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science & technology (547) 547
life sciences & biomedicine (468) 468
pu.1 (352) 352
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Nature immunology, ISSN 1529-2908, 2017, Volume 18, Issue 6, pp. 694 - 704
The transcription factor STAT5 has a critical role in B cell acute lymphoblastic leukemia (B-ALL). How STAT5 mediates this effect is unclear. Here we found... 
Life Sciences & Biomedicine | Immunology | Science & Technology | Prognosis | Humans | Gene Expression Regulation, Neoplastic | Protein Kinase C beta - genetics | B-Lymphocytes | Precursor Cell Lymphoblastic Leukemia-Lymphoma - mortality | NF-kappa B p50 Subunit - genetics | Ikaros Transcription Factor - genetics | Precursor Cell Lymphoblastic Leukemia-Lymphoma - metabolism | STAT5 Transcription Factor - metabolism | Protein-Tyrosine Kinases - genetics | Flow Cytometry | Chromatin Immunoprecipitation | Trans-Activators - genetics | Pre-B Cell Receptors - genetics | Real-Time Polymerase Chain Reaction | Signal Transduction | PAX5 Transcription Factor - genetics | Interferon Regulatory Factors - genetics | Proto-Oncogene Proteins - genetics | Survival Rate | Multiplex Polymerase Chain Reaction | Precursor Cell Lymphoblastic Leukemia-Lymphoma - genetics | Animals | Agammaglobulinaemia Tyrosine Kinase | Adaptor Proteins, Signal Transducing - genetics | Mice | Care and treatment | Immune response | Development and progression | STAT5 | Genetic aspects | Acute lymphocytic leukemia | Health aspects | Protein kinase C | Transcription factors | Ikaros protein | Acute lymphatic leukemia | Cell survival | Leukemia | Gene regulation | T cell receptors | Stat5 protein | Lymphatic leukemia | B-cell receptor | Gene expression | Pax5 protein | Bruton's tyrosine kinase | Defects | Enhancers | PU.1 protein | BLNK protein | Lymphocytes B | Interferon regulatory factor 4 | Index Medicus
Journal Article
Oncogene, ISSN 0950-9232, 12/2000, Volume 19, Issue 55, pp. 6443 - 6454
The Ets family of transcription factors is one of a growing number of master regulators of development. This family was originally defined by the presence of a... 
Angiogenesis | Ets family | Targeted disruption | Hematopoiesis | Knockout | Biochemistry & Molecular Biology | Oncology | Genetics & Heredity | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Mice, Knockout - genetics | Multigene Family | Proto-Oncogene Protein c-fli-1 | Mice, Knockout - embryology | Embryonic and Fetal Development - genetics | Repressor Proteins | Transcription Factors - deficiency | DNA-Binding Proteins - deficiency | Trans-Activators - physiology | Genes, Lethal | Cell Differentiation - genetics | Gene Expression Regulation, Developmental | Gene Deletion | Trans-Activators - genetics | Models, Animal | Proto-Oncogene Proteins c-ets | Protein Structure, Tertiary | Gene Targeting | DNA-Binding Proteins - physiology | Transcription Factors - physiology | Neovascularization, Physiologic - genetics | Mice, Inbred C57BL | Genotype | Hematopoiesis - genetics | Proto-Oncogene Proteins - genetics | Radiation Chimera | Fungal Proteins - genetics | Proto-Oncogene Proteins - deficiency | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Fungal Proteins - physiology | Animals | Trans-Activators - deficiency | Proto-Oncogene Proteins - physiology | Mice | Proto-Oncogene Protein c-ets-2 | Proto-Oncogene Protein c-ets-1 | Physiological aspects | Genetic aspects | Research | DNA binding proteins | Cell differentiation | Properties | Gene expression | Fli1 protein | PU.1 protein | Ets protein | TEL1 protein | Ets2 protein | Spi-B protein | ER81 gene | Index Medicus
Journal Article
Leukemia, ISSN 1476-5551, 08/2014, Volume 29, Issue 3, pp. 535 - 547
Journal Article
Cell research, ISSN 1001-0602, 2008, Volume 18, Issue 8, pp. 834 - 845
... 4.1R protein and, therefore, for red blood cell membrane integrity. This report demonstrates that Spi-1/PU... 
基因表达 | 细胞分化 | 蛋白质 | 致癌基因 | Alternative splicing | Protein 4.1R | Erythroid differentiation | Oncogene | HERF1 | Spi-1/PU.1 | TRIM10 | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Histocompatibility Antigens - genetics | Cell Survival - genetics | Dimethyl Sulfoxide - pharmacology | Erythroid Cells - cytology | Blood Proteins - metabolism | Cell Differentiation - genetics | RNA Interference | Blood Proteins - genetics | Proto-Oncogene Protein c-fli-1 - metabolism | RNA Splicing - genetics | Trans-Activators - genetics | Erythrocytes - cytology | Hematopoiesis - physiology | Hemoglobins - biosynthesis | Histocompatibility Antigens - metabolism | Proto-Oncogene Proteins - metabolism | Proto-Oncogene Protein c-fli-1 - genetics | Erythroid Cells - metabolism | Exons - genetics | Hematopoiesis - genetics | Proto-Oncogene Proteins - genetics | Up-Regulation - genetics | Signal Transduction - genetics | Down-Regulation - genetics | Carrier Proteins - genetics | Animals | Carrier Proteins - metabolism | Microfilament Proteins | Erythrocytes - metabolism | Cell Line, Tumor | Trans-Activators - metabolism | Mice | Index Medicus | Up-Regulation | Proto-Oncogene Protein c-fli-1 | Exons | Erythrocytes | Erythroid Cells | Life Sciences | RNA Splicing | Hematopoiesis | Dimethyl Sulfoxide | Proto-Oncogene Proteins | Cell Differentiation | Hemoglobins | Carrier Proteins | Signal Transduction | Cell Survival | Trans-Activators | Down-Regulation | Biochemistry, Molecular Biology | Histocompatibility Antigens | Blood Proteins | Molecular biology
Journal Article
The Journal of biological chemistry, ISSN 1083-351X, 09/2010, Volume 285, Issue 36, pp. 27601 - 27608
Mammalian colon harbors trillions of bacteria, yet there is no undue inflammatory response by the host against these bacteria under normal conditions. The... 
Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | Propionates - pharmacology | Proto-Oncogene Proteins - metabolism | Propionates - metabolism | Bacteria - metabolism | Butyrates - metabolism | Receptors, Nicotinic - metabolism | Receptors, G-Protein-Coupled - metabolism | Bone Marrow Cells - cytology | Butyrates - pharmacology | Mice, Inbred C57BL | Granulocyte-Macrophage Colony-Stimulating Factor - pharmacology | Histone Deacetylases - metabolism | Fermentation | Animals | Transcription Factor RelB - metabolism | Cation Transport Proteins - metabolism | Cell Differentiation - drug effects | Dendritic Cells - drug effects | Histone Deacetylase Inhibitors - pharmacology | Trans-Activators - metabolism | Dendritic Cells - cytology | Mice | Histone Deacetylase Inhibitors - metabolism | Dendritic Cells - metabolism | Index Medicus | Antigen presentation | Histone deacetylase | Transcription factors | G protein-coupled receptors | Dendritic cells | Propionic acid | Inflammation | Lymphocytes T | double prime G protein-coupled receptors | RelB protein | Immunosuppression | PU.1 protein | Metabolites | Stem cells | Bone marrow | Colon | Acetic acid | Leukocytes (granulocytic) | Immunosupressor | Immunology | Slc5a8 | Colon Cancer | Propionate | Bacteria | Bone Marrow | Cell Biology | Histone Deacetylase | Butyrate
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 07/2011, Volume 108, Issue 36, pp. 14902 - 14907
Epstein-Barr virus nuclear antigen 2 (EBNA2) regulation of transcription through the cell transcription factor RBPJ is essential for resting B-lymphocyte (RBL)... 
T lymphocytes | Up regulation | Chromatin | B lymphocytes | DNA | Nucleosomes | Gene expression regulation | Epstein Barr virus infections | Human herpesvirus 4 | Binding sites | Development | Lymphoma | Leukemia | Notch | Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Cell Proliferation | Herpesvirus 4, Human - genetics | Proto-Oncogene Proteins c-ets - genetics | Genome, Viral - genetics | Humans | Transcription Factor RelA - genetics | Viral Proteins - metabolism | Trans-Activators - genetics | B-Lymphocytes - virology | Epstein-Barr Virus Infections - genetics | Core Binding Factor alpha Subunits - metabolism | Transcription, Genetic | Proto-Oncogene Proteins c-ets - metabolism | Epstein-Barr Virus Nuclear Antigens - metabolism | Immunoglobulin J Recombination Signal Sequence-Binding Protein - genetics | B-Lymphocytes - metabolism | Proto-Oncogene Proteins - metabolism | Immunoglobulin J Recombination Signal Sequence-Binding Protein - metabolism | Epstein-Barr Virus Nuclear Antigens - genetics | Response Elements | Nucleosomes - metabolism | Viral Proteins - genetics | Nucleosomes - genetics | Proto-Oncogene Proteins - genetics | Proto-Oncogene Proteins c-myc - metabolism | Core Binding Factor alpha Subunits - genetics | Transcription Factor RelA - metabolism | Cell Line, Tumor | Trans-Activators - metabolism | Proto-Oncogene Proteins c-myc - genetics | Herpesvirus 4, Human - metabolism | Epstein-Barr Virus Infections - metabolism | Physiological aspects | Transcription factors | Epstein-Barr virus | Genetic aspects | Lymphocytes | Health aspects | Cell growth | Deoxyribonucleic acid--DNA | Index Medicus | ETS protein | Lymphoblasts | Introns | Data processing | Genomes | Myc protein | Promoters | Early B-cell factor | Infection | Enhancers | PU.1 protein | Lymphocytes B | Fluorescence in situ hybridization | RelA protein | Conformation | Biological Sciences | leukemia | development | lymphoma
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