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Molecular and Cellular Neuroscience, ISSN 1044-7431, 10/2016, Volume 76, pp. 52 - 58
The transcription factor Nrf2 and its repressor protein Keap1 play key roles in the regulation of antioxidant stress responses and both Keap1-Nrf2 signalling... 
ALS-FTLD | Oxidative stress | Keap1-Nrf2 | SQSTM1/p62 | ACTIVATE NRF2 | PHOSPHORYLATION | AUTOPHAGIC DEGRADATION | P62 | NEUROSCIENCES | UBIQUITINATION | TRANSCRIPTION FACTOR NRF2 | PATHWAY | ANTIOXIDANT RESPONSE | PAGETS-DISEASE | BONE | Response Elements | Signal Transduction | Amyotrophic Lateral Sclerosis - genetics | Humans | Kelch-Like ECH-Associated Protein 1 - metabolism | Mutation, Missense | Sequestosome-1 Protein - genetics | NF-E2-Related Factor 2 - metabolism | HEK293 Cells | Protein Binding | Frontotemporal Lobar Degeneration - genetics | Sequestosome-1 Protein - chemistry | Binding Sites | Sequestosome-1 Protein - metabolism | Antioxidants | Nervous system diseases | Genetic aspects | Analysis | Protein binding | ARE, Antioxidant response element | NFE2L2, Nuclear factor erythroid 2-like 2 | p62, Sequestosome 1 | IKKβ, Inhibitor of nuclear factor kappa-B kinase subunit beta | RBM45, RNA binding motif protein 45 | KIR, Keap1-interacting region | SALS, Sporadic ALS | UBA, Ubiquitin-associated (domain) | Keap1, Kelch-like ECH-associated protein 1 | Nrf2, Nuclear erythroid 2-related factor 2 | p62 protein | ALS, Amyotrophic lateral sclerosis | p62 | mTORC1, Mammalian target of rapamycin complex 1 | LIR, LC3-interacting region | NQO1, NAD(P)H dehydrogenase, Quinone 1 | FTLD, Frontotemporal lobar degeneration | SOD1, Superoxide dismutase 1 | SQSTM1 | PDB, Paget's disease of bone | TDP-43, TAR DNA-binding protein 43
Journal Article
Cell, ISSN 0092-8674, 06/2009, Volume 137, Issue 6, pp. 1001 - 1004
Journal Article
Journal of Cancer, ISSN 1837-9664, 2018, Volume 9, Issue 21, pp. 4072 - 4086
Aims: SQSTMI/p62, as an autophagy marker, is a key molecule involved in the autophagy process. Recent studies have demonstrated that p62 has a close... 
SQSTM1/p62 | Prognosis | Malignant tumor | BECLIN 1 | P62/SQSTM1 | MOLECULAR SUBTYPE | SQSTMl/p62 | HIGH EXPRESSION | P62 EXPRESSION | prognosis | CANCER | AUTOPHAGY-RELATED PROTEINS | IMPACT | ONCOLOGY | DISEASE | malignant tumor | CARCINOMA
Journal Article
Journal Article
Journal of cell science, ISSN 0021-9533, 11/2018, Volume 131, Issue 21, p. jcs222836
SQSTM1 (also known as p62) is a multifunctional stress-inducible scaffold protein involved in diverse cellular processes. Its functions are tightly regulated... 
Sqstm1 | Selective autophagy | Keap1–Nrf2 system | p62 | Proteinopathies | Phase separation | SUBCELLULAR-LOCALIZATION | SQSTM1/P62 CONTRIBUTES | CISPLATIN RESISTANCE | Keap1-Nrf2 system | PAGET-DISEASE | SELECTIVE AUTOPHAGY SUBSTRATE | CELL BIOLOGY | UBA DOMAIN | HEPATOCELLULAR-CARCINOMA | TRANSCRIPTION FACTOR NRF2 | INTERACTING PROTEIN P62 | ADIPOSE-TISSUE
Journal Article
Cell Death and Differentiation, ISSN 1350-9047, 01/2013, Volume 20, Issue 1, pp. 21 - 30
Ubiquitination has long been recognised as a key determinator of protein fate by tagging proteins for proteasomal degradation. Most recently, the ability of... 
mitophagy | autophagy | Optineurin | phosphorylation | ubiquitin | BIOCHEMISTRY & MOLECULAR BIOLOGY | MITOCHONDRIA | P62/SQSTM1 | P62 | CONJUGATION SYSTEMS | CELL-DEATH | CELL BIOLOGY | PARKIN | PROTEIN-DEGRADATION | PROTEASOME | PAGETS-DISEASE | BINDING | Autophagy - physiology | Ubiquitination - physiology | Signal Transduction | Humans | Ubiquitin - metabolism | Review
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 8/2012, Volume 109, Issue 34, pp. 13561 - 13566
Journal Article
Molecular Cell, ISSN 1097-2765, 01/2014, Volume 53, Issue 2, pp. 167 - 178
Selective autophagy ensures recognition and removal of various cytosolic cargos. Hence, aggregated proteins, damaged organelles, or pathogens are enclosed into... 
TRANSCRIPTION FACTOR NRF2 | BIOGENESIS | MECHANISM | STRUCTURAL BASIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | P62/SQSTM1 | DEGRADATION | ATG8-PE DECONJUGATION | TAGS PEROXISOMES | P62 | LC3 | CELL BIOLOGY | Ubiquitination | Signal Transduction | Models, Biological | Models, Molecular | Autophagy - physiology | Ubiquitins - physiology | Ubiquitins - metabolism | Peroxisomes - metabolism | Ubiquitin | Analysis
Journal Article