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Gut, ISSN 0017-5749, 02/2012, Volume 61, Issue 2, pp. 172 - 178
  In 2006, it was reported that BM is a source of myofibroblast-like cells in fibrotic liver tissue, but the involvement of these cells in the progression of... 
FAT-STORING CELLS | PROGENITOR CELLS | CANCER CELLS | DUCTAL ADENOCARCINOMA | ALCOHOLIC CHRONIC-PANCREATITIS | MOUSE | RATS | LIVER DEVELOPMENT | IDENTIFICATION | GASTROENTEROLOGY & HEPATOLOGY | INDUCE FIBROSIS | Liver Regeneration | Pancreatic Diseases - metabolism | Pancreatic Neoplasms - metabolism | Signal Transduction | Humans | Pancreas - cytology | Pancreatic Neoplasms - pathology | Pancreas - pathology | Pancreatic Stellate Cells - physiology | Pancreatic Diseases - pathology | Pancreatitis, Chronic - pathology | Regeneration | Pancreatic Stellate Cells - cytology | Fibrosis | Liver Cirrhosis - metabolism | Liver Cirrhosis - pathology | Pancreas - physiology | Pancreatic Stellate Cells - pathology | Pancreatitis, Chronic - metabolism | Studies | Rodents | Morphology | Pancreatic cancer | Fibroblasts | Research | Gene expression | molecular biology | chronic pancreatitis | experimental pancreatitis | stellate cells | pancreatic cancer | cancer genetics | Helicobacter pylori | pancreas | cell migration | hepatic surgery | pancreatic surgery | pancreatic physiology | hepatic stellate cell | signalling | Leading | Abdominal surgery | alcohol | pancreatic stellate cells | stem cells | liver | signal transduction | pancreatic pathology | adenocarcinoma | endoscopy | pancreatic enzymes | consensus | pancreatic function | cell biology | pancreatic fibrosis | marker | acute pancreatitis | cancer | fibrosis | pancreatitis | adjuvant treatment | pancreatic disease | hepatic fibrosis | 1506 | extracellular matrix | gene expression
Journal Article
by Bailey, Peter and Chang, David K and Nones, Katia and Johns, Amber L and Patch, Ann-Marie and Gingras, Marie-Claude and Miller, David K and Christ, Angelika N and Bruxner, Tim J. C and Quinn, Michael C and Nourse, Craig and Murtaugh, L. Charles and Harliwong, Ivon and Idrisoglu, Senel and Manning, Suzanne and Nourbakhsh, Ehsan and Wani, Shivangi and Fink, Lynn and Holmes, Oliver and Chin, Venessa and Anderson, Matthew J and Kazakoff, Stephen and Leonard, Conrad and Newell, Felicity and Waddell, Nick and Wood, Scott and Xu, Qinying and Wilson, Peter J and Cloonan, Nicole and Kassahn, Karin S and Taylor, Darrin and Quek, Kelly and Robertson, Alan and Pantano, Lorena and Mincarelli, Laura and Sanchez, Luis N and Evers, Lisa and Wu, Jianmin and Pinese, Mark and Cowley, Mark J and Jones, Marc D and Colvin, Emily K and Nagrial, Adnan M and Humphrey, Emily S and Chantrill, Lorraine A and Mawson, Amanda and Humphris, Jeremy and Chou, Angela and Pajic, Marina and Scarlett, Christopher J and Pinho, Andreia V and Giry-Laterriere, Marc and Rooman, Ilse and Samra, Jaswinder S and Kench, James G and Lovell, Jessica A and Merrett, Neil D and Toon, Christopher W and Epari, Krishna and Nguyen, Nam Q and Barbour, Andrew and Zeps, Nikolajs and Moran-Jones, Kim and Jamieson, Nigel B and Graham, Janet S and Duthie, Fraser and Oien, Karin and Hair, Jane and Grützmann, Robert and Maitra, Anirban and Iacobuzio-Donahue, Christine A and Wolfgang, Christopher L and Morgan, Richard A and Lawlor, Rita T and Corbo, Vincenzo and Bassi, Claudio and Rusev, Borislav and Capelli, Paola and Salvia, Roberto and Tortora, Giampaolo and Mukhopadhyay, Debabrata and Petersen, Gloria M and Munzy, Donna M and Fisher, William E and Karim, Saadia A and Eshleman, James R and Hruban, Ralph H and Pilarsky, Christian and Morton, Jennifer P and Sansom, Owen J and Scarpa, Aldo and Musgrove, Elizabeth A and Bailey, Ulla-Maja Hagbo and Hofmann, Oliver and Sutherland, Robert L and Wheeler, David A and Gill, Anthony J and Gibbs, Richard A and Pearson, John V and Waddell, Nicola and ... and Australian Pancreatic Canc Genome and Australian Pancreatic Cancer Genome Initiative
Nature, ISSN 0028-0836, 03/2016, Volume 531, Issue 7592, pp. 47 - 52
Integrated genomic analysis of 456 pancreatic ductal adenocarcinomas identified 32 recurrently mutated genes that aggregate into 10 pathways: KRAS, TGF-beta,... 
PATHWAYS | METASTASIS | DUCTAL ADENOCARCINOMA | PACKAGE | MULTIDISCIPLINARY SCIENCES | TUMOR | MUTATIONS | DIFFERENTIATION | Pancreatic Neoplasms - metabolism | Prognosis | Hepatocyte Nuclear Factor 3-beta - genetics | Genomics | Humans | Carcinoma, Pancreatic Ductal - metabolism | Gene Expression Regulation, Neoplastic | Transcriptome | Hepatocyte Nuclear Factor 3-gamma - genetics | Gene Regulatory Networks | Histone Demethylases - genetics | Tumor Suppressor Protein p53 - genetics | Carcinoma, Pancreatic Ductal - genetics | DNA Methylation | Tumor Suppressor Proteins - genetics | Carcinoma, Pancreatic Ductal - classification | Trans-Activators - genetics | Pancreatic Neoplasms - classification | Transcription, Genetic | Nuclear Proteins - genetics | Carcinoma, Pancreatic Ductal - immunology | Genes, Neoplasm - genetics | Basic Helix-Loop-Helix Transcription Factors - genetics | Pancreatic Neoplasms - pathology | Pancreatic Neoplasms - genetics | Receptors, Cytoplasmic and Nuclear - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Mutation - genetics | Carcinoma, Pancreatic Ductal - pathology | Genome, Human - genetics | Homeodomain Proteins - genetics | Animals | Pancreatic Neoplasms - immunology | Survival Analysis | Cell Line, Tumor | Mice | Physiological aspects | Pancreatic cancer | Methods | Genes | Genomes | Mutation | Gene expression | Tumors
Journal Article
Pancreas, ISSN 0885-3177, 07/2015, Volume 44, Issue 5, pp. 693 - 712
Journal Article
Gut, ISSN 0017-5749, 01/2013, Volume 62, Issue 1, pp. 112 - 120
Objective Pancreatic ductal adenocarcinoma (PDA) is characterised by stromal desmoplasia and vascular dysfunction, which critically impair drug delivery. This... 
INTERSTITIAL FLUID PRESSURE | CELLS | THERAPY | SOLID TUMORS | MICROENVIRONMENT | PERMEABILITY | DUCTAL ADENOCARCINOMA | GASTROENTEROLOGY & HEPATOLOGY | GEMCITABINE | CHEMOTHERAPY | ENDOTHELIAL GROWTH-FACTOR | Immunohistochemistry | Cell Adhesion Molecules - administration & dosage | Tissue Array Analysis | Pancreatic Neoplasms - blood supply | Antineoplastic Agents - administration & dosage | Hyaluronic Acid - physiology | Antineoplastic Combined Chemotherapy Protocols - pharmacology | Drug Delivery Systems | Pancreatic Neoplasms - drug therapy | Hyaluronoglucosaminidase - administration & dosage | Carcinoma, Pancreatic Ductal - mortality | Cell Adhesion Molecules - pharmacology | Pancreatic Neoplasms - mortality | Doxorubicin - administration & dosage | Deoxycytidine - administration & dosage | Kaplan-Meier Estimate | Mice, Transgenic | Treatment Outcome | Biomarkers, Tumor - physiology | Recombinant Proteins - pharmacology | Recombinant Proteins - administration & dosage | Hyaluronoglucosaminidase - pharmacology | Carcinoma, Pancreatic Ductal - drug therapy | Animals | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Carcinoma, Pancreatic Ductal - physiopathology | Mice | Carcinoma, Pancreatic Ductal - blood supply | Pancreatic Neoplasms - physiopathology | Drug Resistance, Neoplasm - physiology | Deoxycytidine - analogs & derivatives | Drug Resistance, Neoplasm - drug effects | Care and treatment | Blood-vessels | Pancreatic cancer | Patient outcomes | Physiological aspects | Models | Research | Hyaluronic acid | Tumors | Studies | Angiogenesis | Medical research | Medical prognosis | Cytotoxicity | Extracellular matrix | Permeability | Kinases | Vascular endothelial growth factor | stem cells | cell death | cancer vaccines | hyaluronan | epithelial cell growth | cancer immunobiology | drug delivery | matrix | epithelial cell adhesion | cancer genetics | carcinogenesis | pancreatic fibrosis | pharmacology | pharmacokinetics | tumour microenvironment | cancer | fibrosis | pancreatic disease | Original | 1506 | pancreatic tumours | extracellular matrix | oxidative stress
Journal Article
Gastroenterology, ISSN 0016-5085, 2012, Volume 142, Issue 4, pp. 796 - 804
Journal Article
Gut, ISSN 0017-5749, 12/2018, Volume 67, Issue 12, pp. 2131 - 2141
ObjectiveDNA-based testing of pancreatic cyst fluid (PCF) is a useful adjunct to the evaluation of pancreatic cysts (PCs). Mutations in KRAS/GNAS are highly... 
pancreatic cancer | pancreato-biliary disorders | pancreatic epidemiology | pancreatic pathology | PAPILLARY MUCINOUS NEOPLASMS | DIAGNOSIS | PIK3CA MUTATIONS | MANAGEMENT | GENES | FUKUOKA CONSENSUS GUIDELINES | GASTROENTEROLOGY & HEPATOLOGY | PRECURSORS | CANCER | FEATURES | Neoplasms, Cystic, Mucinous, and Serous - surgery | Prospective Studies | Follow-Up Studies | Pancreatic Neoplasms - diagnosis | Proto-Oncogene Proteins p21(ras) - genetics | Humans | Middle Aged | Male | Carcinoma, Pancreatic Ductal - surgery | Carcinoma, Pancreatic Ductal - genetics | GTP-Binding Protein alpha Subunits, Gs - genetics | Neoplasms, Cystic, Mucinous, and Serous - genetics | Young Adult | Pancreatic Cyst - pathology | Pancreatic Cyst - surgery | Sensitivity and Specificity | Aged, 80 and over | Carcinoma, Pancreatic Ductal - diagnosis | Adult | Chromogranins - genetics | Female | High-Throughput Nucleotide Sequencing - methods | Neoplasm Proteins - genetics | Endoscopic Ultrasound-Guided Fine Needle Aspiration | Cyst Fluid - chemistry | Pancreatic Neoplasms - pathology | Pancreatic Neoplasms - surgery | Pancreatic Cyst - diagnosis | Pancreatic Neoplasms - genetics | Pancreatic Cyst - genetics | Preoperative Care | Adolescent | Aged | Biomarkers, Tumor - genetics | DNA, Neoplasm - genetics | Mutation | Neoplasms, Cystic, Mucinous, and Serous - diagnosis | Antigens | Laboratories | p53 Protein | Studies | Pathology | Next-generation sequencing | Cysts | Pancreatic cancer | Cytopathology | Clinical medicine | Endoscopy | Pancreas | Ultrasound | Deoxyribonucleic acid--DNA | PTEN protein | Tumors | 2312 | 1506
Journal Article
Journal Article
Gut, ISSN 0017-5749, 07/2013, Volume 62, Issue 7, pp. 1024 - 1033
Objective Pancreatic cysts are commonly detected in patients undergoing pancreatic imaging. Better approaches are needed to characterise these lesions. In this... 
PAPILLARY MUCINOUS NEOPLASMS | HIGH-RISK INDIVIDUALS | K-RAS MUTATIONS | METHYLATION | EARLY-DIAGNOSIS | TELOMERASE ACTIVITY | SAMPLES | PREVALENCE | FAMILY-HISTORY | GASTROENTEROLOGY & HEPATOLOGY | CANCER | Early Detection of Cancer - methods | Pancreatic Neoplasms - diagnosis | Humans | Middle Aged | Male | Secretin | Case-Control Studies | Carcinoma, Pancreatic Ductal - genetics | GTP-Binding Protein alpha Subunits, Gs - genetics | Chromogranins | Neoplasm Proteins - secretion | Pancreatic Cyst - pathology | GTP-Binding Protein alpha Subunits, Gs - secretion | Duodenum - metabolism | Aged, 80 and over | Carcinoma, Pancreatic Ductal - diagnosis | Pancreatic Juice - metabolism | Female | Neoplasm Proteins - genetics | Genetic Predisposition to Disease | Pancreatic Neoplasms - pathology | Pancreatic Cyst - diagnosis | Pancreatic Neoplasms - genetics | Carcinoma, Pancreatic Ductal - pathology | Pancreatic Cyst - genetics | Aged | Biomarkers, Tumor - genetics | DNA, Neoplasm - genetics | Mutation | Early Diagnosis | Genetic aspects | Diagnosis | Research | Gene mutations | Pancreatic cancer | Studies | Hogs | Ultrasonic imaging | Surveillance | Cysts | Rodents | Family medical history | Endoscopy | Deoxyribonucleic acid--DNA | Tumors | GNAS | pancreatic juice | intraductal papillary mucinous neoplasm | pancreatic cyst
Journal Article
World Journal of Gastroenterology, ISSN 1007-9327, 01/2017, Volume 23, Issue 3, pp. 382 - 405
Journal Article