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Nature cell biology, ISSN 1476-4679, 07/2018, Volume 20, Issue 8, pp. 954 - 965
BRCA1 deficiencies cause breast, ovarian, prostate and other cancers, and render tumours hypersensitive to poly(ADP-ribose) polymerase (PARP) inhibitors. To... 
Life Sciences & Biomedicine | Science & Technology | Cell Biology | Osteosarcoma - drug therapy | Mad2 Proteins - metabolism | Humans | Multiprotein Complexes | Ovarian Neoplasms - pathology | Bone Neoplasms - pathology | DNA Breaks, Double-Stranded | Bone Neoplasms - metabolism | Breast Neoplasms - metabolism | Dose-Response Relationship, Drug | Ovarian Neoplasms - genetics | Telomere-Binding Proteins - genetics | DNA End-Joining Repair | HEK293 Cells | Female | Bone Neoplasms - genetics | Ovarian Neoplasms - metabolism | Bone Neoplasms - drug therapy | BRCA1 Protein - deficiency | Telomere-Binding Proteins - metabolism | Ovarian Neoplasms - drug therapy | Osteosarcoma - metabolism | DNA-Binding Proteins | Tumor Suppressor p53-Binding Protein 1 - metabolism | Recombinational DNA Repair | Tumor Suppressor p53-Binding Protein 1 - genetics | Cisplatin - pharmacology | Breast Neoplasms - drug therapy | Proteins - genetics | Xenograft Model Antitumor Assays | BRCA1 Protein - genetics | Poly(ADP-ribose) Polymerase Inhibitors - pharmacology | Drug Resistance, Neoplasm - genetics | Animals | Breast Neoplasms - genetics | Proteins - metabolism | Breast Neoplasms - pathology | Mad2 Proteins - genetics | Cell Line, Tumor | Mice | Osteosarcoma - genetics | Cell Cycle Proteins | Osteosarcoma - pathology | Care and treatment | DNA | Cancer cells | Breast cancer | Genetic aspects | Research | Gene expression | Single-stranded DNA | DNA damage | Homologous recombination | Poly(ADP-ribose) | Homology | Genomes | Inactivation | Proteins | Ribose | Null cells | Deoxyribonucleic acid--DNA | BRCA2 protein | CRISPR | Deactivation | BRCA1 protein | Poly(ADP-ribose) polymerase | Adenosine diphosphate | Oligosaccharides | Double-strand break repair | Screens | Cisplatin | Inhibitors | Prostate | Viability | Tumors | Index Medicus | Telomere-Binding Proteins / metabolism | Osteosarcoma / genetics | Telomere-Binding Proteins / genetics | BRCA1 Protein / genetics | Cellular Biology | Genetics | Proteins / genetics | Osteosarcoma / drug therapy | Ovarian Neoplasms / genetics | Mad2 Proteins / genetics | Proteins / metabolism | Breast Neoplasms / drug therapy | Breast Neoplasms / metabolism | Tumor Suppressor p53-Binding Protein 1 / genetics | BRCA1 Protein / deficiency | Ovarian Neoplasms / metabolism | Mad2 Proteins / metabolism | Breast Neoplasms / pathology | Bone Neoplasms / genetics | Ovarian Neoplasms / pathology | Bone Neoplasms / pathology | Life Sciences | Bone Neoplasms / drug therapy | Ovarian Neoplasms / drug therapy | Osteosarcoma / metabolism | Biochemistry, Molecular Biology | Breast Neoplasms / genetics | Drug Resistance, Neoplasm / genetics | Osteosarcoma / pathology | Bone Neoplasms / metabolism | Poly(ADP-ribose) Polymerase Inhibitors / pharmacology | Cisplatin / pharmacology | Molecular biology | Tumor Suppressor p53-Binding Protein 1 / metabolism | Cancer
Journal Article
PLoS pathogens, ISSN 1553-7374, 09/2014, Volume 10, Issue 9, pp. e1004400 - e1004400
Journal Article
Cellular physiology and biochemistry, ISSN 1421-9778, 2018, Volume 45, Issue 2, pp. 844 - 855
Background/Aims: LncRNA GAS5, a growth suppressor, has been reported to exert anti-tumor actions in various cancers, whereas the exact mechanism underling the... 
Original Paper | NF-κB signaling | LncRNA GAS5 | TIMP2 | PI3K/AKT/GSK3β signaling | MiR-203a | Osteosarcoma | Physiology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Cyclin D1 - metabolism | Up-Regulation | Cell Proliferation | MicroRNAs - antagonists & inhibitors | Humans | MicroRNAs - metabolism | NF-kappa B - metabolism | Cyclin B1 - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Bone Neoplasms - pathology | Tissue Inhibitor of Metalloproteinase-2 - genetics | Bone Neoplasms - metabolism | CDC2 Protein Kinase - metabolism | Tissue Inhibitor of Metalloproteinase-2 - metabolism | RNA Interference | Proto-Oncogene Proteins c-akt - metabolism | Osteosarcoma - metabolism | CDC2 Protein Kinase - genetics | Signal Transduction | Cell Survival | Cyclin B1 - genetics | RNA, Long Noncoding - genetics | Glycogen Synthase Kinase 3 beta - metabolism | Cyclin D1 - genetics | Cell Line, Tumor | MicroRNAs - genetics | Antagomirs - metabolism | RNA, Long Noncoding - antagonists & inhibitors | RNA, Long Noncoding - metabolism | Osteosarcoma - pathology | Tissue Inhibitor of Metalloproteinase-2 - antagonists & inhibitors | Disease | Pathogenesis | Lung cancer | Colorectal cancer | Breast cancer | Metastasis | Gene expression | Cancer therapies | Bone cancer | Cell growth | Tuberculosis | Pancreatic cancer | Medical prognosis | Cell cycle | Apoptosis | Index Medicus
Journal Article
American Journal of Pathology, The, ISSN 0002-9440, 2012, Volume 181, Issue 5, pp. 1782 - 1795
Journal Article
Journal Article
Oncogene, ISSN 0950-9232, 01/2009, Volume 28, Issue 3, pp. 334 - 344
Sequestosome 1 (SQSTM1/p62) is a multifunctional protein involved in signal transduction, protein degradation and cell transformation. Hypoxia is a common... 
ATG8 | Prolyl hydroxylase | HIF | Autophagy | Sequestosome 1 | PVHL | Biochemistry & Molecular Biology | Oncology | Genetics & Heredity | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Phosphorylation | Sequestosome-1 Protein | Carcinoma, Squamous Cell - metabolism | Carcinoma, Squamous Cell - pathology | Humans | Hypoxia-Inducible Factor 1, alpha Subunit - antagonists & inhibitors | Bone Neoplasms - pathology | Head and Neck Neoplasms - metabolism | Oxygen - metabolism | Bone Neoplasms - metabolism | Hypoxia - metabolism | Procollagen-Proline Dioxygenase - metabolism | Kidney - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Procollagen-Proline Dioxygenase - antagonists & inhibitors | Von Hippel-Lindau Tumor Suppressor Protein - genetics | Von Hippel-Lindau Tumor Suppressor Protein - antagonists & inhibitors | Von Hippel-Lindau Tumor Suppressor Protein - metabolism | Osteosarcoma - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | RNA, Small Interfering - pharmacology | Enzyme Inhibitors - pharmacology | Keratinocytes - cytology | Kidney - cytology | Blotting, Western | Head and Neck Neoplasms - pathology | Mitogen-Activated Protein Kinase 3 - metabolism | Procollagen-Proline Dioxygenase - genetics | Keratinocytes - metabolism | Adaptor Proteins, Signal Transducing - genetics | HeLa Cells | Adaptor Proteins, Signal Transducing - metabolism | Osteosarcoma - pathology | Mitogen-Activated Protein Kinase 1 - metabolism | Control | Physiological aspects | Hypoxia | Genetic aspects | Research | Health aspects | Phagocytosis | Cancer | Signal transduction | Genetics | Cellular biology | Index Medicus
Journal Article