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Bioimpacts, ISSN 2228-5660, 12/2018, Volume 8, Issue Suppl 1, pp. S1 - S129
Journal Article
PloS one, ISSN 1932-6203, 2010, Volume 5, Issue 5, p. e10431
Prostate epithelial cells from both normal and cancer tissues, grown in three-dimensional (3D... 
EPITHELIAL-MESENCHYMAL TRANSITION | BREAST-CANCER | GENE-EXPRESSION SIGNATURE | STEM-CELLS | MAMMARY EPITHELIA | METASTASIS | BIOLOGY | TUMOR-CELL INVASION | DIFFERENTIATION | CULTURE MODEL | LINES | Laminin - pharmacology | Epithelial Cells - drug effects | Humans | Mesoderm - drug effects | Spheroids, Cellular - pathology | Male | Antineoplastic Agents - therapeutic use | Phosphatidylinositol 3-Kinases - metabolism | Spheroids, Cellular - enzymology | Neoplasm Proteins - metabolism | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | RNA, Messenger - metabolism | Prostate - pathology | Prostatic Neoplasms - genetics | Cell Transformation, Neoplastic - genetics | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Prostate - drug effects | Antineoplastic Agents - pharmacology | Collagen - pharmacology | Spheroids, Cellular - drug effects | Gene Expression Regulation, Neoplastic - drug effects | Tumor Cells, Cultured | Proto-Oncogene Proteins c-akt - metabolism | Principal Component Analysis | Prostatic Neoplasms - drug therapy | Epithelium - drug effects | Prostatic Neoplasms - pathology | Epithelium - pathology | Neoplasm Invasiveness | Intracellular Signaling Peptides and Proteins - antagonists & inhibitors | RNA, Messenger - genetics | Epithelial Cells - pathology | Cell Shape - drug effects | Phenotype | Proteoglycans - pharmacology | Signal Transduction - drug effects | Models, Biological | Prostatic Neoplasms - enzymology | Cell Proliferation - drug effects | TOR Serine-Threonine Kinases | Cell Transformation, Neoplastic - pathology | Mesoderm - pathology | Drug Combinations | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Epigenetic inheritance | Growth | Oncology, Experimental | Genes | Research | Gene expression | Ionizing radiation | Stem cells | Physiological aspects | Models | Drug discovery | Drug therapy | Prostate cancer | Integrins | Cancer | Cell culture | Biotechnology | Transformation | Motility | Leukocyte migration | Mesenchyme | Epithelial cells | Homeostasis | AKT protein | Metastasis | Drug resistance | Tissues | Ovarian cancer | Cell adhesion & migration | Metastases | Rodents | Fibroblasts | Extracellular matrix | Basal lamina | Lipid metabolism | Medical research | Invasiveness | Phenotypic plasticity | Tumor cell lines | Metabolism | Spheroids | 1-Phosphatidylinositol 3-kinase | Signaling | Interferon | Three dimensional models | Prostate | Cell migration
Journal Article
The Journal of experimental medicine, ISSN 1540-9538, 2011, Volume 208, Issue 10, pp. 2005 - 2016
Despite lack of tumor control in many models, spontaneous T cell priming occurs frequently in response to a growing tumor... 
MEDICINE, RESEARCH & EXPERIMENTAL | METASTATIC MELANOMA | INTERFERON-ALPHA | DYING CELLS | COLONY-STIMULATING FACTOR | TUMOR-ANTIGENS | REJECTION | PROSTATE-CANCER | INNATE IMMUNE-RESPONSE | RECEPTOR | IMMUNOLOGY | LYMPHOCYTES-T
Journal Article
PloS one, ISSN 1932-6203, 2014, Volume 9, Issue 1, p. e84941
Cells with sphere forming capacity, spheroid cells, are present in the malignant ascites of patients with epithelial ovarian cancer (EOC... 
INITIATING CELLS | PROGENITOR CELLS | CARCINOMA ASCITES SPHEROIDS | INVASION | PROSPECTIVE IDENTIFICATION | MULTIDISCIPLINARY SCIENCES | IN-VITRO MODEL | PROSTATE-CANCER | GROWTH | GENE-EXPRESSION | MASS ISOTOPOMER | Cell Proliferation | Neoplastic Stem Cells - drug effects | Humans | Ovarian Neoplasms - pathology | Drug Resistance, Neoplasm | Antineoplastic Agents - therapeutic use | Cell Movement - genetics | Hypoxia - metabolism | Ovarian Neoplasms - genetics | Neoplasm Metastasis | Neoplastic Stem Cells - metabolism | Neoplastic Stem Cells - pathology | Female | Tumor Burden - genetics | Antineoplastic Agents - pharmacology | Ovarian Neoplasms - metabolism | Tumor Cells, Cultured | Spheroids, Cellular | Aldehyde Dehydrogenase - metabolism | Ovarian Neoplasms - drug therapy | Disease Models, Animal | Cell Transformation, Neoplastic - metabolism | Xenograft Model Antitumor Assays | Cell Movement - drug effects | Animals | Tumor Burden - drug effects | Cell Line, Tumor | Immunocompromised Host | Tumor Burden - immunology | Glucose metabolism | Chemotherapy | Stem cells | Development and progression | Transplantation | Metastasis | Drug resistance | Health aspects | Ovarian cancer | Cancer | Cell culture | Ovarian carcinoma | Stem cell transplantation | Oncology | Glucose | Aldehyde dehydrogenase | Metastases | Allografts | Immunology | Immunotherapy | Pentose | Surgery | Tumorigenesis | Growth factors | Tumor cells | Routing | Forming | Tumor cell lines | Metabolism | Gene expression | Spheroids | Glycolysis | Hypoxia | Ascites | Cell migration | Apoptosis | Tumors
Journal Article
Nature (London), ISSN 1476-4687, 2015, Volume 521, Issue 7550, pp. 94 - 98
Cancer-associated genetic alterations induce expression of tumour antigens that can activate CD8(+) cytotoxic T cells (CTLs... 
IMMUNITY | METASTASIS | THERAPY | TRANSGENIC MOUSE | MULTIDISCIPLINARY SCIENCES | MICE | INFILTRATING B-CELLS | RESISTANT PROSTATE-CANCER | GENE-EXPRESSION ANALYSIS | TARGETED DELETION | PROGRESSION | Humans | Organoplatinum Compounds - immunology | Male | Adoptive Transfer | Organoplatinum Compounds - pharmacology | Prostatic Neoplasms - immunology | Plasma Cells - drug effects | T-Lymphocytes, Cytotoxic - drug effects | Organoplatinum Compounds - administration & dosage | I-kappa B Kinase - metabolism | Neoplastic Stem Cells - pathology | Antineoplastic Agents - pharmacology | Prostatic Neoplasms - drug therapy | T-Lymphocytes, Cytotoxic - immunology | Transforming Growth Factor beta - immunology | Prostatic Neoplasms - pathology | Antibodies, Neoplasm - immunology | Signal Transduction | Antineoplastic Agents - immunology | Mice, Inbred C57BL | Cells, Cultured | B7-H1 Antigen - metabolism | Animals | Receptors, Transforming Growth Factor beta - metabolism | Lymphocyte Activation - drug effects | Chemokine CXCL13 - metabolism | Immunoglobulin A - immunology | Mice | Interleukin-10 - immunology | Organoplatinum Compounds - therapeutic use | T-Lymphocytes, Cytotoxic - cytology | Plasma Cells - cytology | Plasma Cells - immunology | Chemotherapy | Immune response | Plasma cells | T cells | Health aspects | Identification and classification | Methods | Cancer | Immunoglobulins | Phosphorylation | Lymphocytes | DNA damage | Metastasis | Cancer therapies | Prostate cancer | Immune system | Tumors | Skin cancer
Journal Article
PloS one, ISSN 1932-6203, 2014, Volume 9, Issue 9, p. e108925
...) progression is poorly understood. In this study, we investigated the ability of prostate tumor-derived exosomes to downregulate NKG2D expression on natural killer (NK) and CD8(+) T cells... 
IMMUNORECEPTOR | APOPTOSIS | CANCER-PATIENTS | LIGANDS | VESICLES | MULTIDISCIPLINARY SCIENCES | MICROVESICLES | RECEPTOR | IMMUNOSURVEILLANCE | RELEASE | CYTOTOXICITY | Exosomes - metabolism | Cell Line | Prostatic Neoplasms - metabolism | Prostatic Neoplasms - pathology | NK Cell Lectin-Like Receptor Subfamily K - chemistry | Antibodies, Monoclonal - pharmacology | Humans | Histocompatibility Antigens Class I - immunology | Intracellular Signaling Peptides and Proteins - immunology | Male | Intracellular Signaling Peptides and Proteins - metabolism | Down-Regulation - drug effects | Histocompatibility Antigens Class I - metabolism | Castration | NK Cell Lectin-Like Receptor Subfamily K - metabolism | Leukocytes, Mononuclear - immunology | Immune Evasion - drug effects | K562 Cells | CD8-Positive T-Lymphocytes - metabolism | Killer Cells, Natural - immunology | Leukocytes, Mononuclear - cytology | Killer Cells, Natural - metabolism | CD8-Positive T-Lymphocytes - immunology | Antibodies, Monoclonal - immunology | Plasma | Flow cytometry | Regulators | Toxicity | CD8 antigen | Cytotoxicity | Lymphocytes T | Exosomes | Cancer therapies | Proteins | Killer cells | Immunology | Lymphocytes | Rodents | Natural killer cells | Incubation | T cell receptors | Patients | Pathology | Cytometry | Surveillance | Pheochromocytoma cells | Ligands | Aberration | Prostate cancer | Prostate | Cancer | Apoptosis | Tumors
Journal Article
PloS one, ISSN 1932-6203, 2013, Volume 8, Issue 6, p. e67466
Background: Epithelial cell adhesion molecule (EpCAM)-based enumeration of circulating tumor cells (CTC... 
BREAST-CANCER | SURVIVAL | IMPACT | MULTIDISCIPLINARY SCIENCES | PERIPHERAL-BLOOD | PROGRESSION | CLINICAL-SIGNIFICANCE | Prognosis | Prospective Studies | Neoplastic Cells, Circulating - pathology | Cell Count | Humans | Lung Neoplasms - metabolism | Middle Aged | Neoplastic Cells, Circulating - metabolism | Lung Neoplasms - pathology | Male | Tissue Array Analysis - methods | Small Cell Lung Carcinoma - blood | Epithelial Cell Adhesion Molecule | Small Cell Lung Carcinoma - metabolism | Aged, 80 and over | Antigens, Neoplasm - metabolism | Adult | Female | Carcinoma, Non-Small-Cell Lung - pathology | Cell Adhesion Molecules - metabolism | Small Cell Lung Carcinoma - pathology | Cell Line, Tumor | Carcinoma, Non-Small-Cell Lung - blood | Aged | Lung Neoplasms - blood | Medical research | Care and treatment | Medicine, Experimental | Metastasis | Comparative analysis | Lung cancer, Non-small cell | Prostate cancer | Biotechnology | Epithelial cells | Lung cancer | Oncology | Statistical methods | Metastases | Cell adhesion molecules | Engineering | Colon cancer | Cell adhesion | Peripheral blood | Clusters | Life sciences | Colon | Enumeration | Statistical analysis | Research & development--R&D | Tumor cells | Non-small cell lung carcinoma | Breast cancer | Statistics | Patients | Chemotherapy | Solid tumors | Median (statistics) | Prostate | Tumors | Cancer | Research & development | R&D
Journal Article
PloS one, ISSN 1932-6203, 2013, Volume 8, Issue 1, p. e53701
.... The aberrant activity of stem cell pathways, and their regulation by the Androgen Receptor (AR), has the potential to provide insight into novel mechanisms and pathways to prevent and treat advanced, castrate-resistant prostate cancers... 
IN-VITRO | STEM-LIKE CELLS | MECHANISM | MULTIDISCIPLINARY SCIENCES | GROWTH | SELF-RENEWAL | NANOG | DIFFERENTIATION | IDENTIFICATION | EXPRESSION | CARCINOMA | Embryonic Stem Cells - metabolism | Epithelial Cells - metabolism | Epithelial Cells - drug effects | Humans | Receptors, Androgen - metabolism | Male | RNA, Messenger - metabolism | Prostate - metabolism | SOXB1 Transcription Factors - metabolism | Prostate - pathology | Neoplasm Metastasis | Prostatic Neoplasms - genetics | SOXB1 Transcription Factors - genetics | Prostate - drug effects | Gene Expression Regulation, Neoplastic - drug effects | Repressor Proteins - metabolism | Phenylthiohydantoin - pharmacology | Prostatic Neoplasms - pathology | Tumor Suppressor Proteins - metabolism | Prostatic Neoplasms - surgery | RNA, Messenger - genetics | Androgen Antagonists - pharmacology | Repressor Proteins - genetics | Epithelial Cells - pathology | Signal Transduction - genetics | Phenylthiohydantoin - analogs & derivatives | Orchiectomy | Transcription Factors - metabolism | Animals | Embryonic Stem Cells - drug effects | Signal Transduction - drug effects | Mice, Nude | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | Drug Resistance, Neoplasm - drug effects | Androgens | Chromatin | Genetic aspects | Fibroblast growth factors | Embryonic stem cells | Prostate cancer | Cancer | Fibroblast growth factor | Epithelial cells | Embryo cells | Tumor cell lines | Kinases | Embryos | Metastases | Fibroblast growth factor 5 | Signal transduction | Signaling | Pathways | Castration | Medical prognosis | Stem cells | Aberration | Prostate | Tumors
Journal Article