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Molecular Nutrition & Food Research, ISSN 1613-4125, 03/2018, Volume 62, Issue 5, pp. 1700754 - n/a
Scope Vitamin A (VA) is an essential nutrient for the development of the brain. We previously found that children with autism spectrum disorder (ASD) have a... 
oxytocin | vitamin A | retinoic acid receptor beta | autism | RISK-FACTORS | FOOD SCIENCE & TECHNOLOGY | SPECTRUM DISORDERS | CELL-LINES | SOCIAL-BEHAVIOR | NUTRITIONAL-STATUS | OXYTOCIN SECRETION | RETINOIC ACID RECEPTOR | CD38 | CENTRAL-NERVOUS-SYSTEM | EXPRESSION | Membrane Glycoproteins - analysis | ADP-ribosyl Cyclase 1 - analysis | ADP-ribosyl Cyclase 1 - genetics | Oxytocin - blood | Hypothalamus - physiology | Vitamin A Deficiency - complications | ADP-ribosyl Cyclase 1 - physiology | Oxytocin - physiology | Receptors, Retinoic Acid - physiology | Rats | ADP-ribosyl Cyclase - physiology | Rats, Sprague-Dawley | Autistic Disorder - etiology | Membrane Glycoproteins - genetics | Depression - etiology | Membrane Glycoproteins - physiology | Animals | ADP-ribosyl Cyclase - analysis | Receptors, Retinoic Acid - analysis | Interpersonal Relations | Anxiety - etiology | Vitamin A - blood | ADP-ribosyl Cyclase - genetics | Brain | Chromatin | Animal models | Immunoprecipitation | Viruses | Hypothalamus | Risk factors | Western blotting | Vitamin A | Signal transduction | Rodents | CD38 antigen | Children | Supplementation | Nutritional status | Recombinant | RNA viruses | Nutrient deficiency | Calcium (intracellular) | Oxytocin | Neurons | RNA-mediated interference | Dietary supplements | Excitability | siRNA | Risk taking | Ribonucleic acid--RNA | Pregnancy | Autism | Progeny | Signaling | Offspring | Acids | Adenoviruses | Regulatory mechanisms (biology) | Retinoic acid | Calcium ions
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 03/2010, Volume 30, Issue 10, pp. 3803 - 3812
Single-minded 1 (SIM1) mutations are one of the few known causes of nonsyndromic monogenic obesity in both humans and mice. Although the role of Sim1 in the... 
ENERGY-EXPENDITURE | RAT | GENE | FOOD-INTAKE | MOUSE | NEURON ORIGIN | MICE | DIET-INDUCED OBESITY | INSULIN SENSITIVITY | NEUROSCIENCES | HYPOTHALAMIC PARAVENTRICULAR NUCLEUS | Male | Receptor, Melanocortin, Type 4 - biosynthesis | Obesity - genetics | Paraventricular Hypothalamic Nucleus - physiopathology | RNA, Messenger - biosynthesis | Repressor Proteins - deficiency | Gene Expression Regulation, Developmental | RNA, Messenger - antagonists & inhibitors | Female | Basic Helix-Loop-Helix Transcription Factors - deficiency | Animals, Newborn | Oxytocin - antagonists & inhibitors | Receptor, Melanocortin, Type 4 - antagonists & inhibitors | Reproducibility of Results | Basic Helix-Loop-Helix Transcription Factors - genetics | Mice, Inbred C57BL | Gene Silencing | Repressor Proteins - genetics | Mice, Transgenic | Obesity - physiopathology | Hypothalamus - pathology | Receptor, Melanocortin, Type 4 - genetics | Signal Transduction - genetics | Oxytocin - biosynthesis | Hyperphagia - metabolism | Eating - genetics | Hypothalamus - physiopathology | Mice, Knockout | Obesity - metabolism | Obesity - pathology | Hyperphagia - genetics | Hyperphagia - pathology | Animals | Hypothalamus - metabolism | Hyperphagia - physiopathology | Paraventricular Hypothalamic Nucleus - metabolism | Oxytocin - genetics | Mice | Paraventricular Hypothalamic Nucleus - pathology
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Journal Article
Journal of Endocrinology, ISSN 0022-0795, 2016, Volume 230, Issue 1, pp. 13 - 26
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