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Clinical and Experimental Pharmacology and Physiology, ISSN 0305-1870, 05/2007, Volume 34, Issue 5, pp. 387 - 392
SUMMARY • Astragaloside IV is a component from the widely used traditional Chinese herb Astragalus membranaceus and its effect on rat aortic ring contraction... 
endothelium | nitric oxide | aortic rings | vessel contraction and relaxation | astragaloside IV | Astragaloside IV | Nitric oxide | Vessel contraction and relaxation | Aortic rings | Endothelium | SMOOTH-MUSCLE | PHYSIOLOGY | CONTRACTION | TONE | L-ARGININE | PHARMACOLOGY & PHARMACY | CHANNELS | Triterpenes - pharmacology | Muscle, Smooth, Vascular - metabolism | Calcium - metabolism | Nitric Oxide Synthase - antagonists & inhibitors | Male | Saponins - chemistry | Endothelium, Vascular - physiology | Dose-Response Relationship, Drug | Quinoxalines - pharmacology | Intracellular Fluid - drug effects | Triterpenes - chemistry | Phenylephrine - pharmacology | Oxadiazoles - pharmacology | Aorta, Thoracic - physiology | Potassium Chloride - pharmacology | Molecular Structure | Aorta, Thoracic - drug effects | Cyclic GMP - biosynthesis | Intracellular Fluid - metabolism | Muscle, Smooth, Vascular - drug effects | NG-Nitroarginine Methyl Ester - pharmacology | Vasoconstrictor Agents - pharmacology | Vasodilator Agents - pharmacology | Acetylcholine - pharmacology | Rats | Vasodilator Agents - isolation & purification | Muscle, Smooth, Vascular - cytology | Rats, Sprague-Dawley | Indomethacin - pharmacology | Nitroarginine - pharmacology | Aorta, Thoracic - cytology | Calcium Chloride - pharmacology | Animals | Saponins - isolation & purification | Triterpenes - isolation & purification | Vasodilator Agents - chemistry | Nitric Oxide Synthase - metabolism | Vasodilation - drug effects | In Vitro Techniques | Saponins - pharmacology | NG-Nitroarginine Methyl Ester, pharmacology | Triterpenes, pharmacology | Calcium, metabolism | Nitric Oxide Synthase, antagonists and inhibitors | Muscle, Smooth, Vascular, metabolism | Vasodilator Agents, chemistry | Quinoxalines, pharmacology | Aorta, Thoracic, drug effects | Triterpenes, chemistry | Vasodilator Agents, pharmacology | Saponins, chemistry | Intracellular Fluid, drug effects | Nitric Oxide Synthase, metabolism | Aorta, Thoracic, cytology | Potassium Chloride, pharmacology | Aorta, Thoracic, physiology | Calcium Chloride, pharmacology | Vasoconstrictor Agents, pharmacology | Muscle, Smooth, Vascular, drug effects | Nitroarginine, pharmacology | Saponins, pharmacology | Vasodilation, drug effects | Triterpenes, isolation and purification | Cyclic GMP, biosynthesis | Vasodilator Agents, isolation and purification | Acetylcholine, pharmacology | Oxadiazoles, pharmacology | Muscle, Smooth, Vascular, cytology | Phenylephrine, pharmacology | Saponins, isolation and purification | Endothelium, Vascular, physiology | Indomethacin, pharmacology | Intracellular Fluid, metabolism
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 01/2009, Volume 29, Issue 2, pp. 393 - 401
Glucocorticoids exert an opposing rapid regulation of glutamate and GABA synaptic inputs to hypothalamic magnocellular neurons via the activation of... 
GABA | Magnocellular neuron | Endocannabinoid | Glucocorticoid | Nitric oxide | SUPRAOPTIC NUCLEUS | endocannabinoid | glucocorticoid | nitric oxide | ACTIVATION | magnocellular neuron | NITRIC-OXIDE SYNTHASE | NEUROSCIENCES | MEMBRANE ESTROGEN-RECEPTORS | ENDOCANNABINOID RELEASE | PARAVENTRICULAR NUCLEUS | G(alpha) | ADRENALECTOMIZED RATS | NEURONS | VASOPRESSIN | OXYTOCIN | G(beta)gamma | Cannabinoid Receptor Modulators - metabolism | Synaptic Transmission - physiology | gamma-Aminobutyric Acid - metabolism | GTP-Binding Proteins - antagonists & inhibitors | Male | Penicillamine - analogs & derivatives | Patch-Clamp Techniques - methods | Quinoxalines - pharmacology | Dexamethasone - pharmacology | Piperidines - pharmacology | Dronabinol - analogs & derivatives | Neurons - physiology | Nitric Oxide Donors - pharmacology | Dronabinol - pharmacology | Neurons - drug effects | Electric Stimulation - methods | Pyrans - pharmacology | Guanosine Diphosphate - pharmacology | Pyrazoles - pharmacology | Cyclic AMP-Dependent Protein Kinases - metabolism | Synapses - drug effects | Synapses - physiology | Penicillamine - pharmacology | Valine - analogs & derivatives | Enzyme Inhibitors - pharmacology | Rats | Excitatory Amino Acid Antagonists - pharmacology | Thionucleotides - pharmacology | Rats, Sprague-Dawley | Animals | Guanosine Diphosphate - analogs & derivatives | Hypothalamus - cytology | Signal Transduction - drug effects | Arginine - pharmacology | Cannabinoid Receptor Modulators - antagonists & inhibitors | Glucocorticoids - pharmacology | Glutamic Acid - metabolism | Valine - pharmacology | In Vitro Techniques | Nitric Oxide - metabolism | GTP-Binding Proteins - metabolism | Gβγ
Journal Article
Clinical and Experimental Pharmacology and Physiology, ISSN 1440-1681, 02/2008, Volume 35, Issue 2, pp. 192 - 200
SUMMARY • The present study evaluated the effect of diabetes, hypercholesterolaemia and their combination on the contribution of nitric oxide (NO) and... 
endothelium-derived hyperpolarising factor | diabetes | endothelium-dependent relaxation | hypercholesterolaemia | superoxide anion | endothelium‐derived hyperpolarising factor | endothelium‐dependent relaxation | Superoxide anion | Diabetes | Hypercholesterolaemia | Endothelium-dependent relaxation | Endothelium-derived hyperpolarising factor | PHYSIOLOGY | NITRO-L-ARGININE | ACETYLCHOLINE | MELLITUS | NAD(P)H OXIDASE | ARTERIES | PHARMACOLOGY & PHARMACY | SMOOTH-MUSCLE-CELLS | CHANNELS | DYSFUNCTION | EDHF | Body Weight | Oxidative Stress | Cholesterol - blood | Diabetes Mellitus, Type 1 - metabolism | Nitric Oxide Synthase - antagonists & inhibitors | Male | Guanylate Cyclase - antagonists & inhibitors | Nitroprusside - pharmacology | Apamin - pharmacology | Dose-Response Relationship, Drug | Quinoxalines - pharmacology | Charybdotoxin - pharmacology | Oxadiazoles - pharmacology | Potassium Channels, Calcium-Activated - metabolism | Superoxides - metabolism | Aorta, Thoracic - drug effects | Biological Factors - metabolism | Diabetes Mellitus, Experimental - metabolism | Potassium Channel Blockers - pharmacology | Diabetes Mellitus, Experimental - physiopathology | Disease Models, Animal | Vasodilator Agents - pharmacology | Acetylcholine - pharmacology | Diabetes Mellitus, Type 1 - physiopathology | Enzyme Inhibitors - pharmacology | Aorta, Thoracic - metabolism | Rats | Guanylate Cyclase - metabolism | Soluble Guanylyl Cyclase | Aorta, Thoracic - physiopathology | Rats, Sprague-Dawley | Indomethacin - pharmacology | Cyclooxygenase Inhibitors - pharmacology | Nitroarginine - pharmacology | Aorta, Thoracic - enzymology | Animals | Receptors, Cytoplasmic and Nuclear - antagonists & inhibitors | Hypercholesterolemia - physiopathology | Nitric Oxide Synthase - metabolism | Potassium Channels, Calcium-Activated - antagonists & inhibitors | Vasodilation - drug effects | Blood Glucose - metabolism | Nitric Oxide - metabolism | Hypercholesterolemia - metabolism | Receptors, Cytoplasmic and Nuclear - metabolism | Charybdotoxin, pharmacology | Diabetes Mellitus, Type 1, physiopathology | Nitric Oxide Synthase, antagonists and inhibitors | Aorta, Thoracic, physiopathology | Potassium Channels, Calcium-Activated, antagonists and inhibitors | Hypercholesterolemia, metabolism | Quinoxalines, pharmacology | Enzyme Inhibitors, pharmacology | Aorta, Thoracic, drug effects | Aorta, Thoracic, metabolism | Receptors, Cytoplasmic and Nuclear, antagonists and inhibitors | Vasodilator Agents, pharmacology | Vasodilation | Nitric Oxide Synthase, metabolism | Cyclooxygenase Inhibitors, pharmacology | Apamin, pharmacology | Nitroarginine, pharmacology | Potassium Channel Blockers, pharmacology | Vasodilation, drug effects | Cholesterol, blood | Receptors, Cytoplasmic and Nuclear, metabolism | Biological Factors, metabolism | Diabetes Mellitus, Experimental, physiopathology | Nitric Oxide, metabolism | Acetylcholine, pharmacology | Diabetes Mellitus, Experimental, metabolism | Nitroprusside, pharmacology | Oxadiazoles, pharmacology | Blood Glucose, metabolism | Potassium Channels, Calcium-Activated, metabolism | Aorta, Thoracic, enzymology | Hypercholesterolemia, physiopathology | Diabetes Mellitus, Type 1, metabolism | Guanylate Cyclase, antagonists and inhibitors | Guanylate Cyclase, metabolism | Superoxides, metabolism | Indomethacin, pharmacology | Superoxide | Hypercholesterolemia | Endothelium-derived relaxing factors | Type 1 diabetes | Endothelium
Journal Article
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 03/2006, Volume 26, Issue 11, pp. 2862 - 2870
Neuronal activity evokes localized changes in blood flow. Although this response, termed neurovascular coupling, is widely used to monitor human brain function... 
Arachidonic acid metabolites | Retina | Calcium | Glia | ATP | Blood flow | arachidonic acid metabolites | 20-HETE | calcium | RAT | ASTROCYTE-MEDIATED CONTROL | CALCIUM WAVES | EPOXYGENASE | ARACHIDONIC-ACID | RELEASE | glia | NEUROSCIENCES | HEMODYNAMIC-RESPONSE | retina | NITRIC-OXIDE | blood flow | BRAIN | Arterioles - physiology | Cytochrome P-450 Enzyme Inhibitors | Rats, Long-Evans | Arterioles - enzymology | Nitric Oxide Synthase - physiology | Male | Vasodilation - radiation effects | Retinal Vessels - enzymology | Retinal Vessels - physiology | Hydroxyeicosatetraenoic Acids - biosynthesis | Vasoconstriction - radiation effects | Photolysis | Cyclic N-Oxides - pharmacology | Inositol 1,4,5-Trisphosphate - analogs & derivatives | Eye Proteins - antagonists & inhibitors | Purinergic Antagonists | Signal Transduction - radiation effects | Cytochrome P-450 Enzyme System - physiology | Hydrazines - pharmacology | Rats | Imidazoles - pharmacology | Retinal Vessels - drug effects | Arterioles - drug effects | Vasoconstriction - drug effects | Cytochrome P-450 CYP4A - physiology | Signal Transduction - drug effects | Miconazole - pharmacology | Eye Proteins - physiology | Retinal Vessels - radiation effects | Vasodilation - drug effects | Arterioles - radiation effects | Nitric Oxide - pharmacology | Caproates - pharmacology | Vasomotor System - drug effects | Nitric Oxide Synthase - antagonists & inhibitors | Inositol 1,4,5-Trisphosphate - pharmacology | Adenosine Triphosphate - pharmacology | Vasoconstriction - physiology | 8,11,14-Eicosatrienoic Acid - analogs & derivatives | Proadifen - pharmacology | Nitric Oxide Donors - pharmacology | Vasodilation - physiology | 8,11,14-Eicosatrienoic Acid - pharmacology | NG-Nitroarginine Methyl Ester - pharmacology | Calcium Signaling - radiation effects | Cytochrome P-450 CYP4A - antagonists & inhibitors | Neuroglia - physiology | Nitric Oxide - physiology | Vasomotor System - radiation effects | Animals | Vasomotor System - physiology | Amidines - pharmacology | In Vitro Techniques
Journal Article
British journal of pharmacology, ISSN 1476-5381, 2002, Volume 137, Issue 7, pp. 1021 - 1030
The mechanism(s) by which vascular endothelial growth factor (VEGF) induces endothelial nitric oxide synthase (eNOS) activation remain(s) unclear up to a... 
eNOS | PAF | PI3K | VEGF | PLC | PLA2 | PKC | PLA | PROTEIN-KINASE-C | PHOSPHORYLATION | NITRIC-OXIDE SYNTHASE | VASCULAR-PERMEABILITY | CALMODULIN | CALCIUM | PAF SYNTHESIS | PHARMACOLOGY & PHARMACY | PLATELET-ACTIVATING-FACTOR | GROWTH-FACTOR | NEOVASCULARIZATION | Endothelium, Vascular - cytology | Vascular Endothelial Growth Factors | Platelet Activating Factor - pharmacology | Protein-Serine-Threonine Kinases | Type C Phospholipases - metabolism | Endothelium, Vascular - drug effects | Endothelial Growth Factors - pharmacology | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Receptors, Cell Surface - antagonists & inhibitors | Lactones - pharmacology | Platelet Membrane Glycoproteins - antagonists & inhibitors | Calcium - antagonists & inhibitors | Time Factors | Cattle | Protein Kinase C - metabolism | Phosphorylation - drug effects | Chromones - pharmacology | Enzyme Inhibitors - pharmacology | Nitric Oxide Synthase Type III | Receptors, G-Protein-Coupled | Chelating Agents - pharmacology | Serine - metabolism | 1-Methyl-3-isobutylxanthine - pharmacology | Ginkgolides | Platelet Activating Factor - metabolism | Cyclic GMP - metabolism | Endothelium, Vascular - metabolism | Intercellular Signaling Peptides and Proteins - pharmacology | Nitric Oxide Synthase - metabolism | Vascular Endothelial Growth Factor A | Maleimides - pharmacology | Nitric Oxide Synthase - antagonists & inhibitors | Phosphatidylinositol 3-Kinases - metabolism | Nitroprusside - pharmacology | Type C Phospholipases - antagonists & inhibitors | Dose-Response Relationship, Drug | Egtazic Acid - analogs & derivatives | Indoles - pharmacology | Lymphokines - pharmacology | Nitric Oxide Donors - pharmacology | Ionophores - pharmacology | NG-Nitroarginine Methyl Ester - pharmacology | Proto-Oncogene Proteins - metabolism | Cells, Cultured | Morpholines - pharmacology | Protein Kinase C - antagonists & inhibitors | Calcimycin - pharmacology | Proto-Oncogene Proteins c-akt | Animals | Egtazic Acid - pharmacology | Androstadienes - pharmacology | Diterpenes | Signal Transduction - physiology | Nitric Oxide - metabolism | Papers
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 08/2014, Volume 73, pp. 51 - 59
Microglia are the resident immune cells in the brain. Microglial activation is characteristic of several inflammatory and neurodegenerative diseases including... 
Peroxynitrite | Free radicals | Protein radical | Inducible nitric oxide synthase | Lipopolysaccharide | Nitrone adducts | Parkinson disease | Microglia | NADPH OXIDASE | OXIDATIVE STRESS | MACROPHAGES | BIOCHEMISTRY & MOLECULAR BIOLOGY | INOS EXPRESSION | KAPPA-B | LIPOPOLYSACCHARIDE-ACTIVATED MICROGLIA | REACTIVE OXYGEN | ENDOCRINOLOGY & METABOLISM | GENERATION | TYROSINE NITRATION | PARKINSONS-DISEASE | Microglia - metabolism | Metalloporphyrins - pharmacology | Lipopolysaccharides | Thiocarbamates - pharmacology | RNA Interference | Nitric Oxide Synthase Type II - antagonists & inhibitors | Spin Trapping | Acetophenones - pharmacology | Free Radicals - metabolism | Proline - pharmacology | Microglia - cytology | NG-Nitroarginine Methyl Ester - pharmacology | NF-kappa B - antagonists & inhibitors | Proline - analogs & derivatives | Enzyme Inhibitors - pharmacology | Imidazoles - pharmacology | Antioxidants - pharmacology | Neurodegenerative Diseases - metabolism | Coumarins - pharmacology | Animals | Nitric Oxide Synthase Type II - genetics | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Peroxynitrous Acid - metabolism | Amidines - pharmacology | Mice | Pyridines - pharmacology | RNA, Small Interfering | Benzylamines - pharmacology | Cell Line, Transformed | Boronic Acids - pharmacology | Nitric Oxide Synthase Type II - metabolism | Nitration | Nervous system diseases | Multiple sclerosis | Analysis | Nitric oxide | Environmental health | Physiological aspects | Alzheimer's disease | Index Medicus | Inducible nitric | oxide synthase | LPS | Parkinson’s disease
Journal Article
Planta, ISSN 1432-2048, 2008, Volume 229, Issue 4, pp. 757 - 765
The effects of chitosan (β-1,4 linked glucosamine, a fungal elicitor), on the patterns of stomatal movement and signaling components were studied. cPTIO (NO... 
Pathogens | Reactive oxygen species | Calcium | Sodium | Fluorescence | Guard cells | Oxides | Stomata | Plants | Tungstates | Life Sciences | Signal transduction | Nitric oxide | Forestry | ROS | Ecology | Agriculture | Chitosan | Plant Sciences | Pea | ARABIDOPSIS-THALIANA | FREE CALCIUM | CULTURED-CELLS | PROTEIN-KINASES | BLUE-LIGHT | PLANT SCIENCES | SIGNAL-TRANSDUCTION | STRESS RESPONSES | HYDROGEN-PEROXIDE | ABSCISIC-ACID | DISEASE RESISTANCE | Free Radical Scavengers - pharmacology | Oxidants - pharmacology | Reactive Oxygen Species - metabolism | Nitric Oxide Synthase - antagonists & inhibitors | Tungsten Compounds - pharmacology | Peas - cytology | Plant Epidermis - metabolism | Dose-Response Relationship, Drug | Time Factors | Cyclic N-Oxides - pharmacology | Egtazic Acid - analogs & derivatives | Chitosan - pharmacology | Plant Epidermis - cytology | Plant Stomata - drug effects | Peas - metabolism | NG-Nitroarginine Methyl Ester - pharmacology | Enzyme Inhibitors - pharmacology | Hydrogen Peroxide - pharmacology | Peas - drug effects | Imidazoles - pharmacology | Chelating Agents - pharmacology | Onium Compounds - pharmacology | Plant Epidermis - drug effects | Abscisic Acid - pharmacology | Microscopy, Confocal | Egtazic Acid - pharmacology | Kinetics | Nitric Oxide - metabolism | Plant Growth Regulators - pharmacology | Plant Stomata - physiology | Oxidases | Legumes | Beans | Mimosaceae | Tungsten compounds | Index Medicus
Journal Article
British journal of pharmacology, ISSN 0007-1188, 2017, Volume 174, Issue 11, pp. 1290 - 1324
... to corresponding entries in http://www.guidetopharmacology.org , the common portal for data from the IUPHAR/BPS Guide to PHARMACOLOGY (Southan et al ., ) and are permanently... 
IN-VITRO | DIETARY BETA-CAROTENE | NITRIC-OXIDE SYNTHASE | HUMORAL IMMUNE-RESPONSE | PROSTATE-CANCER | PHARMACOLOGY & PHARMACY | MAJOR DEPRESSIVE DISORDER | HUMAN BREAST-CANCER | NF-KAPPA-B | VITAMIN-A | CANCER CELL-PROLIFERATION | Fruit - chemistry | Humans | Neoplasms - prevention & control | Antioxidants - pharmacology | Carotenoids - pharmacology | Dose-Response Relationship, Drug | Vegetables - chemistry | Animals | Neoplasms - diet therapy | Diet | Time Factors | Antioxidants - administration & dosage | Carotenoids - administration & dosage | Dietary Supplements | Retinoids - pharmacology | Colorectal cancer | Biochemistry | Retinoids | T cells | Gene expression | Epidemiology | Lycopene | Prevention | Proteins | Antioxidants | Eye diseases | Cancer | Beta carotene | Intercellular signalling | Transcription factors | Transcription | Effector cells | Cytotoxicity | Lymphocytes T | Cell interactions | Macrophages | Eye | Signal transduction | β-catenin | Time dependence | Receptors | Carotenoids | Immunology | Lymphocytes | Cell cycle | Curcumin | Growth factors | Nutraceuticals | Immune response | Cytokines | Carotene | Gap junctions | Dietary supplements | Lutein | Inflammation | Pharmacology | Cervix | Lymphocytes B | Correlation analysis | Risk management | Tumors | Apoptosis | Themed Section | Review
Journal Article