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Gastroenterology (New York, N.Y. 1943), ISSN 0016-5085, 06/2014, Volume 146, Issue 7, pp. 1763 - 1774
Background & Aims The NACHT, LRR, and pyrin domain–containing protein 3 (NLRP3) inflammasome induces inflammation in response to organ injury, but little is... 
Gastroenterology and Hepatology | Innate Immune Response | Immune Regulation | Pancreas | Mouse Model | Gastroenterology & Hepatology | Life Sciences & Biomedicine | Science & Technology | Liver - pathology | Inflammasomes - metabolism | Receptors, G-Protein-Coupled - metabolism | NLR Family, Pyrin Domain-Containing 3 Protein | Humans | Male | NF-kappa B - metabolism | Monocytes - immunology | Lipopolysaccharides | Liver - immunology | Liver - drug effects | RNA Interference | Interleukin-1beta - metabolism | Toll-Like Receptors - drug effects | Anti-Inflammatory Agents - administration & dosage | Toll-Like Receptors - metabolism | Cytoprotection | Disease Models, Animal | Galactosamine | Chemical and Drug Induced Liver Injury - prevention & control | Anti-Inflammatory Agents - pharmacology | Down-Regulation | Liver - metabolism | Injections, Intraperitoneal | Pancreas - pathology | Pancreas - metabolism | Pancreas - immunology | Toll-Like Receptor 4 - metabolism | Chemical and Drug Induced Liver Injury - immunology | Monocytes - drug effects | Macrophages - metabolism | Signal Transduction - drug effects | Chemical and Drug Induced Liver Injury - metabolism | Sodium Lactate - pharmacology | beta-Arrestins | Mice | Receptors, G-Protein-Coupled - genetics | RNA, Small Interfering - metabolism | Monocytes - metabolism | Pancreatitis - prevention & control | Arrestins - metabolism | Dose-Response Relationship, Drug | Pancreatitis - genetics | Transfection | Inflammasomes - drug effects | Sodium Lactate - administration & dosage | Pancreatitis - immunology | Chemical and Drug Induced Liver Injury - pathology | Chemical and Drug Induced Liver Injury - etiology | Macrophages - immunology | Cell Line | Immunity, Innate - drug effects | Toll-Like Receptor 4 - drug effects | Mice, Inbred C57BL | Pancreas - drug effects | Chemical and Drug Induced Liver Injury - genetics | Pancreatitis - chemically induced | Animals | Carrier Proteins - metabolism | beta-Arrestin 2 | Inflammasomes - immunology | Macrophages - drug effects | Pancreatitis - pathology | Pancreatitis - metabolism | Ceruletide | Lactates | Gastrointestinal diseases | Inflammation | Index Medicus | Abridged Index Medicus
Journal Article
Diabetes (New York, N.Y.), ISSN 0012-1797, 11/2011, Volume 60, Issue 11, pp. 2872 - 2882
OBJECTIVE-To evaluate whether healthy or diabetic adult mice can tolerate an extreme loss of pancreatic a-cells and how this sudden massive depletion affects... 
Life Sciences & Biomedicine | Endocrinology & Metabolism | Science & Technology | Insulin-Secreting Cells - secretion | Apoptosis - drug effects | Cell Count | Glucagon - genetics | Male | Diphtheria Toxin - toxicity | Insulin - blood | Glucagon - blood | Diabetes Mellitus, Experimental - blood | Hypoglycemia - prevention & control | Intercellular Signaling Peptides and Proteins - metabolism | Glucagon-Secreting Cells - drug effects | Insulin-Secreting Cells - metabolism | Hyperglycemia - chemically induced | Glucagon-Secreting Cells - metabolism | Diabetes Mellitus, Experimental - chemically induced | Diabetes Mellitus, Experimental - metabolism | Glucagon-Secreting Cells - secretion | Hyperglycemia - prevention & control | Promoter Regions, Genetic | Signal Transduction | Glucagon-Secreting Cells - pathology | Intercellular Signaling Peptides and Proteins - genetics | Pancreas - drug effects | Pancreas - pathology | Receptors, Glucagon - metabolism | Mice, Transgenic | Pancreas - metabolism | Heparin-binding EGF-like Growth Factor | Insulin - metabolism | Animals | Insulin-Secreting Cells - drug effects | Tamoxifen - pharmacology | Diabetes Mellitus, Experimental - pathology | Glucagon - metabolism | Mice | Streptozocin - toxicity | Insulin-Secreting Cells - pathology | Selective Estrogen Receptor Modulators - pharmacology | Index Medicus | Abridged Index Medicus | Islet Studies
Journal Article
Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 12/2010, Volume 56, Issue 25, pp. 2115 - 2125
Objectives In this study, we have investigated the effects of cannabidiol (CBD) on myocardial dysfunction, inflammation, oxidative/nitrative stress, cell death... 
Cardiovascular | Internal Medicine | diabetic complications | cannabinoids | inflammation | oxidative stress | Cardiac & Cardiovascular Systems | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Diabetic Cardiomyopathies - metabolism | Reactive Oxygen Species - metabolism | Diabetic Cardiomyopathies - drug therapy | Apoptosis - drug effects | Humans | Body Weight - drug effects | Male | NF-kappa B - metabolism | Glucose | Cannabidiol - therapeutic use | Drug Evaluation, Preclinical | Disease Models, Animal | Mice, Inbred C57BL | Cells, Cultured | Pancreas - drug effects | Cannabidiol - pharmacology | Myocardium - pathology | Pancreas - metabolism | Blood Glucose - drug effects | Insulin - metabolism | Animals | MAP Kinase Signaling System - drug effects | Myocytes, Cardiac - drug effects | Fibrosis | Diabetic Cardiomyopathies - pathology | Hemodynamics - drug effects | Mice | Oxidative Stress - drug effects | Phosphates | Oxidative stress | Medical colleges | Niacinamide | Cardiomyopathy | Alcoholism | Radiation | Superoxide | Biochemistry | Dextrose | Purines | Cannabinoids | Cell death | Analysis | Nitric oxide | Protein kinases | Heart diseases | Adenosine triphosphatase | Jewish schools | Mortality | Cardiovascular disease | Cardiomyocytes | Kinases | Experiments | Variance analysis | Antioxidants | Proteins | Polymerase chain reaction | Rodents | Atherosclerosis | Diabetes | Pancreas | Apoptosis | Index Medicus | Abridged Index Medicus
Journal Article
Cell stem cell, ISSN 1934-5909, 02/2014, Volume 14, Issue 2, pp. 237 - 252
Journal Article
Journal Article
PloS one, ISSN 1932-6203, 03/2011, Volume 6, Issue 3, pp. e17850 - e17850
Background: The existence of cancer stem cells (CSCs) or cancer stem-like cells in a tumor mass is believed to be responsible for tumor recurrence because of their intrinsic and extrinsic drug-resistance characteristics... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Curcumin - analogs & derivatives | Humans | Cell Aggregation - drug effects | Deoxycytidine - pharmacology | MicroRNAs - metabolism | NF-kappa B - metabolism | Antineoplastic Agents - therapeutic use | Neoplasm Proteins - metabolism | RNA, Messenger - metabolism | Antineoplastic Combined Chemotherapy Protocols - pharmacology | Epithelial Cell Adhesion Molecule | Pancreatic Neoplasms - drug therapy | Hyaluronan Receptors - metabolism | Antigens, Neoplasm - metabolism | Antineoplastic Agents - pharmacology | Clone Cells | Gene Expression Regulation, Neoplastic - drug effects | Cell Survival - drug effects | PTEN Phosphohydrolase - genetics | Curcumin - therapeutic use | Neoplasm Invasiveness | Pancreatic Neoplasms - pathology | RNA, Messenger - genetics | Curcumin - pharmacology | Pancreatic Neoplasms - enzymology | PTEN Phosphohydrolase - metabolism | Pancreatic Neoplasms - genetics | Reverse Transcriptase Polymerase Chain Reaction | Cell Adhesion Molecules - metabolism | Animals | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | MicroRNAs - genetics | Deoxycytidine - analogs & derivatives | Drug Resistance, Neoplasm - drug effects | Analysis | Oncology, Experimental | Stem cells | Research | Drug resistance | Vascular endothelial growth factor | Tumors | Apoptosis | Cancer | Gemcitabine | Disintegration | Colorectal cancer | Stem cell transplantation | Oncology | Inactivation | Cancer therapies | Anticancer properties | Prevention | Allografts | Antitumor agents | Mouse devices | CD44 antigen | Animal tissues | Xenografts | Inhibition | Pancreas | Drug dosages | Deoxyribonucleic acid--DNA | Killing | Enzymes | NF-κB protein | Deactivation | Hematology | Cloning | Bioavailability | Gene expression | Pathology | Signaling | Chemotherapy | MicroRNAs | Medical prognosis | Pancreatic cancer | Pheochromocytoma cells | Cyclooxygenase-2 | Prostate cancer | PTEN protein | Animal models | Signal transduction | NF- Kappa B protein | DNA | Index Medicus | Deoxyribonucleic acid
Journal Article