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EMBO Molecular Medicine, ISSN 1757-4676, 03/2018, Volume 10, Issue 3, p. n/a
.... These effects coincided with decreased brown adipocyte lipid content and increased nutrient uptake by BAT, confirming increased BAT activity... 
GPR120 | brown adipose tissue | Ca2 | mitochondria | lipid metabolism | Ca | MEDICINE, RESEARCH & EXPERIMENTAL | CELLS | ACTIVATION | PROTEIN | INSULIN-SECRETION | THERMOGENESIS | POTENT | GLUCOSE-METABOLISM | GENE-EXPRESSION | ACID RECEPTOR GPR40 | ADIPOSE-TISSUE | Adipocytes, White - cytology | Adipocytes, Brown - metabolism | Receptors, G-Protein-Coupled - metabolism | Adipose Tissue, White - metabolism | Body Weight - drug effects | Lipids | Male | Receptors, G-Protein-Coupled - agonists | Adipocytes, White - drug effects | Biphenyl Compounds - pharmacology | Cell Respiration - drug effects | Adiposity - drug effects | Oxidation-Reduction | Adipocytes, Brown - drug effects | Uncoupling Protein 1 - metabolism | Mice, Inbred C57BL | Mitochondria - metabolism | Mitochondria - drug effects | Gene Expression Regulation - drug effects | Phenylpropionates - pharmacology | Animals | Receptors, G-Protein-Coupled - deficiency | Cell Differentiation - drug effects | Models, Biological | Oxygen Consumption - drug effects | Glucose - metabolism | Adipose Tissue, Brown - drug effects | Adipose Tissue, Brown - metabolism | Adipocytes, Brown - cytology | Adipocytes, White - metabolism | Adipose Tissue, White - drug effects | Physiological aspects | Obesity | G proteins | Body weight | Membrane proteins | Pharmacology & Drug Discovery | Metabolism
Journal Article
Autophagy, ISSN 1554-8627, 10/2012, Volume 8, Issue 10, pp. 1477 - 1493
...-dependent metabolism and oxygen consumption Tsung- c hin Lin, 1,7,† Yun-Ru c hen, 1,† e lizabeth Kensicki, 2 Angela Ying-Jian Li, 1 Mei Kong, 3,8 Yang Li, 5 Robert P... 
altered metabolism | autophagy | glutamine | ATP | transcriptional reprogramming | ATG5 | Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | Transcriptional reprogramming | Altered metabolism | Autophagy | Glutamine | Metabolomics | Glutamine - deficiency | Microtubule-Associated Proteins - genetics | Microtubule-Associated Proteins - metabolism | Glutamine - metabolism | RNA, Messenger - metabolism | Autophagy - drug effects | Oxidative Phosphorylation - drug effects | Adenosine Triphosphate - metabolism | Autophagy - genetics | Glutamine - pharmacology | Microtubule-Associated Proteins - deficiency | Fibroblasts - metabolism | Intracellular Space - drug effects | RNA, Messenger - genetics | Citric Acid Cycle - drug effects | Mitochondria - metabolism | Mitochondria - drug effects | Metabolome - genetics | Gene Expression Regulation - drug effects | Animals | Autophagy-Related Protein 5 | Metabolome - drug effects | Embryo, Mammalian - cytology | Oxygen Consumption - drug effects | Oxygen Consumption - genetics | Fibroblasts - drug effects | Intracellular Space - metabolism | Cell Proliferation - drug effects | Fibroblasts - cytology | Mice | PROTEIN | PROLIFERATION | CELL-GROWTH | HYPOXIA | CANCER | FIBROBLASTS | CELL BIOLOGY | DISSECTION | DEMETHYLATION | GENERATION | STARVATION | Basic Research Paper
Journal Article
Journal Article
Journal Article
The Journal of clinical investigation, ISSN 0021-9738, 2013, Volume 123, Issue 8, pp. 3272 - 3291
Journal Article
Journal Article
Free radical biology & medicine, ISSN 0891-5849, 2012, Volume 52, Issue 1, pp. 198 - 207
Statins, the widely prescribed cholesterol-lowering drugs for the treatment of cardiovascular disease, cause adverse skeletal muscle side effects ranging from fatigue to fatal rhabdomyolysis... 
Oxidative stress | Mitochondria | Hydrogen peroxide | Simvastatin | Mitochondrial respiration | Superoxide | Human skeletal muscle | Apoptosis | COA REDUCTASE INHIBITORS | COENZYME-A REDUCTASE | RAT | BIOCHEMISTRY & MOLECULAR BIOLOGY | MECHANISMS | INDUCE APOPTOSIS | MUSCLE-CELLS | STATIN-ASSOCIATED MYOPATHY | PERMEABILITY TRANSITION | LIPID-LOWERING DRUGS | MYOTOXICITY | ENDOCRINOLOGY & METABOLISM | Simvastatin - adverse effects | Gene Expression - drug effects | Oxidative Stress | Apoptosis - drug effects | Humans | Muscle Fibers, Skeletal - drug effects | Muscle Fibers, Skeletal - metabolism | Electron Transport Complex I - metabolism | Oxygen Consumption - physiology | Dose-Response Relationship, Drug | Myoblasts - drug effects | Proto-Oncogene Proteins c-bcl-2 - metabolism | Myoblasts - metabolism | Electron Transport Complex I - genetics | Cell Respiration - drug effects | Myoblasts - cytology | Superoxides - metabolism | Muscular Diseases - chemically induced | Respiratory System Agents - pharmacology | Muscular Diseases - metabolism | Mitochondria - metabolism | Muscular Diseases - pathology | Mitochondria - drug effects | Anticholesteremic Agents - adverse effects | Adenosine Diphosphate - pharmacology | Hydrogen Peroxide - metabolism | Signal Transduction - drug effects | Cell Differentiation - drug effects | Oxygen Consumption - drug effects | Muscle Fibers, Skeletal - cytology | Primary Cell Culture | Proto-Oncogene Proteins c-bcl-2 - genetics | Enzymes | Low density lipoproteins | Dimethyl sulfoxide | Dosage and administration | Mitochondrial DNA | Permeability | Glutamate | Cells | Muscles | Drugs | Cell culture | Myotubes | Bcl-2 protein | Fatigue | Oxygen consumption | Nuclei | Skeletal muscle | Myoblasts | Atrophy | Electron transport chain | statins | Cardiovascular diseases | Electron transport | mitochondria | hydrogen peroxide | simvastatin | apoptosis | superoxide | oxidative stress | mitochondrial respiration | human skeletal muscle
Journal Article
Circulation research, ISSN 1524-4571, 2007, Volume 100, Issue 3, pp. 328 - 341
... (eg, deprivation for glucose or oxygen) or that accelerate ATP consumption (eg, muscle contraction... 
Calcium signaling | Signaling pathways | Diabetes | Metabolism | Insulin | RAT-LIVER | CARDIAC & CARDIOVASCULAR SYSTEMS | insulin | 5-AMINOIMIDAZOLE-4-CARBOXAMIDE RIBONUCLEOSIDE | STIMULATED GLUCOSE-TRANSPORT | calcium signaling | ACETYL-COA CARBOXYLASE | COENZYME-A CARBOXYLASE | SKELETAL-MUSCLE | signaling pathways | FATTY-ACID OXIDATION | ENDOTHELIAL-CELLS | CELL-PERMEABLE ACTIVATOR | PANCREATIC BETA-CELLS | PERIPHERAL VASCULAR DISEASE | metabolism | HEMATOLOGY | diabetes | Consensus Sequence | Protein Subunits | Protein-Serine-Threonine Kinases - deficiency | Insulin - physiology | Obesity - drug therapy | Humans | Muscle Cells - drug effects | Adipocytes - drug effects | AMP-Activated Protein Kinases | Aminoimidazole Carboxamide - pharmacology | Multienzyme Complexes - deficiency | Calcium - physiology | Adenosine Triphosphate - metabolism | Energy Metabolism - physiology | Binding Sites | Peptide Hormones - physiology | Hypoglycemic Agents - therapeutic use | Amino Acid Sequence | Diabetes Mellitus - drug therapy | Models, Molecular | Rats | Hypoglycemic Agents - pharmacology | Mice, Knockout | Calcium-Calmodulin-Dependent Protein Kinase Kinase | Carbohydrate Metabolism - physiology | Aminoimidazole Carboxamide - analogs & derivatives | Diabetes Mellitus - therapy | Protein-Serine-Threonine Kinases - drug effects | Mice | Protein-Serine-Threonine Kinases - chemistry | Cell Cycle - drug effects | Energy Metabolism - drug effects | Lipid Metabolism - physiology | Multienzyme Complexes - drug effects | Carbohydrate Metabolism - drug effects | Phosphorylation | Metformin - therapeutic use | Molecular Sequence Data | Hepatocytes - metabolism | Ribonucleotides - pharmacology | Hepatocytes - drug effects | Adenosine Monophosphate - metabolism | Protein-Serine-Threonine Kinases - physiology | Metformin - pharmacology | Muscle Cells - metabolism | Protein-Serine-Threonine Kinases - genetics | Diabetes Mellitus - metabolism | Neoplasms - enzymology | Multienzyme Complexes - genetics | Enzyme Activation - drug effects | Protein Processing, Post-Translational - physiology | Multienzyme Complexes - chemistry | Obesity - metabolism | Sequence Homology, Amino Acid | Sequence Alignment | Animals | Oxygen Consumption - drug effects | Adipocytes - metabolism | Multienzyme Complexes - physiology | Lipid Metabolism - drug effects | Cell Cycle - physiology | Neoplasms - pathology
Journal Article
Hypertension, ISSN 0194-911X, 11/2013, Volume 62, Issue 5, pp. 914 - 919
Journal Article