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Journal of Neurochemistry, ISSN 0022-3042, 05/2013, Volume 125, Issue 3, pp. 410 - 419
Huntington's disease (HD) is a devastating genetic neurodegenerative disease caused by CAG trinucleotide expansion in the exon‐1 region of the huntingtin gene.... 
PPAR‐γ | BDNF | glucose metabolism | Sirt6 | huntingtin | PGC‐1α | PGC-1α | PPAR-γ | BDNF LEVELS | TRANSGENIC MOUSE | NEUROTROPHIC FACTOR | PGC-1-ALPHA | BIOCHEMISTRY & MOLECULAR BIOLOGY | AMYOTROPHIC-LATERAL-SCLEROSIS | NEUROSCIENCES | TRANSCRIPTIONAL DYSREGULATION | PPAR | PGC-1 | ACTIVATED-RECEPTOR-GAMMA | MITOCHONDRIAL DYSFUNCTION | ENERGY-METABOLISM | PARKINSONS-DISEASE | L-Lactate Dehydrogenase - metabolism | Neuroprotective Agents - therapeutic use | Humans | Huntington Disease - pathology | Brain - enzymology | Male | Intracellular Signaling Peptides and Proteins - metabolism | RNA, Messenger - metabolism | Hyperglycemia - drug therapy | Thiazolidinediones - therapeutic use | Neuroprotective Agents - pharmacology | Transfection | Adenosine Triphosphate - metabolism | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Trans-Activators - genetics | Brain-Derived Neurotrophic Factor - metabolism | Neurons - metabolism | Huntington Disease - drug therapy | Neurons - drug effects | Thiazolidinediones - pharmacology | Disease Models, Animal | Cell Line | Orexins | Gene Expression Regulation - genetics | Glutamates - genetics | Mice, Transgenic | Neuropeptides - metabolism | Nerve Tissue Proteins - genetics | Brain - drug effects | Gene Expression Regulation - drug effects | Huntington Disease - complications | Huntingtin Protein | Animals | PPAR gamma - antagonists & inhibitors | Movement Disorders - etiology | Anilides - pharmacology | Brain - pathology | Huntington Disease - genetics | Trinucleotide Repeat Expansion - genetics | Trans-Activators - metabolism | Hyperglycemia - etiology | Mice | Transcription Factors | Sirtuins - metabolism | Movement Disorders - drug therapy | Index Medicus
Journal Article
Biochemical Pharmacology, ISSN 0006-2952, 11/2014, Volume 92, Issue 1, pp. 73 - 89
Agonists of the nuclear receptor PPARγ are therapeutically used to combat hyperglycaemia associated with the metabolic syndrome and type 2 diabetes. In spite... 
Natural product | Nuclear receptor | Diabetes | Nutrition | PPAR gamma | INSULIN ACTION ENHANCER | NECROSIS-FACTOR-ALPHA | LOWERS BLOOD-PRESSURE | IMPROVES GLYCEMIC CONTROL | ADIPOCYTE DIFFERENTIATION | PHARMACOLOGY & PHARMACY | RED-CLOVER EXTRACT | GLUCOSE-UPTAKE | NF-KAPPA-B | LIVER-X-RECEPTOR | ADIPOSE-TISSUE | Biological Products - chemistry | Humans | PPAR gamma - agonists | Models, Molecular | Protein Binding | Biological Products - pharmacology | Molecular Structure | PPAR gamma - metabolism | Energy Metabolism - drug effects | DIO, diet-induced obesity | PPAR, peroxisome proliferator-activated receptor | TNF-α, tumor necrosis factor alpha | RXR, retinoid X receptor | CAP, c-Cbl-associated protein | DCM, dichloromethane | NF-κB, nuclear factor-kappaB | 9-(S)-HODE, (9S,10E,12Z)-9-hydroxyoctadeca-10,12-dienoic acid | AF-2, activation function-2 | Part of the Special Issue | MeOH, methanol | LDL, low-density lipoprotein | Cdk5, cyclin-dependent kinase 5 | DPP-4, dipeptidylpeptidase 4 | HUVEC, human umbilical vein endothelial cells | Glut4, glucose transporter type 4 | FDA, Food and Drug Administration | LBD, ligand-binding domain | EMA, European Medicines Agency | TCM, traditional Chinese medicine | PPRE, peroxisome proliferator response element | Metabolism 2014 – Alterations of metabolic pathways as therapeutic targets | PDB, protein data bank | SPPARMs, selective PPARγ modulators | HDL, high-density lipoprotein | MAPK, mitogen-activated protein kinase
Journal Article
Nature, ISSN 0028-0836, 09/2011, Volume 477, Issue 7365, pp. 477 - 481
Journal Article
Journal Article