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Nature cell biology, ISSN 1476-4679, 2018, Volume 20, Issue 8, pp. 954 - 965
BRCA1 deficiencies cause breast, ovarian, prostate and other cancers, and render tumours hypersensitive to poly(ADP-ribose) polymerase (PARP) inhibitors. To... 
PATHWAY CHOICE | STRAND BREAK REPAIR | RESECTION | DAMAGE-RESPONSE | 53BP1 | CLASS-SWITCH RECOMBINATION | FANCONI-ANEMIA | DIFFERENTIAL EXPRESSION ANALYSIS | POLYMERASE-ZETA | TELOMERES | CELL BIOLOGY | Osteosarcoma - drug therapy | Mad2 Proteins - metabolism | Humans | Multiprotein Complexes | Ovarian Neoplasms - pathology | Bone Neoplasms - pathology | DNA Breaks, Double-Stranded | Bone Neoplasms - metabolism | Breast Neoplasms - metabolism | Dose-Response Relationship, Drug | Ovarian Neoplasms - genetics | Telomere-Binding Proteins - genetics | DNA End-Joining Repair | HEK293 Cells | Female | Bone Neoplasms - genetics | Ovarian Neoplasms - metabolism | Bone Neoplasms - drug therapy | BRCA1 Protein - deficiency | Telomere-Binding Proteins - metabolism | Ovarian Neoplasms - drug therapy | Osteosarcoma - metabolism | DNA-Binding Proteins | Tumor Suppressor p53-Binding Protein 1 - metabolism | Recombinational DNA Repair | Tumor Suppressor p53-Binding Protein 1 - genetics | Cisplatin - pharmacology | Breast Neoplasms - drug therapy | Proteins - genetics | Xenograft Model Antitumor Assays | BRCA1 Protein - genetics | Poly(ADP-ribose) Polymerase Inhibitors - pharmacology | Drug Resistance, Neoplasm - genetics | Animals | Breast Neoplasms - genetics | Proteins - metabolism | Breast Neoplasms - pathology | Mad2 Proteins - genetics | Cell Line, Tumor | Mice | Osteosarcoma - genetics | Cell Cycle Proteins | Osteosarcoma - pathology | Care and treatment | DNA | Cancer cells | Breast cancer | Genetic aspects | Research | Gene expression | Single-stranded DNA | DNA damage | Homologous recombination | Poly(ADP-ribose) | Homology | Genomes | Inactivation | Proteins | Ribose | Null cells | Deoxyribonucleic acid--DNA | BRCA2 protein | CRISPR | Deactivation | BRCA1 protein | Poly(ADP-ribose) polymerase | Adenosine diphosphate | Oligosaccharides | Double-strand break repair | Screens | Cisplatin | Polymerase | Inhibitors | Prostate | Viability | Tumors | Telomere-Binding Proteins / metabolism | Osteosarcoma / genetics | Telomere-Binding Proteins / genetics | BRCA1 Protein / genetics | Cellular Biology | Genetics | Proteins / genetics | Osteosarcoma / drug therapy | Ovarian Neoplasms / genetics | Mad2 Proteins / genetics | Proteins / metabolism | Breast Neoplasms / drug therapy | Breast Neoplasms / metabolism | Tumor Suppressor p53-Binding Protein 1 / genetics | BRCA1 Protein / deficiency | Ovarian Neoplasms / metabolism | Mad2 Proteins / metabolism | Breast Neoplasms / pathology | Bone Neoplasms / genetics | Ovarian Neoplasms / pathology | Bone Neoplasms / pathology | Life Sciences | Bone Neoplasms / drug therapy | Ovarian Neoplasms / drug therapy | Osteosarcoma / metabolism | Biochemistry, Molecular Biology | Breast Neoplasms / genetics | Drug Resistance, Neoplasm / genetics | Osteosarcoma / pathology | Bone Neoplasms / metabolism | Poly(ADP-ribose) Polymerase Inhibitors / pharmacology | Cisplatin / pharmacology | Molecular biology | Tumor Suppressor p53-Binding Protein 1 / metabolism | Cancer
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2016, Volume 11, Issue 5, p. e0154680
The receptor-activator of nuclear kappaB ligand (RANKL) signaling pathway plays an important role in the regulation of bone growth and mediates the formation... 
GIANT-CELL TUMOR | DENOSUMAB | MULTIDISCIPLINARY SCIENCES | Osteosarcoma - drug therapy | RANK Ligand - metabolism | Humans | Male | Antineoplastic Agents - therapeutic use | Fibrous Dysplasia of Bone - drug therapy | RNA, Messenger - metabolism | Bone Neoplasms - pathology | Giant Cell Tumor of Bone - pathology | Chondrosarcoma - genetics | Chondrosarcoma - pathology | Bone Cysts, Aneurysmal - metabolism | Bone Neoplasms - genetics | Fibrous Dysplasia of Bone - genetics | Chondroma - drug therapy | Osteoclasts - pathology | Bone Cysts, Aneurysmal - pathology | Multiple Myeloma - genetics | Osteosarcoma - pathology | Bone Cysts, Aneurysmal - drug therapy | Chondroma - genetics | Gene Expression Regulation, Neoplastic | Chondroma - metabolism | Chondroma - pathology | Bone Neoplasms - metabolism | Denosumab - therapeutic use | Multiple Myeloma - drug therapy | Giant Cell Tumor of Bone - drug therapy | RNA, Messenger - antagonists & inhibitors | Female | Bone Neoplasms - drug therapy | Giant Cell Tumor of Bone - genetics | Osteosarcoma - metabolism | Fibrous Dysplasia of Bone - pathology | RNA, Messenger - genetics | Multiple Myeloma - metabolism | Organ Specificity | Chondrosarcoma - drug therapy | Fibrous Dysplasia of Bone - metabolism | Osteoclasts - metabolism | Bone Resorption - prevention & control | RANK Ligand - genetics | Multiple Myeloma - pathology | Giant Cell Tumor of Bone - metabolism | RANK Ligand - antagonists & inhibitors | Chondrosarcoma - metabolism | Osteosarcoma - genetics | Osteoclasts - drug effects | Bone Cysts, Aneurysmal - genetics | Bone resorption | Bones | Dosage and administration | Denosumab | Growth | Analysis | Immunohistochemistry | Chondrosarcoma | Multiple myeloma | Aneurysm | Bone tumors | Metastasis | Bone surgery | Metastases | Osteoprotegerin | Ewings sarcoma | Signal transduction | Bone growth | Biomedical materials | Osteosarcoma | Fibrous dysplasia | Bone dysplasia | Biocompatibility | Lesions | TRANCE protein | University graduates | Statistical analysis | Bone lesions | Gene expression | Osteoclastogenesis | Signaling | Bone cancer | Hospitals | Biopsy | Stromal cells | Orthopedics | Monoclonal antibodies | Ligands | Osteoclasts | Prostate cancer | Tumors
Journal Article
Surgical pathology clinics, 09/2019, Volume 12, Issue 3, p. 831
Journal Article
Oncotarget, ISSN 1949-2553, 2016, Volume 7, Issue 48, pp. 78343 - 78354
Journal Article
Medicine (United States), ISSN 0025-7974, 01/2015, Volume 94, Issue 3, p. e369
Journal Article
International Journal of Radiation Oncology, Biology, Physics, ISSN 0360-3016, 2015, Volume 91, Issue 4, pp. 866 - 875
Purpose To perform a preliminary exploration with a simplistic mathematical cancer stem cell (CSC) interaction model to determine whether the tumor-intrinsic... 
Radiology | Hematology, Oncology and Palliative Medicine | INITIATING CELLS | LUNG-CANCER | SOLID TUMORS | ONCOLOGY | 4 RS | UNIFIED SURVIVAL-CURVE | RADIORESISTANCE | RADIOBIOLOGY | PROSTATE-CANCER | RADIOTHERAPY | RADIOSENSITIVITY | RADIOLOGY, NUCLEAR MEDICINE & MEDICAL IMAGING | Carcinoma, Non-Small-Cell Lung - radiotherapy | Prostatic Neoplasms - radiotherapy | Humans | Radiation Tolerance | Brain Neoplasms - pathology | Male | Osteosarcoma - radiotherapy | Uterine Cervical Neoplasms - pathology | Bone Neoplasms - pathology | Glioblastoma - radiotherapy | Female | Brain Neoplasms - radiotherapy | Neoplastic Stem Cells - radiation effects | Carcinoma, Non-Small-Cell Lung - pathology | Prostatic Neoplasms - pathology | Uterine Cervical Neoplasms - radiotherapy | Cell Survival | Melanoma - radiotherapy | Linear Models | Melanoma - pathology | Bone Neoplasms - radiotherapy | Breast Neoplasms - radiotherapy | Breast Neoplasms - pathology | Glioblastoma - pathology | Cell Line, Tumor | Osteosarcoma - pathology | Care and treatment | Gliomas | Analysis | Stem cells | Radiation | Models | Transplantation | Radiotherapy | Cancer | DIFFERENTIAL EQUATIONS | GLIOMAS | PROSTATE | DOSE EQUIVALENTS | MAMMARY GLANDS | LUNGS | ANIMAL TISSUES | COMPARTMENTS | MELANOMAS | RADIATION DOSES | COMPARATIVE EVALUATIONS | SIMULATION | FRACTIONATED IRRADIATION | RADIOLOGY AND NUCLEAR MEDICINE | STEM CELLS | OSTEOSARCOMAS | SURVIVAL CURVES
Journal Article
The lancet oncology, ISSN 1470-2045, 2016, Volume 17, Issue 8, pp. 1070 - 1080
Journal Article
Journal of clinical oncology, ISSN 1527-7755, 2007, Volume 25, Issue 34, pp. 5435 - 5441
Journal Article
Journal Article
Oncogene, ISSN 1476-5594, 2010, Volume 29, Issue 42, pp. 5687 - 5699
The development of cancer is due to the growth and proliferation of transformed normal cells. Recent evidence suggests that the nature of oncogenic stress and... 
differentiation | animal model | osteosarcoma | PPARγ | PROTEIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | OSTEOBLAST LINEAGE | PPAR gamma | CANCER | MESENCHYMAL STEM-CELLS | CELL BIOLOGY | PATHOGENESIS | GENE | ONCOLOGY | GENETICS & HEREDITY | MICE | TRANSCRIPTIONAL REGULATION | EXPRESSION | Immunohistochemistry | Stromal Cells - pathology | Humans | Immunoblotting | Male | Gene Expression Profiling | Stem Cells - metabolism | Adipogenesis - physiology | Bone Neoplasms - metabolism | Proto-Oncogene Proteins c-myc - biosynthesis | RNA Interference | Cell Transformation, Neoplastic - genetics | Cell Differentiation - physiology | Chondrocytes - metabolism | Osteosarcoma - metabolism | PPAR gamma - genetics | Chondrocytes - pathology | Cyclin-Dependent Kinase Inhibitor p16 - deficiency | Gene Expression | Mice, Inbred C57BL | Stromal Cells - metabolism | Bone Marrow Cells - pathology | Cyclin-Dependent Kinase Inhibitor p16 - genetics | Mesenchymal Stromal Cells - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Adipocytes - pathology | Cell Transformation, Neoplastic - metabolism | PPAR gamma - biosynthesis | Mice, Knockout | Animals | Adipocytes - metabolism | Stem Cells - pathology | Mice | Mesenchymal Stromal Cells - pathology | Osteosarcoma - genetics | Cell Transformation, Neoplastic - pathology | Bone Marrow Cells - metabolism | Adipose tissues | Care and treatment | Osteosarcoma | Stem cells | Genetic aspects | Research | Health aspects | Risk factors | Bone marrow | Bone cancer | Cellular biology | Gene expression | Rodents
Journal Article