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Journal Article
Lipids in Health and Disease, ISSN 1476-511X, 04/2017, Volume 16, Issue 1, pp. 77 - 10
Background: Atherosclerosis is a common cardiovascular disease that causes myocardial infarction, heart failure, and stroke. Increased oxidized low density... 
Oxidative stress | Oxidized low density lipoprotein | Human umbilical vein endothelial cells | Klotho | Lectin-like ox-LDL receptor | APOPTOSIS | ATHEROGENESIS | ACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | PROTECTS ENDOTHELIAL-CELLS | ATHEROSCLEROSIS | MECHANISMS | NUTRITION & DIETETICS | DISEASE | REQUIRES | DYSFUNCTION | SUPEROXIDE-DISMUTASE | Endothelium, Vascular - cytology | Reactive Oxygen Species - metabolism | Human Umbilical Vein Endothelial Cells - metabolism | Oxidative Stress | Phosphatidylinositol 3-Kinase - antagonists & inhibitors | Humans | Glucuronidase - metabolism | Superoxide Dismutase - antagonists & inhibitors | Proto-Oncogene Proteins c-akt - genetics | Reactive Oxygen Species - agonists | Nitric Oxide Synthase Type III - chemistry | Malondialdehyde - agonists | Human Umbilical Vein Endothelial Cells - cytology | Lipoproteins, LDL - metabolism | Nitric Oxide Synthase Type III - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Phosphatidylinositol 3-Kinase - metabolism | Second Messenger Systems | Superoxide Dismutase - metabolism | Malondialdehyde - metabolism | Recombinant Proteins - metabolism | Atherosclerosis - pathology | Scavenger Receptors, Class E - agonists | Cell Survival | Nitric Oxide - antagonists & inhibitors | Cells, Cultured | Scavenger Receptors, Class E - antagonists & inhibitors | Nitric Oxide Synthase Type III - genetics | Atherosclerosis - metabolism | Nitric Oxide - agonists | Phosphatidylinositol 3-Kinase - chemistry | Superoxide Dismutase - chemistry | Scavenger Receptors, Class E - metabolism | Reactive Oxygen Species - antagonists & inhibitors | Human Umbilical Vein Endothelial Cells - enzymology | Glucuronidase - genetics | Lipoproteins, LDL - antagonists & inhibitors | Phosphatidylinositol 3-Kinase - genetics | Endothelium, Vascular - metabolism | Proto-Oncogene Proteins c-akt - agonists | Endothelium, Vascular - pathology | Lipid Peroxidation | Malondialdehyde - antagonists & inhibitors | Nitric Oxide - metabolism | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Myocardial infarction | Lipoproteins (low density) | Atherogenesis | Superoxide dismutase | Cardiovascular disease | AKT protein | Cell adhesion & migration | Proteins | Lysates | Cell growth | Atherosclerosis | Aging | Tumor necrosis factor-TNF | Heart diseases | Cerebral infarction | Stroke | Inositol | Cytokines | LOX-1 protein | Low density lipoprotein | Nitric-oxide synthase | Malondialdehyde | Endothelial cells | 1-Phosphatidylinositol 3-kinase | Endothelium | Polymerase chain reaction | Klotho protein | Arteriosclerosis | Nitric oxide | Calcification | Diabetes | Umbilical vein | Apoptosis
Journal Article
by Di, XH and Tang, XL and Di, XT
BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS, ISSN 0006-291X, 04/2017, Volume 486, Issue 1, pp. 58 - 62
Recruitment of monocytes to endothelial cells is important during early stages of atherosclerosis development, which is activated in response to a number of... 
Vascular cell adhesion molecule-1 (VCAM-1) | CELLS | WALL | Oxidized low-density lipoprotein (ox-LDL) | BIOPHYSICS | INFLAMMATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | DISEASE | Endothelial inflammation | Montelukast Kruppel-like factor 2 | EXPRESSION | KLF2
Journal Article
Journal Article
Journal Article
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 04/2017, Volume 486, Issue 1, pp. 58 - 62
Recruitment of monocytes to endothelial cells is important during early stages of atherosclerosis development, which is activated in response to a number of... 
Vascular cell adhesion molecule-1 (VCAM-1) | Endothelial inflammation | Montelukast | Kruppel-like factor 2 | Oxidized low-density lipoprotein (ox-LDL) | Gene Expression - drug effects | Human Umbilical Vein Endothelial Cells - metabolism | Humans | Acetates - chemistry | Endothelium, Vascular - drug effects | Monocytes - metabolism | Quinolines - pharmacology | Dose-Response Relationship, Drug | RNA Interference | E-Selectin - genetics | Kruppel-Like Transcription Factors - metabolism | Protective Agents - pharmacology | Vascular Cell Adhesion Molecule-1 - genetics | Molecular Structure | Anti-Asthmatic Agents - chemistry | Anti-Asthmatic Agents - pharmacology | Atherosclerosis - drug therapy | Human Umbilical Vein Endothelial Cells - drug effects | Quinolines - chemistry | Cells, Cultured | Lipoproteins, LDL - pharmacology | E-Selectin - metabolism | Mitogen-Activated Protein Kinase 7 - genetics | Mitogen-Activated Protein Kinase 7 - metabolism | Cell Adhesion - drug effects | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Monocytes - drug effects | Endothelium, Vascular - metabolism | Cell Line, Tumor | Acetates - pharmacology | Vascular Cell Adhesion Molecule-1 - metabolism | Kruppel-Like Transcription Factors - genetics | Atherosclerosis - prevention & control | Care and treatment | Low density lipoproteins | Atherosclerosis | Development and progression | Blood lipids | Drug approval | Asthma | Endothelium | Drug therapy
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 04/2001, Volume 276, Issue 17, pp. 13750 - 13755
Journal Article
Journal Article
European Journal of Pharmacology, ISSN 0014-2999, 06/2019, Volume 853, pp. 84 - 92
Overexpression of connexin 43 (Cx43) was related to dysfunction of vascular smooth muscle cells (VSMCs). Our previous study reported that rutaecarpine, an... 
Vascular smooth muscle cells (VSMCs) | Oxidized low-density lipoprotein (ox-LDL) | Connexin 43 (Cx43) | Rutaecarpine | Transient receptor potential vanilloid subtype 1 (TRPV1) | ACTIVATION | PROLIFERATION | PEPTIDE | GAP-JUNCTIONS | PHARMACOLOGY & PHARMACY | SMOOTH-MUSCLE-CELLS | UP-REGULATION | EXPRESSION
Journal Article
by Yin, J and Hou, XH and Yang, SB
JOURNAL OF CELLULAR PHYSIOLOGY, ISSN 0021-9541, 07/2019, Volume 234, Issue 7, pp. 11577 - 11586
microRNAs (miRNAs) have been revealed to participate in the pathological process of atherosclerosis (AS). However, the exact role of miR-338-3p, a target miRNA... 
PHYSIOLOGY | BMP and activin membrane-bound inhibitor (BAMBI) | Smad pathway | INVOLVEMENT | atherosclerosis (AS) | PROTECTS | BETA | CELL BIOLOGY | HEART | TGF | microRNA-338-3p | DYSFUNCTION | STRESS | EXPRESSION | oxidized low-density lipoprotein (ox-LDL) | PSORIASIS | Bone morphogenetic proteins | MicroRNA | Transforming growth factors | Low density lipoproteins | Atherosclerosis | Endothelium
Journal Article
Annals of the New York Academy of Sciences, ISSN 0077-8923, 08/2007, Volume 1109, Issue 1, pp. 303 - 310
:  The immunolocalization of oxidized low-density lipoproteins (ox-LDL), β2-glycoprotein I (β2GPI), CD4+/CD8+ immunoreactive lymphocytes, and immunoglobulins... 
atherosclerosis | autoimmunity | β2-glycoprotein I (β2GPI | oxidized low-density lipoprotein (ox-LDL) | oxidative stress | ELISA
Journal Article
Annals of the New York Academy of Sciences, ISSN 0077-8923, 08/2007, Volume 1109, Issue 1, pp. 303 - 310
Abstract :  The immunolocalization of oxidized low‐density lipoproteins (ox‐LDL), β2‐glycoprotein I (β 2 GPI), CD4 + /CD8 + immunoreactive lymphocytes, and... 
atherosclerosis | GPI | autoimmunity | β2‐glycoprotein I (β | oxidized low‐density lipoprotein (ox‐LDL) | oxidative stress | ELISA | Autoimmunity | Oxidative stress | Oxidized low-density lipoprotein (ox-LDL) | Atherosclerosis | β2-glycoprotein I (β
Journal Article