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The Journal of experimental medicine, ISSN 1540-9538, 2011, Volume 208, Issue 7, pp. 1459 - 1471
Journal Article
The Journal of immunology (1950), ISSN 1550-6606, 2014, Volume 193, Issue 7, pp. 3755 - 3768
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 09/2009, Volume 119, Issue 9, pp. 2550 - 2563
Uncontrolled activation of the coagulation cascade contributes to the pathophysiology of several conditions, including acute and chronic lung diseases.... 
MEDICINE, RESEARCH & EXPERIMENTAL | GROWTH-FACTOR-BETA | BLOOD-COAGULATION | THROMBUS FORMATION | GENE-EXPRESSION | IDIOPATHIC PULMONARY-FIBROSIS | PROTEASE-ACTIVATED RECEPTOR-1 | TISSUE FACTOR EXPRESSION | SUSCEPTIBILITY GENES | RESPIRATORY-DISTRESS-SYNDROME | BRONCHOALVEOLAR LAVAGE | Up-Regulation | Lung Injury - pathology | Factor Xa Inhibitors | Receptor, PAR-1 - metabolism | Humans | Middle Aged | Actins - metabolism | Male | Pulmonary Fibrosis - blood | Idiopathic Pulmonary Fibrosis - blood | RNA, Messenger - metabolism | Case-Control Studies | Idiopathic Pulmonary Fibrosis - metabolism | Idiopathic Pulmonary Fibrosis - etiology | Base Sequence | Lung Injury - metabolism | Adult | Female | Cell Differentiation | Pulmonary Fibrosis - metabolism | Receptors, Vitronectin - metabolism | Pulmonary Fibrosis - etiology | Bleomycin - toxicity | Fibroblasts - metabolism | Gene Expression | Factor Xa - genetics | Mice, Inbred C57BL | RNA, Messenger - genetics | Cells, Cultured | Pulmonary Fibrosis - pathology | Fibroblasts - pathology | Factor Xa - metabolism | Animals | Transforming Growth Factor beta - genetics | Models, Biological | Fibroblasts - drug effects | Idiopathic Pulmonary Fibrosis - pathology | Aged | Mice | Transforming Growth Factor beta - metabolism | Transforming growth factors | Lung diseases | Integrins | Index Medicus | Abridged Index Medicus
Journal Article
PloS one, ISSN 1932-6203, 2013, Volume 8, Issue 10, p. e76451
Rationale: Idiopathic pulmonary fibrosis (IPF) is a fatal interstitial lung disease characterized by progressive scarring and matrix deposition. Recent reports... 
LUNG | GROWTH-FACTOR-BETA | INHIBITION | MESSENGER-RNA | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | IN-VIVO | MYOFIBROBLAST | AUTOREACTIVITY | IDENTIFICATION | FIBROBLASTS | T-Lymphocyte Subsets - immunology | Transcription, Genetic - drug effects | Idiopathic Pulmonary Fibrosis - genetics | Collagen Type V - genetics | Autoantibodies - blood | Humans | Collagen Type V - immunology | Pulmonary Fibrosis - genetics | RNA, Messenger - metabolism | Transforming Growth Factor beta - biosynthesis | Collagen Type V - administration & dosage | Idiopathic Pulmonary Fibrosis - metabolism | Lymphocyte Activation - immunology | Inflammation Mediators - metabolism | Female | Pulmonary Fibrosis - metabolism | Collagen Type V - metabolism | Cytokines - genetics | Disease Models, Animal | Gene Expression | RNA, Messenger - genetics | Pulmonary Fibrosis - immunology | Immune Tolerance | Pulmonary Fibrosis - pathology | Bleomycin - adverse effects | Idiopathic Pulmonary Fibrosis - immunology | Collagen Type I - immunology | Gene Expression Regulation - drug effects | Autoantibodies - immunology | Animals | Transforming Growth Factor beta - genetics | T-Lymphocyte Subsets - metabolism | Idiopathic Pulmonary Fibrosis - pathology | Mice | Pulmonary Fibrosis - drug therapy | Cytokines - biosynthesis | Idiopathic Pulmonary Fibrosis - drug therapy | Nebulizers and Vaporizers | Animal models | Disease | Transcription | Pathogenesis | Antibodies | Immunity | Interleukin 6 | Bleomycin | Consent | Lymphocytes | Smad2 protein | Rodents | Animal tissues | Collagen (type V) | Extracellular matrix | Growth factors | Deposition | NF-κB protein | Lung diseases | Tumor necrosis factor-α | Gene expression | Sp1 protein | Patients | Immunological tolerance | Medicine | Pathology | Signaling | Scars | Pulmonary fibrosis | Lungs | Liquid oxygen | Collagen | γ-Interferon | Fibrosis | Interferon | Binding sites
Journal Article
Journal Article
The Journal of experimental medicine, ISSN 1540-9538, 2010, Volume 207, Issue 8, pp. 1589 - 1597
Uncontrolled extracellular matrix production by fibroblasts in response to tissue injury contributes to fibrotic diseases, such as idiopathic pulmonary... 
MEDICINE, RESEARCH & EXPERIMENTAL | PHOSPHATASE | MECHANISMS | GROWTH-FACTOR | IMMUNOLOGY | MYOFIBROBLAST DIFFERENTIATION | EXPRESSION | MICRORNA | Oligonucleotides - genetics | Pulmonary Fibrosis - therapy | Idiopathic Pulmonary Fibrosis - genetics | Gene Expression - drug effects | Gene Expression - genetics | Humans | Actins - metabolism | MicroRNAs - metabolism | Pulmonary Fibrosis - genetics | Idiopathic Pulmonary Fibrosis - metabolism | Actins - genetics | Antisense Elements (Genetics) - therapeutic use | Collagen - genetics | Lung - metabolism | Phosphorylation - drug effects | Smad7 Protein - genetics | Extracellular Matrix Proteins - metabolism | Transforming Growth Factor beta1 - pharmacology | Fibroblasts - metabolism | Smad7 Protein - metabolism | Cell Line | Lung - pathology | Extracellular Matrix Proteins - genetics | Mice, Inbred C57BL | Smad2 Protein - metabolism | Mice, Transgenic | Pulmonary Fibrosis - pathology | Transforming Growth Factor beta1 - genetics | Fibroblasts - pathology | Fibronectins - metabolism | Collagen - metabolism | Animals | Fibroblasts - drug effects | Antisense Elements (Genetics) - genetics | Idiopathic Pulmonary Fibrosis - pathology | Pulmonary Fibrosis - chemically induced | Bleomycin - pharmacology | Fibronectins - genetics | Mice | MicroRNAs - genetics | Brief Definitive Report
Journal Article
Respirology (Carlton, Vic.), ISSN 1323-7799, 2016, Volume 21, Issue 5, pp. 920 - 926
Journal Article
Nature medicine, ISSN 1546-170X, 2016, Volume 22, Issue 11, pp. 1285 - 1293
Successful recovery from lung injury requires the repair and regeneration of alveolar epithelial cells to restore the integrity of gas-exchanging regions... 
MEDICINE, RESEARCH & EXPERIMENTAL | STEM-CELLS | REGENERATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | INJURY | PROLIFERATION | LUNG EPITHELIUM | CELL BIOLOGY | REPAIR | CAPILLARY VASCULAR NICHE | INFLAMMATION | TOLL-LIKE RECEPTORS | EXPRESSION | Cell Self Renewal - immunology | Stem Cells - immunology | Antibiotics, Antineoplastic - toxicity | Cell Proliferation | Humans | Stem Cells - metabolism | Idiopathic Pulmonary Fibrosis - metabolism | Alveolar Epithelial Cells - immunology | Cell Self Renewal - genetics | Glucuronosyltransferase - genetics | Flow Cytometry | Organoids | Lung Injury - metabolism | Hyaluronan Synthases | Pulmonary Fibrosis - metabolism | Real-Time Polymerase Chain Reaction | Bleomycin - toxicity | Severity of Illness Index | Cell Line | Hydroxyproline - metabolism | Pulmonary Fibrosis - immunology | Pulmonary Surfactant-Associated Protein C - metabolism | Toll-Like Receptor 4 - genetics | Alveolar Epithelial Cells - metabolism | Pulmonary Fibrosis - pathology | Toll-Like Receptor 4 - immunology | Idiopathic Pulmonary Fibrosis - immunology | Immunity, Innate | Toll-Like Receptor 4 - metabolism | Mice, Knockout | Animals | Hyaluronic Acid - metabolism | Interleukin-6 - immunology | Idiopathic Pulmonary Fibrosis - pathology | Mice | Lung Injury - chemically induced | Lung Injury - immunology | Prevention | Toll-like receptors | Genetic aspects | Pulmonary fibrosis | Hyaluronic acid | Health aspects | Proteins | Surfactants | Rodents | Cells
Journal Article
Thorax, ISSN 0040-6376, 05/2013, Volume 68, Issue 5, pp. 436 - 441
Journal Article