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American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 04/2018, Volume 314, Issue 4, pp. H812 - H838
Myocardial infarction is a prevalent major cardiovascular event that arises from myocardial ischemia with or without reperfusion, and basic and translational... 
Heart failure | Myocardial infarction | Animal models | Rigor and reproducibility | Reperfusion | Cardiac remodeling | heart failure | CHRONIC HEART-FAILURE | animal models | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | POSTMYOCARDIAL INFARCTION | myocardial infarction | HIBERNATING MYOCARDIUM | REPERFUSION INJURY | NO-REFLOW PHENOMENON | CELL-DERIVED CARDIOMYOCYTES | BLOOD-FLOW | reperfuson | CORONARY COLLATERAL CIRCULATION | CARDIOPROTECTIVE THERAPIES CAESAR | LEFT-VENTRICULAR DYSFUNCTION | cardiac remodeling | PERIPHERAL VASCULAR DISEASE | rigor and reproducibility | Myocardial Ischemia - metabolism | Data Accuracy | Cells, Cultured | Myocardium - pathology | Cardiology - standards | Myocardial Infarction - metabolism | Periodicals as Topic - standards | Consensus | Myocardial Ischemia - pathology | Isolated Heart Preparation - standards | Animals | Myocardial Infarction - pathology | Myocardium - metabolism | Physiology - standards | Myocardial Ischemia - physiopathology | Myocardial Infarction - physiopathology | Mice | Biomedical Research - standards | Disease Models, Animal | Quality Control | Myocardial ischemia | Biological models | Usage | Models | Heart attack | Occlusion | Heart attacks | Cardiovascular disease | Angina | Guidelines | Salvage | Ischemia | Reproducibility | Research methodology | Hibernation | Infarction | Best practice | Structure-function relationships | Index Medicus | Review | reperfusion
Journal Article
Journal Article
Journal Article
Basic Research in Cardiology, ISSN 0300-8428, 7/2018, Volume 113, Issue 4, pp. 1 - 18
In response to myocardial infarction (MI), cardiac macrophages regulate inflammation and scar formation. We hypothesized that macrophages undergo polarization... 
Myocardial infarction | Medicine & Public Health | RNA-Seq | Transcriptome | LV remodeling | Cardiology | Macrophage | PRO-INFLAMMATORY CYTOKINES | OXIDATIVE STRESS | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | POSTMYOCARDIAL INFARCTION | CARDIAC DYSFUNCTION | GENE-EXPRESSION | TISSUE MACROPHAGES | STEADY-STATE | EXTRACELLULAR-MATRIX | MICE | Myocardial Infarction - genetics | Cell Proliferation | Ventricular Function, Left - genetics | Extracellular Matrix - metabolism | Male | Gene Expression Profiling | Extracellular Matrix - genetics | Flow Cytometry | Time Factors | Myocardial Infarction - pathology | Myocardium - metabolism | Inflammation Mediators - metabolism | Ventricular Remodeling - genetics | Myocardial Infarction - physiopathology | Extracellular Matrix Proteins - metabolism | Disease Models, Animal | Macrophages - pathology | Extracellular Matrix Proteins - genetics | Mice, Inbred C57BL | Cells, Cultured | Genotype | Myocardium - pathology | Myocardial Infarction - metabolism | Cell Plasticity - genetics | Macrophages - metabolism | Phenotype | Animals | Energy Metabolism | Extracellular Matrix - pathology | Phagocytosis | RNA sequencing | RNA | Collagen | Analysis | Mice | Macrophages | Gene expression | Heart attack | Heart | Polarization | Flow cytometry | Phosphorylation | Collagen (type I) | Heart attacks | Genes | Coronary artery | Inflammation | Remodeling | Metabolism | Gene sequencing | Monocytes | Mitochondria | Oxidative phosphorylation | Ribonucleic acids | Rodents | Extracellular matrix | Infarction | Heart diseases | Index Medicus | Original Contribution
Journal Article
American Heart Journal, ISSN 0002-8703, 06/2018, Volume 200, pp. 110 - 117
Journal Article
Basic Research in Cardiology, ISSN 0300-8428, 7/2017, Volume 112, Issue 4, pp. 1 - 13
Optimal healing after myocardial infarction requires not only the induction of inflammation, but also its timely resolution. In patients, 30 days post... 
Heart failure | Myocardial infarction | Inflammation | Medicine & Public Health | Cardiology | Semaphorin3A | CARDIAC & CARDIOVASCULAR SYSTEMS | ANGIOGENESIS | POSTMYOCARDIAL INFARCTION | MACROPHAGES | PHENOTYPE | MONOCYTES | VASCULAR-PERMEABILITY | SEMAPHORIN 3A | REPAIR | THERAPY | STEM-CELL | Chemotaxis, Leukocyte | Myocardial Infarction - genetics | Male | Monocytes - metabolism | Semaphorin-3A - metabolism | Wound Healing | Recovery of Function | Semaphorin-3A - deficiency | Time Factors | Myocardial Infarction - pathology | Myocardium - metabolism | Female | Myocardial Infarction - physiopathology | Disease Models, Animal | Myocarditis - genetics | Semaphorin-3A - genetics | Macrophages - pathology | Signal Transduction | Cells, Cultured | Myocardium - pathology | Myocardial Infarction - metabolism | Macrophage Activation | Mice, Knockout | Macrophages - metabolism | Phenotype | Animals | Myocarditis - metabolism | Heterozygote | Myocarditis - pathology | Myocarditis - physiopathology | Apoptosis | Heart | Macrophages | Heart attack | Polarization | Heart attacks | Leukocyte migration | Migration | CX3CR1 protein | Myocardial ischemia | Leukocytes | Tissues | Optimization | Recruitment | Ischemia | Rodents | Bone marrow | Heart diseases | Genotypes | Deposition | Recombinant | Wound healing | Patients | Adhesion | Monocytes | Semaphorins | Collagen | Myocardium | Healing | Infarction | Mice | Surface area | In vitro methods and tests | Index Medicus | Original Contribution
Journal Article
Circulation, ISSN 0009-7322, 04/2007, Volume 115, Issue 15, pp. 2006 - 2014
Journal Article
Journal Article
Journal Article