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EUROPEAN JOURNAL OF IMMUNOLOGY, ISSN 0014-2980, 09/2017, Volume 47, Issue 9, pp. 1457 - 1467
The chronic, destructive autoimmune arthritis in SKG mice, which closely resembles human rheumatoid arthritis, is the result of self-reactive T cells escaping... 
Inhibitory receptors | MHC CLASS-I | RHEUMATOID-ARTHRITIS | RESPONSES | ILT2 | PIR-B | INHIBITORY RECEPTOR | Arthritis | IMMUNOLOGY | CD4(+) T cells | SKG | T-CELLS
Journal Article
Journal Article
Journal Article
Oncogene, ISSN 0950-9232, 04/1999, Volume 18, Issue 14, pp. 2291 - 2297
Journal Article
Biochemical Journal, ISSN 0264-6021, 02/2007, Volume 402, Issue 1, pp. 1 - 15
It is now well established that the members of the PTP (protein tyrosine phosphatase) superfamily play critical roles in fundamental biological processes.... 
Substrate identification | Tyrosine phosphorylation | Protein tyrosine phosphatase (PTP) | Substrate-trapping | KINASE-ACTIVITY | BIOCHEMISTRY & MOLECULAR BIOLOGY | FACTOR RECEPTOR | NEGATIVE REGULATOR | SIGNAL-TRANSDUCTION | INSULIN-RECEPTOR | protein tyrosine phosphatase (PTP) | EPIDERMAL-GROWTH-FACTOR | IN-VIVO | HELICOBACTER-PYLORI CAGA | substrate-trapping | tyrosine phosphorylation | PTP-PEST | substrate identification | T-CELLS | Protein Structure, Tertiary | Phosphorylation | Protein Tyrosine Phosphatase, Non-Receptor Type 11 | Humans | Substrate Specificity | Protein Tyrosine Phosphatases - metabolism | Intracellular Signaling Peptides and Proteins - metabolism | Protein Tyrosine Phosphatases - genetics | Tyrosine - metabolism | Animals | Models, Biological | Protein Tyrosine Phosphatase, Non-Receptor Type 1 | Binding Sites | serine | SH, Src homology | CSK, C-terminal Src kinase | RNAi, RNA interference | PTK, protein tyrosine kinase | MKP, MAPK phosphatase | IRS, IR substrate | CSF-1, colony-stimulating factor 1 | SHP, SH2-domain-containing protein tyrosine phosphatase | ERK, extracellular-signal-regulated kinase | PIR-B, paired immunoglobulin-like receptor B | ZAP-70, ζ-chain-associated protein kinase of 70 kDa | KIM, kinase-interacting motif | FAK, focal adhesion kinase | EGFR, epidermal growth factor receptor | STEP, striatal-enriched PTP | LYP, lymphoid phosphatase | MAPK, mitogen-activated protein kinase | PTP, protein tyrosine phosphatase | SNARE, NSF-attachment protein receptor | TCR, T-cell receptor | PI3K, phosphoinositide 3-kinase | Gab1, Grb2-associated binder-1 | PAG, phosphoprotein associated with glycosphingolipid-enriched membrane microdomains | Cbp, C-terminal Src kinase-binding protein | SNP, single-nucleotide polymorphism | Review | WASP, Wiskott–Aldrich syndrome protein | NSF, N-ethylmaleimide-sensitive factor | PDGFR, platelet-derived growth factor receptor | AP-1, activator protein 1 | PEP, PEST (Pro-Glu-Ser-Thr) domain phosphatase | SFK, Src family kinase | DSP, dual-specificity phosphatase | PSTPIP, proline | NS, Noonan syndrome | IR, insulin receptor | IFN, interferon | STAT, signal transducer and activator of transcription | NFAT, nuclear factor of activated T-cells | IGF-1, insulin-like growth factor 1 | BIT, brain immunoglobulin-like molecule with tyrosine-based activation motifs | threonine-phosphatase-interacting protein | TCPTP, T-cell PTP | JAK, Janus kinase | MEF, mouse embryonic fibroblast | BCR, B-cell receptor | GAP, GTPase-activating protein | HGF, hepatocyte growth factor
Journal Article
Nature Immunology, ISSN 1529-2908, 01/2014, Volume 15, Issue 1, pp. 36 - 44
Eosinophilia is a hallmark characteristic of T helper type 2 (T(H)2) cell-associated diseases and is critically regulated by the central eosinophil growth... 
RESPONSES | ALTERNATIVELY ACTIVATED MACROPHAGES | DENDRITIC CELLS | NK CELL RECOGNITION | PIR-B | BONE-MARROW | IN-VIVO | DISEASE | INHIBITORY RECEPTOR | MICE | IMMUNOLOGY | Stem Cells - immunology | GRB2 Adaptor Protein - immunology | Apoptosis - genetics | Male | Eosinophils - immunology | Extracellular Signal-Regulated MAP Kinases - immunology | Asthma - immunology | Gene Expression - immunology | Apoptosis Regulatory Proteins - immunology | Membrane Proteins - genetics | Mice, Transgenic | Signal Transduction - genetics | Reverse Transcriptase Polymerase Chain Reaction | Apoptosis Regulatory Proteins - metabolism | Mice, Knockout | Interleukin-5 - immunology | Cell Differentiation - immunology | Mice | Receptors, Immunologic - genetics | Asthma - metabolism | Eosinophils - metabolism | Stem Cells - cytology | Extracellular Signal-Regulated MAP Kinases - metabolism | Extracellular Signal-Regulated MAP Kinases - genetics | Colony-Forming Units Assay - methods | Cell Differentiation - genetics | Signal Transduction - immunology | Flow Cytometry | Bcl-2-Like Protein 11 | GRB2 Adaptor Protein - genetics | Proto-Oncogene Proteins - immunology | Apoptosis Regulatory Proteins - genetics | Female | Membrane Proteins - metabolism | Receptors, Immunologic - immunology | Proto-Oncogene Proteins - metabolism | Interleukin-5 - pharmacology | Mice, Inbred C57BL | Cells, Cultured | Membrane Proteins - immunology | Proto-Oncogene Proteins - genetics | Asthma - genetics | Animals | Apoptosis - immunology | Cell Differentiation - drug effects | Stem Cells - drug effects | Eosinophils - cytology | GRB2 Adaptor Protein - metabolism | Receptors, Immunologic - metabolism | Biological research | Immune response | Research | Properties | Identification and classification | Interleukin-5 | Eosinophils | Biology, Experimental
Journal Article
Journal Article
Cancer Immunology, Immunotherapy, ISSN 0340-7004, 8/2017, Volume 66, Issue 8, pp. 1079 - 1087
Journal Article