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eLife, ISSN 2050-084X, 09/2014, Volume 3, p. e03696
Journal Article
eLife, ISSN 2050-084X, 03/2015, Volume 2015, Issue 4, pp. 1 - 25
Recent breakthroughs in 3-dimensional (3D) organoid cultures for many organ systems have led to new physiologically complex in vitro models to study human... 
EXPRESSION PATTERNS | PROGENITOR CELLS | HUMAN AIRWAY EPITHELIUM | VENTRAL FOREGUT | BIOLOGY | BRANCHING MORPHOGENESIS | DEFINITIVE ENDODERM | EXTRACELLULAR-MATRIX | MOUSE LUNG | CLUSTER-ANALYSIS | BASAL-CELLS | Embryonic Stem Cells - metabolism | Embryonic Stem Cells - cytology | Humans | Gene Expression Profiling | Spheroids, Cellular - cytology | Lung - cytology | Cell Culture Techniques - methods | Cell Differentiation - genetics | Endoderm - cytology | Organoids - metabolism | Organoids - ultrastructure | Lung - metabolism | Induced Pluripotent Stem Cells - cytology | Induced Pluripotent Stem Cells - metabolism | Cell Line | Microscopy, Electron, Transmission | Reproducibility of Results | Pluripotent Stem Cells - cytology | Tissue Engineering - methods | Spheroids, Cellular - metabolism | Cells, Cultured | Organoids - cytology | Reverse Transcriptase Polymerase Chain Reaction | Endoderm - metabolism | Organogenesis | Pluripotent Stem Cells - metabolism | Microscopy, Confocal | Lung - embryology | Cluster analysis | Mesenchyme | Transcription | Developmental biology | Lung diseases | Fetuses | Embryo cells | Basal cells | Principal components analysis | Smooth muscle | Genomes | Epithelium | Ribonucleic acid--RNA | Spheroids | Children & youth | Respiratory tract | Hospitals | Organoids | Stem cells | Alveoli | Pluripotency | Foregut
Journal Article
Nature, ISSN 0028-0836, 10/2015, Volume 526, Issue 7571, pp. 131 - 135
Despite major advances in understanding the molecular and genetic basis of cancer, metastasis remains the cause of >90% of cancer-related mortality(1).... 
INHIBITION | COMMITMENT | MULTIDISCIPLINARY SCIENCES | GROWTH | GENES | MARKERS | IDENTIFICATION | FATE | TUMORS | TRANSCRIPTOME ANALYSIS | MAMMARY-GLAND | Neoplastic Stem Cells - drug effects | Epithelial Cells - drug effects | Humans | Gene Expression Profiling | Epithelial-Mesenchymal Transition - genetics | Cell Differentiation - genetics | Flow Cytometry | Neoplasm Metastasis - drug therapy | Neoplastic Stem Cells - metabolism | Neoplastic Stem Cells - pathology | Cyclin-Dependent Kinases - antagonists & inhibitors | Single-Cell Analysis | Disease Models, Animal | Cell Separation | Epithelial Cells - pathology | Mice, SCID | Breast Neoplasms - drug therapy | Disease Progression | Xenograft Model Antitumor Assays | Animals | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Cell Differentiation - drug effects | Neoplasm Metastasis - pathology | Genes, myc - genetics | Cell Line, Tumor | Mice, Inbred NOD | Cell Proliferation - drug effects | Mesoderm - metabolism | Mice | Cell Transformation, Neoplastic - drug effects | Cell Cycle - drug effects | Cell Transformation, Neoplastic - pathology | Mesoderm - pathology | Stem cells | Cancer cells | Development and progression | Breast cancer | Metastasis | Observations | Health aspects | Bone marrow | Principal components analysis | Gene expression | Tumors | Apoptosis
Journal Article
Immunity, ISSN 1074-7613, 2012, Volume 36, Issue 1, pp. 142 - 152
Cytotoxic CD8 T lymphocytes directly kill infected or aberrant cells and secrete proinflammatory cytokines. By using metal-labeled probes and mass... 
IMMUNE | RESPONSES | EFFECTOR FUNCTIONS | MEMORY | CLASS-I LIGANDS | SUBSETS | PEPTIDE COMPLEXES | IMMUNOLOGY | MHC TETRAMERS | ANTIGEN | LYMPHOCYTES | Antigens | Flow cytometry | Peptides | Cytokines | Cytotoxicity | Principal components analysis | Software | Viral infections
Journal Article
Molecular Systems Biology, ISSN 1744-4292, 03/2015, Volume 11, Issue 3, pp. 790 - n/a
Journal Article
PLoS ONE, ISSN 1932-6203, 2010, Volume 5, Issue 5, p. e10431
Prostate epithelial cells from both normal and cancer tissues, grown in three-dimensional (3D) culture as spheroids, represent promising in vitro models for... 
EPITHELIAL-MESENCHYMAL TRANSITION | BREAST-CANCER | GENE-EXPRESSION SIGNATURE | STEM-CELLS | MAMMARY EPITHELIA | METASTASIS | BIOLOGY | TUMOR-CELL INVASION | DIFFERENTIATION | CULTURE MODEL | LINES | Laminin - pharmacology | Epithelial Cells - drug effects | Humans | Mesoderm - drug effects | Spheroids, Cellular - pathology | Male | Antineoplastic Agents - therapeutic use | Phosphatidylinositol 3-Kinases - metabolism | Spheroids, Cellular - enzymology | Neoplasm Proteins - metabolism | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | RNA, Messenger - metabolism | Prostate - pathology | Prostatic Neoplasms - genetics | Cell Transformation, Neoplastic - genetics | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Prostate - drug effects | Antineoplastic Agents - pharmacology | Collagen - pharmacology | Spheroids, Cellular - drug effects | Gene Expression Regulation, Neoplastic - drug effects | Tumor Cells, Cultured | Proto-Oncogene Proteins c-akt - metabolism | Principal Component Analysis | Prostatic Neoplasms - drug therapy | Epithelium - drug effects | Prostatic Neoplasms - pathology | Epithelium - pathology | Neoplasm Invasiveness | Intracellular Signaling Peptides and Proteins - antagonists & inhibitors | RNA, Messenger - genetics | Epithelial Cells - pathology | Cell Shape - drug effects | Phenotype | Proteoglycans - pharmacology | Signal Transduction - drug effects | Models, Biological | Prostatic Neoplasms - enzymology | Cell Proliferation - drug effects | TOR Serine-Threonine Kinases | Cell Transformation, Neoplastic - pathology | Mesoderm - pathology | Drug Combinations | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Epigenetic inheritance | Growth | Oncology, Experimental | Genes | Research | Gene expression | Ionizing radiation | Stem cells | Physiological aspects | Models | Drug discovery | Drug therapy | Prostate cancer | Integrins | Cancer | Cell culture | Biotechnology | Transformation | Motility | Leukocyte migration | Mesenchyme | Epithelial cells | Homeostasis | AKT protein | Metastasis | Drug resistance | Tissues | Ovarian cancer | Cell adhesion & migration | Metastases | Rodents | Fibroblasts | Extracellular matrix | Basal lamina | Lipid metabolism | Medical research | Invasiveness | Phenotypic plasticity | Cultures | Tumor cell lines | Metabolism | Spheroids | 1-Phosphatidylinositol 3-kinase | Signaling | Interferon | Three dimensional models | Prostate | Cell migration
Journal Article
Stem Cells: the international journal of cell differentiation and proliferation, ISSN 1066-5099, 03/2017, Volume 35, Issue 3, pp. 611 - 625
textabstractIn human embryonic stem cells (ESCs) the transcription factor Zeb2 regulates neuroectoderm versus mesendoderm formation, but it is unclear how Zeb2... 
Transcription factors | Repressors | Transcriptom | DNA‐methylation | Cell differentiation | Embryonic stem cells | Pluripotent stem cells | RNA‐sequencing | RNA-sequencing | DNA-methylation | NERVOUS-SYSTEM | MENTAL-RETARDATION | GROUND-STATE | SMAD-INTERACTING PROTEIN-1 | SELF-RENEWAL | PLURIPOTENCY | CELL & TISSUE ENGINEERING | CELL BIOLOGY | SIP1 | ONCOLOGY | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | GERM-CELLS | DNA | HIRSCHSPRUNG-DISEASE | HEMATOLOGY | Proto-Oncogene Proteins - metabolism | Pluripotent Stem Cells - cytology | Germ Layers - metabolism | Mouse Embryonic Stem Cells - cytology | Neurons - cytology | DNA Methylation - genetics | Down-Regulation - genetics | Mouse Embryonic Stem Cells - metabolism | Mice, Knockout | DNA-Binding Proteins - metabolism | Embryoid Bodies - metabolism | Pluripotent Stem Cells - metabolism | Cell Lineage | Phenotype | Animals | Embryoid Bodies - cytology | Sequence Analysis, RNA | Zinc Finger E-box Binding Homeobox 2 - metabolism | Transcription, Genetic | Cell Differentiation | Mice | Germ Layers - cytology | Principal Component Analysis | Repressor Proteins - metabolism | Sulfites | RNA | Analysis | Genetic transcription | DNA binding proteins | Methylation | Correlation | Deregulation | Bisulfite | Mesenchyme | Genes | Embryo cells | Stem cell transplantation | Decisions | Ribonucleic acid--RNA | Embryos | Gene sequencing | Cell fate | Neuroectoderm | Stem cells | DNA methylation | Links | Differentiation | Mesendoderm | Pluripotency | Binding sites | Deoxyribonucleic acid--DNA | Embryonic Stem Cells | Induced Pluripotent Stem Cells
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2016, Volume 11, Issue 11, p. e0166316
Journal Article