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Nature cell biology, ISSN 1476-4679, 2018, Volume 20, Issue 8, pp. 954 - 965
BRCA1 deficiencies cause breast, ovarian, prostate and other cancers, and render tumours hypersensitive to poly(ADP-ribose) polymerase (PARP) inhibitors. To... 
PATHWAY CHOICE | STRAND BREAK REPAIR | RESECTION | DAMAGE-RESPONSE | 53BP1 | CLASS-SWITCH RECOMBINATION | FANCONI-ANEMIA | DIFFERENTIAL EXPRESSION ANALYSIS | POLYMERASE-ZETA | TELOMERES | CELL BIOLOGY | Osteosarcoma - drug therapy | Mad2 Proteins - metabolism | Humans | Multiprotein Complexes | Ovarian Neoplasms - pathology | Bone Neoplasms - pathology | DNA Breaks, Double-Stranded | Bone Neoplasms - metabolism | Breast Neoplasms - metabolism | Dose-Response Relationship, Drug | Ovarian Neoplasms - genetics | Telomere-Binding Proteins - genetics | DNA End-Joining Repair | HEK293 Cells | Female | Bone Neoplasms - genetics | Ovarian Neoplasms - metabolism | Bone Neoplasms - drug therapy | BRCA1 Protein - deficiency | Telomere-Binding Proteins - metabolism | Ovarian Neoplasms - drug therapy | Osteosarcoma - metabolism | DNA-Binding Proteins | Tumor Suppressor p53-Binding Protein 1 - metabolism | Recombinational DNA Repair | Tumor Suppressor p53-Binding Protein 1 - genetics | Cisplatin - pharmacology | Breast Neoplasms - drug therapy | Proteins - genetics | Xenograft Model Antitumor Assays | BRCA1 Protein - genetics | Poly(ADP-ribose) Polymerase Inhibitors - pharmacology | Drug Resistance, Neoplasm - genetics | Animals | Breast Neoplasms - genetics | Proteins - metabolism | Breast Neoplasms - pathology | Mad2 Proteins - genetics | Cell Line, Tumor | Mice | Osteosarcoma - genetics | Cell Cycle Proteins | Osteosarcoma - pathology | Care and treatment | DNA | Cancer cells | Breast cancer | Genetic aspects | Research | Gene expression | Single-stranded DNA | DNA damage | Homologous recombination | Poly(ADP-ribose) | Homology | Genomes | Inactivation | Proteins | Ribose | Null cells | Deoxyribonucleic acid--DNA | BRCA2 protein | CRISPR | Deactivation | BRCA1 protein | Poly(ADP-ribose) polymerase | Adenosine diphosphate | Oligosaccharides | Double-strand break repair | Screens | Cisplatin | Polymerase | Inhibitors | Prostate | Viability | Tumors | Telomere-Binding Proteins / metabolism | Osteosarcoma / genetics | Telomere-Binding Proteins / genetics | BRCA1 Protein / genetics | Cellular Biology | Genetics | Proteins / genetics | Osteosarcoma / drug therapy | Ovarian Neoplasms / genetics | Mad2 Proteins / genetics | Proteins / metabolism | Breast Neoplasms / drug therapy | Breast Neoplasms / metabolism | Tumor Suppressor p53-Binding Protein 1 / genetics | BRCA1 Protein / deficiency | Ovarian Neoplasms / metabolism | Mad2 Proteins / metabolism | Breast Neoplasms / pathology | Bone Neoplasms / genetics | Ovarian Neoplasms / pathology | Bone Neoplasms / pathology | Life Sciences | Bone Neoplasms / drug therapy | Ovarian Neoplasms / drug therapy | Osteosarcoma / metabolism | Biochemistry, Molecular Biology | Breast Neoplasms / genetics | Drug Resistance, Neoplasm / genetics | Osteosarcoma / pathology | Bone Neoplasms / metabolism | Poly(ADP-ribose) Polymerase Inhibitors / pharmacology | Cisplatin / pharmacology | Molecular biology | Tumor Suppressor p53-Binding Protein 1 / metabolism | Cancer
Journal Article
CA: a cancer journal for clinicians, ISSN 0007-9235, 2016, Volume 66, Issue 5, pp. 408 - 436
Answer questions and earn CME/CNE In this report, a team of surgical pathologists has provided a review of intraepithelial neoplasia in a host of (but not all)... 
carcinoma in situ | dysplasia | intraepithelial neoplasia | preinvasive neoplasia | precancer | HIGH-GRADE DYSPLASIA | URINARY-BLADDER | BARRETTS-ESOPHAGUS | HUMAN-PAPILLOMAVIRUS | INTRADUCTAL CARCINOMA | CARCINOMA IN-SITU | PAPILLARY UROTHELIAL HYPERPLASIA | ONCOLOGY | SINGLE ACADEMIC CENTER | TERM-FOLLOW-UP | NECK-CANCER IMPLICATIONS | Humans | Lung Neoplasms - metabolism | Ovarian Neoplasms - pathology | Lung Neoplasms - pathology | Male | Uterine Cervical Neoplasms - pathology | Head and Neck Neoplasms - metabolism | Breast Neoplasms - metabolism | Esophageal Neoplasms - pathology | Breast Neoplasms - therapy | American Cancer Society | Uterine Cervical Neoplasms - metabolism | Skin Neoplasms - diagnosis | Urinary Bladder Neoplasms - pathology | Female | Ovarian Neoplasms - metabolism | Uterine Cervical Neoplasms - therapy | Skin Neoplasms - pathology | Prostatic Neoplasms - pathology | Uterine Cervical Neoplasms - diagnosis | Skin Neoplasms - therapy | Carcinoma in Situ - pathology | Ovarian Neoplasms - diagnosis | Pancreatic Neoplasms - pathology | Risk Factors | Head and Neck Neoplasms - therapy | Lung Neoplasms - therapy | Carcinoma in Situ - therapy | Head and Neck Neoplasms - pathology | Skin Neoplasms - metabolism | Breast Neoplasms - pathology | Ovarian Neoplasms - therapy | Head and Neck Neoplasms - diagnosis | Breast Neoplasms - diagnosis | Carcinoma in Situ - diagnosis | Carcinoma in Situ - metabolism | Lung Neoplasms - diagnosis | Population Surveillance | Prevention | Care and treatment | Gastrointestinal system | Practice | Pathologists | Metastasis | Patients | Health aspects | Cervical cancer | Risk factors | Pathology | Usage | Diagnosis | Tumors | Medical diagnosis | Cancer
Journal Article
Human Pathology, ISSN 0046-8177, 2013, Volume 44, Issue 11, pp. 2563 - 2570
Summary BRAF mutation is seen in a variety of human neoplasms including cutaneous malignant melanoma, papillary thyroid carcinoma, colorectal carcinoma, non... 
Pathology | Immunohistochemistry | Anti–B-Raf | BRAF V600E | VE1 | Glioma | Melanoma | Thyroid carcinoma | Anti-B-Raf | PROTEIN | IMPROVED SURVIVAL | PATHOLOGY | CANCER | TUMORS | THERAPY | MEK INHIBITION | GENE | MUTANT BRAF | Neoplasms - metabolism | Gastrointestinal Neoplasms - genetics | Humans | Lung Neoplasms - metabolism | Ovarian Neoplasms - pathology | Lung Neoplasms - pathology | Mutation, Missense | Breast Neoplasms - metabolism | Glioma - metabolism | Glioma - genetics | Ovarian Neoplasms - genetics | Gastrointestinal Neoplasms - pathology | Neoplasms - genetics | DNA Mutational Analysis | Melanoma - genetics | Glioma - pathology | Sensitivity and Specificity | Biomarkers, Tumor - metabolism | Female | Ovarian Neoplasms - metabolism | Carcinoma - pathology | Antibodies, Monoclonal - immunology | Proto-Oncogene Proteins B-raf - metabolism | Gastrointestinal Neoplasms - metabolism | Lung Neoplasms - genetics | Melanoma - metabolism | Mutant Proteins - genetics | Mutant Proteins - metabolism | Melanoma - pathology | Thyroid Neoplasms - genetics | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Proto-Oncogene Proteins B-raf - genetics | Carcinoma - genetics | Biomarkers, Tumor - genetics | Carcinoma - metabolism | Neoplasms - pathology | Amino Acid Substitution | Thyroid Neoplasms - metabolism | Thyroid Neoplasms - pathology | Carcinoma | Gene mutations | Gliomas | Thyroid diseases | Monoclonal antibodies | Genetic aspects | Comparative analysis | Lung cancer, Non-small cell | Cancer | Breast cancer | Mutation | Tumors | Skin cancer
Journal Article
BMC Genomics, ISSN 1471-2164, 04/2006, Volume 7, Issue 1, pp. 90 - 90
Background: Post-translational modification of histones resulting in chromatin remodelling plays a key role in the regulation of gene expression. Here we... 
BREAST-CANCER | CBP | METHYLATION | INHIBITION | BOOTSTRAP | ACETYLATION | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | GENETICS & HEREDITY | PATTERNS | MUTATIONS | REPRODUCIBILITY | PROGRESSION | Neoplasms - metabolism | Protein Methyltransferases | Pancreatic Neoplasms - metabolism | Colorectal Neoplasms - genetics | Histone Acetyltransferases - biosynthesis | Humans | Lung Neoplasms - metabolism | Protein Processing, Post-Translational - genetics | DNA, Complementary - genetics | Gene Expression Regulation, Neoplastic | Histone Acetyltransferases - genetics | Male | Histone-Lysine N-Methyltransferase - biosynthesis | Breast Neoplasms - metabolism | Ovarian Neoplasms - genetics | Polymorphism, Single-Stranded Conformational | Neoplasms - genetics | Cell Transformation, Neoplastic - genetics | Polymerase Chain Reaction | Female | Ovarian Neoplasms - metabolism | Neoplasm Proteins - genetics | Colorectal Neoplasms - metabolism | Lung Neoplasms - genetics | Histone-Lysine N-Methyltransferase - genetics | Histone Deacetylases - genetics | Neoplasm Proteins - biosynthesis | Linear Models | Pancreatic Neoplasms - genetics | Gene Expression Regulation, Enzymologic | Breast Neoplasms - genetics | Carcinoma - genetics | Carcinoma - metabolism | DNA, Neoplasm - genetics | Histones - metabolism | Histone Code - genetics | Cluster Analysis
Journal Article
Journal Article
Nature communications, ISSN 2041-1723, 2017, Volume 8, Issue 1, p. 13923
Tumour metastasis, the spread of cancer cells from the original tumour site followed by growth of secondary tumours at distant organs, is the primary cause of... 
EPITHELIAL-MESENCHYMAL TRANSITION | KINASE 4/6 INHIBITOR | PHOSPHORYLATION | MULTIDISCIPLINARY SCIENCES | TUMOR-METASTASIS | TRANSCRIPTION FACTOR SNAIL | HUMAN BREAST-CANCER | CELL | E-CADHERIN REPRESSION | PROGRESSION | PLASTICITY | Cyclin-Dependent Kinase 6 - antagonists & inhibitors | RNA, Small Interfering - genetics | Cyclin-Dependent Kinase 4 - genetics | Humans | Lung Neoplasms - metabolism | Gene Expression Regulation, Neoplastic | Ovarian Neoplasms - genetics | Ovarian Neoplasms - secondary | Snail Family Transcription Factors - genetics | Lung Neoplasms - secondary | MCF-7 Cells | Triple Negative Breast Neoplasms - pathology | Female | Antineoplastic Agents - pharmacology | Ovarian Neoplasms - metabolism | Cyclin-Dependent Kinase 4 - antagonists & inhibitors | Liver Neoplasms - secondary | Disease Models, Animal | Liver Neoplasms - prevention & control | Lung Neoplasms - genetics | Endopeptidases - metabolism | Snail Family Transcription Factors - metabolism | Liver Neoplasms - genetics | Ovarian Neoplasms - prevention & control | Signal Transduction | Cyclin-Dependent Kinase 6 - genetics | Cyclin-Dependent Kinase 6 - metabolism | Cyclin-Dependent Kinase 4 - metabolism | Piperazines - pharmacology | Xenograft Model Antitumor Assays | Leupeptins - pharmacology | Animals | Endopeptidases - genetics | Triple Negative Breast Neoplasms - genetics | Lung Neoplasms - prevention & control | Triple Negative Breast Neoplasms - metabolism | Liver Neoplasms - metabolism | Cell Line, Tumor | Mice | Pyridines - pharmacology | Cell Movement | RNA, Small Interfering - metabolism
Journal Article
Cancer cell, ISSN 1535-6108, 2014, Volume 26, Issue 1, pp. 77 - 91
Journal Article
Cancer chemotherapy and pharmacology, ISSN 1432-0843, 2019, Volume 84, Issue 3, pp. 487 - 499
Enzymes of the cytochrome P450 (CYP) subfamily 3A and 2C play a major role in the metabolism of taxane anticancer agents... 
Intratumoral | Ritonavir | Paclitaxel | Docetaxel | CYP | Review | Journal Article | Medicine & Public Health | Oncology | Cancer Research | Pharmacology/Toxicology | PREGNANE-X RECEPTOR | IMMUNOHISTOCHEMICAL LOCALIZATION | DOCETAXEL RESISTANCE | CYP3A4 EXPRESSION | TARGETED THERAPIES | MESSENGER-RNA | ONCOLOGY | P450-MEDIATED METABOLISM | BIOLOGICAL-ACTIVITY | PHARMACOLOGY & PHARMACY | HIV-1 PROTEASE INHIBITOR | METABOLIZING-ENZYMES | Lung Neoplasms - drug therapy | Taxoids - pharmacology | Humans | Ovarian Neoplasms - pathology | Drug Resistance, Neoplasm | Lung Neoplasms - pathology | Male | Breast Neoplasms - enzymology | Female | Antineoplastic Agents - pharmacology | Cytochrome P-450 CYP2C8 - metabolism | Ovarian Neoplasms - drug therapy | Prostatic Neoplasms - drug therapy | Prostatic Neoplasms - pathology | Endometrial Neoplasms - enzymology | Lung Neoplasms - enzymology | Neoplasms - enzymology | Breast Neoplasms - drug therapy | Ovarian Neoplasms - enzymology | Neoplasms - drug therapy | Breast Neoplasms - pathology | Cytochrome P-450 CYP3A - metabolism | Prostatic Neoplasms - enzymology | Endometrial Neoplasms - drug therapy | Endometrial Neoplasms - pathology | Neoplasms - pathology | Antimitotic agents | Protease inhibitors | Proteases | Pharmacy | Cytochrome P-450 | Physiological aspects | Drugstores | Antineoplastic agents | Ovarian cancer | Cytochrome | Enzymes | Cytochrome P450 | Proteinase inhibitors | Taxane | Metabolism | Anticancer properties | Antitumor agents | Lungs | Drug metabolism | Breast | Solid tumors | Taxanes | Prostate | Endometrium | Tumors | Cytochromes P450
Journal Article