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Journal Article
Circulation, ISSN 0009-7322, 2013, Volume 128, Issue 13, pp. 1420 - 1432
Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 09/2017, Volume 70, Issue 13, pp. 1616 - 1617
The protomeric heptad repeats are followed by multiple epidermal growth factor-like domains, a string of fibronectin type III repeats, and a carboxy-terminal... 
tenascin C | cardiac hypertrophy | cardiac fibrosis | pressure overload | angiotensin II | CARDIAC & CARDIOVASCULAR SYSTEMS | HEART-FAILURE | CARDIOMYOPATHY | Tenascin | Cardiomegaly | Inflammation | Bone Marrow | Humans | Fibrosis | Heart failure | Proteins | Heart attacks | Tenascin C | Cardiomyopathy | Rodents | Bone marrow | Extracellular matrix | Heart diseases | Hypertrophy | Index Medicus | Abridged Index Medicus
Journal Article
Circulation research, ISSN 0009-7330, 02/2015, Volume 116, Issue 5, pp. E28 - E39
Rationale: Sustained activation of Gq transgenic (Gq) signaling during pressure overload causes cardiac hypertrophy that ultimately progresses to dilated... 
Heart failure | Mitochondrial uncoupling protein 3 | Oxidative stress | G-protein | Calcium-calmodulin-dependent protein kinase type 2 | heart failure | OXYGEN SPECIES PRODUCTION | PRESSURE-OVERLOAD | CARDIAC & CARDIOVASCULAR SYSTEMS | UNCOUPLING PROTEINS | HEART-FAILURE | calcium-calmodulin-dependent protein kinase type 2 | KINASE-II | CARDIAC-HYPERTROPHY | MYOCARDIAL HYPERTROPHY | PERIPHERAL VASCULAR DISEASE | MICE | mitochondrial uncoupling protein 3 | PPAR-ALPHA AGONIST | DYSFUNCTION | HEMATOLOGY | oxidative stress | Reactive Oxygen Species | Oxidative Stress | GTP-Binding Protein alpha Subunits, Gq-G11 - deficiency | Heart Failure - physiopathology | Ion Channels - genetics | Male | Gene Expression Profiling | Ion Channels - physiology | Mitochondrial Proteins - genetics | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - deficiency | Cardiomyopathy, Dilated - prevention & control | RNA, Messenger - biosynthesis | Transfection | RNA Interference | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - physiology | Heart Failure - etiology | GTP-Binding Protein alpha Subunits, Gq-G11 - genetics | Ion Channels - biosynthesis | PPAR alpha - biosynthesis | RNA, Messenger - genetics | RNA, Small Interfering - pharmacology | Cells, Cultured | Cardiomegaly - physiopathology | Mitochondrial Proteins - physiology | Rats | Mice, Transgenic | PPAR alpha - genetics | GTP-Binding Protein alpha Subunits, Gq-G11 - physiology | Mitochondrial Proteins - biosynthesis | Cardiomegaly - enzymology | Sulfonamides - pharmacology | Pressure | Disease Progression | Mice, Knockout | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - genetics | Point Mutation | Animals | Sequence Analysis, RNA | Cardiomyopathy, Dilated - etiology | Cardiomyopathy, Dilated - physiopathology | Mitochondria, Heart - physiology | Acetylcysteine - pharmacology | Myocytes, Cardiac - metabolism | Mice | Benzylamines - pharmacology | Uncoupling Protein 3 | Apoptosis | Index Medicus | UCP | calcium | Gq | calmodulin-dependent protein kinase II | peroxisome proliferator-activated receptor alpha and gamma
Journal Article
Circulation, ISSN 0009-7322, 08/2010, Volume 122, Issue 7, pp. 717 - 728
Journal Article
Journal Article
Journal Article
Cardiovascular Research, ISSN 0008-6363, 4/2008, Volume 78, Issue 1, pp. 79 - 89
Aims Peroxisome proliferator-activated receptor-α (PPARα) is a nuclear receptor regulating cardiac metabolism that also has anti-inflammatory properties. Since... 
Inflammation | Pressure-overload | Metabolism | PPARα | Fibrosis | Hypertrophy | TRANSCRIPTION FACTORS | CARDIAC & CARDIOVASCULAR SYSTEMS | MYOCARDIAL-INFARCTION | hypertrophy | PROLIFERATOR-ACTIVATED RECEPTORS | NUCLEAR RECEPTORS | PPAR alpha | FIBROBLAST PROLIFERATION | ALPHA PPAR-ALPHA | pressure-overload | inflammation | IN-VIVO | metabolism | fibrosis | TNF-ALPHA | NF-KAPPA-B | FATTY-ACID HOMEOSTASIS | Tumor Necrosis Factor-alpha - metabolism | Ventricular Function, Left | 3-Hydroxyacyl CoA Dehydrogenases - metabolism | Actins - metabolism | Male | RNA, Messenger - metabolism | Coenzyme A Ligases - metabolism | Hypertrophy, Left Ventricular - genetics | Ligation | Myocardium - metabolism | Ultrasonography | Ventricular Remodeling - genetics | Interleukin-6 - metabolism | Disease Models, Animal | Myocardial Contraction | Collagen Type I - metabolism | Atrial Natriuretic Factor - metabolism | Matrix Metalloproteinase 2 - metabolism | Hypertrophy, Left Ventricular - metabolism | Mice, Inbred C57BL | Myocardium - pathology | PPAR alpha - genetics | Mice, Knockout | Stroke Volume | Myocardium - enzymology | PPAR alpha - deficiency | Animals | Aorta, Thoracic - surgery | Cyclooxygenase 2 - metabolism | Mice | Hypertrophy, Left Ventricular - physiopathology | PPAR alpha - metabolism | Hypertrophy, Left Ventricular - diagnostic imaging | Index Medicus
Journal Article
Circulation, ISSN 0009-7322, 09/2010, Volume 122, Issue 11, pp. S23 - S28
Journal Article
Journal Article
Nature Medicine, ISSN 1078-8956, 10/2016, Volume 22, Issue 10, pp. 1131 - 1139
Epigenetic reprogramming is a critical process of pathological gene induction during cardiac hypertrophy and remodeling, but the underlying regulatory... 
MEDICINE, RESEARCH & EXPERIMENTAL | CARDIOMYOCYTE HYPERTROPHY | PRESSURE-OVERLOAD | PATHOLOGICAL HYPERTROPHY | BIOCHEMISTRY & MOLECULAR BIOLOGY | HEART-FAILURE | HISTONE METHYLATION | MTOR | CELL BIOLOGY | RAPTOR | GENE-EXPRESSION | BINDING | EZH2 | TOR Serine-Threonine Kinases - metabolism | Humans | Immunoblotting | Gene Expression Profiling | Gene Knockdown Techniques | Mechanistic Target of Rapamycin Complex 1 | Multiprotein Complexes - metabolism | Chromatin Immunoprecipitation | Computer Simulation | Myocardium - metabolism | Real-Time Polymerase Chain Reaction | Echocardiography | Rats | In Situ Hybridization, Fluorescence | RNA, Long Noncoding - genetics | Reverse Transcriptase Polymerase Chain Reaction | Heart - diagnostic imaging | Mice, Knockout | Blotting, Northern | Animals | Epigenesis, Genetic - genetics | Myocytes, Cardiac - metabolism | Mice | Cardiomegaly - genetics | In Vitro Techniques | Methylation | Polycomb Repressive Complex 2 - metabolism | Induced Pluripotent Stem Cells | Histone Code - genetics | Cardiomegaly - metabolism | Genetic aspects | Research | RNA | Heart enlargement | TOR protein | Heart | Chromatin | Genes | Rapamycin | Gene expression | Ribonucleic acid--RNA | Polycomb group proteins | Lysine | Regulatory mechanisms (biology) | DNA methylation | Epigenetics | Catalysis | Inhibition | Histone H3 | Hypertrophy | Index Medicus
Journal Article