | Craniofacial Abnormalities - metabolism | Calcium-Binding Proteins - metabolism | Haploinsufficiency - genetics | Osteocalcin - metabolism | Tissue Culture Techniques | Osteogenesis - drug effects | Cells, Cultured | Transcription Factors - genetics | Mice, Inbred Strains | Cleidocranial Dysplasia - pathology | Skull - embryology | Animals | Heterozygote | Protein Kinase Inhibitors - pharmacology | Osteoblasts - metabolism | Core Binding Factor Alpha 1 Subunit - genetics | Original
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1079-5642, 02/2015, Volume 35, Issue 2, pp. 430 - 438
OBJECTIVE—Blood vessel hemodynamics have profound influences on function and structure of vascular cells. One of the main mechanical forces influencing... 
MEF2B | vascular remodeling | oxidative stress | Nox1 | NADPH OXIDASE | AORTIC-ANEURYSMS | PROTEIN-KINASE | CONTRACTILE PHENOTYPE | MEDIATED ACTIVATION | TRANSCRIPTIONAL CONTROL | NAD(P)H OXIDASES | MOLECULAR REGULATION | PERIPHERAL VASCULAR DISEASE | MECHANICAL STRETCH | TISSUE PERFUSION | HEMATOLOGY | MEF2 Transcription Factors - genetics | Reactive Oxygen Species - metabolism | Mechanotransduction, Cellular - drug effects | Muscle, Smooth, Vascular - metabolism | Osteopontin - metabolism | RNA, Messenger - metabolism | Matrix Metalloproteinase 9 - metabolism | Transfection | RNA Interference | Time Factors | Muscle Proteins - metabolism | Cytoskeletal Proteins - metabolism | Microfilament Proteins - metabolism | Pressoreceptors - metabolism | Vascular Remodeling - drug effects | Myocytes, Smooth Muscle - drug effects | NADH, NADPH Oxidoreductases - metabolism | Myocytes, Smooth Muscle - metabolism | Muscle, Smooth, Vascular - drug effects | Biomarkers - metabolism | Calcium-Binding Proteins - metabolism | NADH, NADPH Oxidoreductases - genetics | Cells, Cultured | Enzyme Inhibitors - pharmacology | Rats | NADPH Oxidase 1 | Phenotype | Animals | MEF2 Transcription Factors - metabolism | NADH, NADPH Oxidoreductases - antagonists & inhibitors | Cell Movement | Vascular Remodeling | Vascular Dysfunction | Oxidative Stress | Cyclic stretch | Smooth Muscle Cell Differentiation
Journal Article
The Journal of clinical investigation, ISSN 1558-8238, 2017, Volume 127, Issue 5, pp. 1600 - 1612
The cardiac extracellular matrix (ECM) not only provides mechanical support, but also transduces essential molecular signals in health and disease. Following... 
ISCHEMIA-REPERFUSION INJURY | MEDICINE, RESEARCH & EXPERIMENTAL | VOLUME OVERLOAD | HEART-FAILURE | CHRONIC PRESSURE-OVERLOAD | LEFT-VENTRICULAR DILATION | MATRICELLULAR PROTEINS | SMOOTH-MUSCLE-CELLS | ANGIOTENSIN-II | CARDIAC FIBROSIS | MICE LACKING OSTEOPONTIN | Inflammation - pathology | Leukocytes - pathology | Myocardium - immunology | Myocytes, Cardiac - immunology | Humans | Extracellular Matrix - immunology | Extracellular Matrix - metabolism | Stress, Mechanical | Heart Failure, Diastolic - metabolism | Myocardial Infarction - immunology | Leukocytes - immunology | Intercellular Signaling Peptides and Proteins - metabolism | Signal Transduction - immunology | Inflammation - metabolism | Myocardial Infarction - pathology | Myocardium - metabolism | Extracellular Matrix Proteins - metabolism | Myocardium - pathology | Inflammation - immunology | Myocardial Infarction - metabolism | Heart Failure, Diastolic - pathology | Myocytes, Cardiac - pathology | Animals | Extracellular Matrix Proteins - immunology | Heart Failure, Diastolic - immunology | Myocytes, Cardiac - metabolism | Extracellular Matrix - pathology | Leukocytes - metabolism | Intercellular Signaling Peptides and Proteins - immunology | Extracellular matrix | Care and treatment | Research | Regeneration (Biology) | Heart attack | Heart failure | Hypertension | Myocardial infarction | Heart attacks | Pathogenesis | Cardiomyocytes | Inflammation | Metabolism | Proteins | Ischemia | Collagen | Rodents | Fibroblasts | Plastics | Growth factors | Heart diseases | Binding sites
Journal Article
International Journal of Radiation Oncology, Biology, Physics, ISSN 0360-3016, 2007, Volume 69, Issue 1, pp. 167 - 175
Purpose To investigate the expression pattern of hypoxia-induced proteins identified as being involved in malignant progression of head-and-neck squamous cell... 
Radiology | Hematology, Oncology and Palliative Medicine | Head-and-neck cancer | Hypoxia | Prognosis | Marker panel | ENDOGENOUS MARKERS | TUMOR HYPOXIA | EPH RECEPTORS | GENE | ONCOLOGY | EPHRIN-A1 | OXYGENATION | GROWTH | RADIOTHERAPY | IDENTIFICATION | CANCER | RADIOLOGY, NUCLEAR MEDICINE & MEDICAL IMAGING | Cell Hypoxia - physiology | Ephrin-A1 - metabolism | Carcinoma, Squamous Cell - metabolism | Carcinoma, Squamous Cell - pathology | Humans | Middle Aged | Galectin 1 - metabolism | Male | Osteopontin - metabolism | Neoplasm Proteins - metabolism | Head and Neck Neoplasms - metabolism | Oxygen - metabolism | Carbonic Anhydrases - metabolism | Carcinoma, Squamous Cell - mortality | I-kappa B Kinase - metabolism | Antigens, Neoplasm - metabolism | Biomarkers, Tumor - metabolism | Female | Membrane Proteins - metabolism | Carbonic Anhydrase IX | Proto-Oncogene Proteins - metabolism | Tumor Suppressor Proteins - metabolism | Disease Progression | Head and Neck Neoplasms - pathology | Tetrahydrofolate Dehydrogenase - metabolism | Analysis of Variance | Partial Pressure | Protein-Lysine 6-Oxidase - metabolism | Cell Line, Tumor | Head and Neck Neoplasms - mortality | Proteins | Development and progression | Squamous cell carcinoma | HEMOGLOBIN | HEAD | CARBONIC ANHYDRASE | OXYGEN | GROWTH FACTORS | MULTIVARIATE ANALYSIS | THERAPY | OXIDASES | ANOXIA | CARCINOMAS | RADIOLOGY AND NUCLEAR MEDICINE | PARTIAL PRESSURE | NECK | CONNECTIVE TISSUE
Journal Article