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Nature (London), ISSN 1476-4687, 03/2010, Volume 464, Issue 7287, pp. 374 - 379
Cellular senescence has been recently shown to have an important role in opposing tumour initiation and promotion. Senescence induced by oncogenes or by loss... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Proto-Oncogene Proteins p21(ras) - genetics | Cellular Senescence - drug effects | Male | Prostate - metabolism | Cyclin-Dependent Kinase Inhibitor p27 - metabolism | S-Phase Kinase-Associated Proteins - antagonists & inhibitors | Adenovirus E1A Proteins - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Fibroblasts | Prostatic Neoplasms - prevention & control | Prostatic Neoplasms - drug therapy | Proto-Oncogene Proteins p21(ras) - metabolism | PTEN Phosphohydrolase - genetics | Prostatic Neoplasms - pathology | Cyclin-Dependent Kinase Inhibitor p16 - deficiency | PTEN Phosphohydrolase - deficiency | Cells, Cultured | Tumor Suppressor Protein p53 - metabolism | Cyclin-Dependent Kinase Inhibitor p16 - genetics | PTEN Phosphohydrolase - metabolism | SKP Cullin F-Box Protein Ligases - metabolism | S-Phase Kinase-Associated Proteins - metabolism | Tumor Suppressor Protein p53 - deficiency | Animals | Activating Transcription Factor 4 - metabolism | Adenovirus E1A Proteins - genetics | Prostate - cytology | Cyclin-Dependent Kinase Inhibitor p16 - metabolism | S-Phase Kinase-Associated Proteins - genetics | Mice | Cell Transformation, Neoplastic - drug effects | Prevention | Care and treatment | Physiological aspects | Aging | Tumor suppressor genes | Genetic aspects | Research | Cells | Tumors | Cell cycle | Apoptosis | Index Medicus
Journal Article
Nature (London), ISSN 1476-4687, 10/2009, Volume 461, Issue 7267, pp. 1084 - 1091
Journal Article
Nature neuroscience, ISSN 1546-1726, 08/2010, Volume 13, Issue 9, pp. 1075 - 1081
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 0027-8424, 3/2010, Volume 107, Issue 12, pp. 5471 - 5476
The phosphatidylinositol 3-kinase (PI3K) signaling pathway is deregulated in many human diseases including cancer, diabetes, obesity, and autoimmunity. PI3K... 
Proteins | Phosphatases | HeLa cells | Lipids | Insulin resistance | Gene expression regulation | Physiological regulation | Insulin | Phosphatidylinositols | Cancer | Explains mouse phenotypes | Lipid phosphatase | Protein:protein interactions | PTEN tumor suppressor | Insulin signaling | Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Protein Subunits | Sequence Deletion | Humans | Cercopithecus aethiops | Phosphatidylinositol 3-Kinases - metabolism | Recombinant Fusion Proteins - metabolism | Phosphatidylinositol 3-Kinases - deficiency | Protein Interaction Domains and Motifs | Peptide Fragments - genetics | PTEN Phosphohydrolase - genetics | PTEN Phosphohydrolase - deficiency | Peptide Fragments - metabolism | Signal Transduction | Phosphatidylinositol 3-Kinases - chemistry | PTEN Phosphohydrolase - metabolism | Epidermal Growth Factor - metabolism | Recombinant Fusion Proteins - chemistry | Mice, Knockout | Phosphatidylinositol 3-Kinases - genetics | Insulin - metabolism | Peptide Fragments - chemistry | Phenotype | Animals | Protein Binding | Recombinant Fusion Proteins - genetics | Mice | HeLa Cells | COS Cells | PTEN Phosphohydrolase - chemistry | Phosphatidylinositol | Chemical properties | Research | Phosphotransferases | Protein-protein interactions | Tumors | Index Medicus | Biological Sciences | protein:protein interactions | explains mouse phenotypes | insulin signaling | lipid phosphatase
Journal Article
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 02/2013, Volume 110, Issue 10, pp. 4039 - 4044
Journal Article
Journal Article