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Bioimpacts, ISSN 2228-5660, 12/2018, Volume 8, Issue Suppl 1, pp. S1 - S129
Journal Article
PloS one, ISSN 1932-6203, 2015, Volume 10, Issue 8, p. e0135111
Exosomes deliver functional proteins and genetic materials to neighboring cells, and have potential applications for tissue regeneration... 
PROGENITOR CELLS | TRAUMATIC BRAIN-INJURY | REGENERATION | MULTIDISCIPLINARY SCIENCES | BONE-MARROW | SCIATIC-NERVE INJURY | SPINAL-CORD-INJURY | PROLIFERATION | MEDIATED TRANSFER | STROMAL CELLS | MICRORNA EXPRESSION | Oligonucleotide Array Sequence Analysis | Melanoma, Experimental - chemistry | Humans | Culture Media, Conditioned - chemistry | Culture Media, Conditioned - pharmacology | MicroRNAs - metabolism | Neurons - cytology | Gene Expression Profiling | PC12 Cells | Mesenchymal Stromal Cells - cytology | Bone Marrow Cells - drug effects | Neurons - metabolism | Neurons - drug effects | Mesenchymal Stromal Cells - drug effects | Bone Marrow Cells - cytology | Gene Expression Regulation | Mesenchymal Stromal Cells - metabolism | Rats | Animals | Cell Differentiation - drug effects | Cell Line, Tumor | Exosomes - chemistry | Mice | MicroRNAs - genetics | Primary Cell Culture | Bone Marrow Cells - metabolism | Physiological aspects | Genetic aspects | Research | Cell differentiation | Neurons | Stem cells | Transplants & implants | Mesenchyme | Amino acids | Exosomes | Kinases | Neurogenesis | Proteins | Cell growth | miRNA | Growth factors | Biomedical engineering | Tissue engineering | Melanoma | Ribonucleic acid--RNA | Progenitor cells | Regeneration | DNA microarrays | Microscopy | MicroRNAs | Cells (biology) | Pheochromocytoma cells | Neural stem cells | Spinal cord injuries | Differentiation | Cancer | RNA | Ribonucleic acid
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2009, Volume 106, Issue 52, pp. 22067 - 22072
Journal Article
PloS one, ISSN 1932-6203, 2010, Volume 5, Issue 12, pp. e15353 - 8
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2014, Volume 9, Issue 3, p. e92917
It has been proposed that differential activation kinetics allows cells to use a common set of signaling pathways to specify distinct cellular outcomes... 
PHEOCHROMOCYTOMA CELLS | SIGNALING PATHWAYS | ACTIVATED PROTEIN-KINASE | MAP KINASE | MULTIDISCIPLINARY SCIENCES | NEURONAL DIFFERENTIATION | MAMMALIAN-CELLS | PLASMA-MEMBRANE | NUCLEAR TRANSLOCATION | NERVE GROWTH-FACTOR | LIVING CELLS | NIH 3T3 Cells | Protein Transport - drug effects | Extracellular Signal-Regulated MAP Kinases - metabolism | raf Kinases - metabolism | PC12 Cells | Light | Cell Membrane - metabolism | Cell Membrane - drug effects | Neurites - drug effects | Cell Membrane - radiation effects | Cryptochromes - metabolism | Cell Differentiation - radiation effects | Protein Structure, Tertiary | MAP Kinase Signaling System - radiation effects | Proto-Oncogene Proteins c-raf | Nerve Growth Factor - pharmacology | Rats | MAP Kinase Kinase Kinases - metabolism | Neurites - metabolism | Enzyme Activation - drug effects | Enzyme Activation - radiation effects | Animals | MAP Kinase Signaling System - drug effects | Cell Differentiation - drug effects | Protein Transport - radiation effects | Cryptochromes - chemistry | Mice | Kinetics | Dose-Response Relationship, Radiation | Neurites - radiation effects | Nerve growth factor | Cellular signal transduction | Epidermal growth factor | Protein-protein interactions | Phosphorylation | Raf protein | Stimulation | Activation | Kinases | Proteins | Signal transduction | Cell growth | Cell cycle | Localization | Growth factors | Axonogenesis | Extracellular signal-regulated kinase | Gene expression | Studies | Signaling | Cell lines | Pheochromocytoma cells | Transduction | Genetic engineering | Protein interaction
Journal Article
Journal of Bioscience and Bioengineering, ISSN 1389-1723, 11/2017, Volume 124, Issue 5, pp. 572 - 582
Journal Article
Brain Research, ISSN 0006-8993, 2009, Volume 1283, pp. 139 - 147
Abstract Amyloid β-peptide (Aβ) has been implicated in the pathogenesis of AD. It can cause cell death in AD by evoking a cascade of oxidative damage to neurons... 
Neurology | Acteoside | Oxidative stress | MMP | ROS | Alzheimer's disease | Amyloid β-peptide | Apoptosis | INDUCED APOPTOSIS | ALZHEIMERS-DISEASE | RAT CORTICAL-NEURONS | PRIMARY CULTURES | NEUROSCIENCES | CYTOCHROME-C | INHIBITS APOPTOSIS | Amyloid beta-peptide | 1-METHYL-4-PHENYLPYRIDINIUM ION | INDUCED OXIDATIVE STRESS | PC12 CELLS | GAMMA-SECRETASE | Neuroprotective Agents - therapeutic use | Neurons - pathology | Reactive Oxygen Species - metabolism | Apoptosis - drug effects | Peptide Fragments - toxicity | Humans | Oxidative Stress - physiology | Caspase 3 - metabolism | Nerve Degeneration - physiopathology | Membrane Potential, Mitochondrial - drug effects | Apoptosis Regulatory Proteins - drug effects | Proto-Oncogene Proteins c-bcl-2 - metabolism | Neuroprotective Agents - pharmacology | Nerve Degeneration - prevention & control | Amyloid beta-Peptides - metabolism | Caspase 3 - drug effects | Membrane Potential, Mitochondrial - physiology | Glucosides - therapeutic use | Neurons - metabolism | Neurons - drug effects | Cell Survival - physiology | Phenols - pharmacology | Cell Survival - drug effects | Alzheimer Disease - physiopathology | Glucosides - pharmacology | Amyloid beta-Peptides - toxicity | Cytochromes c - metabolism | Alzheimer Disease - drug therapy | Antioxidants - pharmacology | Apoptosis Regulatory Proteins - metabolism | Amyloid beta-Peptides - antagonists & inhibitors | Antioxidants - therapeutic use | Peptide Fragments - antagonists & inhibitors | Phenols - therapeutic use | Alzheimer Disease - metabolism | Proto-Oncogene Proteins c-bcl-2 - drug effects | Cell Line, Tumor | Apoptosis - physiology | Oxidative Stress - drug effects | Nerve Degeneration - drug therapy | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2014, Volume 9, Issue 3, p. e90667
Journal Article
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 12/2009, Volume 284, Issue 51, pp. 35973 - 35986
...) remains biologically inert. Furthermore, N1 protects retinal ganglion cells from hypoxia-induced apoptosis, reduces the number of terminal deoxynucleotidyltransferase-mediated biotinylated UTP nick end labeling-positive... 
HEK293 HUMAN-CELLS | WILD-TYPE | CASPASE-3 ACTIVATION | ISCHEMIC TOLERANCE | SERUM DEPRIVATION | TUMOR SUPPRESSION | BIOCHEMISTRY & MOLECULAR BIOLOGY | PRIMARY CULTURED NEURONS | APOPTOTIC STIMULI | MUSCARINIC ACETYLCHOLINE-RECEPTORS | TRANSGENIC MICE | Recombinant Proteins - metabolism | Cell Survival - drug effects | Transcription, Genetic - drug effects | Cell Hypoxia - drug effects | Humans | Caspase 3 - metabolism | Cells, Cultured | Enzyme Inhibitors - pharmacology | Tumor Suppressor Protein p53 - metabolism | Cell Survival - genetics | Rats | Recombinant Proteins - genetics | Retinal Ganglion Cells - metabolism | Caspase Inhibitors | Tumor Suppressor Protein p53 - genetics | Animals | Mice | PrPC Proteins - metabolism | Cell Hypoxia - genetics | PrPC Proteins - genetics | Staurosporine - pharmacology | Apoptosis | Disease Models, Animal | Enzyme Inhibitors | Staurosporine | Neurons and Cognition | Male | Recombinant Proteins | Cellular Biology | PC12 Cells | Retinal Ganglion Cells | Cell Hypoxia | Dose-Response Relationship, Drug | Life Sciences | Caspase 3 | Brain-Derived Neurotrophic Factor | Transcription, Genetic | Signal Transduction | Cell Survival | Antibodies, Monoclonal | Receptor, Nerve Growth Factor | Receptors, Nerve Growth Factor | Tumor Suppressor Protein p53 | Nerve Growth Factors | PrPC Proteins | Kinetics | Amyloid beta-Protein Precursor | Molecular Basis of Cell and Developmental Biology
Journal Article