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Clinical and Experimental Pharmacology and Physiology, ISSN 0305-1870, 05/2007, Volume 34, Issue 5, pp. 425 - 431
SUMMARY • It is known that stress causes disruption of homeostasis and an imbalanced anti-oxidant status in several organs. The aim of the present study was to... 
immobilization-cold stress | rat | glutathione | protein oxidation | cold stress | anti-oxidant enzymes | lipid peroxidation | immobilization stress | anti‐oxidant enzymes | immobilization–cold stress | Cold stress | Protein oxidation | Rat | Anti-oxidant enzymes | Immobilization-cold stress | Lipid peroxidation | Immobilization stress | Glutathione | CATALASE | ENZYME-ACTIVITIES | DEFENSE | PHYSIOLOGY | GLUCOCORTICOIDS | GLUTATHIONE-PEROXIDASE | BODY-WEIGHT | METABOLISM | AGE-RELATED-CHANGES | PHARMACOLOGY & PHARMACY | BRAIN | EXPOSURE | Protein Carbonylation | Rats, Wistar | Glutathione - metabolism | Zinc - metabolism | Antioxidants - metabolism | Male | Stress, Psychological - psychology | Immobilization - psychology | Kidney - metabolism | Myocardium - metabolism | Copper - metabolism | Corticosterone - blood | Weight Gain | Superoxide Dismutase - metabolism | Glutathione Peroxidase - metabolism | Cold Temperature | Liver - metabolism | Rats | Thiobarbituric Acid Reactive Substances - metabolism | Kidney - cytology | Liver - chemistry | Lipid Peroxidation - physiology | Myocardium - cytology | Catalase - metabolism | Animals | Selenium - metabolism | Models, Biological | Weight Loss | Kidney - chemistry | Liver - cytology | Liver, cytology | Thiobarbituric Acid Reactive Substances, metabolism | Superoxide Dismutase, metabolism | Zinc, metabolism | Antioxidants, metabolism | Kidney, metabolism | Glutathione, metabolism | Myocardium, metabolism | Immobilization, psychology | Corticosterone, blood | Catalase, metabolism | Selenium, metabolism | Liver, chemistry | Cold | Copper, metabolism | Lipid Peroxidation, physiology | Myocardium, cytology | Kidney, chemistry | Liver, metabolism | Glutathione Peroxidase, metabolism | Stress, Psychological, psychology | Kidney, cytology | Antioxidants | Comparative analysis | Oxidation-reduction reaction
Journal Article
Antioxidants & redox signaling, ISSN 1557-7716, 2016, Volume 25, Issue 4, pp. 208 - 216
Journal Article
The Journal of biological chemistry, ISSN 1083-351X, 2002, Volume 277, Issue 47, pp. 44784 - 44790
Journal Article
European journal of immunology, ISSN 0014-2980, 2011, Volume 41, Issue 7, pp. 2040 - 2051
Journal Article
The Journal of biological chemistry, ISSN 0021-9258, 2017, Volume 292, Issue 37, pp. 15577 - 15586
The budding yeast Saccharomyces cerevisiae stores iron in the vacuole, which is a major resistance mechanism against iron toxicity. One key protein involved in... 
PATHWAYS | TRANSPORTER | METABOLISM | MECHANISM | PROTEIN-KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | SACCHAROMYCES-CEREVISIAE | EXPRESSION | AMP-Activated Protein Kinases - metabolism | Basic-Leucine Zipper Transcription Factors - metabolism | Oxidants - metabolism | Saccharomyces cerevisiae - drug effects | RNA Polymerase II - metabolism | DNA-Binding Proteins - metabolism | Gene Expression Regulation, Fungal - drug effects | Cation Transport Proteins - metabolism | Membrane Transport Proteins - genetics | Gene Deletion | Cation Transport Proteins - genetics | Membrane Transport Proteins - metabolism | Biological Transport - drug effects | Protein-Serine-Threonine Kinases - metabolism | Repressor Proteins - metabolism | Saccharomyces cerevisiae - physiology | Oxidants - toxicity | Protein-Serine-Threonine Kinases - genetics | Repressor Proteins - genetics | Basic-Leucine Zipper Transcription Factors - genetics | Microbial Viability | Saccharomyces cerevisiae Proteins - genetics | Transcription Factors - genetics | Iron - metabolism | Transcription Factors - metabolism | Signal Transduction - drug effects | Saccharomyces cerevisiae Proteins - metabolism | Vacuoles - metabolism | RNA Polymerase II - genetics | Mutation | Oxidative Stress - drug effects | Iron - toxicity | AMP-Activated Protein Kinases - genetics | Saccharomyces cerevisiae - growth & development | Index Medicus | iron metabolism | toxicity | iron | transcription | vacuole | Metabolism
Journal Article
The Journal of biological chemistry, ISSN 0021-9258, 2018, Volume 293, Issue 8, pp. 2640 - 2649
Transglutaminase 2 (TG2) is a ubiquitously expressed, intracellular as well as extracellular protein with multiple modes of post-translational regulation,... 
SELECTIVE-INHIBITION | HUMAN THIOREDOXIN-1 | ACTIVE-SITE | THROMBUS FORMATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | ISOMERASE | THIOL-DISULFIDE EXCHANGE | EXTRACELLULAR-MATRIX | QUIESCIN-SULFHYDRYL OXIDASE | TISSUE TRANSGLUTAMINASE | CELIAC-DISEASE | Enzymes, Immobilized - metabolism | Oxidants - pharmacology | Human Umbilical Vein Endothelial Cells - metabolism | Membrane Glycoproteins - metabolism | Glutathione - metabolism | Protein Disulfide-Isomerases - metabolism | GTP-Binding Proteins - antagonists & inhibitors | Humans | Membrane Glycoproteins - chemistry | Oxidants - metabolism | Extracellular Matrix - metabolism | Transglutaminases - genetics | Protein Disulfide-Isomerases - antagonists & inhibitors | Protein Transport - drug effects | GTP-Binding Proteins - genetics | Thioredoxins - genetics | Oxidoreductases Acting on Sulfur Group Donors - chemistry | Protein Processing, Post-Translational - drug effects | RNA Interference | Biocatalysis - drug effects | Human Umbilical Vein Endothelial Cells - cytology | Thioredoxins - metabolism | Peptide Fragments - genetics | Transglutaminases - antagonists & inhibitors | Transglutaminases - chemistry | Allosteric Regulation - drug effects | Oxidoreductases Acting on Sulfur Group Donors - metabolism | Recombinant Proteins - metabolism | Peptide Fragments - metabolism | GTP-Binding Proteins - chemistry | Oxidation-Reduction | Extracellular Matrix - drug effects | Cells, Cultured | Hydrogen Peroxide - pharmacology | Recombinant Proteins - chemistry | Membrane Glycoproteins - genetics | Extracellular Matrix - enzymology | Peptide Fragments - chemistry | Protein Disulfide-Isomerases - genetics | Human Umbilical Vein Endothelial Cells - enzymology | Cystine - metabolism | Transglutaminases - metabolism | Oxidoreductases Acting on Sulfur Group Donors - genetics | Enzymes, Immobilized - antagonists & inhibitors | GTP-Binding Proteins - metabolism | Editors' Picks | post-translational modification (PTM) | PDIA3 | transglutaminase | thioredoxin | disulfide | oxidation-reduction (redox) | ERp57 | redox switch | celiac disease | transglutaminase 2 | protein disulfide isomerase family A member 3
Journal Article
The Journal of biological chemistry, ISSN 0021-9258, 12/2007, Volume 282, Issue 51, pp. 37006 - 37015
Peroxisome proliferator-activated receptor gamma(PPAR gamma) has been proposed as a therapeutic target for neurodegenerative diseases because of its... 
SURVIVAL | SIGNALING PATHWAY | ALZHEIMERS-DISEASE | INSULIN-RESISTANCE | BIOCHEMISTRY & MOLECULAR BIOLOGY | PPAR-GAMMA | TROGLITAZONE | LIGAND | AGONISTS | NERVE GROWTH-FACTOR | CELL-DEATH | Neurons - pathology | Oxidants - pharmacology | Apoptosis - drug effects | Neuroglia - pathology | Oxidants - metabolism | Apoptosis - genetics | Neurodegenerative Diseases - drug therapy | PPAR gamma - metabolism | PC12 Cells | Proto-Oncogene Proteins c-akt - genetics | Cell Differentiation - genetics | Proto-Oncogene Proteins c-bcl-2 - metabolism | Mitochondria - genetics | Amyloid beta-Peptides - genetics | Amyloid beta-Peptides - metabolism | Neurons - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Thiazolidinediones - pharmacology | PPAR gamma - genetics | Cell Survival - drug effects | Nerve Growth Factor - metabolism | Neurodegenerative Diseases - pathology | RNA, Small Interfering - pharmacology | Hydrogen Peroxide - pharmacology | Oxidative Stress - genetics | Rats | Nerve Growth Factor - genetics | Neurodegenerative Diseases - genetics | Hippocampus - pathology | Mitochondria - metabolism | Extracellular Signal-Regulated MAP Kinases | Mitochondria - pathology | Neurodegenerative Diseases - metabolism | Hydrogen Peroxide - metabolism | Hypoglycemic Agents - pharmacology | Hippocampus - metabolism | Animals | PPAR gamma - antagonists & inhibitors | Cell Differentiation - drug effects | Ganglia, Spinal - pathology | PPAR gamma - agonists | Neuroglia - metabolism | Mice | Oxidative Stress - drug effects | Proto-Oncogene Proteins c-bcl-2 - genetics | Ganglia, Spinal - metabolism
Journal Article
Cell (Cambridge), ISSN 0092-8674, 2006, Volume 127, Issue 2, pp. 397 - 408
Journal Article
Annual review of biochemistry, ISSN 0066-4154, 6/2017, Volume 86, Issue 1, pp. 715 - 748
Journal Article