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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 6/2007, Volume 104, Issue 25, pp. 10589 - 10594
Vessel loss precipitates many diseases. In particular, vessel loss resulting in hypoxia induces retinal neovascularization in diabetic retinopathy and in... 
Diabetic angiopathies | Diabetic retinopathy | Oxygen | Messenger RNA | Blood vessels | Retina | Regrowth | Hypoxia | Mice | Infants | Angiogenesis | Retinopathy of prematurity | Insulin-like growth factor | Binding protein-3 | Insulin-like growth factor 1 | Stem cells | SURVIVAL | stem cells | FACTOR-BINDING PROTEIN-3 | angiogenesis | MULTIDISCIPLINARY SCIENCES | MOUSE | binding protein-3 | DNA-SYNTHESIS | retinopathy of prematurity | BREAST-CANCER | ENDOTHELIAL-CELLS | DISEASE | LINE | insulin-like growth factor | insulin-like growth factor 1 | PREMATURITY | GROWTH-FACTOR-I | Insulin-Like Growth Factor Binding Protein 3 - genetics | Retinopathy of Prematurity - classification | Humans | Insulin-Like Growth Factor Binding Protein 3 - blood | Insulin-Like Growth Factor Binding Protein 3 - deficiency | Insulin-Like Growth Factor Binding Protein 3 - pharmacology | Retinal Neovascularization - metabolism | RNA, Messenger - metabolism | Retinopathy of Prematurity - physiopathology | Dose-Response Relationship, Drug | Retina - cytology | Hyperoxia - complications | Infant, Newborn | Disease Models, Animal | Retinopathy of Prematurity - etiology | Gene Expression Regulation | Mice, Transgenic | Gestational Age | Mice, Knockout | Animals | Infant, Premature | Retinal Neovascularization - chemically induced | Insulin-Like Growth Factor Binding Protein 1 - blood | Retinopathy of Prematurity - metabolism | Retinopathy of Prematurity - pathology | Angiogenesis inhibitors | Research | Retrolental fibroplasia | Binding proteins | Biological Sciences | like growth factor 1 | insulin | like growth factor binding protein | Premature | Disease Models | RNA | Transgenic | Retinal Neovascularization/chemically induced/metabolism | Infant | MEDICIN OCH HÄLSOVETENSKAP | Insulin-Like Growth Factor Binding Protein 1/blood | Prematurity/classification/etiology/metabolism/pathology/physiopathology | Hyperoxia/complications | MEDICAL AND HEALTH SCIENCES | Drug | Insulin-Like Growth Factor Binding Protein | Retinopathy of | Knockout | Messenger/metabolism | Retina/cytology | 3/blood/deficiency/genetics/pharmacology | Newborn | Animal | Dose-Response Relationship
Journal Article
Journal of Pediatrics, The, ISSN 0022-3476, 2015, Volume 167, Issue 5, pp. 982 - 986.e2
Journal Article
Journal Article
Predicting the risk of preterm labor by second trimester measurement and a risk factor scoring system, 12/2005
Background: Preterm labor (delivery after 20 weeks and before 37 weeks of gestation) is the leading cause of prenatal mortality in developed countries. Many... 
α-fetoprotein, prematurity, preterm delivery
Journal
02/2014
Objective: To evaluate early aggressive vs. conservative nutrition and its effect on Retinopathy of Prematurity (ROP) in <32 weeks of gestation neonates.... 
Retinopathy of prematurity | Prematurity | IGFBP3 | Parenteral nutrition | IGF-1
Web Resource
Journal Article
Journal Article
RETINA-THE JOURNAL OF RETINAL AND VITREOUS DISEASES, ISSN 0275-004X, 12/2015, Volume 35, Issue 12, pp. E76 - E76
Journal Article
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 09/2018, Volume 503, Issue 4, pp. 2792 - 2799
Retinopathy of prematurity (ROP) is an eye disease that causes blindness due to delayed vascular growth, retinal ischemia, and resulting abnormal angiogenesis.... 
Oxygen-induced ROP | Pericyte | Retinopathy of prematurity | Propranolol | Vascular permeability | DIABETIC-RETINOPATHY | CELLS | ANGIOGENESIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | PROLIFERATION | VEGF-A | INHIBITION | BIOPHYSICS | BLOCKADE | EXPRESSION | PREMATURITY | ENDOTHELIAL GROWTH-FACTOR | Human Umbilical Vein Endothelial Cells - metabolism | Pericytes - drug effects | Apoptosis - drug effects | Coculture Techniques | Humans | Pericytes - cytology | Capillary Permeability - drug effects | Apoptosis - genetics | Retinal Neovascularization - metabolism | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Retinopathy of Prematurity - genetics | Vascular Endothelial Growth Factor A - genetics | Proto-Oncogene Proteins c-akt - genetics | Retinal Vessels - pathology | Propranolol - pharmacology | Human Umbilical Vein Endothelial Cells - cytology | Hyperoxia - metabolism | Retinal Neovascularization - genetics | Proto-Oncogene Proteins c-akt - metabolism | Disease Models, Animal | Tight Junctions - drug effects | Animals, Newborn | Tight Junctions - metabolism | Human Umbilical Vein Endothelial Cells - drug effects | Vasodilator Agents - pharmacology | Retinal Vessels - metabolism | Signal Transduction | Mice, Inbred C57BL | Pericytes - metabolism | Retinopathy of Prematurity - chemically induced | Gene Expression Regulation | Retinal Vessels - drug effects | Hyperoxia - chemically induced | Hyperoxia - pathology | Tight Junctions - ultrastructure | Oxygen - administration & dosage | Animals | Hyperoxia - genetics | Retinal Neovascularization - chemically induced | Mice | Retinopathy of Prematurity - metabolism | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Retinal Neovascularization - pathology | Retinopathy of Prematurity - pathology | Medical colleges | Endothelial growth factors | Analysis | Blindness | Permeability | Apoptosis | Endothelium
Journal Article