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The New England journal of medicine, ISSN 1533-4406, 2012, Volume 366, Issue 22, pp. 2055 - 2064
Journal Article
Pediatric research, ISSN 1530-0447, 2015, Volume 79, Issue 1, pp. 81 - 86
BACKGROUND: The early diagnosis of inherited thrombophilia in children is challenging because of the rarity and hemostatic maturation. METHODS: We explored... 
VENOUS THROMBOEMBOLISM | LABORATORY CHARACTERISTICS | PLASMA-LEVELS | THROMBOSIS | INHERITED THROMBOPHILIA | ANTITHROMBIN | PEDIATRICS | PROTEIN-S DEFICIENCY | C-DEFICIENCY | GENOME-WIDE ASSOCIATION | CHILDREN | Activated Protein C Resistance - epidemiology | Japan - epidemiology | Protein S - analysis | Humans | Child, Preschool | Infant | Male | Antithrombin III Deficiency - genetics | Thromboembolism - epidemiology | Antithrombin III Deficiency - blood | Thrombophilia - epidemiology | Promoter Regions, Genetic - genetics | Antithrombin III - genetics | Protein S Deficiency - genetics | Thrombophilia - diagnosis | Protein C Deficiency - epidemiology | DNA Mutational Analysis | Activated Protein C Resistance - blood | Female | Cerebrovascular Disorders - epidemiology | Activated Protein C Resistance - genetics | Child | Antithrombin III Deficiency - diagnosis | Protein C Deficiency - blood | Thrombophilia - genetics | Protein S - genetics | Antithrombin III Deficiency - epidemiology | Antithrombin III - analysis | Genotype | Protein C - analysis | Thromboembolism - etiology | Cerebrovascular Disorders - etiology | Activated Protein C Resistance - diagnosis | Thrombophilia - blood | Adolescent | Age of Onset | Protein C Deficiency - diagnosis | Protein S Deficiency - epidemiology | Prothrombin - genetics | Factor V - genetics | Protein C Deficiency - genetics | Protein S Deficiency - blood | Protein C - genetics | Protein S Deficiency - diagnosis
Journal Article
Journal Article
Journal Article
Journal Article
PloS one, ISSN 1932-6203, 08/2016, Volume 11, Issue 8, p. e0160382
Journal Article
PloS one, ISSN 1932-6203, 04/2015, Volume 10, Issue 4, p. e0122410
Journal Article
Journal Article
PLoS pathogens, ISSN 1553-7374, 2018, Volume 14, Issue 4, p. e1006981
Purpura fulminans is a deadly complication of Neisseria meningitidis infections due to extensive thrombosis of microvessels. Although a Disseminated... 
ACTIVATED PROTEIN-C | NEISSERIA-MENINGITIDIS | BRAIN ENDOTHELIUM | MICROBIOLOGY | NECROSIS-FACTOR-ALPHA | CUTANEOUS LESIONS | VIROLOGY | EPITHELIAL-CELLS | REPLACEMENT THERAPY | SEPTIC SHOCK | IV PILI | PARASITOLOGY | SEVERE SEPSIS | Amyloid Precursor Protein Secretases - genetics | Purpura Fulminans - etiology | ADAM10 Protein - genetics | Humans | Microvessels - metabolism | ADAM10 Protein - metabolism | Meningococcal Infections - complications | Microvessels - microbiology | Microvessels - pathology | Endothelial Protein C Receptor - genetics | Meningococcal Infections - microbiology | Membrane Proteins - metabolism | Purpura Fulminans - metabolism | Neisseria meningitidis - physiology | Endothelial Protein C Receptor - metabolism | Membrane Proteins - genetics | Cells, Cultured | Purpura Fulminans - pathology | Endothelium, Vascular - microbiology | Bacterial Adhesion | Amyloid Precursor Protein Secretases - metabolism | Endothelium, Vascular - metabolism | Endothelium, Vascular - pathology | Blood Coagulation - physiology | Protein C - metabolism | Protein C - genetics | Purpura (Pathology) | Glycocalyx | Genetic aspects | Research | Meningococcal infections | CRISPR | Shedding | Anticoagulants | Target recognition | Funding | Therapeutic applications | Pathogenesis | Coagulation | Protein C | Activation | Genomes | siRNA | Thrombosis | Adhesion | Endothelial cells | Proteins | Receptors | Pili | Purpura | Sepsis | Thromboembolism | Life Sciences | Human health and pathology | Infectious diseases | Cardiology and cardiovascular system
Journal Article