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Chemical Senses, ISSN 0379-864X, 3/2008, Volume 33, Issue 3, pp. 243 - 254
The transient receptor potential channel, PKD2L1, is reported to be a candidate receptor for sour taste based on molecular biological and functional studies.... 
Mouse | Taste bud cells | Gustation | Polycystic kidney disease - Like ion channel (PKD2L1) | Sour taste | LOCALIZATION | NERVE-FIBERS | PHYSIOLOGY | FINE-STRUCTURE | RAT | IMMUNOREACTIVITY | FOOD SCIENCE & TECHNOLOGY | polycystic kidney disease-like ion channel (PKD2L1) | NEUROSCIENCES | TRANSIENT RECEPTOR | CIRCUMVALLATE PAPILLAE | mouse | ADHESION MOLECULE | sour taste | gustation | taste bud cells | BEHAVIORAL SCIENCES | CHANNELS | BUDS | Larynx - metabolism | Calcium Channels - metabolism | Chemoreceptor Cells - metabolism | Green Fluorescent Proteins - genetics | Recombinant Fusion Proteins - metabolism | Microscopy, Immunoelectron | Chemoreceptor Cells - cytology | Taste Buds - ultrastructure | Ubiquitin Thiolesterase - metabolism | Chromogranin A - metabolism | 5-Hydroxytryptophan - metabolism | Palate - metabolism | Phospholipase C beta - metabolism | Green Fluorescent Proteins - metabolism | Mice, Inbred C57BL | Adenosine Triphosphatases - metabolism | Receptors, Cell Surface - metabolism | Taste Buds - cytology | Mice, Transgenic | Pharynx - metabolism | Microscopy, Confocal | Taste Buds - metabolism | Animals | Models, Biological | Neural Cell Adhesion Molecules - metabolism | Recombinant Fusion Proteins - genetics | TRPM Cation Channels - genetics | Mice | TRPM Cation Channels - metabolism | Index Medicus | Polycystic-kidney disease-like ion channel (PKD2L1)
Journal Article
Modern Pathology, ISSN 0893-3952, 02/2008, Volume 21, Issue 2, pp. 192 - 200
Journal Article
Science, ISSN 0036-8075, 6/2012, Volume 336, Issue 6085, pp. 1150 - 1153
Protein phosphorylation is a fundamental mechanism regulating nearly every aspect of cellular life. Several secreted proteins are phosphorylated, but the... 
Proteins | T lymphocytes | Phosphorylation | Biomineralogy | Secretion | Phosphoproteins | REPORTS | Immunoblotting | Cellular immunity | Physiological regulation | Siblings | OSTEOPONTIN | INHIBITION | MULTIDISCIPLINARY SCIENCES | PHOSPHOPROTEOME | PURIFICATION | SEQUENCE | GOLGI-APPARATUS | SPECIFICITY DETERMINANTS | MASS-SPECTROMETRY | APPARATUS CASEIN KINASE | LACTATING MAMMARY-GLAND | Abnormalities, Multiple - metabolism | Golgi Apparatus - enzymology | Microcephaly - genetics | Humans | Secretory Pathway | Caseins - metabolism | Glycoproteins - metabolism | Molecular Sequence Data | Substrate Specificity | Cleft Palate - genetics | Casein Kinase I | Recombinant Fusion Proteins - metabolism | Extracellular Matrix Proteins - secretion | Cattle | Exophthalmos - genetics | HEK293 Cells | Osteosclerosis - metabolism | Osteosclerosis - genetics | Abnormalities, Multiple - genetics | Extracellular Matrix Proteins - metabolism | Amino Acid Sequence | Extracellular Matrix Proteins - chemistry | Milk - enzymology | Extracellular Matrix Proteins - genetics | Microcephaly - metabolism | Osteopontin | Recombinant Fusion Proteins - chemistry | Protein Sorting Signals | Amino Acid Motifs | Animals | Calcification, Physiologic | Casein Kinases - metabolism | Exophthalmos - metabolism | Cell Line, Tumor | Recombinant Fusion Proteins - secretion | Cleft Palate - metabolism | HeLa Cells | Mutation | Physiological aspects | Biomineralization | Extracellular matrix | Research | Phosphotransferases | Mineralogy | Cells | Index Medicus | Mutations | Casein | Genes | Bones | Kinases
Journal Article
Science Signaling, ISSN 1945-0877, 04/2012, Volume 5, Issue 218, pp. ra28 - ra28
Dimers of conventional transforming growth factor-beta (TGF-beta) and bone morphogenetic protein (BMP) ligands are composed of two 100- to 140-amino acid... 
BETA FAMILY-MEMBER | PATTERN-FORMATION | TGF-BETA | DPP | TRANSFORMING GROWTH-FACTOR-BETA-2 | CRYSTAL-STRUCTURE | BIOCHEMISTRY & MOLECULAR BIOLOGY | ACTIVITY GRADIENT | DROSOPHILA DEVELOPMENT | BONE MORPHOGENETIC PROTEIN-4 | PRO-DOMAIN | CELL BIOLOGY | Bone Morphogenetic Protein 4 - genetics | Humans | Male | Cleft Palate - genetics | Phylogeny | Drosophila Proteins - metabolism | Bone Morphogenetic Protein 4 - metabolism | Drosophila melanogaster - genetics | Drosophila melanogaster - metabolism | Anti-Mullerian Hormone - genetics | Bone Morphogenetic Proteins - metabolism | Protein Isoforms - metabolism | Wings, Animal - growth & development | Bone Morphogenetic Protein 15 - genetics | Female | Mullerian Ducts - metabolism | Bone Morphogenetic Proteins - genetics | Cell Line | Protein Precursors - genetics | Signal Transduction | Animals, Genetically Modified | Proprotein Convertases - metabolism | Bone Morphogenetic Protein 15 - metabolism | Cleft Lip - metabolism | Blotting, Western | Protein Precursors - metabolism | Wings, Animal - metabolism | Cleft Lip - genetics | Mullerian Ducts - abnormalities | Animals | Transforming Growth Factor beta - genetics | Drosophila melanogaster - growth & development | Ligands | Cleft Palate - metabolism | Anti-Mullerian Hormone - metabolism | Drosophila Proteins - genetics | Mutation | Transforming Growth Factor beta - metabolism | Protein Isoforms - genetics | Index Medicus
Journal Article
Stem Cells and Development, ISSN 1547-3287, 12/2011, Volume 20, Issue 12, pp. 253 - 2064
Adult neural crest related-stem cells persist in adulthood, making them an ideal and easily accessible source of multipotent cells for potential clinical use.... 
Original Research Reports | MEDICINE, RESEARCH & EXPERIMENTAL | CREST | CHICKEN RETINA | ASTROCYTES | CELL & TISSUE ENGINEERING | TRANSPLANTATION | PERIPHERAL-NERVE | NEURAL STEM-CELLS | IN-VITRO | GROWTH-FACTOR | DIFFERENTIATION | HEMATOLOGY | PRECURSOR CELLS | MULLER GLIA | Intermediate Filament Proteins - ultrastructure | Nestin | Multipotent Stem Cells - metabolism | Ki-67 Antigen - metabolism | NF-kappa B - metabolism | Receptors, Nerve Growth Factor - metabolism | Neural Stem Cells - cytology | Cell Culture Techniques - methods | Schwann Cells - cytology | Cellular Reprogramming | Myelin Sheath - metabolism | Mucous Membrane - cytology | Sciatic Nerve - metabolism | Synapses - metabolism | Sciatic Nerve - cytology | Cell Differentiation | Clone Cells | Palate - cytology | Nerve Tissue Proteins - ultrastructure | Nerve Fibers - metabolism | Neural Crest - cytology | Pluripotent Stem Cells - cytology | Cell Separation | Cells, Cultured | Rats | S100 Proteins - metabolism | Schwann Cells - metabolism | Nerve Tissue Proteins - metabolism | Pluripotent Stem Cells - metabolism | Animals | Schwann Cells - ultrastructure | Multipotent Stem Cells - cytology | Cell Aggregation | Myelin Sheath - ultrastructure | Myelin Proteins - metabolism | Intermediate Filament Proteins - metabolism | Neural Stem Cells - metabolism | Index Medicus
Journal Article
BioEssays, ISSN 0265-9247, 05/2016, Volume 38, Issue 5, pp. 427 - 439
We propose that inhibition of GRINA and its interacting partners by the 33‐mer gliadin fragment provokes phenotypes that are found in extraintestinal... 
gluten ataxia | osteopenia | cleft lip and palate | thyroid | celiac disease | dermatitis herpetiformis | GRINA | Celiac disease | Gluten ataxia | Dermatitis herpetiformis | Osteopenia | Cleft lip and palate | Thyroid | BAX INHIBITOR MOTIF | SENSORINEURAL HEARING-LOSS | BONE-MINERAL DENSITY | CEREBRAL CALCIFICATIONS | BIOCHEMISTRY & MOLECULAR BIOLOGY | CLEFT-LIP | TRANSGLUTAMINASE ANTIBODIES | GLUTEN-FREE DIET | PROTEIN-INTERACTION NETWORKS | RESTLESS LEGS SYNDROME | SMALL-INTESTINAL MUCOSA | BIOLOGY | Coenzyme A Ligases - genetics | Humans | Protein Multimerization | Receptors, N-Methyl-D-Aspartate - metabolism | Bone Diseases, Metabolic - pathology | Cleft Palate - genetics | Coenzyme A Ligases - metabolism | Receptors, N-Methyl-D-Aspartate - genetics | Thyroiditis - pathology | Gliadin - metabolism | Thyroiditis - genetics | Cleft Lip - pathology | Membrane Proteins - metabolism | Sterol Regulatory Element Binding Protein 2 - genetics | Sterol Regulatory Element Binding Protein 2 - metabolism | Ataxia - genetics | Ataxia - metabolism | Ataxia - pathology | Amino Acid Sequence | Celiac Disease - chemically induced | Gliadin - genetics | Celiac Disease - genetics | Signal Transduction | Membrane Proteins - genetics | Thyroiditis - metabolism | Gene Expression Regulation | Dermatitis Herpetiformis - metabolism | Cleft Lip - metabolism | Dermatitis Herpetiformis - pathology | Bone Diseases, Metabolic - metabolism | Proteins - genetics | Sequence Homology, Amino Acid | Celiac Disease - pathology | Cleft Lip - genetics | Animals | Proteins - metabolism | Cleft Palate - pathology | Adaptor Proteins, Signal Transducing - genetics | Protein Binding | Glutens - adverse effects | Cleft Palate - metabolism | Models, Genetic | Bone Diseases, Metabolic - genetics | Adaptor Proteins, Signal Transducing - metabolism | Dermatitis Herpetiformis - genetics | Celiac Disease - metabolism | Cleft lip | Research | Binding proteins | Neuroglia | Restless legs syndrome | Hypotheses | Rodents | Index Medicus
Journal Article
Histopathology, ISSN 0309-0167, 07/2013, Volume 63, Issue 1, pp. 36 - 49
Journal Article
Cellular Signalling, ISSN 0898-6568, 07/2015, Volume 27, Issue 7, pp. 1509 - 1516
IRF6 and RIPK4 are critical regulators of keratinocyte differentiation and their mutation cause the developmental syndromes Van der Woude syndrome (VWS) and... 
Protein degradation | IRF6 | Mutation | RIPK4 | Cleft lip/palate | TRANSCRIPTION FACTORS | REGULATORY FACTOR 6 | KERATINOCYTE DIFFERENTIATION | WOUDE SYNDROME PATIENTS | PROTEIN-KINASE | POPLITEAL PTERYGIUM SYNDROME | INDEPENDENT MANNER | CELL BIOLOGY | BARTSOCAS-PAPAS SYNDROME | C-ASSOCIATED KINASE | NF-KAPPA-B | Abnormalities, Multiple - metabolism | Abnormalities, Multiple - pathology | Lip - abnormalities | Phosphorylation | Humans | Transcriptional Activation | Interferon Regulatory Factors - metabolism | Knee - pathology | Half-Life | Knee - abnormalities | Cytoplasm - metabolism | JNK Mitogen-Activated Protein Kinases - metabolism | NF-kappa B - metabolism | Syndactyly - pathology | Mutation, Missense | Lip - metabolism | Cysts - metabolism | Cleft Lip - pathology | HEK293 Cells | Eye Abnormalities - metabolism | Protein-Serine-Threonine Kinases - metabolism | Lip - pathology | Protein Structure, Tertiary | Recombinant Fusion Proteins - biosynthesis | Cell Line | Promoter Regions, Genetic | Signal Transduction | Syndactyly - metabolism | Protein-Serine-Threonine Kinases - genetics | Interferon Regulatory Factors - genetics | Recombinant Fusion Proteins - chemistry | Cleft Lip - metabolism | Cysts - pathology | beta Catenin - metabolism | Eye Abnormalities - pathology | Cleft Palate - pathology | Recombinant Fusion Proteins - genetics | Cleft Palate - metabolism | Genetic aspects | Interferon | Biological response modifiers | Proteolysis | Protein kinases | Index Medicus
Journal Article
Journal Article
Journal of Bone and Mineral Research, ISSN 0884-0431, 04/2017, Volume 32, Issue 4, pp. 757 - 769
In 1985, we briefly characterized “congenital sclerosing osteomalacia with cerebral calcification” (CSOCC) in infant sisters. Later, Raine syndrome (RNS)... 
CRANIOFACIAL DYSOSTOSIS | OSTEOMALACIA | RICKETS | DENTIN MATRIX PROTEIN | KINASE | PHOSPHOPROTEOME | CROUZON SYNDROME | CRANIOSYNOSTOSIS | HYPOPHOSPHATEMIA | SIBLING PROTEINS | OSTEOPONTIN | METOPIC SUTURE | MINERALIZATION | OSTEOSCLEROSIS | OSTEOPETROSIS | TRIGONOCEPHALY | AUTOSOMAL RECESSIVE SYNDROME | INTRACRANIAL CALCIFICATION | HYPOPLASTIC NOSE | SECRETED PROTEINS | ENDOCRINOLOGY & METABOLISM | OSTEOSCLEROTIC BONE DYSPLASIA | ANHYDRASE-II DEFICIENCY | CASEIN KINASE | RENAL TUBULAR-ACIDOSIS | Abnormalities, Multiple - metabolism | Calcinosis - genetics | Microcephaly - genetics | Cerebrum - diagnostic imaging | Casein Kinase I - genetics | Osteomalacia - diagnostic imaging | Humans | Calcinosis - diagnostic imaging | Male | Cleft Palate - genetics | Exophthalmos - genetics | Osteosclerosis - metabolism | Adult | Cerebrum - metabolism | Female | Osteomalacia - metabolism | Osteosclerosis - genetics | Calcinosis - metabolism | Cleft Palate - diagnostic imaging | Cerebrum - pathology | Osteosclerosis - diagnostic imaging | Abnormalities, Multiple - genetics | Extracellular Matrix Proteins - metabolism | Infant, Newborn | Extracellular Matrix Proteins - genetics | Microcephaly - metabolism | Abnormalities, Multiple - diagnostic imaging | Exophthalmos - diagnostic imaging | Microcephaly - diagnostic imaging | Osteomalacia - genetics | Exophthalmos - metabolism | Casein Kinase I - metabolism | Cleft Palate - metabolism | Medical research | Dysplasia | Genetic disorders | Calcification | Medicine, Experimental | Genetics | Genetic aspects | Histochemistry | Index Medicus
Journal Article