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Journal Article
Gastroenterology, ISSN 0016-5085, 2011, Volume 141, Issue 1, pp. 150 - 156
Journal Article
International Journal of Cancer, ISSN 0020-7136, 09/2015, Volume 137, Issue 5, pp. 1167 - 1175
Journal Article
Cell Stem Cell, ISSN 1934-5909, 04/2016, Volume 18, Issue 4, pp. 441 - 455
Journal Article
Occupational and Environmental Medicine, ISSN 1351-0711, 02/2012, Volume 69, Issue 2, pp. 153 - 156
ObjectiveThis study examined prospective data from the Third National Health and Nutrition Examination Survey (NHANES III) cohort to investigate the... 
Cadmium | Smoking cessation | Short reports | Nutrition | Mortality | Men | Cigar smoking | Death | Cigarette smoking | Non Hodgkin lymphoma | Lung neoplasms | UNITED-STATES | POPULATION | LUNG | PUBLIC, ENVIRONMENTAL & OCCUPATIONAL HEALTH | WORKERS | PANCREATIC-CANCER | ESTROGEN | RISK | SMOKING | Lymphoma, Non-Hodgkin - chemically induced | United States - epidemiology | Cadmium - urine | Prospective Studies | Lung Neoplasms - chemically induced | Nutrition Surveys | Lung Neoplasms - mortality | Humans | Metals, Heavy - urine | Male | Cause of Death | Cadmium - adverse effects | Neoplasms - chemically induced | Adult | Female | Pancreatic Neoplasms - chemically induced | Pancreatic Neoplasms - mortality | Metals, Heavy - adverse effects | Proportional Hazards Models | Neoplasms - mortality | Creatinine - urine | Environmental Exposure - adverse effects | Adolescent | Sex Factors | Lymphoma, Non-Hodgkin - mortality | Leukemia - chemically induced | Leukemia - mortality | Smoking | Causes of | Risk factors | Urine | Laboratories | Lung cancer | Melanoma | Breast cancer | Cigarettes | Medical diagnosis | Skin cancer | Ovarian cancer | Studies | Variables | Mens health | Hypotheses | Tobacco | Womens health | Population | Health risk assessment | Index Medicus | National Health and Nutrition Examination Survey | cadmium | cancer mortality
Journal Article
Gastroenterology, ISSN 0016-5085, 2014, Volume 147, Issue 5, pp. 1119 - 1133.e4
Background & Aims Although smoking is a leading risk factor for pancreatic ductal adenocarcinoma (PDAC), little is known about the mechanisms by which smoking... 
Gastroenterology and Hepatology | Metastasis | Pancreas | Progenitor Cells | Mouse Model | METFORMIN | STEM-CELLS | DUCTAL ADENOCARCINOMA | PROLIFERATION | EPITHELIAL-MESENCHYMAL TRANSITION | ADULT-MOUSE PANCREAS | ACETYLCHOLINE-RECEPTORS | K-RAS | GROWTH | DIFFERENTIATION | GASTROENTEROLOGY & HEPATOLOGY | Pancreatic Neoplasms - metabolism | Neoplastic Stem Cells - drug effects | Proto-Oncogene Proteins p21(ras) - genetics | Humans | Carcinoma, Pancreatic Ductal - metabolism | Epithelial-Mesenchymal Transition - drug effects | Mice, 129 Strain | Carcinoma, Pancreatic Ductal - genetics | Neoplastic Stem Cells - metabolism | Time Factors | Cell Transformation, Neoplastic - genetics | Neoplastic Stem Cells - pathology | Pancreatic Neoplasms - chemically induced | Liver Neoplasms - secondary | Nicotinic Agonists - toxicity | Proto-Oncogene Proteins c-akt - metabolism | Neoplastic Cells, Circulating - drug effects | GATA6 Transcription Factor - deficiency | Liver Neoplasms - genetics | Pancreatic Neoplasms - pathology | Pancreas - pathology | alpha7 Nicotinic Acetylcholine Receptor - genetics | Pancreas - metabolism | Mice, Knockout | Acinar Cells - pathology | Signal Transduction - drug effects | Mice, Nude | Liver Neoplasms - metabolism | Cell Line, Tumor | alpha7 Nicotinic Acetylcholine Receptor - metabolism | Mutation | Carcinoma, Pancreatic Ductal - secondary | Carcinoma, Pancreatic Ductal - chemically induced | Neoplastic Cells, Circulating - pathology | Neoplastic Cells, Circulating - metabolism | Cell Transformation, Neoplastic - chemically induced | Extracellular Signal-Regulated MAP Kinases - metabolism | Carcinoma, Pancreatic Ductal - prevention & control | Tumor Suppressor Protein p53 - genetics | Transfection | Proto-Oncogene Proteins p21(ras) - deficiency | Gene Expression Regulation, Neoplastic - drug effects | Pancreatic Neoplasms - prevention & control | Tumor Cells, Cultured | Proto-Oncogene Proteins p21(ras) - metabolism | Acinar Cells - metabolism | Cell Dedifferentiation - drug effects | Metformin - pharmacology | Mice, Inbred C57BL | Tumor Suppressor Protein p53 - metabolism | Pancreas - drug effects | Pancreatic Neoplasms - genetics | Nicotine - toxicity | Cell Transformation, Neoplastic - metabolism | Proto-Oncogene Proteins c-myc - metabolism | GATA6 Transcription Factor - metabolism | Animals | Acinar Cells - drug effects | GATA6 Transcription Factor - genetics | Cell Transformation, Neoplastic - pathology | Pancreatic cancer | Stem cells | Nicotine | Index Medicus | Abridged Index Medicus
Journal Article
Nature Cell Biology, ISSN 1465-7392, 2014, Volume 16, Issue 5, pp. 469 - 478
Understanding how malignancies arise within normal tissues requires identification of the cancer cell of origin and knowledge of the cellular and tissue... 
EPITHELIAL STEM-CELLS | OF-ORIGIN | CIGARETTE-SMOKING | PANCREATIC-CANCER | MONOCLONAL ORIGIN | URINARY-TRACT | TUMOR | PROSTATIC INTRAEPITHELIAL NEOPLASIA | UROTHELIAL CARCINOMAS | CLONAL ANALYSIS | CELL BIOLOGY | Cell Proliferation | Urothelium - metabolism | Butylhydroxybutylnitrosamine | Humans | Hedgehog Proteins - metabolism | Male | Urinary Bladder - pathology | Recombinant Fusion Proteins - metabolism | Urinary Bladder Neoplasms - chemically induced | Urothelium - pathology | Urinary Bladder Neoplasms - genetics | Hedgehog Proteins - genetics | Neoplastic Stem Cells - metabolism | Time Factors | Cell Transformation, Neoplastic - genetics | Urinary Bladder Neoplasms - pathology | Neoplastic Stem Cells - pathology | Carcinoma - pathology | Urinary Bladder Neoplasms - metabolism | Disease Models, Animal | Urinary Bladder - metabolism | Neoplasm Invasiveness | Carcinoma - chemically induced | Genotype | Mice, Transgenic | Tumor Burden | Cell Transformation, Neoplastic - metabolism | Cell Lineage | Phenotype | Animals | Recombinant Fusion Proteins - genetics | Carcinoma - genetics | Mice | Carcinoma - metabolism | Cell Transformation, Neoplastic - pathology | Oncology, Experimental | Cancer cells | Physiological aspects | Development and progression | Research | Bladder cancer | Carcinogenesis | Cancer | Index Medicus
Journal Article
Toxicology, ISSN 0300-483X, 2015, Volume 333, pp. 195 - 205
Journal Article
Carcinogenesis, ISSN 0143-3334, 12/2014, Volume 35, Issue 12, pp. 2798 - 2806
Journal Article
Diabetes, Obesity and Metabolism, ISSN 1462-8902, 09/2017, Volume 19, Issue 9, pp. 1233 - 1241
Journal Article
Oncogene, ISSN 0950-9232, 08/2009, Volume 28, Issue 32, pp. 2882 - 2893
The function of the class III histone deacetylase, Sir2, in promoting lifespan extension is well established in small model organisms. By analogy, SirT1, the... 
Sirtuins | Oncogenesis | Resveratrol | LIFE-SPAN | COLON-CANCER GROWTH | REGULATES SIRT1 | sirtuins | BIOCHEMISTRY & MOLECULAR BIOLOGY | INSULIN-SECRETION | SACCHAROMYCES-CEREVISIAE | CELLULAR SENESCENCE | CELL BIOLOGY | ONCOLOGY | oncogenesis | GENETICS & HEREDITY | PANCREATIC BETA-CELLS | resveratrol | NF-KAPPA-B | MOUSE SKIN | CALORIE RESTRICTION | Immunohistochemistry | NIH 3T3 Cells | Tetradecanoylphorbol Acetate | Blood Vessels - metabolism | Blood Vessels - pathology | Male | Neoplasms, Experimental - chemically induced | Stilbenes - pharmacology | Carcinogens | Intestinal Polyps - genetics | Skin Neoplasms - prevention & control | Transfection | Cell Transformation, Neoplastic - genetics | Sirtuin 1 | Female | Skin Neoplasms - blood supply | Sirtuins - genetics | Mice, Inbred C57BL | Neoplasms, Experimental - prevention & control | Angiogenesis Inhibitors - pharmacology | Genotype | Skin Neoplasms - chemically induced | Mice, Inbred Strains | Mice, Knockout | Animals | Blood Vessels - drug effects | Intestinal Polyps - pathology | 9,10-Dimethyl-1,2-benzanthracene | Mice | Cell Transformation, Neoplastic - drug effects | Intestinal Polyps - metabolism | Sirtuins - metabolism | Neoplasms, Experimental - blood supply | Gene mutations | Physiological aspects | Tumor suppressor genes | Dosage and administration | Genetic aspects |