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Journal of molecular and cellular cardiology, ISSN 0022-2828, 07/2014, Volume 72, pp. 85 - 94
Abstract Tumor necrosis factor-α (TNF-α) is an established pro-atherosclerotic factor, but the mechanism is not completely understood. We explored whether... 
Cardiovascular | NF-κB | TNF-α | LDL | PPAR-γ | Atherosclerosis | Cardiac & Cardiovascular Systems | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Cell Biology | Tumor Necrosis Factor-alpha - metabolism | RNA, Small Interfering - genetics | Human Umbilical Vein Endothelial Cells - metabolism | Nitriles - pharmacology | Atherosclerosis - genetics | Humans | NF-kappa B - metabolism | Filipin - pharmacology | PPAR gamma - metabolism | Sulfones - pharmacology | Caveolin 2 - antagonists & inhibitors | Thiocarbamates - pharmacology | Human Umbilical Vein Endothelial Cells - cytology | Proline - pharmacology | Benzamides - pharmacology | Lipoproteins, LDL - metabolism | PPAR gamma - genetics | Caveolin 1 - antagonists & inhibitors | Receptors, LDL - genetics | Atherosclerosis - pathology | NF-kappa B - antagonists & inhibitors | Human Umbilical Vein Endothelial Cells - drug effects | Proline - analogs & derivatives | Signal Transduction | Atherosclerosis - chemically induced | Receptors, LDL - antagonists & inhibitors | Gene Expression Regulation | Caveolin 1 - genetics | Receptors, LDL - metabolism | Mice, Knockout | Caveolin 1 - metabolism | Caveolin 2 - metabolism | Cinchona Alkaloids - pharmacology | Tumor Necrosis Factor-alpha - pharmacology | Animals | PPAR gamma - antagonists & inhibitors | Lipoproteins, LDL - antagonists & inhibitors | NF-kappa B - genetics | Anilides - pharmacology | Mice | Pyridines - pharmacology | Transcytosis - drug effects | Caveolin 2 - genetics | Atherosclerosis - prevention & control | RNA, Small Interfering - metabolism | Index Medicus
Journal Article
Neurotoxicity research, ISSN 1029-8428, 1/2017, Volume 31, Issue 1, pp. 77 - 89
Journal Article
The Journal of biological chemistry, ISSN 0021-9258, 2012, Volume 287, Issue 3, pp. 1847 - 1860
...) and, even more importantly, anti-inflammatory interleukin 1 receptor antagonist (IL1Rn). Overexpression of PGC-1 alpha and induction of PGC-1 alpha by fasting, physical... 
necrosis-factor-alpha | skeletal-muscle | metabolic syndrome | nuclear receptors | hepatic gluconeogenesis | insulin-resistance | caloric restriction | diabetes-mellitus | endoplasmic-reticulum stress | human obesity | Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | Enzyme Activators - pharmacology | RNA-Binding Proteins - genetics | AMP-Activated Protein Kinases - metabolism | Liver - pathology | Receptors, Interleukin-15 - biosynthesis | Haptoglobins - biosynthesis | Caloric Restriction | Haptoglobins - genetics | Hepatocytes - pathology | Interleukin 1 Receptor Antagonist Protein - genetics | Male | Hepatocytes - metabolism | Obesity - genetics | Gene Knockdown Techniques | Receptors, Interleukin-15 - genetics | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Inflammation Mediators - metabolism | Trans-Activators - genetics | Mice, Inbred DBA | Fasting - metabolism | Interleukin 1 Receptor Antagonist Protein - biosynthesis | C-Reactive Protein - biosynthesis | Physical Conditioning, Animal | Gene Expression Regulation - genetics | Liver - metabolism | Metformin - pharmacology | Cells, Cultured | Rats | PPAR alpha - genetics | C-Reactive Protein - genetics | Transcription Factors - genetics | Hypoglycemic Agents - pharmacology | Obesity - metabolism | Gene Expression Regulation - drug effects | Transcription Factors - metabolism | Animals | Energy Metabolism | Insulin Resistance - genetics | Obesity - therapy | Trans-Activators - metabolism | Mice | PPAR alpha - metabolism | AMP-Activated Protein Kinases - genetics | RNA-Binding Proteins - metabolism | Index Medicus | IL15Rα | IL1Rn | Coactivator Transcription | Liver | Inflammation | Cellular Immune Response | Diabetes | Tweak | Metformin | Metabolism | Nuclear Receptors
Journal Article
Gut, ISSN 0017-5749, 08/2014, Volume 63, Issue 8, pp. 1300 - 1312
Objective Enteric glia activation has been reported to amplify intestinal inflammation via the enteroglial-specific S100B protein. This neurotrophin promotes... 
Gastroenterology & Hepatology | Life Sciences & Biomedicine | Science & Technology | Tumor Necrosis Factor-alpha - metabolism | Dextran Sulfate | Ethanolamines - pharmacology | Endocannabinoids - pharmacology | Rectum - pathology | Humans | Middle Aged | Palmitic Acids - therapeutic use | Male | NF-kappa B - metabolism | Glial Fibrillary Acidic Protein - metabolism | Nitric Oxide - analysis | Colon, Sigmoid - chemistry | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Neuroglia - drug effects | Colitis - chemically induced | Female | Indoles - pharmacology | Colitis - drug therapy | S100 Calcium Binding Protein beta Subunit - metabolism | Colitis, Ulcerative - drug therapy | Palmitic Acids - pharmacology | Colon, Sigmoid - pathology | Endocannabinoids - therapeutic use | Neutrophil Infiltration - drug effects | Severity of Illness Index | PPAR alpha - antagonists & inhibitors | Signal Transduction | Colitis, Ulcerative - metabolism | Cells, Cultured | Colitis, Ulcerative - pathology | Toll-Like Receptor 4 - metabolism | Dinoprostone - metabolism | Nerve Tissue Proteins - metabolism | Animals | PPAR gamma - antagonists & inhibitors | Anti-Inflammatory Agents, Non-Steroidal - therapeutic use | Anilides - pharmacology | Colitis - metabolism | Cyclooxygenase 2 - metabolism | Rectum - chemistry | Ethanolamines - therapeutic use | Neuroglia - metabolism | Mice | PPAR alpha - metabolism | Nitric Oxide - metabolism | Nitric Oxide Synthase Type II - metabolism | Influence | Amides | Inflammation | Neuroglia | Analysis | Risk factors | Index Medicus | Abridged Index Medicus
Journal Article
Experimental cell research, ISSN 0014-4827, 01/2015, Volume 330, Issue 2, pp. 287 - 299
The present study deals with the molecular mechanisms involved in the regulation of E-cadherin expression under hypoxia, because the adjustment of the amount... 
Wwox | HIF-1 | Hypoxia | Bone metastasis | E-cadherin | Oncology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Oxidoreductases - antagonists & inhibitors | Cell Hypoxia - physiology | Cadherins - metabolism | Tumor Suppressor Proteins - antagonists & inhibitors | Epithelial-Mesenchymal Transition - physiology | Humans | Transcriptional Activation | Gene Expression Regulation, Neoplastic | Bone Neoplasms - secondary | PPAR gamma - metabolism | Cadherins - biosynthesis | WW Domain-Containing Oxidoreductase | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Tumor Suppressor Proteins - genetics | Female | Cadherins - genetics | Proto-Oncogene Proteins c-mdm2 - antagonists & inhibitors | Proto-Oncogene Proteins c-mdm2 - metabolism | Carcinoma, Ductal, Breast - secondary | Protein-Serine-Threonine Kinases - metabolism | Oxidoreductases - biosynthesis | Oxidoreductases - genetics | Ubiquitin-Protein Ligases - metabolism | Transcription Factors - antagonists & inhibitors | Transcription Factors - metabolism | Breast Neoplasms - pathology | Cell Line, Tumor | Tumor Suppressor Proteins - biosynthesis | Index Medicus | TRANSCRIPTION FACTORS | MAMMARY GLANDS | PHOSPHORYLATION | METASTASES | PHENOTYPE | BONE MARROW | 60 APPLIED LIFE SCIENCES | SKELETON | ANOXIA | IN VITRO | CARCINOMAS | LIGASES | RECEPTORS
Journal Article
Canadian journal of physiology and pharmacology, ISSN 1205-7541, 08/2012, Volume 90, Issue 8, pp. 1135 - 1144
Peroxisome proliferator-activated receptors (PPAR) regulate the expression of genes involved in lipid metabolism, energy production, and inflammation. Their... 
WY-14643 | activation du PPAR-α | ischémie–reperfusion du myocarde | myocardial ischaemia–reperfusion | PPAR-α activation | cardioprotection | PI3K–Akt | metabolic genes | gènes du métabolisme | Pharmacology & Pharmacy | Physiology | Life Sciences & Biomedicine | Science & Technology | Phosphorylation | Pyrimidines - antagonists & inhibitors | Phosphatidylinositol 3-Kinase - physiology | Peroxisome Proliferators - therapeutic use | Rats, Wistar | Myocardial Reperfusion Injury - complications | Phosphatidylinositol 3-Kinase - antagonists & inhibitors | Male | Arrhythmias, Cardiac - physiopathology | Peroxisome Proliferators - antagonists & inhibitors | Up-Regulation - physiology | Myocardial Infarction - physiopathology | Protein Kinases - biosynthesis | Proto-Oncogene Proteins c-akt - metabolism | Disease Models, Animal | Arrhythmias, Cardiac - prevention & control | PPAR alpha - biosynthesis | Rats | Peroxisome Proliferators - pharmacology | Chymases - biosynthesis | Proto-Oncogene Proteins c-akt - physiology | Pyrimidines - pharmacology | Myocardial Reperfusion Injury - physiopathology | Up-Regulation - drug effects | Myocardial Infarction - complications | Myocardial Reperfusion Injury - metabolism | Animals | Androstadienes - pharmacology | Signal Transduction - drug effects | Pyrimidines - therapeutic use | PPAR alpha - physiology | Signal Transduction - physiology | Arrhythmias, Cardiac - complications | Myocardial Infarction - prevention & control | Protein Kinase Inhibitors - pharmacology | Myocardial Reperfusion Injury - prevention & control | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Cardiovascular system | Prevention | Transcription factors | Ischemia | Cardiotonic agents | Analysis | Research | Cardiac glycosides | Index Medicus
Journal Article
Molecular and cellular endocrinology, ISSN 0303-7207, 08/2011, Volume 343, Issue 1-2, pp. 55 - 62
Journal Article