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Journal Article
Thrombosis Research, ISSN 0049-3848, 01/2017, Volume 149, pp. 88 - 94
Journal Article
Circulation, ISSN 0009-7322, 2018, Volume 137, Issue 3, pp. 307 - 309
Journal Article
Circulation, ISSN 0009-7322, 09/2010, Volume 122, Issue 11, pp. 1056 - 1067
Background-Reduced renal function is associated with a poorer prognosis and increased bleeding risk in patients with acute coronary syndromes and may therefore... 
clopidogrel | mortality | renal function | acute coronary syndrome | bleeding | myocardial infarction | CARDIAC & CARDIOVASCULAR SYSTEMS | EVENTS | MYOCARDIAL-INFARCTION | INSUFFICIENCY | RISK | CLASSIFICATION | IMPACT | EPTIFIBATIDE | PERIPHERAL VASCULAR DISEASE | DYSFUNCTION | CHRONIC KIDNEY-DISEASE | HEMATOLOGY | ARTERY-DISEASE | Kidney Diseases - physiopathology | Outcome Assessment (Health Care) | Ticlopidine - therapeutic use | Humans | Middle Aged | Acute Coronary Syndrome - physiopathology | Male | Adenosine - adverse effects | Adenosine - therapeutic use | Receptors, Purinergic P2Y12 | Ticlopidine - adverse effects | Cardiovascular Diseases - epidemiology | Female | Kidney Diseases - complications | Platelet Aggregation Inhibitors - therapeutic use | Platelet Aggregation Inhibitors - adverse effects | Cardiovascular Diseases - physiopathology | Hemorrhage - epidemiology | Hemorrhage - physiopathology | Risk Factors | Kaplan-Meier Estimate | Purinergic P2 Receptor Antagonists | Treatment Outcome | Ticlopidine - analogs & derivatives | Acute Coronary Syndrome - drug therapy | Adenosine - analogs & derivatives | Aged | Chronic Disease | Aggregation | Usage | Blood platelets | Patient outcomes | Clopidogrel | Drug therapy | Comparative analysis | Health aspects | Heart attack | Index Medicus | Abridged Index Medicus | Medical and Health Sciences | Medicin och hälsovetenskap | MEDICIN | MEDICINE
Journal Article
Circulation, ISSN 0009-7322, 11/2010, Volume 122, Issue 21, pp. 2131 - 2141
Background-Aspirin and clopidogrel are recommended for patients with acute coronary syndromes (ACS) or undergoing coronary stenting. Ticagrelor, a reversible... 
acute coronary syndrome | thrombosis | platelets | myocardial infarction | hemorrhage | 2007 FOCUSED UPDATE | AZD6140 | CARDIAC & CARDIOVASCULAR SYSTEMS | SEGMENT ELEVATION | MANAGEMENT | EVENTS | P2Y RECEPTOR ANTAGONIST | ASPIRIN | ACUTE MYOCARDIAL-INFARCTION | DOUBLE-BLIND | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | ASSOCIATION | Purinergic P2Y Receptor Antagonists - administration & dosage | Coronary Thrombosis - epidemiology | Humans | Middle Aged | Male | Angioplasty, Balloon, Coronary | Purinergic P2Y Receptor Antagonists - adverse effects | Adenosine - adverse effects | Platelet Aggregation Inhibitors - administration & dosage | Coronary Thrombosis - prevention & control | Ticlopidine - adverse effects | Electrocardiography | Ticlopidine - administration & dosage | Female | Platelet Aggregation Inhibitors - adverse effects | Acute Coronary Syndrome - epidemiology | Hemorrhage - epidemiology | Risk Factors | Kaplan-Meier Estimate | Treatment Outcome | Adenosine - administration & dosage | Ticlopidine - analogs & derivatives | Acute Coronary Syndrome - drug therapy | Adenosine - analogs & derivatives | Acute Coronary Syndrome - diagnosis | Aged | Hemorrhage - chemically induced | Usage | Angioplasty | Patient outcomes | Clopidogrel | Drug therapy | Ticagrelor | Health aspects | Heart attack | Index Medicus | Abridged Index Medicus | Medical and Health Sciences | Medicin och hälsovetenskap | MEDICIN | MEDICINE
Journal Article
Circulation, ISSN 0009-7322, 07/2012, Volume 126, Issue 4, pp. 455 - 467
Background-Epigenetic programming, dynamically regulated by histone acetylation, is a key mechanism regulating cell proliferation and survival. Little is known... 
genetic | epigenesis | vorinostat | histone deacetylation | pulmonary | valproic acid | hypertension | TARGET | CELLS | APOPTOSIS | CARDIAC & CARDIOVASCULAR SYSTEMS | ACETYLATION | hypertension, pulmonary | epigenesis, genetic | PROLIFERATION | EMERGENCE | ARTERIAL-HYPERTENSION | PROSTATE-CANCER | IN-VIVO | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | EXPRESSION | Muscle, Smooth, Vascular - metabolism | Humans | Male | Valproic Acid - pharmacology | Pulmonary Artery - metabolism | Histone Deacetylase 1 - antagonists & inhibitors | Valproic Acid - therapeutic use | Hypertension, Pulmonary - drug therapy | Lung - metabolism | Hydroxamic Acids - pharmacology | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Lung - pathology | Cells, Cultured | Rats | Histone Deacetylases - metabolism | Hypertension, Pulmonary - metabolism | Hypoxia - complications | Platelet-Derived Growth Factor - pharmacology | Pulmonary Artery - drug effects | Rats, Sprague-Dawley | Muscle, Smooth, Vascular - pathology | Animals | Lung - drug effects | Histone Deacetylase Inhibitors - pharmacology | Histone Deacetylases - drug effects | Hydroxamic Acids - therapeutic use | Cell Proliferation - drug effects | Histone Deacetylase Inhibitors - therapeutic use | Hypertension, Pulmonary - etiology | Pulmonary Artery - pathology | Histone Deacetylase 1 - metabolism | Cell proliferation | Divalproex | Care and treatment | Usage | Enzyme inhibitors | Physiological aspects | Research | Valproic acid | Health aspects | Pulmonary hypertension | Index Medicus | Abridged Index Medicus
Journal Article
Circulation, ISSN 0009-7322, 12/2007, Volume 116, Issue 25, pp. 2923 - 2932
Journal Article
Circulation, ISSN 0009-7322, 06/2011, Volume 123, Issue 23, pp. 2681 - 2689
Background-The balance between benefit (ischemia protection) and risk (bleeding) is a key consideration in choosing the intensity of antiplatelet therapy for... 
clopidogrel | mortality | prasugrel | hemorrhage | CARDIAC & CARDIOVASCULAR SYSTEMS | METAANALYSIS | ELEVATION | PREVENTION | RISK | ACUTE CORONARY SYNDROMES | ASPIRIN | IMPACT | RESIDUALS | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | INTERVENTION | Piperazines - administration & dosage | Prasugrel Hydrochloride | Predictive Value of Tests | Myocardial Infarction - mortality | Purinergic P2Y Receptor Antagonists - administration & dosage | Thiophenes - adverse effects | Humans | Middle Aged | Hemorrhage - mortality | Male | Thiophenes - administration & dosage | Aspirin - administration & dosage | Purinergic P2Y Receptor Antagonists - adverse effects | Aspirin - adverse effects | Platelet Aggregation Inhibitors - administration & dosage | Thrombolytic Therapy - methods | Ticlopidine - adverse effects | Ticlopidine - administration & dosage | Female | Drug Therapy, Combination | Platelet Aggregation Inhibitors - adverse effects | Risk Factors | Proportional Hazards Models | Piperazines - adverse effects | Ticlopidine - analogs & derivatives | Hemorrhage - diagnosis | Myocardial Infarction - drug therapy | Aged | Hemorrhage - chemically induced | Thrombolytic Therapy - adverse effects | Aggregation | Usage | Prognosis | Blood platelets | Myocardial ischemia | Diagnosis | Health aspects | Risk factors | Heart attack | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 11/2004, Volume 44, Issue 9, pp. 1792 - 1800
Journal Article
The American Journal of Cardiology, ISSN 0002-9149, 04/2017, Volume 119, Issue 7, pp. 991 - 995
Journal Article
American Heart Journal, ISSN 0002-8703, 2012, Volume 164, Issue 3, pp. 334 - 342.e1
Journal Article
Nature, ISSN 0028-0836, 2014, Volume 508, Issue 1, pp. 118 - 122
Treatment of BRAF(V600E) mutant melanoma by small molecule drugs that target the BRAF or MEK kinases can be effective, but resistance develops invariably(1,2).... 
GROWTH-FACTOR RECEPTOR | BRAF INHIBITOR | RAF INHIBITION | CELLS | MULTIDISCIPLINARY SCIENCES | IMPROVED SURVIVAL | DIFFERENTIATION | C-JUN | CANCER | EXPRESSION | EGFR | Receptor, Epidermal Growth Factor - genetics | Humans | Receptor Protein-Tyrosine Kinases - biosynthesis | Cellular Senescence - drug effects | Melanoma - enzymology | Antineoplastic Agents - administration & dosage | Receptor, Platelet-Derived Growth Factor beta - genetics | Indoles - administration & dosage | Mitogen-Activated Protein Kinase Kinases - metabolism | Receptor, Epidermal Growth Factor - metabolism | Flow Cytometry | Melanoma - genetics | Female | Indoles - pharmacology | Antineoplastic Agents - pharmacology | Gene Expression Regulation, Neoplastic - drug effects | SOXE Transcription Factors - deficiency | Proto-Oncogene Proteins B-raf - metabolism | Receptor, Platelet-Derived Growth Factor beta - metabolism | Receptor, Epidermal Growth Factor - biosynthesis | Gene Library | Mitogen-Activated Protein Kinase Kinases - antagonists & inhibitors | Melanoma - pathology | Receptor Protein-Tyrosine Kinases - metabolism | Sulfonamides - pharmacology | Proto-Oncogene Proteins B-raf - antagonists & inhibitors | Protein Kinase Inhibitors - administration & dosage | Transforming Growth Factor beta - pharmacology | Drug Resistance, Neoplasm - genetics | Animals | Receptor Protein-Tyrosine Kinases - genetics | Signal Transduction - drug effects | Proto-Oncogene Proteins B-raf - genetics | Melanoma - drug therapy | Cell Proliferation - drug effects | Mice | Protein Kinase Inhibitors - pharmacology | RNA, Small Interfering | Transforming Growth Factor beta - metabolism | Receptor, Platelet-Derived Growth Factor beta - biosynthesis | Sulfonamides - administration & dosage | Drug Resistance, Neoplasm - drug effects | SOXE Transcription Factors - genetics | Proteins | Biopsy | Rodents | Genes | Melanoma | Mutation | Kinases | Drug resistance | Patients | Tumors | Index Medicus
Journal Article