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Molecular and Cellular Biology, ISSN 0270-7306, 05/2005, Volume 25, Issue 9, pp. 3553 - 3562
Article Usage Stats Services MCB Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley... 
EARLY EMBRYONIC LETHALITY | DAMAGE CHECKPOINT | CELL-DIVISION | XENOPUS EGG EXTRACTS | ACTIVATION | COMPLEX | CYCLIN-E | BIOCHEMISTRY & MOLECULAR BIOLOGY | IONIZING-RADIATION | S-PHASE CHECKPOINT | CDC25A | CELL BIOLOGY | RNA, Small Interfering - genetics | Phosphorylation | Protein Kinases - genetics | DNA Replication - drug effects | Humans | Staurosporine - analogs & derivatives | DNA-Binding Proteins - metabolism | DNA Damage - physiology | Caffeine - pharmacology | Cell Cycle Proteins - genetics | Roscovitine | Replication Protein A | Protein Kinases - pharmacology | Chromosomal Proteins, Non-Histone - metabolism | Protein-Serine-Threonine Kinases - physiology | Purines - pharmacology | DNA, Single-Stranded - metabolism | RNA, Small Interfering - pharmacology | Cell Cycle Proteins - metabolism | Tumor Suppressor Protein p53 - metabolism | Ataxia Telangiectasia Mutated Proteins | DNA Replication - physiology | Protein Kinases - physiology | Protein-Serine-Threonine Kinases - drug effects | Checkpoint Kinase 2 | Checkpoint Kinase 1 | Protein Kinase Inhibitors - pharmacology | Histones - metabolism | Cell Cycle Proteins - physiology | Staurosporine - pharmacology | Index Medicus | Gene Expression | RNA; Small Interfering/genetics/pharmacology | Caffeine/pharmacology | Protein Kinases/genetics/pharmacology/physiology | Protein-Serine-Threonine Kinases/drug effects/physiology | Purines/pharmacology | Tumor Suppressor Protein p53/metabolism | DNA Replication/drug effects/physiology | DNA-Binding Proteins/metabolism | Histones/metabolism | Cell Cycle Proteins/genetics/metabolism/physiology | DNA; Single-Stranded/metabolism | Chromosomal Proteins; Non-Histone/metabolism | Protein Kinase Inhibitors/pharmacology | Staurosporine/analogs & derivatives/pharmacology | DNA Damage/physiology
Journal Article
Nature, ISSN 0028-0836, 01/2016, Volume 529, Issue 7587, pp. 541 - 545
Journal Article
Structure, ISSN 0969-2126, 04/2012, Volume 20, Issue 4, pp. 604 - 617
The viral oncoprotein E6 is an essential factor for cervical cancers induced by “high-risk” mucosal HPV. Among other oncogenic activities, E6 recruits the... 
BIOPHYSICAL CHARACTERIZATION | LOCALIZATION | CELLS | HUMAN-PAPILLOMAVIRUS TYPE-16 | TERMINAL DOMAIN | UBIQUITIN-PROTEIN LIGASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | CERVICAL-CANCER | NMR STRUCTURE DETERMINATION | CELL BIOLOGY | BIOPHYSICS | TUMOR-SUPPRESSOR | BINDING | Oncogene Proteins, Viral - chemistry | Escherichia coli | Humans | Protein Multimerization | Ubiquitin - metabolism | Molecular Sequence Data | Tumor Suppressor Protein p53 - genetics | Ubiquitination | Nuclear Magnetic Resonance, Biomolecular | Oncogene Proteins, Viral - genetics | Binding Sites | Repressor Proteins - metabolism | Oncogene Proteins, Viral - metabolism | Protein Structure, Tertiary | Recombinant Proteins - metabolism | Amino Acid Sequence | Repressor Proteins - chemistry | Tumor Suppressor Protein p53 - metabolism | Ubiquitin-Protein Ligases - metabolism | Models, Molecular | Recombinant Proteins - chemistry | Repressor Proteins - genetics | Recombinant Proteins - genetics | Ubiquitin-Protein Ligases - chemistry | Protein Binding | Tumor Suppressor Protein p53 - chemistry | Mutation | Proteasome Endopeptidase Complex - metabolism | Ubiquitin-Protein Ligases - genetics | Ubiquitin | Tumor proteins | Ligases | Papillomavirus infections | Cervical cancer | Index Medicus | p53 degradation | self-association | E6 | cervical cancer | oncoprotein | HPV
Journal Article
Nature Communications, ISSN 2041-1723, 12/2019, Volume 10, Issue 1, pp. 353 - 14
Mucosal melanoma is a rare and poorly characterized subtype of human melanoma. Here we perform a cross-species analysis by sequencing tumor-germline pairs from... 
POINT MUTATIONS | CANCER PATIENTS | BREAST-CANCER | MUTATIONAL PROCESSES | ARRAY-CGH | MULTIDISCIPLINARY SCIENCES | COPY NUMBER | TUMOR | OVARIAN-CANCER | MOUSE MODELS | REVEALS | Proto-Oncogene Proteins c-mdm2 - genetics | Cadherins - metabolism | Humans | Carcinogenesis - metabolism | DNA Copy Number Variations | Germ-Line Mutation | Cadherins - genetics | Neoplasm Proteins - genetics | Mouth Neoplasms - genetics | Protein-Serine-Threonine Kinases - metabolism | PTEN Phosphohydrolase - genetics | Tumor Suppressor Proteins - metabolism | Membrane Proteins - genetics | Carcinogenesis - genetics | Cell Cycle Proteins - metabolism | Melanoma - pathology | Mucous Membrane - pathology | GTP Phosphohydrolases - metabolism | GTP Phosphohydrolases - genetics | Microtubule-Associated Proteins - genetics | Species Specificity | Microtubule-Associated Proteins - metabolism | Gene Expression Regulation, Neoplastic | Mouth Neoplasms - metabolism | Receptor-Like Protein Tyrosine Phosphatases, Class 3 - genetics | Neoplasm Proteins - metabolism | Tumor Suppressor Protein p53 - genetics | Melanoma - genetics | BRCA1 Protein - metabolism | Tumor Suppressor Proteins - genetics | Cell Cycle Proteins - genetics | Membrane Proteins - metabolism | Proto-Oncogene Proteins c-mdm2 - metabolism | Melanoma - metabolism | Skin Neoplasms - pathology | Neoplasm Recurrence, Local | Protein-Serine-Threonine Kinases - genetics | Tumor Suppressor Protein p53 - metabolism | PTEN Phosphohydrolase - metabolism | Mucous Membrane - metabolism | Skin Neoplasms - metabolism | Carcinogenesis - pathology | BRCA1 Protein - genetics | Animals | BRCA2 Protein - metabolism | Receptor-Like Protein Tyrosine Phosphatases, Class 3 - metabolism | Skin Neoplasms - genetics | Dogs | Horses | Mouth Neoplasms - pathology | BRCA2 Protein - genetics | BRCA2 protein | MDM2 protein | BRCA1 protein | Copy number | p53 Protein | Mucosa | Melanoma | Data processing | Breast cancer | Metastases | Heterogeneity | Human performance | Tumorigenesis | Mutation | Species | PTEN protein | Tumors | Index Medicus
Journal Article
Journal Article
Oncogene, ISSN 0950-9232, 09/2011, Volume 30, Issue 37, pp. 3918 - 3929
The BH3 mimetic ABT737 induces autophagy by competitively disrupting the inhibitory interaction between the BH3 domain of Beclin 1 and the anti-apoptotic... 
autophagy | ABT737 | Bcl-2 family protein | Beclin 1 | CELLS | COMPLEX | BIOCHEMISTRY & MOLECULAR BIOLOGY | IKK | INDUCTION | CELL BIOLOGY | ONCOLOGY | GENETICS & HEREDITY | BCL-2 FAMILY | BH3-ONLY PROTEINS | Phosphorylation | Nitriles - pharmacology | Ribosomal Protein S6 Kinases - metabolism | TOR Serine-Threonine Kinases - metabolism | Humans | Autophagy - drug effects | Biphenyl Compounds - pharmacology | I-kappa B Kinase - metabolism | Nitrophenols - pharmacology | Phosphotransferases (Phosphate Group Acceptor) - metabolism | Benzopyrans - pharmacology | Oncogene Protein v-akt - metabolism | Proto-Oncogene Proteins c-mdm2 - metabolism | Beclin-1 | Tumor Suppressor Protein p53 - metabolism | Glycogen Synthase Kinase 3 - metabolism | Sulfonamides - pharmacology | Membrane Proteins - agonists | Piperazines - pharmacology | Signal Transduction - drug effects | Apoptosis Regulatory Proteins - agonists | Cell Line, Tumor | Proto-Oncogene Proteins c-bcl-2 - antagonists & inhibitors | Sirtuins - metabolism | Acetyl-CoA Carboxylase - metabolism | Autophagy (Cytology) | Cellular proteins | Care and treatment | Physiological aspects | Genetic aspects | Cellular signal transduction | Research | Cancer | Proteins | Signal transduction | Biomimetics | Cellular biology | Gene expression | Sirtuins | TOR protein | MDM2 protein | Bcl-2 protein | Glycogen synthase kinase 3 | AKT protein | Dephosphorylation | IKK protein | p53 protein | Allosteric properties | NF- Kappa B protein | Bcl-x protein | I Kappa B kinase | Lipid kinase | Phagocytosis | Ubiquitin-protein ligase | Index Medicus
Journal Article
Biochemistry, ISSN 0006-2960, 05/2017, Volume 56, Issue 18, pp. 2379 - 2384
Appropriate integration of cellular signals requires a delicate balance of ligand target binding affinities. Increasing the level of residual structure in... 
UNSTRUCTURED PROTEINS | IMPROVED SENSITIVITY | SPECTROSCOPY | BINDING | BIOCHEMISTRY & MOLECULAR BIOLOGY | KIX DOMAIN | Myeloid-Lymphoid Leukemia Protein - metabolism | Proline - metabolism | Humans | Myeloid-Lymphoid Leukemia Protein - chemistry | Phosphoproteins - metabolism | Phosphoproteins - chemistry | Tumor Suppressor Protein p53 - genetics | Cloning, Molecular | Escherichia coli - metabolism | Conserved Sequence | Membrane Proteins - metabolism | Protein Interaction Domains and Motifs | Binding Sites | Recombinant Proteins - metabolism | Amino Acid Sequence | Protein Conformation, alpha-Helical | Gene Expression | Histone-Lysine N-Methyltransferase - genetics | Membrane Proteins - genetics | Tumor Suppressor Protein p53 - metabolism | Models, Molecular | Recombinant Proteins - chemistry | Recombinant Proteins - genetics | Phosphoproteins - genetics | Intrinsically Disordered Proteins - genetics | Protein Folding | Proline - chemistry | Histone-Lysine N-Methyltransferase - chemistry | Sequence Homology, Amino Acid | Sequence Alignment | Animals | Histone-Lysine N-Methyltransferase - metabolism | Membrane Proteins - chemistry | Escherichia coli - genetics | Intrinsically Disordered Proteins - chemistry | Myeloid-Lymphoid Leukemia Protein - genetics | Protein Binding | Mice | Tumor Suppressor Protein p53 - chemistry | Mutation | Intrinsically Disordered Proteins - metabolism | Helix-loop-helix motif | Protein deficiency | Research | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 03/2011, Volume 471, Issue 7337, pp. 189 - 195
B-cell non-Hodgkin's lymphoma comprises biologically and clinically distinct diseases the pathogenesis of which is associated with genetic lesions affecting... 
CBP | RUBINSTEIN-TAYBI SYNDROME | HISTONE ACETYLTRANSFERASES | ACETYLATION | P300 | MULTIDISCIPLINARY SCIENCES | CREB-BINDING-PROTEIN | CYCLE ARREST | ACUTE MYELOID-LEUKEMIA | TRANSCRIPTIONAL COACTIVATOR | P53 | Histone Acetyltransferases - deficiency | Recurrence | Acetyltransferases - metabolism | Histone Acetyltransferases - chemistry | Humans | E1A-Associated p300 Protein - genetics | Gene Expression Regulation, Neoplastic | Histone Acetyltransferases - genetics | CREB-Binding Protein - chemistry | E1A-Associated p300 Protein - metabolism | Lymphoma, B-Cell - genetics | Acetyltransferases - genetics | Mutation, Missense - genetics | CREB-Binding Protein - genetics | DNA-Binding Proteins - metabolism | CREB-Binding Protein - metabolism | Lymphoma, Follicular - genetics | Acetyltransferases - deficiency | Base Sequence | Histone Acetyltransferases - metabolism | Lymphoma, B-Cell - enzymology | HEK293 Cells | Acetylation | CREB-Binding Protein - deficiency | Lymphoma, Large B-Cell, Diffuse - pathology | Lymphoma, Follicular - pathology | Cells, Cultured | E1A-Associated p300 Protein - chemistry | Lymphoma, Large B-Cell, Diffuse - enzymology | Tumor Suppressor Protein p53 - metabolism | Protein Structure, Tertiary - genetics | Mutation - genetics | Acetyl Coenzyme A - metabolism | Acetyltransferases - chemistry | Animals | Lymphoma, B-Cell - pathology | Polymorphism, Single Nucleotide - genetics | Protein Binding | Lymphoma, Follicular - enzymology | Mice | E1A-Associated p300 Protein - deficiency | Lymphoma, Large B-Cell, Diffuse - genetics | Proto-Oncogene Proteins c-bcl-6 | Sequence Deletion - genetics | Lymphomas | Genetic aspects | B cells | Gene mutations | Health aspects | Abnormalities | Proteins | Congenital diseases | Pathogenesis | Genes | Cell cycle | Genomes | Genetic engineering | Mutation | Binding sites | Index Medicus
Journal Article