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British Journal of Cancer, ISSN 0007-0920, 02/2018, Volume 118, Issue 3, pp. 428 - 434
Background: Periodontal pathogens have been linked to oral and gastrointestinal (orodigestive) carcinogenesis. However, the exact mechanisms remain unknown.... 
chymotrypsin-like proteinase | Treponema denticola | periodontopathogen | COLLAGENASE-2 MMP-8 | PORPHYROMONAS-GINGIVALIS | PANCREATIC-CANCER | TISSUE INHIBITORS | PERIODONTAL-DISEASE | METALLOPROTEINASES | ONCOLOGY | gastrointestinal cancer | ALPHA-1-ANTITRYPSIN | EXPRESSION | oral cancer | HUMAN NEUTROPHIL | PROGRESSION | Digestive System Neoplasms - metabolism | Pancreatic Neoplasms - chemistry | Adenocarcinoma - pathology | Pancreatic Neoplasms - metabolism | Carcinoma, Squamous Cell - metabolism | Carcinoma, Squamous Cell - pathology | Humans | Treponema denticola - enzymology | Mouth Neoplasms - metabolism | Stomach Neoplasms - pathology | Tonsillar Neoplasms - chemistry | Complement C1q - metabolism | Colonic Neoplasms - metabolism | Head and Neck Neoplasms - metabolism | Esophageal Neoplasms - pathology | Tonsillar Neoplasms - metabolism | Tissue Inhibitor of Metalloproteinase-1 - metabolism | Tissue Inhibitor of Metalloproteinase-2 - metabolism | Head and Neck Neoplasms - chemistry | Matrix Metalloproteinase 9 - metabolism | Adenocarcinoma - metabolism | Matrix Metalloproteinase 8 - metabolism | Esophageal Neoplasms - metabolism | Digestive System Neoplasms - pathology | Colonic Neoplasms - chemistry | Stomach Neoplasms - chemistry | Adenocarcinoma - chemistry | Esophageal Neoplasms - chemistry | Tonsillar Neoplasms - pathology | Pancreatic Neoplasms - pathology | Carcinoma, Squamous Cell - chemistry | Mouth Neoplasms - chemistry | Head and Neck Neoplasms - pathology | Cell Transformation, Neoplastic - immunology | Digestive System Neoplasms - chemistry | alpha 1-Antichymotrypsin - metabolism | Colonic Neoplasms - pathology | Chymases - analysis | Mouth Neoplasms - pathology | Molecular Diagnostics
Journal Article
CA: a cancer journal for clinicians, ISSN 0007-9235, 2016, Volume 66, Issue 5, pp. 408 - 436
Answer questions and earn CME/CNE In this report, a team of surgical pathologists has provided a review of intraepithelial neoplasia in a host of (but not all)... 
carcinoma in situ | dysplasia | intraepithelial neoplasia | preinvasive neoplasia | precancer | HIGH-GRADE DYSPLASIA | URINARY-BLADDER | BARRETTS-ESOPHAGUS | HUMAN-PAPILLOMAVIRUS | INTRADUCTAL CARCINOMA | CARCINOMA IN-SITU | PAPILLARY UROTHELIAL HYPERPLASIA | ONCOLOGY | SINGLE ACADEMIC CENTER | TERM-FOLLOW-UP | NECK-CANCER IMPLICATIONS | Humans | Lung Neoplasms - metabolism | Ovarian Neoplasms - pathology | Lung Neoplasms - pathology | Male | Uterine Cervical Neoplasms - pathology | Head and Neck Neoplasms - metabolism | Breast Neoplasms - metabolism | Esophageal Neoplasms - pathology | Breast Neoplasms - therapy | American Cancer Society | Uterine Cervical Neoplasms - metabolism | Skin Neoplasms - diagnosis | Urinary Bladder Neoplasms - pathology | Female | Ovarian Neoplasms - metabolism | Uterine Cervical Neoplasms - therapy | Skin Neoplasms - pathology | Prostatic Neoplasms - pathology | Uterine Cervical Neoplasms - diagnosis | Skin Neoplasms - therapy | Carcinoma in Situ - pathology | Ovarian Neoplasms - diagnosis | Pancreatic Neoplasms - pathology | Risk Factors | Head and Neck Neoplasms - therapy | Lung Neoplasms - therapy | Carcinoma in Situ - therapy | Head and Neck Neoplasms - pathology | Skin Neoplasms - metabolism | Breast Neoplasms - pathology | Ovarian Neoplasms - therapy | Head and Neck Neoplasms - diagnosis | Breast Neoplasms - diagnosis | Carcinoma in Situ - diagnosis | Carcinoma in Situ - metabolism | Lung Neoplasms - diagnosis | Population Surveillance | Prevention | Care and treatment | Gastrointestinal system | Practice | Pathologists | Metastasis | Patients | Health aspects | Cervical cancer | Risk factors | Pathology | Usage | Diagnosis | Tumors | Medical diagnosis | Cancer
Journal Article
BMC Genomics, ISSN 1471-2164, 04/2006, Volume 7, Issue 1, pp. 90 - 90
Background: Post-translational modification of histones resulting in chromatin remodelling plays a key role in the regulation of gene expression. Here we... 
BREAST-CANCER | CBP | METHYLATION | INHIBITION | BOOTSTRAP | ACETYLATION | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | GENETICS & HEREDITY | PATTERNS | MUTATIONS | REPRODUCIBILITY | PROGRESSION | Neoplasms - metabolism | Protein Methyltransferases | Pancreatic Neoplasms - metabolism | Colorectal Neoplasms - genetics | Histone Acetyltransferases - biosynthesis | Humans | Lung Neoplasms - metabolism | Protein Processing, Post-Translational - genetics | DNA, Complementary - genetics | Gene Expression Regulation, Neoplastic | Histone Acetyltransferases - genetics | Male | Histone-Lysine N-Methyltransferase - biosynthesis | Breast Neoplasms - metabolism | Ovarian Neoplasms - genetics | Polymorphism, Single-Stranded Conformational | Neoplasms - genetics | Cell Transformation, Neoplastic - genetics | Polymerase Chain Reaction | Female | Ovarian Neoplasms - metabolism | Neoplasm Proteins - genetics | Colorectal Neoplasms - metabolism | Lung Neoplasms - genetics | Histone-Lysine N-Methyltransferase - genetics | Histone Deacetylases - genetics | Neoplasm Proteins - biosynthesis | Linear Models | Pancreatic Neoplasms - genetics | Gene Expression Regulation, Enzymologic | Breast Neoplasms - genetics | Carcinoma - genetics | Carcinoma - metabolism | DNA, Neoplasm - genetics | Histones - metabolism | Histone Code - genetics | Cluster Analysis
Journal Article
Metabolism, ISSN 0026-0495, 2015, Volume 64, Issue 2, pp. 182 - 189
Journal Article
Annals of the Royal College of Surgeons of England, ISSN 0035-8843, 03/2017, Volume 99, Issue 3, pp. 193 - 197
INTRODUCTION Neuroendocrine tumours (NETs) are a heterogeneous group of tumours with a highly variable presentation and prognosis. Management decisions are... 
Ki-67 | Pathology | Neuroendocrine tumours | Diagnostic techniques and procedures | General surgery | SURVIVAL | SURGERY | PROGNOSTIC-FACTORS | NEOPLASMS | ENDOCRINE TUMORS | RGETNE | DISEASE | REGISTRY | Colorectal Neoplasms - surgery | Digestive System Neoplasms - metabolism | Pancreatic Neoplasms - metabolism | Prognosis | Humans | Lung Neoplasms - metabolism | Middle Aged | Stomach Neoplasms - metabolism | Ki-67 Antigen - metabolism | Lung Neoplasms - pathology | Male | Stomach Neoplasms - pathology | Neoplasm Grading | Digestive System Neoplasms - pathology | Adult | Female | Liver Neoplasms - pathology | Retrospective Studies | Biopsy, Large-Core Needle | Colorectal Neoplasms - metabolism | Ileal Neoplasms - pathology | Neuroendocrine Tumors - pathology | Ileal Neoplasms - surgery | Neuroendocrine Tumors - metabolism | Pancreatic Neoplasms - pathology | Pancreatic Neoplasms - surgery | Ileal Neoplasms - metabolism | Liver Neoplasms - surgery | Biopsy, Fine-Needle | Digestive System Neoplasms - surgery | Liver Neoplasms - metabolism | Lung Neoplasms - surgery | Neuroendocrine Tumors - surgery | Aged | Colorectal Neoplasms - pathology | Stomach Neoplasms - surgery | Cohort Studies | Ultrasonic imaging | Laboratories | Medical prognosis | Cell cycle | Hormones | Metastasis | Endoscopy | Epidemiology | Cancer therapies | Tumors | Oncology
Journal Article
by Ma, YC and Chen, HB and Ma, HY and Yao, ZH and Hu, JW and Ma, J and Zhang, XQ and Chen, GW and Liu, YC
SAUDI MEDICAL JOURNAL, ISSN 0379-5284, 08/2019, Volume 40, Issue 8, pp. 755 - 765
Objectives: To analyze the heterogeneous functions of secreted protein acidic and rich in cysteine (SPARC) from different origins and in different tumor... 
SURVIVAL | LUNG-CANCER | PREDICTS | MEDICINE, GENERAL & INTERNAL | GASTRIC-CANCER | PANCREATIC-CANCER | MARKERS | SPARC EXPRESSION | GEMCITABINE | PROGRESSION | PACLITAXEL | Esophageal Squamous Cell Carcinoma - metabolism | Neoplasms - metabolism | Nasopharyngeal Neoplasms - metabolism | Nasopharyngeal Neoplasms - mortality | Pancreatic Neoplasms - metabolism | Prognosis | Lung Neoplasms - mortality | Humans | Lung Neoplasms - metabolism | Stomach Neoplasms - metabolism | Tumor Microenvironment | Biliary Tract Neoplasms - mortality | Lymphoma, Large B-Cell, Diffuse - metabolism | Breast Neoplasms - metabolism | Esophageal Neoplasms - metabolism | Biliary Tract Neoplasms - metabolism | Esophageal Neoplasms - mortality | Pancreatic Neoplasms - mortality | Colorectal Neoplasms - metabolism | Colorectal Neoplasms - mortality | Nasopharyngeal Carcinoma - mortality | Proportional Hazards Models | Neoplasms - mortality | Osteonectin - metabolism | Nasopharyngeal Carcinoma - metabolism | Survival Rate | Lymphoma, Large B-Cell, Diffuse - mortality | Disease-Free Survival | Esophageal Squamous Cell Carcinoma - mortality | Breast Neoplasms - mortality | Stomach Neoplasms - mortality | Medical research | Cysteine | Care and treatment | Analysis | Pancreatic cancer | Colorectal cancer | Medicine, Experimental | Development and progression | Cystine | Proteins | Angiogenesis | Medical prognosis | Bias | Fibroblasts | Lymphomas | Metastasis | Stomach cancer | Tumors | Cell adhesion & migration | Meta-analysis | Systematic Review | meta-analysis | SPARC | tumor microenvironment | prognosis | stroma
Journal Article
Cell stem cell, ISSN 1934-5909, 2010, Volume 6, Issue 6, pp. 603 - 615
Recent evidence suggests that a subpopulation of cancer cells, cancer stem cells (CSCs), is responsible for tumor growth in colorectal cancer. However,... 
STEMCELL | CELLCYCLE | BREAST-CANCER | INITIATING CELLS | DIPEPTIDYL-PEPTIDASE-IV | CARCINOMA CELLS | EXTRACELLULAR-MATRIX | ACUTE MYELOID-LEUKEMIA | E-CADHERIN | IDENTIFICATION | TUMOR-GROWTH | HUMAN PANCREATIC-CANCER | CELL & TISSUE ENGINEERING | CELL BIOLOGY | Neoplasm Transplantation | RNA, Small Interfering - genetics | Prognosis | Follow-Up Studies | Apoptosis - drug effects | Neoplastic Stem Cells - drug effects | Colorectal Neoplasms - genetics | Humans | Apoptosis - genetics | Gene Expression Profiling | Colorectal Neoplasms - diagnosis | Organoplatinum Compounds - pharmacology | Neoplastic Stem Cells - metabolism | Cell Transformation, Neoplastic - genetics | Colorectal Neoplasms - drug therapy | Liver Neoplasms - physiopathology | Neoplastic Stem Cells - pathology | Tumor Burden - genetics | Tumor Cells, Cultured | Liver Neoplasms - secondary | Colorectal Neoplasms - metabolism | Carcinoma - secondary | Carcinoma - drug therapy | Liver Neoplasms - genetics | Dipeptidyl Peptidase 4 - biosynthesis | Liver Neoplasms - drug therapy | Carcinoma - diagnosis | Dipeptidyl Peptidase 4 - genetics | Mice, SCID | Carcinoma - physiopathology | Disease Progression | Colorectal Neoplasms - physiopathology | Drug Resistance, Neoplasm - genetics | Animals | Liver Neoplasms - diagnosis | Tumor Burden - drug effects | Liver Neoplasms - metabolism | Carcinoma - genetics | Biomarkers, Tumor - genetics | Fluorouracil - pharmacology | Mice | Carcinoma - metabolism | Colorectal Neoplasms - pathology | Neoplasm Invasiveness - genetics | Cell Migration Assays | Biomarkers, Tumor - biosynthesis
Journal Article
Oncotarget, ISSN 1949-2553, 05/2016, Volume 7, Issue 19, pp. 28592 - 28611
Journal Article
Journal Article
Molecular cell, ISSN 1097-2765, 2017, Volume 67, Issue 4, pp. 685 - 701.e6
Metabolic deregulation is a hallmark of human cancers, and the glycolytic and glutamine metabolism pathways were shown to be deregulated in pancreatic ductal adenocarcinoma (PDAC... 
GLUT1 | anoikis | glucose | pancreatic cancer | metabolism | PON2 | PATHWAYS | CELLS | ACTIVATED PROTEIN-KINASE | GENE | ONCOGENIC KRAS | BIOCHEMISTRY & MOLECULAR BIOLOGY | SENSITIVITY | ENERGY SENSOR | EXPRESSION | PROGRESSION | P53 | CELL BIOLOGY | Proto-Oncogene Proteins p21(ras) - genetics | Humans | AMP-Activated Protein Kinases - analysis | Glucose Transporter Type 1 - metabolism | Male | Carcinoma, Pancreatic Ductal - genetics | RNA Interference | Time Factors | Transcription, Genetic | Liver Neoplasms - enzymology | Liver Neoplasms - secondary | Lung Neoplasms - enzymology | Liver Neoplasms - genetics | Signal Transduction | Pancreatic Neoplasms - pathology | Tumor Burden | Aryldialkylphosphatase - genetics | Aryldialkylphosphatase - metabolism | Forkhead Box Protein O3 - metabolism | Apoptosis Regulatory Proteins - metabolism | Cell Movement - drug effects | Lung Neoplasms - prevention & control | Mice, Nude | Cell Line, Tumor | Glucose - metabolism | Mutation | AMP-Activated Protein Kinases - genetics | Energy Metabolism - drug effects | AMP-Activated Protein Kinases - metabolism | Gene Expression Regulation, Neoplastic | Tumor Suppressor Protein p53 - genetics | Forkhead Box Protein O3 - genetics | Pancreatic Neoplasms - drug therapy | Transfection | Lung Neoplasms - secondary | Apoptosis Regulatory Proteins - genetics | Female | Antineoplastic Agents - pharmacology | Liver Neoplasms - prevention & control | Lung Neoplasms - genetics | Proto-Oncogene Proteins - metabolism | Pancreatic Neoplasms - enzymology | Tumor Suppressor Protein p53 - metabolism | Pancreatic Neoplasms - genetics | Proto-Oncogene Proteins - genetics | Carcinoma, Pancreatic Ductal - pathology | Carcinoma, Pancreatic Ductal - enzymology | Xenograft Model Antitumor Assays | Carcinoma, Pancreatic Ductal - drug therapy | Animals | Glucose Transporter Type 1 - genetics | Cell Proliferation - drug effects | Protein Kinase Inhibitors - pharmacology | Deregulation | Growth | Pancreatic cancer | Physiological aspects | Genetic transcription | Metastasis | Glucose | Tumor proteins | Dextrose | Glutamine | Glucose metabolism | Medical colleges | Protein kinases
Journal Article