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Cell cycle (Georgetown, Tex.), ISSN 1551-4005, 10/2014, Volume 8, Issue 24, pp. 4112 - 4118
Cellular senescence is currently viewed as a response to DNA damage. In this report, we showed that non-damaging agents such as sodium butyrate-induced p21 and... 
Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | DDR | DNA damage | γH2AX | Cell cycle | Cellular senescence | Aging | p21 | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Cyclin-Dependent Kinase Inhibitor p21 - drug effects | Humans | Intracellular Signaling Peptides and Proteins - metabolism | Neoplasm Proteins - metabolism | Cyclin-Dependent Kinase Inhibitor p16 | Neoplasm Proteins - drug effects | Cell Nucleus - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | DNA Damage - genetics | Neoplasm Proteins - genetics | Protein-Serine-Threonine Kinases - metabolism | Biomarkers - metabolism | Cellular Senescence - genetics | Histones - ultrastructure | Stress, Physiological - genetics | Intracellular Signaling Peptides and Proteins - antagonists & inhibitors | Biomarkers - analysis | Cells, Cultured | Rats | Signal Transduction - genetics | DNA - metabolism | Cell Nucleus - ultrastructure | Sirolimus - pharmacology | DNA - genetics | Animals | Histones - genetics | Cell Nucleus - genetics | Cell Line, Tumor | Mice | TOR Serine-Threonine Kinases | Histones - metabolism | Index Medicus | cellular senescence | cell cycle | aging
Journal Article
Tumor biology, ISSN 1010-4283, 6/2016, Volume 37, Issue 6, pp. 7431 - 7440
Journal Article
Nature (London), ISSN 1476-4687, 03/2010, Volume 464, Issue 7287, pp. 374 - 379
Cellular senescence has been recently shown to have an important role in opposing tumour initiation and promotion. Senescence induced by oncogenes or by loss... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Proto-Oncogene Proteins p21(ras) - genetics | Cellular Senescence - drug effects | Male | Prostate - metabolism | Cyclin-Dependent Kinase Inhibitor p27 - metabolism | S-Phase Kinase-Associated Proteins - antagonists & inhibitors | Adenovirus E1A Proteins - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Fibroblasts | Prostatic Neoplasms - prevention & control | Prostatic Neoplasms - drug therapy | Proto-Oncogene Proteins p21(ras) - metabolism | PTEN Phosphohydrolase - genetics | Prostatic Neoplasms - pathology | Cyclin-Dependent Kinase Inhibitor p16 - deficiency | PTEN Phosphohydrolase - deficiency | Cells, Cultured | Tumor Suppressor Protein p53 - metabolism | Cyclin-Dependent Kinase Inhibitor p16 - genetics | PTEN Phosphohydrolase - metabolism | SKP Cullin F-Box Protein Ligases - metabolism | S-Phase Kinase-Associated Proteins - metabolism | Tumor Suppressor Protein p53 - deficiency | Animals | Activating Transcription Factor 4 - metabolism | Adenovirus E1A Proteins - genetics | Prostate - cytology | Cyclin-Dependent Kinase Inhibitor p16 - metabolism | S-Phase Kinase-Associated Proteins - genetics | Mice | Cell Transformation, Neoplastic - drug effects | Prevention | Care and treatment | Physiological aspects | Aging | Tumor suppressor genes | Genetic aspects | Research | Cells | Tumors | Cell cycle | Apoptosis | Index Medicus
Journal Article
Cell cycle (Georgetown, Tex.), ISSN 1551-4005, 06/2006, Volume 5, Issue 12, pp. 1313 - 1319
Journal Article
Archives of toxicology, ISSN 1432-0738, 12/2014, Volume 89, Issue 2, pp. 155 - 178
The cell cycle inhibitor p21CDKN1A is a protein playing multiple roles not only in the DNA damage response, but also in many cellular processes during... 
Cell cycle inhibition | Biomedicine | Environmental Health | Occupational Medicine/Industrial Medicine | DNA damage response | Chemical toxicity | Pharmacology/Toxicology | p21 CDKN1A/WAF1/CIP1 | Genotoxic compounds | DNA repair | Biomedicine general | p21
Journal Article