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Journal Article
The Journal of Pathology, ISSN 0022-3417, 08/2011, Volume 224, Issue 4, pp. 553 - 563
IL‐32 is a described pro‐inflammatory cytokine produced by T lymphocytes, natural killer cells, monocytes, and epithelial cells. However, the specific... 
allergic rhinitis | inflammatory cytokines | caspase‐1 | interleukin‐32 | granulocyte‐macrophage colony‐stimulating factor | granulocyte-macrophage colony-stimulating factor | caspase-1 | interleukin-32 | RHEUMATOID-ARTHRITIS | ACTIVATION | COLONY-STIMULATING FACTOR | INNATE | PATHOLOGY | PROINFLAMMATORY CYTOKINE | ONCOLOGY | GM-CSF | EOSINOPHIL | UPPER AIRWAYS | T-CELLS | Humans | Immunoglobulin E - biosynthesis | Middle Aged | Child, Preschool | Caspase 1 - metabolism | Male | Enzyme Activation - immunology | Interleukins - immunology | Reverse Transcriptase Polymerase Chain Reaction - methods | Inflammation Mediators - metabolism | Adenoids - immunology | Adult | Female | Interleukins - biosynthesis | Tumor Cells, Cultured | Child | Disease Models, Animal | Rhinitis - immunology | Interleukins - blood | Respiratory Hypersensitivity - immunology | Animals | Granulocyte-Macrophage Colony-Stimulating Factor - immunology | Up-Regulation - immunology | Nasal Mucosa - immunology | Adolescent | Mice | Mice, Inbred BALB C | Hypereosinophilic Syndrome - immunology | Cytokines - biosynthesis | Animal models | Epithelial cells | Granulocyte-macrophage colony-stimulating factor | Leukocytes (eosinophilic) | Mucosa | Immunoglobulin E | Inflammation | Lymphocytes T | mRNA | Caspase-1 | Interleukin 18 | Cell activation | Monocytes | Allergic rhinitis | Natural killer cells
Journal Article
Journal of Molecular Medicine, ISSN 0946-2716, 6/2011, Volume 89, Issue 6, pp. 577 - 593
The environmental, genetic, and/or age-related changes in proteostasis induce inflammation, oxidative stress, and apoptosis. We quantified the correlation of... 
Human Genetics | VCP | HDAC2 | Biomedicine | NFκB | Nrf2 | Internal Medicine | Molecular Medicine | Proteasome | MEDICINE, RESEARCH & EXPERIMENTAL | NRF2 ENHANCES SUSCEPTIBILITY | RETICULUM-ASSOCIATED DEGRADATION | TRANSMEMBRANE CONDUCTANCE REGULATOR | OBSTRUCTIVE PULMONARY-DISEASE | INDUCED LUNG INFLAMMATION | REDOX REGULATION | AAA ATPASE P97/VCP | GENETICS & HEREDITY | PROINFLAMMATORY CYTOKINE RELEASE | CIGARETTE-SMOKE | NF-KAPPA-B | NF kappa B | Proteostasis Deficiencies - metabolism | Thiourea - therapeutic use | Oxidative Stress | Humans | Middle Aged | Valosin Containing Protein | Male | NF-kappa B - metabolism | Pulmonary Emphysema - metabolism | Pulmonary Disease, Chronic Obstructive - pathology | Thiourea - pharmacology | Receptors, Autocrine Motility Factor | Adenosine Triphosphatases - biosynthesis | HEK293 Cells | Cell Cycle Proteins - genetics | Ubiquitin Thiolesterase - metabolism | Female | Pulmonary Disease, Chronic Obstructive - metabolism | Transcription Factor CHOP - biosynthesis | Bronchi - cytology | Receptors, Cytokine - genetics | Endopeptidases - metabolism | Receptors, Cytokine - biosynthesis | Proteostasis Deficiencies - pathology | Pulmonary Emphysema - pathology | Cell Cycle Proteins - biosynthesis | Epithelial Cells | Ubiquitinated Proteins - analysis | Cinnamates - pharmacology | Ubiquitin-Protein Ligases - biosynthesis | Animals | Proteins - metabolism | NF-E2-Related Factor 2 - metabolism | Adenosine Triphosphatases - genetics | Aged | Mice | Histone Deacetylase 2 - metabolism | Ubiquitin-Protein Ligases - genetics | Cinnamates - therapeutic use | Thiourea - analogs & derivatives | Apoptosis | Smoking | Lung diseases, Obstructive | Emphysema, Pulmonary | Physiological aspects | Development and progression | Genetic aspects | Research | Gene expression
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 08/2006, Volume 203, Issue 8, pp. 1883 - 1889
Journal Article
Journal Article
European Journal of Immunology, ISSN 0014-2980, 12/2005, Volume 35, Issue 12, pp. 3405 - 3413
The pro‐inflammatory cytokine macrophage migration inhibitory factor (MIF) acts as a physiological counter‐regulator of the immuno‐suppressive effects of... 
Macrophage migration inhibitory factor | Innate immunity | Mitogen‐activated protein kinase phosphatase‐1 | Glucocorticoids | Macrophages | Mitogen-activated protein kinase phosphatase-1 | mitogen-activated protein kinase phosphatase-1 | IMMEDIATE-EARLY GENE | MAP KINASE | FACTOR MIF | KAPPA-B-ALPHA | IMMUNOLOGY | TUMOR-NECROSIS-FACTOR | TLR4 GENE | SEPTIC SHOCK | STIMULATED MACROPHAGES | migration inhibitory factor macrophages | glucocorticoids | REGULATORY ROLE | PROINFLAMMATORY CYTOKINE BIOSYNTHESIS | innate immunity. macrophage | Dual Specificity Phosphatase 1 | Humans | Cell Cycle Proteins - antagonists & inhibitors | Protein Tyrosine Phosphatases - antagonists & inhibitors | Protein Tyrosine Phosphatases - genetics | Phosphoprotein Phosphatases - biosynthesis | U937 Cells | Cell Cycle Proteins - genetics | Glucocorticoids - physiology | Protein Phosphatase 1 | Cell Line | Protein Tyrosine Phosphatases - biosynthesis | Immediate-Early Proteins - biosynthesis | Cell Cycle Proteins - biosynthesis | Phosphoprotein Phosphatases - antagonists & inhibitors | Immunity, Innate | Animals | Immediate-Early Proteins - genetics | Phosphoprotein Phosphatases - genetics | Glucocorticoids - antagonists & inhibitors | Macrophage Migration-Inhibitory Factors - physiology | Mice | Cytokines - antagonists & inhibitors | Cytokines - biosynthesis | Immediate-Early Proteins - antagonists & inhibitors
Journal Article
Clinical & Experimental Immunology, ISSN 0009-9104, 12/2013, Volume 174, Issue 3, pp. 459 - 471
Summary Numerous reports have shown that a diet containing large amounts of trans fatty acids (TFAs) is a major risk factor for metabolic disorders. Although... 
macrophages | proinflammatory cytokines | trans fatty acids | inflammatory bowel disease | Inflammatory bowel disease | Proinflammatory cytokines | Macrophages | METABOLIC SYNDROME | EUROPEAN PROSPECTIVE COHORT | CROHNS-DISEASE | LINOLEIC-ACID | ULCERATIVE-COLITIS | IMMUNOLOGY | DIETARY-INTAKE | SYSTEMIC INFLAMMATION | INFLAMMATORY-BOWEL-DISEASE | DSS-INDUCED COLITIS | MICE | Inflammation - chemically induced | Dextran Sulfate | Interleukin-12 Subunit p40 - genetics | Up-Regulation | Oleic Acid | Lipopolysaccharides | RNA, Messenger - biosynthesis | Interleukin-23 Subunit p19 - biosynthesis | Th17 Cells - metabolism | Colitis - chemically induced | Interleukin-23 Subunit p19 - genetics | Female | Colitis - immunology | Cytokines - genetics | Interleukin-12 Subunit p40 - biosynthesis | Nuclear Receptor Subfamily 1, Group F, Member 3 - genetics | Cell Line | Interleukin-6 - genetics | Mice, Inbred C57BL | Interleukin-17 - genetics | Inflammation - immunology | Interleukin-17 - biosynthesis | Cell Differentiation - immunology | Macrophages - metabolism | Animals | Trans Fatty Acids | Interleukin-6 - biosynthesis | Nuclear Receptor Subfamily 1, Group F, Member 3 - biosynthesis | Th17 Cells - immunology | Linoleic Acid | Mice | Cytokines - biosynthesis | Dextran | Cytokines | RNA | Gastrointestinal diseases | Colitis | Mitogens | Trans fatty acids | Original
Journal Article