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EMBO Molecular Medicine, ISSN 1757-4676, 03/2018, Volume 10, Issue 3, p. n/a
.... These effects coincided with decreased brown adipocyte lipid content and increased nutrient uptake by BAT, confirming increased BAT activity... 
GPR120 | brown adipose tissue | Ca2 | mitochondria | lipid metabolism | Ca | MEDICINE, RESEARCH & EXPERIMENTAL | CELLS | ACTIVATION | PROTEIN | INSULIN-SECRETION | THERMOGENESIS | POTENT | GLUCOSE-METABOLISM | GENE-EXPRESSION | ACID RECEPTOR GPR40 | ADIPOSE-TISSUE | Adipocytes, White - cytology | Adipocytes, Brown - metabolism | Receptors, G-Protein-Coupled - metabolism | Adipose Tissue, White - metabolism | Body Weight - drug effects | Lipids | Male | Receptors, G-Protein-Coupled - agonists | Adipocytes, White - drug effects | Biphenyl Compounds - pharmacology | Cell Respiration - drug effects | Adiposity - drug effects | Oxidation-Reduction | Adipocytes, Brown - drug effects | Uncoupling Protein 1 - metabolism | Mice, Inbred C57BL | Mitochondria - metabolism | Mitochondria - drug effects | Gene Expression Regulation - drug effects | Phenylpropionates - pharmacology | Animals | Receptors, G-Protein-Coupled - deficiency | Cell Differentiation - drug effects | Models, Biological | Oxygen Consumption - drug effects | Glucose - metabolism | Adipose Tissue, Brown - drug effects | Adipose Tissue, Brown - metabolism | Adipocytes, Brown - cytology | Adipocytes, White - metabolism | Adipose Tissue, White - drug effects | Physiological aspects | Obesity | G proteins | Body weight | Membrane proteins | Pharmacology & Drug Discovery | Metabolism
Journal Article
Cell metabolism, ISSN 1550-4131, 2014, Volume 19, Issue 1, pp. 96 - 108
The transcriptional coactivator peroxisome proliferator-activated receptor-gamma coactivator-1α (PGC-1α) regulates metabolic genes in skeletal muscle and... 
SKELETAL-MUSCLE | PPAR-ALPHA | ACTIVATION | MITOCHONDRIAL UNCOUPLING PROTEIN | PGC-1-ALPHA | IN-VIVO | ENDOCRINOLOGY & METABOLISM | MICE | EXPRESSION | EXERCISE | GENOME-WIDE ASSOCIATION | CELL BIOLOGY | Organ Specificity - drug effects | Transcription, Genetic - drug effects | Metabolic Diseases - pathology | Adipocytes, Brown - metabolism | Humans | Adipose Tissue, White - metabolism | Cardiovascular Diseases - pathology | Organ Specificity - genetics | Adipocytes, White - drug effects | Adipose Tissue, White - cytology | Exercise | Liver - drug effects | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Oxidation-Reduction - drug effects | Weight Gain - drug effects | Physical Conditioning, Animal | Aminoisobutyric Acids - pharmacology | Induced Pluripotent Stem Cells - metabolism | Adipocytes, Brown - pathology | Glucose Tolerance Test | Cardiovascular Diseases - metabolism | Induced Pluripotent Stem Cells - drug effects | Adipocytes, Brown - drug effects | Liver - metabolism | Risk Factors | Aminoisobutyric Acids - blood | Gene Expression Regulation - drug effects | Transcription Factors - metabolism | Adipose Tissue, Brown - cytology | Phenotype | Adipocytes, White - pathology | Animals | Metabolic Diseases - metabolism | Cell Differentiation - drug effects | Adipose Tissue, Brown - drug effects | Adipose Tissue, Brown - metabolism | Mice | PPAR alpha - metabolism | Adipocytes, White - metabolism | Adipose Tissue, White - drug effects
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2012, Volume 7, Issue 10, p. e47792
Despite increased risk of a recurrent stroke following a minor stroke, information is minimal regarding the interaction between injurious mild cerebral... 
CELLS | OXIDATIVE STRESS | DISEASES | MULTIDISCIPLINARY SCIENCES | INJURY | TRANS-RESVERATROL | RAT-BRAIN | MODEL | ISCHEMIA-REPERFUSION | BLOOD-BRAIN-BARRIER | EXPRESSION | Inflammation - pathology | Recurrence | Stilbenes - administration & dosage | Stilbenes - therapeutic use | Body Temperature - drug effects | Endothelium, Vascular - drug effects | Stilbenes - pharmacology | Stroke - physiopathology | Brain - metabolism | Stroke - pathology | Heart Rate - drug effects | Inflammation - complications | Liver - drug effects | Oxidation-Reduction - drug effects | Blood Pressure - drug effects | Stress, Physiological - drug effects | Cell Death - drug effects | Biomarkers - metabolism | Cell Hypoxia - drug effects | Oxygen | Stroke - prevention & control | Administration, Oral | Cerebrovascular Circulation - drug effects | Liver - metabolism | Blood Gas Analysis | Rats | Treatment Outcome | Stroke - drug therapy | Blood-Brain Barrier - drug effects | Brain - drug effects | Blood-Brain Barrier - pathology | Glucose - deficiency | Animals | Nitrosation - drug effects | Brain - pathology | Endothelium, Vascular - pathology | Stilbenes - blood | Endothelial Cells - pathology | Endothelial Cells - drug effects | Stroke (Disease) | Usage | Care and treatment | Health aspects | Resveratrol | Cell culture | Oxidative stress | Neurosciences | Pressure effects | Glucose | Health physics | Toxicology | Ischemia | Blood-brain barrier | Cerebral blood flow | Temperature effects | Rodents | Gangrene | Physiology | Blood pressure | Inhibition | Drug dosages | Informatics | Edema | Deprivation | Stroke | Body temperature | Health risks | Pharmacology | Inflammation | Chromatography | Endothelial cells | Blood flow | Endothelium | Medicine | Cell death | Nitric oxide | Brain damage | In vivo methods and tests | Brain injury | Apoptosis
Journal Article
Journal Article
Journal of Hepatology, ISSN 0168-8278, 2011, Volume 54, Issue 4, pp. 773 - 794
Numerous investigations have shown that mitochondrial dysfunction is a major mechanism of drug-induced liver injury, which involves the parent drug or a reactive metabolite generated through cytochromes P450... 
Gastroenterology and Hepatology | Drugs | Obesity | Oxidative stress | Mitochondria | Cell death | Lipids | Hepatotoxicity | Steatosis | TRIGLYCERIDE TRANSFER PROTEIN | DNA-POLYMERASE-GAMMA | PREGNANE X-RECEPTOR | PROLIFERATOR-ACTIVATED RECEPTOR | HEPATIC CYTOCHROME-P450 2E1 | ELEMENT-BINDING PROTEINS | FATTY-ACID OXIDATION | CONSTITUTIVE ANDROSTANE RECEPTOR | MANGANESE SUPEROXIDE-DISMUTASE | STRESS-RELATED PARAMETERS | GASTROENTEROLOGY & HEPATOLOGY | Carbohydrate Metabolism - drug effects | Reactive Oxygen Species - metabolism | Mitochondria, Liver - metabolism | Humans | Leptin - metabolism | Oxidative Phosphorylation - drug effects | Adipose Tissue - metabolism | Adiponectin - metabolism | Hepatitis C - complications | Cell Death - drug effects | Fatty Acids - metabolism | Chemical and Drug Induced Liver Injury - etiology | Diabetes Mellitus, Type 2 - complications | Genetic Predisposition to Disease | Fatty Liver - metabolism | Oxidation-Reduction | Obesity - complications | Insulin Resistance | Mitochondrial Membrane Transport Proteins - drug effects | Chemical and Drug Induced Liver Injury - genetics | Mitochondria, Liver - drug effects | Animals | Chemical and Drug Induced Liver Injury - metabolism | Models, Biological | Lipid Metabolism - drug effects | Alcoholic Intoxication - complications | Genome, Mitochondrial | Adipose Tissue - drug effects | Energy Metabolism - drug effects | Fatty Liver - etiology | Divalproex | Liver diseases | Thiols | Liver | Cytochrome P-450 | Mitochondrial DNA | Triglycerides | Stavudine | Permeability | Valproic acid | Fatty acids | Cells | Carnitine | Metabolites | Glutathione transferase | Acetaminophen | Physiological aspects | DNA polymerases | Health aspects | Zidovudine | Index Medicus | Reactive Oxygen Species | Fatty Acids | Adiponectin | Mitochondria, Liver | Life Sciences | Mitochondrial Membrane Transport Proteins | Cell Death | Diabetes Mellitus, Type 2 | Adipose Tissue | Alcoholic Intoxication | Hépatology and Gastroenterology | Oxidative Phosphorylation | Carbohydrate Metabolism | Lipid Metabolism | Drug-Induced Liver Injury | Fatty Liver | Human health and pathology | Energy Metabolism | Leptin | Hepatitis C
Journal Article
Cell stem cell, ISSN 1934-5909, 2016, Volume 19, Issue 1, pp. 23 - 37
Journal Article
Psychopharmacology (Berlin, Germany), ISSN 1432-2072, 2018, Volume 236, Issue 2, pp. 641 - 655
.... The present study investigated the effects of 50 and 100 mg/kg berberine (BRB) on recognition memory, oxidative stress, and purinergic neurotransmission, in a model of sporadic dementia... 
Neurosciences | Biomedicine | Ectoenzymes | Reactive species | Pharmacology/Toxicology | Antioxidant | Psychiatry | Alzheimer | HIGH-FAT | PSYCHIATRY | ALZHEIMERS-DISEASE | ACID DEHYDRATASE | COGNITIVE IMPAIRMENT | DELTA-AMINOLEVULINATE DEHYDRATASE | NEUROSCIENCES | AMYLOID-BETA | MOUSE MODEL | MITOCHONDRIAL DYSFUNCTION | PHARMACOLOGY & PHARMACY | ANXIOGENIC-LIKE BEHAVIOR | BRAIN | Memory - drug effects | Synaptosomes - drug effects | Antibiotics, Antineoplastic - toxicity | Reactive Oxygen Species - metabolism | Rats, Wistar | Adenosine Deaminase - metabolism | Injections, Intraventricular | Male | Hippocampus - drug effects | Cerebral Cortex - metabolism | Antioxidants | Brain - metabolism | Adenosine Deaminase - drug effects | Neuroprotective Agents - pharmacology | 5'-Nucleotidase - drug effects | Oxidation-Reduction - drug effects | Cerebral Cortex - drug effects | Alzheimer Disease - psychology | Disease Models, Animal | Glutathione | Synaptosomes - enzymology | Glutathione Transferase - metabolism | Rats | Pyrophosphatases - drug effects | Brain - drug effects | Pyrophosphatases - metabolism | Berberine - pharmacology | Hippocampus - metabolism | Animals | Recognition (Psychology) - drug effects | Glutathione Transferase - drug effects | Lipid Metabolism - drug effects | Streptozocin - toxicity | Oxidative Stress - drug effects | 5'-Nucleotidase - metabolism | Memory Disorders - chemically induced | Prevention | Complications and side effects | Oxidative stress | Care and treatment | Streptozocin | Causes of | Physiological aspects | Cellular signal transduction | Purine nucleotides | Memory, Disorders of | Health aspects | Nucleotidase | Neuroprotection | Reactive oxygen species | Cerebral cortex | Thiols | Memory | Impairment | Lipid peroxidation | Lipids | Dehydration | Proteins | Glutathione transferase | Rodents | Dementia disorders | Oxidation | Synaptosomes | Alzheimer's disease | Berberine | Adenosine | Adenosine deaminase | Injection | Hippocampus | Neurotransmission | Recognition
Journal Article
Molecular Endocrinology, ISSN 1944-9917, 2009, Volume 23, Issue 6, pp. 893 - 900
Journal Article
International Journal of Pediatric Otorhinolaryngology, ISSN 0165-5876, 2016, Volume 92, pp. 61 - 66
Abstract Objective Cisplatin is commonly used to treat solid tumors. However, permanent hearing loss is a major side effect of cisplatin chemotherapy and often results in dose reduction of the cisplatin chemotherapy... 
Otolaryngology | Pediatrics | Peanut sprout | Cell death | Cisplatin | Ototoxicity | Apoptosis | RESVERATROL | CELLS | ACTIVATION | PROTECTION | MECHANISMS | TOXICITY | ANTIOXIDANT | OTORHINOLARYNGOLOGY | PEDIATRICS |