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Nature (London), ISSN 1476-4687, 2019, Volume 567, Issue 7746, pp. 43 - 48
Cell-identity switches, in which terminally differentiated cells are converted into different cell types when stressed, represent a widespread regenerative... 
SURVIVAL | THERAPY | ISLETS | DECOY SEARCH STRATEGY | CONVERSION | MULTIDISCIPLINARY SCIENCES | TISSUE | PANCREATIC BETA-CELLS | PROTEIN-PHOSPHORYLATION | EXPRESSION | EXOCRINE | Diabetes Mellitus - pathology | Organ Specificity - drug effects | Islets of Langerhans - drug effects | Homeodomain Proteins - metabolism | Humans | Transcriptome | Male | Cell Lineage - drug effects | Pancreatic Polypeptide-Secreting Cells - metabolism | Pancreatic Polypeptide-Secreting Cells - cytology | Maf Transcription Factors, Large - metabolism | Glucagon-Secreting Cells - drug effects | Maf Transcription Factors, Large - genetics | Islets of Langerhans - metabolism | Glucagon-Secreting Cells - metabolism | Trans-Activators - genetics | Female | Disease Models, Animal | Glucagon-Secreting Cells - cytology | Pancreatic Polypeptide - metabolism | Transduction, Genetic | Islets of Langerhans - pathology | Biomarkers - analysis | Diabetes Mellitus - metabolism | Islets of Langerhans - immunology | Glucose - pharmacology | Pancreatic Polypeptide-Secreting Cells - drug effects | Homeodomain Proteins - genetics | Cellular Reprogramming - drug effects | Insulin - metabolism | Animals | Sequence Analysis, RNA | Diabetes Mellitus - immunology | Diabetes Mellitus - therapy | Glucagon-Secreting Cells - transplantation | Proteomics | Glucose - metabolism | Glucagon - metabolism | Trans-Activators - metabolism | Mice
Journal Article
Diabetes, obesity & metabolism, ISSN 1462-8902, 2017, Volume 19, Issue Suppl 1, pp. 124 - 136
The progressive loss of pancreatic β‐cell mass that occurs in both type 1 and type 2 diabetes is a primary factor driving efforts to identify strategies for... 
beta‐cell | islets | beta-cell | Glucagon-Secreting Cells - immunology | Cell Proliferation | Species Specificity | Humans | Somatostatin-Secreting Cells - cytology | Extracellular Matrix - immunology | Diabetes Mellitus, Type 1 - metabolism | Extracellular Matrix - metabolism | Islets of Langerhans - innervation | Diabetes Mellitus, Type 2 - metabolism | Pancreatic Polypeptide-Secreting Cells - metabolism | Pancreatic Polypeptide-Secreting Cells - cytology | Insulin-Secreting Cells - metabolism | Glucagon-Secreting Cells - metabolism | Islets of Langerhans - cytology | Pancreatic Polypeptide-Secreting Cells - immunology | Somatostatin-Secreting Cells - immunology | Insulin-Secreting Cells - cytology | Pancreatic Polypeptide-Secreting Cells - pathology | Macrophages - immunology | Glucagon-Secreting Cells - cytology | Islets of Langerhans - blood supply | Somatostatin-Secreting Cells - pathology | Islets of Langerhans - pathology | Macrophages - pathology | Glucagon-Secreting Cells - pathology | Diabetes Mellitus, Type 1 - pathology | Cell Communication | Cellular Microenvironment | Macrophages - cytology | Insulin-Secreting Cells - immunology | Macrophages - metabolism | Animals | Models, Biological | Diabetes Mellitus, Type 2 - pathology | Insulin-Secreting Cells - pathology | Extracellular Matrix - pathology | Somatostatin-Secreting Cells - metabolism | Apoptosis | beta cell
Journal Article
Journal Article
Molecular and Cellular Biology, ISSN 0270-7306, 10/2008, Volume 28, Issue 20, pp. 6373 - 6383
Journal Article
Journal Article
Journal Article