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The Journal of pharmacology and experimental therapeutics, ISSN 1521-0103, 06/2015, Volume 353, Issue 3, pp. 446 - 457
Recent findings indicate that a major mechanism by which poly (ADP-ribose) polymerase (PARP) inhibitors kill cancer cells is by trapping PARP1 and PARP2 to the sites of DNA damage... 
Life Sciences & Biomedicine | Pharmacology & Pharmacy | Science & Technology | Poly(ADP-ribose) Polymerases - metabolism | Antineoplastic Agents - pharmacology | Animals | Poly(ADP-ribose) Polymerase Inhibitors | Humans | Enzyme Inhibitors - pharmacology | Index Medicus
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Journal Article
The Lancet (British edition), ISSN 0140-6736, 2010, Volume 376, Issue 9737, pp. 245 - 251
Journal Article
Journal Article
Cancer science, ISSN 1347-9032, 11/2018, Volume 109, Issue 11, pp. 3383 - 3392
...‐directed therapies for other solid organ malignancies. Poly (ADP‐ribose) polymerase inhibitors (PARPi... 
lysine‐specific histone demethylase‐1A | poly (ADP‐ribose) polymerase inhibitor | immune checkpoint inhibitor | cancer stem cell | triple‐negative breast cancer | triple-negative breast cancer | lysine-specific histone demethylase-1A | poly (ADP-ribose) polymerase inhibitor | Life Sciences & Biomedicine | Oncology | Science & Technology | Homologous Recombination - drug effects | Neoplastic Stem Cells - drug effects | Humans | Clinical Trials as Topic | Triple Negative Breast Neoplasms - drug therapy | B7-H1 Antigen - genetics | Poly(ADP-ribose) Polymerase Inhibitors - therapeutic use | Up-Regulation - drug effects | Triple Negative Breast Neoplasms - genetics | Neoplastic Stem Cells - metabolism | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Triple Negative Breast Neoplasms - metabolism | Immunotherapy | Female | Histone Deacetylase Inhibitors - pharmacology | Epigenesis, Genetic - drug effects | Histone Deacetylase Inhibitors - therapeutic use | Gene Expression Regulation, Neoplastic - drug effects | Drug Resistance, Neoplasm - drug effects | Epigenetic inheritance | Lysine | Cell death | Stem cells | Sugars | Monosaccharides | Drugs | Histone deacetylase | Transcription factors | DNA damage | Homologous recombination | Cytotoxicity | Cancer therapies | DNA repair | Immunosuppressive agents | Ovarian cancer | Proteins | Epidermal growth factor | Antitumor agents | Ribose | Cell cycle | Deoxyribonucleic acid--DNA | Phenotypes | Breast cancer | Chemotherapy | Immunogenicity | Response rates | Epigenetics | Ligands | Tumors | Apoptosis | Index Medicus | Review
Journal Article