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The Journal of Physiology, ISSN 0022-3751, 2004, Volume 556, Issue 3, pp. 983 - 1000
Muscular adaptation to physical exercise has previously been described as a repair process following tissue damage. Recently, evidence has been published to... 
INDUCED INJURY | PHYSIOLOGY | IMMUNOREACTIVITY | ECCENTRIC EXERCISE | REGENERATION | LEUKEMIA INHIBITORY FACTOR | DENERVATED HUMAN MUSCLE | SATELLITE CELLS | STRENGTH | FACTOR-I | EXPRESSION | NEUROSCIENCES | Immunohistochemistry | Granulocytes - cytology | Cytokines - analysis | Humans | Middle Aged | DNA-Binding Proteins - analysis | Ki-67 Antigen - metabolism | Male | Muscle, Skeletal - metabolism | Proteins - analysis | Pain - metabolism | fas Receptor - metabolism | Fascia - chemistry | Oxygen Consumption - physiology | fas Receptor - analysis | Antigens, CD - metabolism | Antigens, CD - analysis | Running - physiology | C-Reactive Protein - metabolism | Aryl Hydrocarbon Receptor Nuclear Translocator | CD11b Antigen - analysis | Receptors, Cytokine - metabolism | Testosterone - blood | C-Reactive Protein - analysis | Hormones - blood | Leukocytes - chemistry | Interleukin-6 - metabolism | Lymphocytes - metabolism | CD56 Antigen - analysis | Lymphocytes - cytology | Insulin-Like Growth Factor I - analysis | Muscle, Skeletal - physiology | CD3 Complex - metabolism | Leukemia Inhibitory Factor Receptor alpha Subunit | Regression Analysis | Pain - physiopathology | Receptors, Cytokine - analysis | Adolescent | Antigens, Differentiation, Myelomonocytic - analysis | Creatine Kinase - metabolism | Transcription Factors - analysis | Leukocytes - metabolism | Insulin-Like Growth Factor I - metabolism | Receptors, OSM-LIF | Interleukin-6 - analysis | Monocytes - cytology | Receptors, Cell Surface - analysis | Monocytes - metabolism | Receptors, Aryl Hydrocarbon - analysis | DNA-Binding Proteins - metabolism | Testosterone - metabolism | Flow Cytometry | Interleukin-6 - blood | Exercise Test - methods | Muscle, Skeletal - chemistry | Fascia - metabolism | Heart Rate - physiology | Receptors, Aryl Hydrocarbon - metabolism | Adult | Female | Leukocyte Count | Leukemia Inhibitory Factor | Isometric Contraction - physiology | Cytokines - blood | Leukocytes - cytology | CD56 Antigen - metabolism | Creatine Kinase - blood | Cytokines - metabolism | Receptors, Cell Surface - metabolism | Transcription Factors - metabolism | CD3 Complex - analysis | Ki-67 Antigen - analysis | Proteins - metabolism | Hormones - metabolism | Antigens, Differentiation, Myelomonocytic - metabolism | CD11b Antigen - metabolism | Growth Substances - metabolism | Pain - diagnosis | Research Papers
Journal Article
The Journal of biological chemistry, ISSN 1083-351X, 2015, Volume 290, Issue 22, pp. 13875 - 13887
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2016, Volume 113, Issue 43, pp. E6686 - E6695
Pain is a critical component hindering recovery and regaining of function after surgery, particularly in the elderly. Understanding the role of pain signaling... 
Delirium | Nerve injury | Postoperative pain | Neurogenesis | Memory | CALCIUM-BINDING PROTEINS | memory | TIBIA FRACTURE MODEL | postoperative pain | NEUROTROPHIC FACTOR | MULTIDISCIPLINARY SCIENCES | DORSAL-ROOT GANGLIA | SENSORY NEURONS | PERIPHERAL-NERVE INJURY | PERSISTENT POSTSURGICAL PAIN | NEUROPATHIC PAIN | WORKING-MEMORY DEFICITS | nerve injury | neurogenesis | POSTOPERATIVE COGNITIVE DYSFUNCTION | delirium | Brain-Derived Neurotrophic Factor - genetics | Glial Fibrillary Acidic Protein - genetics | Sciatic Nerve - injuries | Spinal Cord - metabolism | Male | Ganglia, Spinal - physiopathology | Neuropeptide Y - genetics | Glial Fibrillary Acidic Protein - metabolism | Pain - metabolism | Dentate Gyrus - physiopathology | Galanin - metabolism | Hyperalgesia - pathology | Neuropeptide Y - metabolism | Tibial Fractures - metabolism | Tibial Fractures - surgery | Sciatic Nerve - metabolism | CX3C Chemokine Receptor 1 - metabolism | Pain - genetics | Tibial Fractures - physiopathology | CX3C Chemokine Receptor 1 - genetics | Pain - etiology | Cognitive Dysfunction - genetics | Brain-Derived Neurotrophic Factor - metabolism | Microfilament Proteins - metabolism | Microfilament Proteins - genetics | Activating Transcription Factor 3 - genetics | Calcium-Binding Proteins - metabolism | Dentate Gyrus - metabolism | Hyperalgesia - metabolism | Signal Transduction | Cognitive Dysfunction - metabolism | Mice, Inbred C57BL | Gene Expression Regulation | Proto-Oncogene Proteins c-fos - metabolism | Cognitive Dysfunction - pathology | Cognitive Dysfunction - etiology | Activating Transcription Factor 3 - metabolism | Pain - pathology | Animals | Hyperalgesia - genetics | Tibial Fractures - genetics | Proto-Oncogene Proteins c-fos - genetics | Mice | Spinal Cord - physiopathology | Galanin - genetics | Fracture Fixation, Intramedullary - adverse effects | Calcium-Binding Proteins - genetics | Ganglia, Spinal - metabolism | Spinal cord | Orthopedic surgery | Brain-derived neurotrophic factor | Neuropeptides | Hippocampus (Brain) | Health aspects | Methods | Biological Sciences | PNAS Plus
Journal Article
PloS one, ISSN 1932-6203, 2012, Volume 7, Issue 10, p. e47977
In recent years a growing debate is about whether botulinum neurotoxins are retrogradely transported from the site of injection. Immunodetection of cleaved... 
ACTIVATION | ACIDIC PROTEIN GFAP | RAT | TOXIN | MULTIDISCIPLINARY SCIENCES | NEURONS | AXONAL-TRANSPORT | NEUROEXOCYTOSIS | GLIAL-CELLS | EXPRESSION | SNAP-25 | Immunohistochemistry | Analgesics - pharmacology | Sciatic Nerve - injuries | Spinal Cord - drug effects | Spinal Cord - metabolism | Neuralgia - metabolism | Analgesics - metabolism | Male | Acetylcholine - metabolism | Glial Fibrillary Acidic Protein - metabolism | Botulinum Toxins, Type A - pharmacology | Hindlimb - physiopathology | Sciatic Nerve - metabolism | Biological Transport | Neuralgia - prevention & control | Neuromuscular Agents - pharmacology | Peripheral Nerves - metabolism | Schwann Cells - drug effects | Neuromuscular Agents - metabolism | Cells, Cultured | Nuclear Proteins - metabolism | Schwann Cells - metabolism | Nerve Tissue Proteins - metabolism | Microscopy, Confocal | Animals | Hindlimb - drug effects | Cell Proliferation - drug effects | Mice | Synaptosomal-Associated Protein 25 - metabolism | Botulinum Toxins, Type A - metabolism | CD11b Antigen - metabolism | Hindlimb - metabolism | Astrocytes - metabolism | Ganglia, Spinal - metabolism | Care and treatment | Analysis | Pain | Cell proliferation | Spinal cord | Neurosciences | Complement receptor 3 | Neurobiology | Bovet, Daniel | SNAP receptors | Biology | Neuropathy | Proteins | Biological effects | Dorsal root ganglia | Analgesics | Botulinum Toxin Type A | Animal tissues | Fibroblasts | Neurotoxin A | Horns | Growth factors | Nerve endings | CD11b antigen | Neurotoxins | Pain perception | Astrocytes | Glial fibrillary acidic protein | Schwann cells | Injection | Retrograde transport | Ganglia | Quality of life | Councils | SNAP-25 protein | Acetylcholine | Toxins | Transport | Immunofluorescence | Sciatic nerve | Combinatorial analysis
Journal Article
Journal Article
Cell (Cambridge), ISSN 0092-8674, 2017, Volume 170, Issue 1, pp. 185 - 198.e16
Journal Article
Spine (Philadelphia, Pa. 1976), ISSN 0362-2436, 2011, Volume 36, Issue 21, pp. E1373 - E1384
Study Design. Descriptive and mechanistic investigation of the anti-inflammatory and anticatabolic effect of resveratrol in intervertebral discs (IVDs) in... 
nucleus pulposusmediated back pain | NF-κB | MAP kinases | anti-inflammatory | anticatabolic | radiculopathy | toll-like receptor | resveratrol | INTERVERTEBRAL DISCS | NECROSIS-FACTOR-ALPHA | NERVE ROOT | CYTOKINE EXPRESSION | CLINICAL NEUROLOGY | EPIDURAL APPLICATION | BACK-PAIN | NF-kappa B | LUMBAR DISC HERNIATION | nucleus pulposus-mediated back pain | TNF-ALPHA | ORTHOPEDICS | NF-KAPPA-B | MOUSE SKIN | Wine | Analgesics - pharmacology | Interleukin-8 - genetics | Toll-Like Receptor 2 - genetics | Radiculopathy - genetics | Humans | Middle Aged | Male | NF-kappa B - metabolism | Interleukin-1beta - genetics | Stilbenes - pharmacology | Pain - metabolism | RNA, Messenger - metabolism | Young Adult | Radiculopathy - drug therapy | Pain - genetics | Time Factors | Interleukin-1beta - metabolism | Toll-Like Receptor 2 - drug effects | Pain - etiology | p38 Mitogen-Activated Protein Kinases - metabolism | Interleukin-8 - metabolism | Interleukin-6 - metabolism | Disease Models, Animal | Interleukin-6 - genetics | Anti-Inflammatory Agents - pharmacology | Matrix Metalloproteinase 13 - genetics | Rats | Matrix Metalloproteinase 3 - metabolism | Toll-Like Receptor 2 - metabolism | Rats, Sprague-Dawley | Matrix Metalloproteinase 13 - metabolism | Signal Transduction - drug effects | Matrix Metalloproteinase 1 - metabolism | Matrix Metalloproteinase 3 - genetics | Sirtuin 1 - metabolism | JNK Mitogen-Activated Protein Kinases - metabolism | Pain - immunology | Extracellular Signal-Regulated MAP Kinases - metabolism | Intervertebral Disc - immunology | Dose-Response Relationship, Drug | Radiculopathy - complications | Pain - drug therapy | Inflammation Mediators - metabolism | Resveratrol | Adult | Female | Radiculopathy - metabolism | Matrix Metalloproteinase 1 - genetics | Cells, Cultured | Intervertebral Disc - metabolism | Gene Expression Regulation - drug effects | Animals | Aged | Intervertebral Disc - drug effects | Pain Measurement | Radiculopathy - immunology
Journal Article
10.