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1. Full Text β-cells are not generated in pancreatic duct ligation-induced injury in adult mice
by Rankin, Matthew M and Wilbur, Christopher J and Rak, Kimberly and Shields, Emily J and Granger, Anne and Kushner, Jake A
Diabetes (New York, N.Y.), ISSN 0012-1797, 05/2013, Volume 62, Issue 5, pp. 1634 - 1645
Life Sciences & Biomedicine | Endocrinology & Metabolism | Science & Technology | Biological and medical sciences | Endocrinopathies | Etiopathogenesis. Screening. Investigations. Target tissue resistance | Diabetes. Impaired glucose tolerance | Medical sciences | Endocrine pancreas. Apud cells (diseases) | Cell Proliferation | Pancreas - cytology | RNA, Messenger - metabolism | Recombinant Fusion Proteins - metabolism | Insulin-Secreting Cells - metabolism | Ligation | Basic Helix-Loop-Helix Transcription Factors - metabolism | Cell Differentiation | Insulin-Secreting Cells - cytology | Insulin - genetics | Genes, Reporter | Adult Stem Cells - cytology | Basic Helix-Loop-Helix Transcription Factors - genetics | Gene Expression Regulation | Pancreatic Ducts - surgery | Mice, Transgenic | Pancreas - metabolism | Nerve Tissue Proteins - genetics | Nerve Tissue Proteins - metabolism | Cell Lineage | Adult Stem Cells - metabolism | Insulin - metabolism | Animals | Biomarkers | Mice | Crosses, Genetic | Index Medicus | Abridged Index Medicus | Original Research
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2. Full Text GLI1 is regulated through Smoothened-independent mechanisms in neoplastic pancreatic ducts and mediates PDAC cell survival and transformation
by Nolan-Stevaux, Olivier and Lau, Janet and Truitt, Morgan L and Chu, Gerald C and Hebrok, Matthias and Fernández-Zapico, Martin E and Hanahan, Douglas
Genes & development, ISSN 0890-9369, 01/2009, Volume 23, Issue 1, pp. 24 - 36
Hedgehog | Smoothened | PDAC | Gli | Pancreatic cancer | Pancreatic ductal adenocarcinoma | Genetics & Heredity | Life Sciences & Biomedicine | Developmental Biology | Science & Technology | Cell Biology | Pancreatic Neoplasms - metabolism | Receptors, G-Protein-Coupled - metabolism | Humans | Carcinoma, Pancreatic Ductal - metabolism | Gene Expression Regulation, Neoplastic | Hedgehog Proteins - metabolism | Pancreatic Ducts - pathology | Pancreatic Ducts - metabolism | Carcinoma, Pancreatic Ductal - genetics | Hedgehog Proteins - genetics | Kruppel-Like Transcription Factors - metabolism | Smoothened Receptor | Proto-Oncogene Proteins p21(ras) - metabolism | Cell Line | Signal Transduction | Cell Survival | Pancreatic Neoplasms - pathology | Cells, Cultured | Pancreatic Neoplasms - genetics | Carcinoma, Pancreatic Ductal - pathology | Cell Transformation, Neoplastic - metabolism | Animals | Mice | Receptors, G-Protein-Coupled - genetics | Zinc Finger Protein GLI1 | Kruppel-Like Transcription Factors - genetics | Transforming Growth Factor beta - metabolism | Index Medicus | pancreatic cancer | hedgehog | Research Paper
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3. Full Text Adult Duct-Lining Cells Can Reprogram into β-like Cells Able to Counter Repeated Cycles of Toxin-Induced Diabetes
by Al-Hasani, Keith and Pfeifer, Anja and Courtney, Monica and Ben-Othman, Nouha and Gjernes, Elisabet and Vieira, Andhira and Druelle, Noémie and Avolio, Fabio and Ravassard, Philippe and Leuckx, Gunter and Lacas-Gervais, Sandra and Ambrosetti, Damien and Benizri, Emmanuel and Hecksher-Sorensen, Jacob and Gounon, Pierre and Ferrer, Jorge and Gradwohl, Gerard and Heimberg, Harry and Mansouri, Ahmed and Collombat, Patrick
Developmental cell, ISSN 1534-5807, 07/2013, Volume 26, Issue 1, pp. 86 - 100
Life Sciences & Biomedicine | Developmental Biology | Science & Technology | Cell Biology | Paired Box Transcription Factors - genetics | Streptozocin | Homeodomain Proteins - metabolism | Pancreatic Ducts - drug effects | Diabetes Mellitus, Experimental - genetics | Pancreatic Ducts - pathology | Hypertrophy - pathology | Hypertrophy - metabolism | Pancreatic Ducts - metabolism | Cellular Reprogramming | Insulin-Secreting Cells - metabolism | Basic Helix-Loop-Helix Transcription Factors - metabolism | Glucagon-Secreting Cells - metabolism | Epithelial-Mesenchymal Transition | Cell Differentiation | Diabetes Mellitus, Experimental - metabolism | Basic Helix-Loop-Helix Transcription Factors - genetics | Blood Glucose - analysis | Glucagon-Secreting Cells - pathology | Gene Expression Regulation | Nerve Tissue Proteins - genetics | Homeodomain Proteins - genetics | Nerve Tissue Proteins - metabolism | Cell Lineage | Animals | Mice | Insulin-Secreting Cells - pathology | Paired Box Transcription Factors - metabolism | Cell Movement | Index Medicus | Life Sciences | Development Biology
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4. Full Text Long-Term Correction of Diabetes in Mice by In Vivo Reprogramming of Pancreatic Ducts
by Wang, Yuhan and Dorrell, Craig and Naugler, Willscott E and Heskett, Michael and Spellman, Paul and Li, Bin and Galivo, Feorillo and Haft, Annelise and Wakefield, Leslie and Grompe, Markus
Molecular therapy, ISSN 1525-0016, 05/2018, Volume 26, Issue 5, pp. 1327 - 1342
reprogramming | Neurog3 | insulin | liver | pancreas | Pdx1 | β cell | gene therapy | Mafa | diabetes | Genetics & Heredity | Life Sciences & Biomedicine | Biotechnology & Applied Microbiology | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Humans | Diabetes Mellitus, Type 1 - metabolism | Gene Expression Profiling | Hepatocytes - metabolism | ATPases Associated with Diverse Cellular Activities - genetics | Maf Transcription Factors, Large - metabolism | Insulin-Secreting Cells - metabolism | Basic Helix-Loop-Helix Transcription Factors - metabolism | Maf Transcription Factors, Large - genetics | ATPases Associated with Diverse Cellular Activities - metabolism | Diabetes Mellitus, Experimental | Adenoviridae - genetics | Insulin-Secreting Cells - cytology | Diabetes Mellitus, Type 1 - immunology | Insulin - genetics | Single-Cell Analysis | Cellular Reprogramming - genetics | Disease Models, Animal | Pancreatic Ducts - cytology | Basic Helix-Loop-Helix Transcription Factors - genetics | Gene Expression Regulation | Diabetes Mellitus, Type 1 - genetics | Transcription Factors - genetics | Nerve Tissue Proteins - genetics | Genetic Vectors - genetics | Nerve Tissue Proteins - metabolism | Transcription Factors - metabolism | Insulin - metabolism | Animals | Biomarkers | Mice | Flow cytometry | Data analysis | Transcription factors | Animal models | Liver | Diabetes mellitus | Genomics | Principal components analysis | Diabetes mellitus (insulin dependent) | Gene expression | Experiments | Insulin | Generalized linear models | Hyperglycemia | Cell activation | Cell fate | Hepatocytes | Software | Genetic engineering | Diabetes | Pancreas | Index Medicus | Original
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5. Full Text Lineage Tracing Evidence for Transdifferentiation of Acinar to Duct Cells and Plasticity of Human Pancreas
by Houbracken, Isabelle and de Waele, Evelien and Lardon, Jessy and Ling, Zhidong and Heimberg, Harry and Rooman, Ilse and Bouwens, Luc
Gastroenterology (New York, N.Y. 1943), ISSN 0016-5085, 2011, Volume 141, Issue 2, pp. 731 - 741.e4
Gastroenterology and Hepatology | Metaplasia | Reprogramming | Exocrine Pancreas | Tissue Engineering | Gastroenterology & Hepatology | Life Sciences & Biomedicine | Science & Technology | Cell Proliferation | Chymotrypsin - metabolism | Humans | Glycoproteins - metabolism | Carbonic Anhydrase II - metabolism | Ki-67 Antigen - metabolism | Cell Transdifferentiation - genetics | RNA, Messenger - metabolism | Pancreatic Ducts - metabolism | Antigens, CD - metabolism | Cell Transdifferentiation - physiology | Peptides - metabolism | Pancreas, Exocrine - cytology | Cell Lineage - genetics | Genes, Reporter | Hepatocyte Nuclear Factor 1-beta - metabolism | Plant Lectins - pharmacokinetics | Biomarkers - metabolism | SOX9 Transcription Factor - metabolism | Green Fluorescent Proteins - metabolism | Promoter Regions, Genetic | Pancreatic Ducts - cytology | Transduction, Genetic | Cell Survival | Cells, Cultured | Cystic Fibrosis Transmembrane Conductance Regulator - metabolism | Signal Transduction - genetics | AC133 Antigen | Cell Lineage - physiology | Phenotype | Signal Transduction - physiology | Pancreas, Exocrine - metabolism | Keratin-19 - metabolism | Mitogen-Activated Protein Kinases - metabolism | Keratin | Epidermal growth factor | Analysis | Fluorescein | Fluorescence | Cystic fibrosis | Diabetes | Universities and colleges | Research | Protein kinases | Index Medicus | Abridged Index Medicus
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6. Full Text Localization of dipeptidyl peptidase-4 (CD26) to human pancreatic ducts and islet alpha cells
by Augstein, Petra and Naselli, Gaetano and Loudovaris, Thomas and Hawthorne, Wayne J and Campbell, Peter and Bandala-Sanchez, Esther and Rogers, Kelly and Heinke, Peter and Thomas, Helen E and Kay, Thomas W and Harrison, Leonard C
Diabetes research and clinical practice, ISSN 0168-8227, 2015, Volume 110, Issue 3, pp. 291 - 300
Endocrinology & Metabolism | Human pancreas | CD26 | Alpha cell | Islet | Beta cell | Dipeptidyl peptidase-4 | Life Sciences & Biomedicine | Science & Technology | Immunohistochemistry | Dipeptidyl Peptidase 4 - metabolism | Glucagon-Like Peptide 1 - metabolism | Proinsulin - metabolism | Humans | Middle Aged | Incretins - metabolism | Male | Diabetes Mellitus, Type 2 - metabolism | Pancreatic Ducts - metabolism | Young Adult | Insulin-Secreting Cells - metabolism | Glucagon-Secreting Cells - metabolism | Adult | Female | Glucagon - metabolism | Aged | Type 1 diabetes | Index Medicus
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7. Full Text Identification and Manipulation of Biliary Metaplasia in Pancreatic Tumors
by DelGiorno, Kathleen E and Hall, Jason C and Takeuchi, Kenneth K and Pan, Fong Cheng and Halbrook, Christopher J and Washington, M. Kay and Olive, Kenneth P and R. Spence, Jason and Sipos, Bence and Wright, Christopher V.E and Wells, James M and Crawford, Howard C
Gastroenterology (New York, N.Y. 1943), ISSN 0016-5085, 2014, Volume 146, Issue 1, pp. 233 - 244.e5
Gastroenterology and Hepatology | Pancreatic Cancer | Signal Transduction | Pathogenesis | Mouse Model | Gastroenterology & Hepatology | Life Sciences & Biomedicine | Science & Technology | Pancreatic Neoplasms - metabolism | Metaplasia - metabolism | Bile Ducts - metabolism | Humans | Carcinoma, Pancreatic Ductal - metabolism | Precancerous Conditions - metabolism | Pancreatic Ducts - pathology | SOXF Transcription Factors - metabolism | Pancreatic Ducts - metabolism | Bile Ducts - cytology | Metaplasia - complications | HMGB Proteins - metabolism | Precancerous Conditions - pathology | Proto-Oncogene Proteins p21(ras) - metabolism | Pancreatic Ducts - cytology | Pancreatic Neoplasms - pathology | Mice, Transgenic | Carcinoma, Pancreatic Ductal - complications | Carcinoma, Pancreatic Ductal - pathology | Cell Transformation, Neoplastic - metabolism | Metaplasia - pathology | Animals | Mice | Pancreatitis - metabolism | Cell Transformation, Neoplastic - pathology | Pancreatic Neoplasms - complications | Molecular genetics | Pancreatic cancer | Pancreatitis | Index Medicus | Abridged Index Medicus | pathogenesis | pancreatic cancer | mouse model | signal transduction
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8. Full Text Intraislet Pancreatic Ducts Can Give Rise to Insulin-Positive Cells
by El-Gohary, Yousef and Wiersch, John and Tulachan, Sidhartha and Xiao, Xiangwei and Guo, Ping and Rymer, Christopher and Fischbach, Shane and Prasadan, Krishna and Shiota, Chiyo and Gaffar, Iljana and Song, Zewen and Galambos, Csaba and Esni, Farzad and Gittes, George K
Endocrinology (Philadelphia), ISSN 0013-7227, 01/2016, Volume 157, Issue 1, pp. 166 - 175
Life Sciences & Biomedicine | Endocrinology & Metabolism | Science & Technology | Receptors, Transforming Growth Factor beta - genetics | Age Factors | Pancreatic Ducts - growth & development | Humans | Child, Preschool | Infant | Male | Insulin - biosynthesis | Mice, Mutant Strains | Islets of Langerhans - cytology | Cell Transdifferentiation | Female | Insulin-Secreting Cells - cytology | Protein-Serine-Threonine Kinases - metabolism | Insulin-Secreting Cells - physiology | Pancreatic Ducts - cytology | Islets of Langerhans - physiology | Mutant Proteins - genetics | Pancreatectomy | Protein-Serine-Threonine Kinases - genetics | Mice, Transgenic | Mutant Proteins - metabolism | Regeneration | Animals | Pancreatic Ducts - physiology | Receptors, Transforming Growth Factor beta - metabolism | Adolescent | Luminescent Proteins - genetics | Islets of Langerhans - growth & development | Cadaver | Luminescent Proteins - metabolism | Index Medicus | Abridged Index Medicus | Original Research
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9. Full Text Pathologic Features of Mucin-producing Bile Duct Tumors: Two Histopathologic Categories as Counterparts of Pancreatic Intraductal Papillary-mucinous Neoplasms
by Shibahara, Hiroaki and Tamada, Shugo and Goto, Masamichi and Oda, Koji and Nagino, Masato and Nagasaka, Tetsuro and Batra, Surinder K and Hollingsworth, Michael A and Imai, Kohzoh and Nimura, Yuji and Yonezawa, Suguru
The American journal of surgical pathology, ISSN 0147-5185, 03/2004, Volume 28, Issue 3, pp. 327 - 338
Immunohistochemistry | Bile duct neoplasms | Prognosis | Nuclear morphometry | Classification | Pathology | Life Sciences & Biomedicine | Surgery | Science & Technology | Biological and medical sciences | Investigative techniques, diagnostic techniques (general aspects) | Medical sciences | Pathology. Cytology. Biochemistry. Spectrometry. Miscellaneous investigative techniques | Pancreatic Neoplasms - metabolism | Adenoma - mortality | Humans | Middle Aged | Cystadenoma, Mucinous - metabolism | Carcinoma - mortality | Male | Pancreatic Ducts - pathology | Immunoenzyme Techniques | Adenoma - metabolism | Cystadenoma, Mucinous - pathology | Bile Ducts, Extrahepatic - metabolism | Bile Ducts, Intrahepatic - metabolism | Adenocarcinoma, Papillary - metabolism | Mucins - metabolism | Aged, 80 and over | Biomarkers, Tumor - metabolism | Adult | Female | Adenocarcinoma, Papillary - pathology | Carcinoma - secondary | Bile Ducts, Intrahepatic - pathology | Bile Duct Neoplasms - metabolism | Pancreatic Neoplasms - pathology | Survival Rate | Bile Duct Neoplasms - mortality | Adenoma - pathology | Aged | Bile Ducts, Extrahepatic - pathology | Bile Duct Neoplasms - pathology | Carcinoma - metabolism | Index Medicus
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