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Science, ISSN 0036-8075, 1/2013, Volume 339, Issue 6116, pp. 211 - 214
Journal Article
Plant Physiology, ISSN 0032-0889, 3/2013, Volume 161, Issue 3, pp. 1202 - 1216
OsTZF1 is a member of the CCCH-type zinc finger gene family in rice (Oryza sativa). Expression of OsTZF1 was induced by drought, high-salt stress, and hydrogen... 
Oxidative stress | RNA | Genes | Stress tolerance | Cellular senescence | Gene expression regulation | Plants | ECOPHYSIOLOGY AND SUSTAINABILITY | Zinc | Rice | Seedlings | LEAF SENESCENCE | SALT STRESS | HIGH-SALINITY STRESSES | ARABIDOPSIS-THALIANA | TRANSGENIC TOBACCO | FUNCTIONAL-ANALYSIS | BETA-GLUCURONIDASE | EXPRESSION PROFILES | ABSCISIC-ACID | TRANSCRIPTION FACTOR | PLANT SCIENCES | Organ Specificity - drug effects | Protein Binding - genetics | Reactive Oxygen Species - metabolism | Sodium Chloride - pharmacology | Gene Expression Profiling | Protein Transport - drug effects | RNA, Plant - metabolism | Organ Specificity - genetics | Oryza - growth & development | Peptides - metabolism | Oryza - genetics | RNA Interference | Adaptation, Physiological - genetics | Protein Binding - drug effects | Plant Proteins - metabolism | Subcellular Fractions - drug effects | Stress, Physiological - genetics | Oxidative Stress - genetics | Oryza - physiology | Transcriptome - drug effects | Transcriptome - genetics | Oryza - drug effects | Subcellular Fractions - metabolism | Plant Proteins - genetics | Gene Expression Regulation, Plant - drug effects | Metals - metabolism | Phenotype | Genes, Plant - genetics | Zinc Fingers - genetics | Oxidative Stress - drug effects | Plant Growth Regulators - pharmacology | Genetic aspects | Stress (Physiology) | Zinc finger proteins | Research | Genetic regulation | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 2014, Volume 507, Issue 7493, pp. 448 - 454
Human neurons are functional over an entire lifetime, yet the mechanisms that preserve function and protect against neurodegeneration during ageing are... 
LIFE-SPAN | CAENORHABDITIS-ELEGANS | PROTEIN | CHROMATIN | MULTIDISCIPLINARY SCIENCES | GENOME-WIDE ANALYSIS | NEURONS | MILD COGNITIVE IMPAIRMENT | SEL-12 PRESENILIN | REPRESSOR | C. ELEGANS | Neurons - pathology | Up-Regulation | Humans | Oxidative Stress - physiology | Caenorhabditis elegans Proteins - metabolism | Phagosomes | Neurons - cytology | Cognition | Autophagy | Neuroprotective Agents - metabolism | Alzheimer Disease - pathology | Brain - metabolism | DNA-Binding Proteins - metabolism | Frontotemporal Dementia - metabolism | Young Adult | Cell Nucleus - metabolism | Aging - genetics | Cell Death - genetics | Chromatin Immunoprecipitation | Repressor Proteins - deficiency | Aged, 80 and over | Neurons - metabolism | Repressor Proteins - metabolism | Wnt Signaling Pathway | Frontotemporal Dementia - pathology | Brain - cytology | Amyloid beta-Peptides - toxicity | Cognitive Dysfunction - metabolism | Down-Regulation | Gene Expression Regulation | Lewy Body Disease - pathology | Oxidative Stress - genetics | Repressor Proteins - genetics | Longevity | Aging - pathology | Lewy Body Disease - metabolism | Transcription Factors - metabolism | Amyloid beta-Peptides - antagonists & inhibitors | Animals | Alzheimer Disease - metabolism | Brain - pathology | Aged | Mice | Alzheimer Disease - genetics | Aging - metabolism | Oxidative stress | Control | Transcription factors | Neurons | Aging | Physiological aspects | Genetic aspects | Research | Alzheimer's disease | Studies | Pathology | Cognitive ability | Epigenetics | Kinases | Stress response | Gene expression | Alzheimers disease | Age | Apoptosis | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 8/2008, Volume 105, Issue 32, pp. 11364 - 11369
Parkinson's disease (PD) is a common neurodegenerative disorder thought to be associated with mitochondrial dysfunction. Loss of function mutations in the... 
Oxidative stress | Mitochondria | Cerebral cortex | Parkinsonian disorders | Reactive oxygen species | Parkinson disease | Mice | Respiration | Genetic mutation | Genotypes | Genetic Parkinson's disease | Parkinsonism | PARK6 | Neurodegeneration | Knockout | knockout | PROTEIN | parkinsonism | MULTIDISCIPLINARY SCIENCES | neurodegeneration | RECESSIVE PARKINSONISM | DAMAGE | genetic Parkinson's disease | COMPLEX-I | DOPAMINE | MUTATIONS | DYSFUNCTION | DROSOPHILA-PINK1 | BRAIN | PARKINSONS-DISEASE | Protein Kinases - metabolism | Protein Kinases - genetics | Cerebral Cortex - pathology | Mitochondrial Diseases - metabolism | Mitochondrial Proteins - genetics | Cerebral Cortex - metabolism | Mitochondria - genetics | Time Factors | Mitochondrial Proteins - metabolism | Gene Deletion | Germ-Line Mutation | Parkinson Disease - metabolism | Mitochondrial Diseases - genetics | Parkinson Disease - pathology | Oxidative Stress - genetics | Mitochondria - metabolism | Mitochondria - pathology | Parkinson Disease - genetics | Heat-Shock Response - genetics | Hydrogen Peroxide - metabolism | Mice, Knockout | Citric Acid Cycle - genetics | Animals | Oxygen Consumption - genetics | Corpus Striatum | Complications and side effects | Cellular proteins | Genetic aspects | Mitochondrial myopathies | Health aspects | Risk factors | Proteins | Mutation | Microscopy | Rodents | Cells | Parkinsons disease | Index Medicus | Biological Sciences
Journal Article
Nature Neuroscience, ISSN 1097-6256, 05/2009, Volume 12, Issue 5, pp. 627 - 636
The mechanisms underlying disease manifestations in neurodegeneration remain unclear, but their understanding is critical to devising effective therapies. We... 
NEURODEGENERATIVE DISEASE | UNFOLDED PROTEIN RESPONSE | MOTOR-NEURON DEGENERATION | WILD-TYPE SOD1 | ALS-LINKED SOD1 | MOUSE MODEL | AMYOTROPHIC-LATERAL-SCLEROSIS | ENDOPLASMIC-RETICULUM STRESS | MUTANT SUPEROXIDE-DISMUTASE | NEUROSCIENCES | TRANSGENIC MICE | Microglia - metabolism | Central Nervous System - metabolism | Amyotrophic Lateral Sclerosis - physiopathology | Ubiquitin - metabolism | Central Nervous System - pathology | Endoplasmic Reticulum - metabolism | Gliosis - physiopathology | Thiourea - pharmacology | Mice, Neurologic Mutants | Motor Neurons - classification | Motor Neurons - pathology | Ubiquitination | Neuroprotective Agents - pharmacology | Disease Models, Animal | Wallerian Degeneration - genetics | Biomarkers - metabolism | Genetic Predisposition to Disease - genetics | Gliosis - genetics | Endoplasmic Reticulum - genetics | Wallerian Degeneration - metabolism | Amyotrophic Lateral Sclerosis - genetics | Gliosis - metabolism | Wallerian Degeneration - physiopathology | Biomarkers - analysis | Oxidative Stress - genetics | Cinnamates - pharmacology | Disease Progression | Protein Folding | Motor Neurons - metabolism | Phenotype | Animals | Amyotrophic Lateral Sclerosis - metabolism | Mice | Central Nervous System - physiopathology | Thiourea - analogs & derivatives | Motor neurons | Physiological aspects | Amyotrophic lateral sclerosis | Nervous system | Degeneration | Research | Endoplasmic reticulum | Index Medicus
Journal Article
by Weinstein, John N and Akbani, Rehan and Broom, Bradley M and Wang, Wenyi and Verhaak, Roeland G. W and McConkey, David and Lerner, Seth and Morgan, Margaret and Creighton, Chad J and Smith, Carolyn and Cherniack, Andrew D and Kim, Jaegil and Pedamallu, Chandra Sekhar and Noble, Michael S and Al-Ahmadie, Hikmat A and Reuter, Victor E and Rosenberg, Jonathan E and F.Bajorin, Dean and Bochner, Bernard H and Solit, David B and Koppie, Theresa and Robinson, Brian and Gordenin, Dmitry A and Fargo, David and Klimczak, Leszek J and Roberts, Steven A and Au, Jessie and Laird, Peter W and Hinoue, Toshinori and Schultz, Nikolaus and Ramirez, Ricardo and Hansel, Donna and Hoadley, Katherine A and Kim, William Y and Damrauer, Jeffrey S and Baylin, Stephen B and Mungall, Andrew J and Robertson, A. Gordon and Chu, Andy and Kwiatkowski, David J and Sougnez, Carrie and Cibulskis, Kristian and Lichtenstein, Lee and Sivachenko, Andrey and Stewart, Chip and Lawrence, Michael S and Getz, Gad and Lander, Eric and Gabrie, Stacey B and Donehower, Lawrence and Carter, Scott L and Saksena, Gordon and Schumacher, Steven E and Freeman, Samuel S and Jung, Joonil and Bhatt, Ami S and Pugh, Trevor and Beroukhim, Rameen and Meyerson, Matthew and Ally, Adrian and Balasundaram, Miruna and Butterfield, Yaron S. N and Dhalla, Noreen and Hirst, Carrie and Holt, Robert A and Jones, Steven J. M and Lee, Darlene and Li, Haiyan I and Marra, Marco A and Mayo, Michael and Moore, Richard A and Schein, Jacqueline E and Sipahimalani, Payal and Tam, Angela and Thiessen, Nina and Wong, Tina and Wye, Natasja and Bowlby, Reanne and Chuah, Eric and Guin, Ranabir and Shen, Hui and Bootwalla, Moiz S and Triche Jr, Timothy and Lai, Phillip H and Van Den Berg, David J and Weisenberger, Daniel J and Balu, Saianand and Bodenheimer, Tom and Hoyle, Alan P and Jefferys, Stuart R and Meng, Shaowu and Mose, Lisle E and Simons, Janae V and Soloway, Mathew G and Wu, Junyuan and Parker, Joel S and Hayes, D. Neil and Roach, Jeffrey and Buda, Elizabeth and Jones, Corbin D and ... and Canc Genome Atlas Res Network and Cancer Genome Atlas Research Network and The Cancer Genome Atlas Research Network
Nature, ISSN 0028-0836, 2014, Volume 507, Issue 7492, pp. 315 - 322
Journal Article
Cell, ISSN 0092-8674, 2007, Volume 128, Issue 2, pp. 325 - 339
Journal Article