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Journal Article
The Journal of neuroscience, ISSN 1529-2401, 10/2012, Volume 32, Issue 40, pp. 13718 - 13728
The symptoms of Parkinson's disease (PD) are related to changes in the frequency and pattern of activity in the reciprocally connected GABAergic external... 
Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Dopaminergic Neurons - pathology | Nerve Tissue Proteins - analysis | Male | Synaptic Transmission | Isonicotinic Acids - pharmacology | Exploratory Behavior - physiology | RNA, Messenger - biosynthesis | Dopamine - physiology | Exploratory Behavior - drug effects | Nerve Tissue Proteins - biosynthesis | Dopaminergic Neurons - physiology | Globus Pallidus - physiopathology | Vesicular Inhibitory Amino Acid Transport Proteins - biosynthesis | Globus Pallidus - pathology | Receptors, GABA-A - biosynthesis | Parkinsonian Disorders - physiopathology | Subthalamic Nucleus - physiopathology | Membrane Proteins - genetics | Carrier Proteins - biosynthesis | Mice, Inbred C57BL | RNA, Messenger - genetics | Inhibitory Postsynaptic Potentials - physiology | Rats | Nerve Tissue Proteins - genetics | Rats, Sprague-Dawley | Carrier Proteins - genetics | Subthalamic Nucleus - pathology | Membrane Proteins - biosynthesis | Oxidopamine - toxicity | Patch-Clamp Techniques | Animals | Parkinsonian Disorders - pathology | Receptors, GABA-A - genetics | Vesicular Inhibitory Amino Acid Transport Proteins - genetics | Mice | Nerve Degeneration | Inhibitory Postsynaptic Potentials - drug effects | Index Medicus | Parkinsonian Disorders | Subthalamic Nucleus | Neurons and Cognition | Neurobiology | Receptors, GABA-A | Oxidopamine | Life Sciences | Inhibitory Postsynaptic Potentials | Vesicular Inhibitory Amino Acid Transport Proteins | Carrier Proteins | Exploratory Behavior | Dopamine | Globus Pallidus | Nerve Tissue Proteins | Membrane Proteins | Dopaminergic Neurons | RNA, Messenger | Isonicotinic Acids
Journal Article
Journal Article
The Journal of neuroscience, ISSN 1529-2401, 03/2005, Volume 25, Issue 11, pp. 2771 - 2780
Acquisition and performance of instrumental actions are assumed to require both action-outcome and stimulus-response (S-R) habit processes. Over the course of... 
Basal ganglia | Lateral striatum | Dopamine | Habit formation | Instrumental learning | Substantia nigra | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Tyrosine 3-Monooxygenase - metabolism | Conditioning, Operant - physiology | Extinction, Psychological - drug effects | Eating - physiology | Behavior, Animal | Conditioning, Operant - drug effects | Time Factors | Reaction Time - drug effects | Corpus Striatum - pathology | Dopamine - metabolism | Extinction, Psychological - physiology | Corpus Striatum - physiopathology | Rats | Reaction Time - physiology | Dopamine and cAMP-Regulated Phosphoprotein 32 - metabolism | Rats, Sprague-Dawley | Corpus Striatum - injuries | Gene Expression Regulation - drug effects | Immunohistochemistry - methods | Sympatholytics - toxicity | Eating - drug effects | Oxidopamine - toxicity | Animals | Reward | Habits | Index Medicus | Immunohistochemistry | Eating | Sympatholytics | Neurons and Cognition | Psychology and behavior | Gene Expression Regulation | Neurobiology | Reaction Time | Oxidopamine | Tyrosine 3-Monooxygenase | Dopamine and cAMP-Regulated Phosphoprotein 32 | Life Sciences | Extinction, Psychological | Corpus Striatum | Conditioning, Operant | Cognitive Sciences | Behavioral | substantia nigra | basal ganglia | instrumental learning | Cognitive | dopamine | Systems | lateral striatum | habit formation
Journal Article
The Journal of neuroscience, ISSN 1529-2401, 05/2005, Volume 25, Issue 20, pp. 5079 - 5086
High-frequency stimulation of the subthalamic nucleus ( HFS-STN) is an effective treatment for alleviating the motor symptoms of parkinsonian patients.... 
Basal ganglia | High-frequency deep brain stimulation | Microdialysis | Parkinson's disease | GABA | Subthalamic nucleus | Substantia nigra pars reticulata | Glutamate | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Tyrosine 3-Monooxygenase - metabolism | Chromatography, High Pressure Liquid - methods | Electric Stimulation | Microdialysis - methods | gamma-Aminobutyric Acid - metabolism | Rats | Male | Functional Laterality | Substantia Nigra - metabolism | Rats, Sprague-Dawley | Electrodes | Immunohistochemistry - methods | Globus Pallidus - metabolism | Oxidopamine - toxicity | Substantia Nigra - drug effects | Subthalamic Nucleus - radiation effects | Animals | Cell Count - methods | Glutamic Acid - metabolism | Electrochemistry - methods | Dose-Response Relationship, Radiation | Substantia Nigra - anatomy & histology | Index Medicus | Immunohistochemistry | gamma-Aminobutyric Acid | Subthalamic Nucleus | Globus Pallidus | Substantia Nigra | Cell Count | Biochemistry, Molecular Biology | Chromatography, High Pressure Liquid | Oxidopamine | Tyrosine 3-Monooxygenase | Glutamic Acid | Life Sciences | Electrochemistry | substantia nigra pars reticulata | basal ganglia | microdialysis | Neurobiology of Disease | subthalamic nucleus | high-frequency deep brain stimulation | glutamate
Journal Article
Neurobiology of aging, ISSN 0197-4580, 09/2012, Volume 33, Issue 9, pp. 2125 - 2137
Abstract This study was aimed to investigate the potential neuroprotective effect of neuropeptide Y (NPY) on the survival of dopaminergic cells in both in... 
Neurology | Internal Medicine | Neuropeptide Y | Neuroprotection | Parkinson's disease | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Geriatrics & Gerontology | Science & Technology | Neuroprotective Agents - therapeutic use | Dopamine Plasma Membrane Transport Proteins - metabolism | Tyrosine 3-Monooxygenase - metabolism | Protein Binding - genetics | Rats, Wistar | Substantia Nigra - pathology | Dopaminergic Neurons - pathology | Neurodegenerative Diseases - etiology | Humans | Neurodegenerative Diseases - prevention & control | Male | Chromatography, High Pressure Liquid | Arginine - analogs & derivatives | Oligopeptides - therapeutic use | Adrenergic Agents - toxicity | Receptors, Neuropeptide Y - deficiency | MAP Kinase Signaling System - genetics | Protein Binding - drug effects | Dopaminergic Neurons - drug effects | Female | Receptors, Neuropeptide Y - antagonists & inhibitors | Receptors, Neuropeptide Y - agonists | Neuroblastoma - pathology | Disease Models, Animal | Animals, Newborn | Cell Survival - drug effects | Parkinson Disease - complications | Mice, Inbred C57BL | Neuropeptide Y - therapeutic use | Nortropanes - pharmacokinetics | Enzyme Inhibitors - pharmacology | Rats | Neurodegenerative Diseases - genetics | Functional Laterality | Mice, Knockout | Autoradiography | Oxidopamine - toxicity | Parkinson Disease - prevention & control | Substantia Nigra - drug effects | Animals | MAP Kinase Signaling System - drug effects | Analysis of Variance | Arginine - pharmacology | Cell Line, Tumor | Mice | Parkinson Disease - etiology | Tyrosine | Analysis | Neuropeptides | Models | Hydroxylases | Index Medicus | AKT protein | Neuroprotective Agents | Receptors, Neuropeptide Y | Enzyme Inhibitors | Oligopeptides | Neurons and Cognition | Neuroblastoma | Oxidopamine | Cellular Biology | Tyrosine 3-Monooxygenase | MAP Kinase Signaling System | Life Sciences | Arginine | Adrenergic Agents | Dopamine Plasma Membrane Transport Proteins | Neurodegenerative Diseases | Substantia Nigra | Cell Survival | Parkinson Disease | Dopaminergic Neurons | Protein Binding | Nortropanes
Journal Article
Journal of neurochemistry, ISSN 0022-3042, 05/2013, Volume 125, Issue 4, pp. 620 - 633
Oxidative stress, induced by various neurodegenerative diseases, initiates a cascade of events leading to apoptosis, and thus plays a critical role in neuronal... 
cerebellar granule neurons | ODN | apoptosis | neuroprotection | oxidative stress | 6‐hydroxydopamine | 6-hydroxydopamine | Biochemistry & Molecular Biology | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Phosphorylation - physiology | Cyclin D1 - metabolism | Neurons - pathology | MAP Kinase Signaling System - physiology | Reactive Oxygen Species - metabolism | Rats, Wistar | Apoptosis - drug effects | Glutathione - metabolism | Oxidative Stress - physiology | Caspase 3 - metabolism | Cerebellum - drug effects | Peptide Fragments - pharmacology | Diazepam Binding Inhibitor - pharmacology | Neuroprotective Agents - pharmacology | Protein Kinase C - metabolism | Neuropeptides - pharmacology | Phosphorylation - drug effects | Neurons - drug effects | Rats | Cerebellum - pathology | Sympatholytics - toxicity | Oxidopamine - toxicity | Animals | MAP Kinase Signaling System - drug effects | Cyclin D1 - genetics | Apoptosis - physiology | Prevention | Nervous system diseases | Neurons | Apoptosis | Neurochemistry | Oxidative stress | Brain | Peptides | Index Medicus | Cerebellum | Neuroprotective Agents | Peptide Fragments | Phosphorylation | Reactive Oxygen Species | Sympatholytics | Oxidative Stress | Neurons and Cognition | Neurobiology | Neuropeptides | Oxidopamine | Cyclin D1 | Cellular Biology | Chemical Sciences | MAP Kinase Signaling System | Life Sciences | Caspase 3 | Pharmaceutical sciences | Glutathione | Cell Behavior | Pharmacology | Diazepam Binding Inhibitor | Medicinal Chemistry | Protein Kinase C
Journal Article
Journal of neurochemistry, ISSN 0022-3042, 10/2010, Volume 115, Issue 1, pp. 178 - 187
J. Neurochem. (2010) 115, 178–187. Recent findings suggest that a defect in the ubiquitin‐proteasome system plays an important role in the pathogenesis of... 
tyrosine inclusion body | proteasome inhibitor | dopamine | hydroxylase | medaka (Oryzias latipes) | Parkinson’s disease | Parkinson's disease | Biochemistry & Molecular Biology | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Brain Chemistry - drug effects | Immunohistochemistry | Oxidopamine - administration & dosage | Dyskinesia, Drug-Induced - physiopathology | Norepinephrine - physiology | Acetylcysteine - cerebrospinal fluid | Oryzias - physiology | Cysteine Proteinase Inhibitors - cerebrospinal fluid | Parkinson Disease, Secondary - pathology | Oxidopamine - pharmacology | Ubiquitination - drug effects | Acetylcysteine - analogs & derivatives | Dopamine - physiology | Sympatholytics - pharmacology | Behavior, Animal - drug effects | Neurons - metabolism | Parkinson Disease, Secondary - chemically induced | Neurons - drug effects | Sympatholytics - cerebrospinal fluid | Microscopy, Electron, Transmission | Dyskinesia, Drug-Induced - pathology | Blotting, Western | Brain Chemistry - physiology | Oxidopamine - cerebrospinal fluid | Swimming - physiology | Animals | Sympatholytics - administration & dosage | Cysteine Proteinase Inhibitors - pharmacology | Acetylcysteine - pharmacology | Parkinson Disease, Secondary - psychology | Inclusion Bodies - pathology | Proteasome Inhibitors | Tyrosine | Medical colleges | Evolutionary biology | Analysis | Neurochemistry | Fish | Neurotransmitters | Inhibitor drugs | Parkinsons disease | Index Medicus
Journal Article