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1. Full Text StellaTUM: Current consensus and discussion on pancreatic stellate cell research
by Erkan, Mert and Adler, Guido and Apte, Minoti V and Bachem, Max G and Buchholz, Malte and Detlefsen, Sönke and Esposito, Irene and Friess, Helmut and Gress, Thomas M and Habisch, Hans-Joerg and Hwang, Rosa F and Jaster, Robert and Kleeff, Jörg and Klöppel, Günter and Kordes, Claus and Logsdon, Craig D and Masamune, Atsushi and Michalski, Christoph W and Oh, Junseo and Phillips, Phoebe A and Pinzani, Massimo and Reiser-Erkan, Carolin and Tsukamoto, Hidekazu and Wilson, Jeremy
Gut, ISSN 0017-5749, 02/2012, Volume 61, Issue 2, pp. 172 - 178
  In 2006, it was reported that BM is a source of myofibroblast-like cells in fibrotic liver tissue, but the involvement of these cells in the progression of... 
FAT-STORING CELLS | PROGENITOR CELLS | CANCER CELLS | DUCTAL ADENOCARCINOMA | ALCOHOLIC CHRONIC-PANCREATITIS | MOUSE | RATS | LIVER DEVELOPMENT | IDENTIFICATION | GASTROENTEROLOGY & HEPATOLOGY | INDUCE FIBROSIS | Liver Regeneration | Pancreatic Diseases - metabolism | Pancreatic Neoplasms - metabolism | Signal Transduction | Humans | Pancreas - cytology | Pancreatic Neoplasms - pathology | Pancreas - pathology | Pancreatic Stellate Cells - physiology | Pancreatic Diseases - pathology | Pancreatitis, Chronic - pathology | Regeneration | Pancreatic Stellate Cells - cytology | Fibrosis | Liver Cirrhosis - metabolism | Liver Cirrhosis - pathology | Pancreas - physiology | Pancreatic Stellate Cells - pathology | Pancreatitis, Chronic - metabolism | Studies | Rodents | Morphology | Pancreatic cancer | Fibroblasts | Research | Gene expression | molecular biology | chronic pancreatitis | experimental pancreatitis | stellate cells | pancreatic cancer | cancer genetics | Helicobacter pylori | pancreas | cell migration | hepatic surgery | pancreatic surgery | pancreatic physiology | hepatic stellate cell | signalling | Leading | Abdominal surgery | alcohol | pancreatic stellate cells | stem cells | liver | signal transduction | pancreatic pathology | adenocarcinoma | endoscopy | pancreatic enzymes | consensus | pancreatic function | cell biology | pancreatic fibrosis | marker | acute pancreatitis | cancer | fibrosis | pancreatitis | adjuvant treatment | pancreatic disease | hepatic fibrosis | 1506 | extracellular matrix | gene expression
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2. Full Text Genomic analyses identify molecular subtypes of pancreatic cancer
by Bailey, Peter and Chang, David K and Nones, Katia and Johns, Amber L and Patch, Ann-Marie and Gingras, Marie-Claude and Miller, David K and Christ, Angelika N and Bruxner, Tim J. C and Quinn, Michael C and Nourse, Craig and Murtaugh, L. Charles and Harliwong, Ivon and Idrisoglu, Senel and Manning, Suzanne and Nourbakhsh, Ehsan and Wani, Shivangi and Fink, Lynn and Holmes, Oliver and Chin, Venessa and Anderson, Matthew J and Kazakoff, Stephen and Leonard, Conrad and Newell, Felicity and Waddell, Nick and Wood, Scott and Xu, Qinying and Wilson, Peter J and Cloonan, Nicole and Kassahn, Karin S and Taylor, Darrin and Quek, Kelly and Robertson, Alan and Pantano, Lorena and Mincarelli, Laura and Sanchez, Luis N and Evers, Lisa and Wu, Jianmin and Pinese, Mark and Cowley, Mark J and Jones, Marc D and Colvin, Emily K and Nagrial, Adnan M and Humphrey, Emily S and Chantrill, Lorraine A and Mawson, Amanda and Humphris, Jeremy and Chou, Angela and Pajic, Marina and Scarlett, Christopher J and Pinho, Andreia V and Giry-Laterriere, Marc and Rooman, Ilse and Samra, Jaswinder S and Kench, James G and Lovell, Jessica A and Merrett, Neil D and Toon, Christopher W and Epari, Krishna and Nguyen, Nam Q and Barbour, Andrew and Zeps, Nikolajs and Moran-Jones, Kim and Jamieson, Nigel B and Graham, Janet S and Duthie, Fraser and Oien, Karin and Hair, Jane and Grützmann, Robert and Maitra, Anirban and Iacobuzio-Donahue, Christine A and Wolfgang, Christopher L and Morgan, Richard A and Lawlor, Rita T and Corbo, Vincenzo and Bassi, Claudio and Rusev, Borislav and Capelli, Paola and Salvia, Roberto and Tortora, Giampaolo and Mukhopadhyay, Debabrata and Petersen, Gloria M and Munzy, Donna M and Fisher, William E and Karim, Saadia A and Eshleman, James R and Hruban, Ralph H and Pilarsky, Christian and Morton, Jennifer P and Sansom, Owen J and Scarpa, Aldo and Musgrove, Elizabeth A and Bailey, Ulla-Maja Hagbo and Hofmann, Oliver and Sutherland, Robert L and Wheeler, David A and Gill, Anthony J and Gibbs, Richard A and Pearson, John V and Waddell, Nicola and ... and Australian Pancreatic Canc Genome and Australian Pancreatic Cancer Genome Initiative
Nature, ISSN 0028-0836, 03/2016, Volume 531, Issue 7592, pp. 47 - 52
Integrated genomic analysis of 456 pancreatic ductal adenocarcinomas identified 32 recurrently mutated genes that aggregate into 10 pathways: KRAS, TGF-beta,... 
PATHWAYS | METASTASIS | DUCTAL ADENOCARCINOMA | PACKAGE | MULTIDISCIPLINARY SCIENCES | TUMOR | MUTATIONS | DIFFERENTIATION | Pancreatic Neoplasms - metabolism | Prognosis | Hepatocyte Nuclear Factor 3-beta - genetics | Genomics | Humans | Carcinoma, Pancreatic Ductal - metabolism | Gene Expression Regulation, Neoplastic | Transcriptome | Hepatocyte Nuclear Factor 3-gamma - genetics | Gene Regulatory Networks | Histone Demethylases - genetics | Tumor Suppressor Protein p53 - genetics | Carcinoma, Pancreatic Ductal - genetics | DNA Methylation | Tumor Suppressor Proteins - genetics | Carcinoma, Pancreatic Ductal - classification | Trans-Activators - genetics | Pancreatic Neoplasms - classification | Transcription, Genetic | Nuclear Proteins - genetics | Carcinoma, Pancreatic Ductal - immunology | Genes, Neoplasm - genetics | Basic Helix-Loop-Helix Transcription Factors - genetics | Pancreatic Neoplasms - pathology | Pancreatic Neoplasms - genetics | Receptors, Cytoplasmic and Nuclear - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Mutation - genetics | Carcinoma, Pancreatic Ductal - pathology | Genome, Human - genetics | Homeodomain Proteins - genetics | Animals | Pancreatic Neoplasms - immunology | Survival Analysis | Cell Line, Tumor | Mice | Physiological aspects | Pancreatic cancer | Methods | Genes | Genomes | Mutation | Gene expression | Tumors
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3. Full Text Early Detection of Sporadic Pancreatic Cancer: Summative Review
by Chari, Suresh T and Kelly, Kimberly and Hollingsworth, Michael A and Thayer, Sarah P and Ahlquist, David A and Andersen, Dana K and Batra, Surinder K and Brentnall, Teresa A and Canto, Marcia and Cleeter, Deborah F and Firpo, Matthew A and Gambhir, Sanjiv Sam and Go, Vay Liang W and Hines, O. Joe and Kenner, Barbara J and Klimstra, David S and Lerch, Markus M and Levy, Michael J and Maitra, Anirban and Mulvihill, Sean J and Petersen, Gloria M and Rhim, Andrew D and Simeone, Diane M and Srivastava, Sudhir and Tanaka, Masao and Vinik, Aaron I and Wong, David
Pancreas, ISSN 0885-3177, 07/2015, Volume 44, Issue 5, pp. 693 - 712
Pancreatic cancer (PC) is estimated to become the second leading cause of cancer death in the United States by 2020. Early detection is the key to improving... 
familial | screening | biomarker | diabetes | imaging | pancreatic ductal adenocarcinoma | GASTROENTEROLOGY & HEPATOLOGY | Predictive Value of Tests | Pancreatic Neoplasms - metabolism | Prognosis | Early Detection of Cancer - methods | Pancreatic Neoplasms - diagnosis | Humans | Carcinoma, Pancreatic Ductal - metabolism | Biomedical Research - organization & administration | Carcinoma, Pancreatic Ductal - genetics | Molecular Diagnostic Techniques | Biomarkers, Tumor - metabolism | Carcinoma, Pancreatic Ductal - diagnosis | Carcinoma, Pancreatic Ductal - mortality | Pancreatic Neoplasms - mortality | Congresses as Topic | Pancreatic Neoplasms - pathology | Cooperative Behavior | Pancreatic Neoplasms - genetics | Carcinoma, Pancreatic Ductal - therapy | International Cooperation | Carcinoma, Pancreatic Ductal - pathology | Diagnostic Imaging | Interdisciplinary Communication | Biopsy | Survival Analysis | Diffusion of Innovation | Biomarkers, Tumor - genetics | Pancreatic Neoplasms - therapy | Reviews
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4. Full Text Hyaluronan impairs vascular function and drug delivery in a mouse model of pancreatic cancer
by Jacobetz, Michael A and Chan, Derek S and Neesse, Albrecht and Bapiro, Tashinga E and Cook, Natalie and Frese, Kristopher K and Feig, Christine and Nakagawa, Tomoaki and Caldwell, Meredith E and Zecchini, Heather I and Lolkema, Martijn P and Jiang, Ping and Kultti, Anne and Thompson, Curtis B and Maneval, Daniel C and Jodrell, Duncan I and Frost, Gregory I and Shepard, H M and Skepper, Jeremy N and Tuveson, David A
Gut, ISSN 0017-5749, 01/2013, Volume 62, Issue 1, pp. 112 - 120
Objective Pancreatic ductal adenocarcinoma (PDA) is characterised by stromal desmoplasia and vascular dysfunction, which critically impair drug delivery. This... 
INTERSTITIAL FLUID PRESSURE | CELLS | THERAPY | SOLID TUMORS | MICROENVIRONMENT | PERMEABILITY | DUCTAL ADENOCARCINOMA | GASTROENTEROLOGY & HEPATOLOGY | GEMCITABINE | CHEMOTHERAPY | ENDOTHELIAL GROWTH-FACTOR | Immunohistochemistry | Cell Adhesion Molecules - administration & dosage | Tissue Array Analysis | Pancreatic Neoplasms - blood supply | Antineoplastic Agents - administration & dosage | Hyaluronic Acid - physiology | Antineoplastic Combined Chemotherapy Protocols - pharmacology | Drug Delivery Systems | Pancreatic Neoplasms - drug therapy | Hyaluronoglucosaminidase - administration & dosage | Carcinoma, Pancreatic Ductal - mortality | Cell Adhesion Molecules - pharmacology | Pancreatic Neoplasms - mortality | Doxorubicin - administration & dosage | Deoxycytidine - administration & dosage | Kaplan-Meier Estimate | Mice, Transgenic | Treatment Outcome | Biomarkers, Tumor - physiology | Recombinant Proteins - pharmacology | Recombinant Proteins - administration & dosage | Hyaluronoglucosaminidase - pharmacology | Carcinoma, Pancreatic Ductal - drug therapy | Animals | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Carcinoma, Pancreatic Ductal - physiopathology | Mice | Carcinoma, Pancreatic Ductal - blood supply | Pancreatic Neoplasms - physiopathology | Drug Resistance, Neoplasm - physiology | Deoxycytidine - analogs & derivatives | Drug Resistance, Neoplasm - drug effects | Care and treatment | Blood-vessels | Pancreatic cancer | Patient outcomes | Physiological aspects | Models | Research | Hyaluronic acid | Tumors | Studies | Angiogenesis | Medical research | Medical prognosis | Cytotoxicity | Extracellular matrix | Permeability | Kinases | Vascular endothelial growth factor | stem cells | cell death | cancer vaccines | hyaluronan | epithelial cell growth | cancer immunobiology | drug delivery | matrix | epithelial cell adhesion | cancer genetics | carcinogenesis | pancreatic fibrosis | pharmacology | pharmacokinetics | tumour microenvironment | cancer | fibrosis | pancreatic disease | Original | 1506 | pancreatic tumours | extracellular matrix | oxidative stress
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5. Full Text Frequent Detection of Pancreatic Lesions in Asymptomatic High-Risk Individuals
by Canto, Marcia Irene and Hruban, Ralph H and Fishman, Elliot K and Kamel, Ihab R and Schulick, Richard and Zhang, Zhe and Topazian, Mark and Takahashi, Naoki and Fletcher, Joel and Petersen, Gloria and Klein, Alison P and Axilbund, Jennifer and Griffin, Constance and Syngal, Sapna and Saltzman, John R and Mortele, Koenraad J and Lee, Jeffrey and Tamm, Eric and Vikram, Raghunandan and Bhosale, Priya and Margolis, Daniel and Farrell, James and Goggins, Michael and Amer Canc Pancreas Screening CAPS and American Cancer of the Pancreas Screening (CAPS) Consortium
Gastroenterology, ISSN 0016-5085, 2012, Volume 142, Issue 4, pp. 796 - 804
Background & Aims The risk of pancreatic cancer is increased in patients with a strong family history of pancreatic cancer or a predisposing germline mutation.... 
Gastroenterology and Hepatology | IPMN | Surveillance | Familial Pancreatic Cancer | PanIN | DIAGNOSIS | BRCA2 MUTATIONS | UPDATED EXPERIENCE | CONSENSUS | CANCER KINDREDS | PREVALENCE | FAMILY-HISTORY | PALB2 | PAPILLARY MUCINOUS NEOPLASMS | ENDOSCOPIC ULTRASONOGRAPHY | GASTROENTEROLOGY & HEPATOLOGY | Multivariate Analysis | Predictive Value of Tests | United States - epidemiology | Pancreatic Neoplasms - epidemiology | Prevalence | Prognosis | Prospective Studies | Endosonography | Pancreatic Neoplasms - diagnosis | Humans | Middle Aged | Male | Tomography, X-Ray Computed | Asymptomatic Diseases | Diagnostic Imaging - methods | Pancreatic Cyst - pathology | Pancreatic Cyst - surgery | Pancreatic Cyst - epidemiology | Female | Genetic Predisposition to Disease | Risk Assessment | Pancreatic Neoplasms - pathology | Pancreatic Neoplasms - surgery | Risk Factors | Logistic Models | Pancreatic Cyst - diagnosis | Pancreatic Neoplasms - genetics | Heredity | Academic Medical Centers | Chi-Square Distribution | Pancreatic Cyst - genetics | Magnetic Resonance Imaging | Pedigree | Aged | CT imaging | Oncology, Experimental | Melanoma | Research | Biometry | Risk factors | Gene mutations | Pancreatic cancer | Universities and colleges | Diagnosis | Health aspects | Cancer | Tumors
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6. Full Text Preoperative next-generation sequencing of pancreatic cyst fluid is highly accurate in cyst classification and detection of advanced neoplasia
by Singhi, Aatur D and McGrath, Kevin and Brand, Randall E and Khalid, Asif and Zeh, Herbert J and Chennat, Jennifer S and Fasanella, Kenneth E and Papachristou, Georgios I and Slivka, Adam and Bartlett, David L and Dasyam, Anil K and Hogg, Melissa and Lee, Kenneth K and Marsh, James Wallis and Monaco, Sara E and Ohori, N Paul and Pingpank, James F and Tsung, Allan and Zureikat, Amer H and Wald, Abigail I and Nikiforova, Marina N
Gut, ISSN 0017-5749, 12/2018, Volume 67, Issue 12, pp. 2131 - 2141
ObjectiveDNA-based testing of pancreatic cyst fluid (PCF) is a useful adjunct to the evaluation of pancreatic cysts (PCs). Mutations in KRAS/GNAS are highly... 
pancreatic cancer | pancreato-biliary disorders | pancreatic epidemiology | pancreatic pathology | PAPILLARY MUCINOUS NEOPLASMS | DIAGNOSIS | PIK3CA MUTATIONS | MANAGEMENT | GENES | FUKUOKA CONSENSUS GUIDELINES | GASTROENTEROLOGY & HEPATOLOGY | PRECURSORS | CANCER | FEATURES | Neoplasms, Cystic, Mucinous, and Serous - surgery | Prospective Studies | Follow-Up Studies | Pancreatic Neoplasms - diagnosis | Proto-Oncogene Proteins p21(ras) - genetics | Humans | Middle Aged | Male | Carcinoma, Pancreatic Ductal - surgery | Carcinoma, Pancreatic Ductal - genetics | GTP-Binding Protein alpha Subunits, Gs - genetics | Neoplasms, Cystic, Mucinous, and Serous - genetics | Young Adult | Pancreatic Cyst - pathology | Pancreatic Cyst - surgery | Sensitivity and Specificity | Aged, 80 and over | Carcinoma, Pancreatic Ductal - diagnosis | Adult | Chromogranins - genetics | Female | High-Throughput Nucleotide Sequencing - methods | Neoplasm Proteins - genetics | Endoscopic Ultrasound-Guided Fine Needle Aspiration | Cyst Fluid - chemistry | Pancreatic Neoplasms - pathology | Pancreatic Neoplasms - surgery | Pancreatic Cyst - diagnosis | Pancreatic Neoplasms - genetics | Pancreatic Cyst - genetics | Preoperative Care | Adolescent | Aged | Biomarkers, Tumor - genetics | DNA, Neoplasm - genetics | Mutation | Neoplasms, Cystic, Mucinous, and Serous - diagnosis | Antigens | Laboratories | p53 Protein | Studies | Pathology | Next-generation sequencing | Cysts | Pancreatic cancer | Cytopathology | Clinical medicine | Endoscopy | Pancreas | Ultrasound | Deoxyribonucleic acid--DNA | PTEN protein | Tumors | 2312 | 1506
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7. Full Text Advances in pancreatic cancer research: Moving towards early detection
by Xiang-Yi He Yao-Zong Yuan
世界胃肠病学杂志：英文版（电子版）, ISSN 1007-9327, 2014, Volume 20, Issue 32, pp. 11241 - 11248
Pancreatic ductal adenocarcinoma(PDAC) is one of the most lethal forms of cancer. Substantial progress has been made in the understanding of the biology of... 
ductal | adenocarcinoma | detection | D | early | Pancreatic | Diabetes mellitus | Pancreatic cancer | Pancreatic ductal adenocarcinoma | Early detection | DIAGNOSTIC-VALUE | DUCTAL ADENOCARCINOMA | SERUM BIOMARKER | DJ-1 | INTRAEPITHELIAL NEOPLASIA | DIABETES-MELLITUS | INVASION | CIRCULATING TUMOR-CELLS | PROTEOMIC ANALYSIS | GASTROENTEROLOGY & HEPATOLOGY | MICRORNA | Pancreatic Neoplasms - blood | Predictive Value of Tests | Genetic Testing | Early Detection of Cancer - methods | Pancreatic Neoplasms - diagnosis | Humans | Pancreatic Neoplasms - pathology | Pancreatic Neoplasms - surgery | Risk Factors | Pancreatic Neoplasms - genetics | Carcinoma, Pancreatic Ductal - pathology | Carcinoma, Pancreatic Ductal - surgery | Carcinoma, Pancreatic Ductal - genetics | Biomarkers, Tumor - blood | Carcinoma, Pancreatic Ductal - blood | Diabetes Mellitus - epidemiology | Carcinoma, Pancreatic Ductal - diagnosis | Carcinoma, Pancreatic Ductal - mortality | Biomarkers, Tumor - genetics | Pancreatic Neoplasms - mortality | Neoplasm Staging | Topic Highlight
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8. Full Text Mutant GNAS detected in duodenal collections of secretin-stimulated pancreatic juice indicates the presence or emergence of pancreatic cysts
by Kanda, Mitsuro and Knight, Spencer and Topazian, Mark and Syngal, Sapna and Farrell, James and Lee, Jeffrey and Kamel, Ihab and Lennon, Anne Marie and Borges, Michael and Young, Angela and Fujiwara, Sho and Seike, Junro and Eshleman, James and Hruban, Ralph H and Canto, Marcia Irene and Goggins, Michael
Gut, ISSN 0017-5749, 07/2013, Volume 62, Issue 7, pp. 1024 - 1033
Objective Pancreatic cysts are commonly detected in patients undergoing pancreatic imaging. Better approaches are needed to characterise these lesions. In this... 
PAPILLARY MUCINOUS NEOPLASMS | HIGH-RISK INDIVIDUALS | K-RAS MUTATIONS | METHYLATION | EARLY-DIAGNOSIS | TELOMERASE ACTIVITY | SAMPLES | PREVALENCE | FAMILY-HISTORY | GASTROENTEROLOGY & HEPATOLOGY | CANCER | Early Detection of Cancer - methods | Pancreatic Neoplasms - diagnosis | Humans | Middle Aged | Male | Secretin | Case-Control Studies | Carcinoma, Pancreatic Ductal - genetics | GTP-Binding Protein alpha Subunits, Gs - genetics | Chromogranins | Neoplasm Proteins - secretion | Pancreatic Cyst - pathology | GTP-Binding Protein alpha Subunits, Gs - secretion | Duodenum - metabolism | Aged, 80 and over | Carcinoma, Pancreatic Ductal - diagnosis | Pancreatic Juice - metabolism | Female | Neoplasm Proteins - genetics | Genetic Predisposition to Disease | Pancreatic Neoplasms - pathology | Pancreatic Cyst - diagnosis | Pancreatic Neoplasms - genetics | Carcinoma, Pancreatic Ductal - pathology | Pancreatic Cyst - genetics | Aged | Biomarkers, Tumor - genetics | DNA, Neoplasm - genetics | Mutation | Early Diagnosis | Genetic aspects | Diagnosis | Research | Gene mutations | Pancreatic cancer | Studies | Hogs | Ultrasonic imaging | Surveillance | Cysts | Rodents | Family medical history | Endoscopy | Deoxyribonucleic acid--DNA | Tumors | GNAS | pancreatic juice | intraductal papillary mucinous neoplasm | pancreatic cyst
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9. Full Text Pancreatic stellate cell: Pandora's box for pancreatic disease biology
by Bynigeri, Ratnakar R and Jakkampudi, Aparna and Jangala, Ramaiah and Subramanyam, Chivukula and Sasikala, Mitnala and Rao, G. Venkat and Reddy, D. Nageshwar and Talukdar, Rupjyoti
World Journal of Gastroenterology, ISSN 1007-9327, 01/2017, Volume 23, Issue 3, pp. 382 - 405
Pancreatic stellate cells (PSCs) were identified in the early 1980s, but received much attention after 1998 when the methods to isolate and culture them from... 
Pancreatic fibrosis | Pancreatic stellate cells | CONVERTING ENZYME-INHIBITOR | CANCER-CELLS | Pancreatic cancer stroma | EXTRACELLULAR-MATRIX PRODUCTION | SUPPRESSES CHRONIC-PANCREATITIS | TRANSFORMING GROWTH FACTOR-BETA | Physiological functions | Therapeutic targets | ACTIVATES ACTIVATOR PROTEIN-1 | EPITHELIAL-MESENCHYMAL TRANSITION | Pancreatic stellate cells-cancer-stromal interactions | ERK-DEPENDENT PATHWAYS | CATENIN SIGNALING PATHWAY | GASTROENTEROLOGY & HEPATOLOGY | TRINITROBENZENE SULFONIC-ACID | Pancreatitis, Chronic - immunology | Cytokines - metabolism | Signal Transduction | Neoplasm Invasiveness | Humans | Pancreatic Stellate Cells - drug effects | Pancreas, Exocrine - pathology | Tumor Microenvironment | MicroRNAs - metabolism | Pancreatitis, Chronic - physiopathology | Pancreatic Neoplasms - drug therapy | Carcinoma, Pancreatic Ductal - drug therapy | Animals | Pancreas, Exocrine - cytology | Pancreatic Stellate Cells - immunology | Pancreatic Neoplasms - immunology | Pancreatitis, Chronic - drug therapy | Fibrosis | Carcinoma, Pancreatic Ductal - physiopathology | Pancreatic Neoplasms - physiopathology | Pancreatic Stellate Cells - pathology | Carcinoma, Pancreatic Ductal - immunology | Extracellular Matrix Proteins - metabolism | Pancreatic Stellate Cells - metabolism | Review
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