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Experimental Neurology, ISSN 0014-4886, 04/2018, Volume 302, pp. 112 - 128
Huntington's disease (HD) is a monogenic inherited polyglutamine-mediated neurodegenerative disorder for which effective therapies are currently unavailable.... 
Neuroprotection | Huntington's disease | Immunomodulation | R6/2 mice | PC12/HttQ103-EGFP cells, NPY-Y2 receptor | PC12/Htt | cells, NPY-Y2 receptor | ALZHEIMER-DISEASE | PC12/Htt(Q103-EGFP) cells, NPY-Y2 receptor | NEURONS IN-VITRO | NEUROTROPHIC FACTOR | SUBVENTRICULAR ZONE | KAPPA-B | NEUROSCIENCES | PANCREATIC-POLYPEPTIDE | NEUROPEPTIDE-Y NPY | STRIATAL NEURONS | GROWTH-FACTOR-I | TRANSGENIC MICE | Muscle Strength - genetics | Motor Activity - drug effects | Receptors, Neuropeptide Y - metabolism | Trinucleotide Repeats - genetics | Brain - metabolism | MAP Kinase Signaling System - genetics | Encephalitis - drug therapy | Huntington Disease - mortality | Huntington Disease - drug therapy | Disease Models, Animal | Fluoresceins - pharmacokinetics | Encephalitis - genetics | Gene Expression Regulation - genetics | Huntingtin Protein - metabolism | Neuropeptide Y - therapeutic use | Enzyme Inhibitors - pharmacology | Rats | Mice, Transgenic | Dopamine and cAMP-Regulated Phosphoprotein 32 - metabolism | Psychomotor Disorders - drug therapy | Psychomotor Disorders - etiology | Gene Expression Regulation - drug effects | Huntington Disease - complications | Motor Activity - genetics | PC12 Cells - drug effects | Animals | MAP Kinase Signaling System - drug effects | Receptors, Neuropeptide Y - genetics | PC12 Cells - metabolism | Brain - pathology | Huntington Disease - genetics | Mice | Muscle Strength - drug effects | Peptide Fragments - therapeutic use | Huntingtin Protein - genetics | Encephalitis - etiology | Neuropeptide Y | Medical research | Nervous system diseases | Analysis | Medicine, Experimental | Nerve growth factor | Models | Index Medicus
Journal Article
Journal Article
11/2010
Mitochondrial function is dependent upon mitochondrial protein import (MPI), a complex process that transports nuclear-encoded proteins into mitochondria.... 
Mitochondrial protein import | 0719 | Reseveratrol | PC12 cells
Dissertation
Environmental Health Perspectives, ISSN 0091-6765, 4/2010, Volume 118, Issue 4, pp. 519 - 525
Background: Brominated flame retardants, including the widely used polybrominated diphenyl ethers (PBDEs), have been detected in humans, raising concern about... 
Mitochondria | Cellular metabolism | Halogenated diphenyl ethers | Calcium | Neurons | Atoms | Flame retardants | Homeostasis | Research | Ethers | PC12 cells | Persistent organic pollutant | PC12 | Neurotoxicity | Intracellular calcium stores | Structure-activity relationship | Fura-2 | Polybrominated diphenyl ether | Brominated flame retardant | Calcium fluctuations | calcium | NEONATAL EXPOSURE | intracellular calcium stores | brominated flame retardant | DNA-DAMAGE | ADULT MICE | POLYBROMINATED DIPHENYL ETHERS | SPONTANEOUS BEHAVIOR | ENVIRONMENTAL SCIENCES | IN-VITRO | PUBLIC, ENVIRONMENTAL & OCCUPATIONAL HEALTH | neurotoxicity | calcium fluctuations | PBDE | persistent organic pollutant | FLAME RETARDANTS | RAT-BRAIN | structure-activity relationship | DEVELOPMENTAL EXPOSURE | polybrominated diphenyl ether | Cell Survival - drug effects | PC12 Cells - drug effects | Animals | Polybrominated Biphenyls - chemistry | Calcium - metabolism | PC12 Cells - metabolism | Halogenation | Polybrominated Biphenyls - pharmacology | Rats | Structure-Activity Relationship | Halogenated Diphenyl Ethers | Metabolites | Fireproofing agents | Physiological aspects | Health aspects | Calcium ions | Cells | structure–activity relationship | Materials Chemistry | materialkemi | Chemical Sciences | Chemistry | Naturvetenskap | Kemi | Natural Sciences
Journal Article
Environmental Health Perspectives, ISSN 0091-6765, 5/2008, Volume 116, Issue 5, pp. 637 - 643
Background: Oxidative metabolism, resulting in the formation of hydroxylated polybrominated diphenyl ether (PBDE) metabolites, may enhance the neurotoxic... 
Chemical hazards | Halogenated diphenyl ethers | Metabolites | Neurons | Flame retardants | Homeostasis | Polychlorinated biphenyls | Catecholamines | Research | Exocytosis | PC12 cells | Neurotoxicity | Neurotransmitter release | Calcium | Intracellular calcium stores | Bioactivation | Brominated flame retardants | Polybrominated diphenyl ether | Catecholamine | Persistent organic pollutants | PHEOCHROMOCYTOMA CELLS | METABOLITES | calcium | intracellular calcium stores | bioactivation | neurotransmitter release | POLYBROMINATED DIPHENYL ETHERS | INTRACELLULAR CA2 | ENVIRONMENTAL SCIENCES | IN-VITRO | PUBLIC, ENVIRONMENTAL & OCCUPATIONAL HEALTH | catecholamine | neurotoxicity | brominated flame retardants | RYANODINE RECEPTORS | persistent organic pollutants | exocytosis | CHANNELS | RAT-BRAIN | polybrominated diphenyl ether | POLYCHLORINATED-BIPHENYLS | Hydroxylation | Calcium - metabolism | Catecholamines - secretion | Rats | Calcium - analysis | Environmental Exposure | Exocytosis - drug effects | PC12 Cells - drug effects | Flame Retardants - toxicity | Animals | PC12 Cells - metabolism | Polybrominated Biphenyls - metabolism | Polybrominated Biphenyls - toxicity | Homeostasis - drug effects | Halogenated Diphenyl Ethers | Health aspects | Index Medicus | poly-brominated diphenyl ether | intra-cellular calcium stores | Naturvetenskap | Natural Sciences | Kemi | Annan kemi | Chemical Sciences | Other Chemistry Topics
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2013, Volume 8, Issue 3, p. e57037
A wide range of growth factors encode information into specific temporal patterns of MAP kinase (MAPK) and CREB phosphorylation, which are further decoded by... 
SIGNALING PATHWAYS | ACTIVATION | SPECIFICITY | MULTIDISCIPLINARY SCIENCES | NEURONAL DIFFERENTIATION | NETWORKS | MECHANISMS | PC12 CELLS | NEGATIVE FEEDBACK | ERK OSCILLATIONS | NERVE GROWTH-FACTOR | PC12 Cells - cytology | Proto-Oncogene Proteins c-fos - metabolism | Nerve Growth Factor - pharmacology | Rats | Transcription Factors - genetics | Gene Expression Regulation - drug effects | Transcription Factors - metabolism | Cyclic AMP Response Element-Binding Protein - genetics | PC12 Cells - drug effects | Animals | Signal Transduction - drug effects | Time Factors | Models, Biological | PC12 Cells - metabolism | Pituitary Adenylate Cyclase-Activating Polypeptide - pharmacology | Cyclic AMP Response Element-Binding Protein - metabolism | Anisomycin - pharmacology | Mitogen-Activated Protein Kinases - genetics | Proto-Oncogene Proteins c-fos - genetics | Genes, Immediate-Early | Phosphorylation - drug effects | Mitogen-Activated Protein Kinases - metabolism | Physiological aspects | Nerve growth factor | Transcription factors | Research | Gene expression | Analysis | Phosphorylation | EGR-1 protein | Extracellular signal-regulated kinase | MAP kinase | Data processing | Decoding | Cyclic AMP response element-binding protein | Kinases | JunB protein | Pituitary adenylate cyclase-activating polypeptide | Filters | Epidermal growth factor | Fos protein | Rodents | Information processing | Signal processing | Growth factors | c-Fos protein
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 06/2005, Volume 93, Issue 5, pp. 1157 - 1167
Journal Article